Ekrinní potní žlázy (Czech Wikipedia)

Analysis of information sources in references of the Wikipedia article "Ekrinní potní žlázy" in Czech language version.

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archive.org

  • JAMES, William; BERGER, Timothy; ELSTON, Dirk. Andrews' Diseases of the Skin: Clinical Dermatology. 10th. vyd. [s.l.]: Saunders, 2005. Dostupné online. ISBN 978-0-7216-2921-6. S. 6–7. 

bbc.com

doi.org

dx.doi.org

  • WILKE, K.; MARTIN, A.; TERSTEGEN, L.; BIEL, S. S. A short history of sweat gland biology. International Journal of Cosmetic Science. June 2007, s. 169–179. ISSN 1468-2494. DOI 10.1111/j.1467-2494.2007.00387.x. PMID 18489347. S2CID 205556581. 
  • CUI, Chang-Yi; SCHLESSINGER, David. Eccrine sweat gland development and sweat secretion. Experimental Dermatology. 2015, s. 644–650. ISSN 0906-6705. DOI 10.1111/exd.12773. PMID 26014472. 
  • Hanukoglu I, Boggula VR, Vaknine H, Sharma S, Kleyman T, Hanukoglu A. Expression of epithelial sodium channel (ENaC) and CFTR in the human epidermis and epidermal appendages. Histochemistry and Cell Biology. January 2017, s. 733–748. Dostupné online. DOI 10.1007/s00418-016-1535-3. PMID 28130590. S2CID 8504408. 
  • Hanukoglu A. Type I pseudohypoaldosteronism includes two clinically and genetically distinct entities with either renal or multiple target organ defects. The Journal of Clinical Endocrinology and Metabolism. Nov 1991, s. 936–44. Dostupné online. DOI 10.1210/jcem-73-5-936. PMID 1939532. 
  • Hanukoglu I, Hanukoglu A. Epithelial sodium channel (ENaC) family: Phylogeny, structure-function, tissue distribution, and associated inherited diseases.. Gene. Jan 2016, s. 95–132. DOI 10.1016/j.gene.2015.12.061. PMID 26772908. 
  • EDELHEIT, Oded; HANUKOGLU, Israel; SHRIKI, Yafit; TFILIN, Matanel; DASCAL, Nathan; GILLIS, David; HANUKOGLU, Aaron. Truncated beta epithelial sodium channel (ENaC) subunits responsible for multi-system pseudohypoaldosteronism (PHA) support partial activity of ENaC. The Journal of Steroid Biochemistry and Molecular Biology. 2010, s. 84–88. DOI 10.1016/j.jsbmb.2010.01.002. PMID 20064610. S2CID 9564777. 
  • NIYONSABA, F; SUZUKI, A; USHIO, H; NAGAOKA, I; OGAWA, H; OKUMURA, K. The human antimicrobial peptide dermcidin activates normal human keratinocytes. The British Journal of Dermatology. 2009, s. 243–9. DOI 10.1111/j.1365-2133.2008.08925.x. PMID 19014393. S2CID 26601547. 

nih.gov

ncbi.nlm.nih.gov

  • WILKE, K.; MARTIN, A.; TERSTEGEN, L.; BIEL, S. S. A short history of sweat gland biology. International Journal of Cosmetic Science. June 2007, s. 169–179. ISSN 1468-2494. DOI 10.1111/j.1467-2494.2007.00387.x. PMID 18489347. S2CID 205556581. 
  • CUI, Chang-Yi; SCHLESSINGER, David. Eccrine sweat gland development and sweat secretion. Experimental Dermatology. 2015, s. 644–650. ISSN 0906-6705. DOI 10.1111/exd.12773. PMID 26014472. 
  • Hanukoglu I, Boggula VR, Vaknine H, Sharma S, Kleyman T, Hanukoglu A. Expression of epithelial sodium channel (ENaC) and CFTR in the human epidermis and epidermal appendages. Histochemistry and Cell Biology. January 2017, s. 733–748. Dostupné online. DOI 10.1007/s00418-016-1535-3. PMID 28130590. S2CID 8504408. 
  • SOKOLOV, VE; SHABADASH, SA; ZELIKINA, TI. Innervation of eccrine sweat glands. Biology Bulletin of the Academy of Sciences of the USSR. 1980, s. 331–46. PMID 7317512. 
  • Hanukoglu A. Type I pseudohypoaldosteronism includes two clinically and genetically distinct entities with either renal or multiple target organ defects. The Journal of Clinical Endocrinology and Metabolism. Nov 1991, s. 936–44. Dostupné online. DOI 10.1210/jcem-73-5-936. PMID 1939532. 
  • Hanukoglu I, Hanukoglu A. Epithelial sodium channel (ENaC) family: Phylogeny, structure-function, tissue distribution, and associated inherited diseases.. Gene. Jan 2016, s. 95–132. DOI 10.1016/j.gene.2015.12.061. PMID 26772908. 
  • EDELHEIT, Oded; HANUKOGLU, Israel; SHRIKI, Yafit; TFILIN, Matanel; DASCAL, Nathan; GILLIS, David; HANUKOGLU, Aaron. Truncated beta epithelial sodium channel (ENaC) subunits responsible for multi-system pseudohypoaldosteronism (PHA) support partial activity of ENaC. The Journal of Steroid Biochemistry and Molecular Biology. 2010, s. 84–88. DOI 10.1016/j.jsbmb.2010.01.002. PMID 20064610. S2CID 9564777. 
  • NIYONSABA, F; SUZUKI, A; USHIO, H; NAGAOKA, I; OGAWA, H; OKUMURA, K. The human antimicrobial peptide dermcidin activates normal human keratinocytes. The British Journal of Dermatology. 2009, s. 243–9. DOI 10.1111/j.1365-2133.2008.08925.x. PMID 19014393. S2CID 26601547. 

semanticscholar.org

api.semanticscholar.org

  • WILKE, K.; MARTIN, A.; TERSTEGEN, L.; BIEL, S. S. A short history of sweat gland biology. International Journal of Cosmetic Science. June 2007, s. 169–179. ISSN 1468-2494. DOI 10.1111/j.1467-2494.2007.00387.x. PMID 18489347. S2CID 205556581. 
  • Hanukoglu I, Boggula VR, Vaknine H, Sharma S, Kleyman T, Hanukoglu A. Expression of epithelial sodium channel (ENaC) and CFTR in the human epidermis and epidermal appendages. Histochemistry and Cell Biology. January 2017, s. 733–748. Dostupné online. DOI 10.1007/s00418-016-1535-3. PMID 28130590. S2CID 8504408. 
  • EDELHEIT, Oded; HANUKOGLU, Israel; SHRIKI, Yafit; TFILIN, Matanel; DASCAL, Nathan; GILLIS, David; HANUKOGLU, Aaron. Truncated beta epithelial sodium channel (ENaC) subunits responsible for multi-system pseudohypoaldosteronism (PHA) support partial activity of ENaC. The Journal of Steroid Biochemistry and Molecular Biology. 2010, s. 84–88. DOI 10.1016/j.jsbmb.2010.01.002. PMID 20064610. S2CID 9564777. 
  • NIYONSABA, F; SUZUKI, A; USHIO, H; NAGAOKA, I; OGAWA, H; OKUMURA, K. The human antimicrobial peptide dermcidin activates normal human keratinocytes. The British Journal of Dermatology. 2009, s. 243–9. DOI 10.1111/j.1365-2133.2008.08925.x. PMID 19014393. S2CID 26601547. 

worldcat.org

zenodo.org

  • Hanukoglu I, Boggula VR, Vaknine H, Sharma S, Kleyman T, Hanukoglu A. Expression of epithelial sodium channel (ENaC) and CFTR in the human epidermis and epidermal appendages. Histochemistry and Cell Biology. January 2017, s. 733–748. Dostupné online. DOI 10.1007/s00418-016-1535-3. PMID 28130590. S2CID 8504408. 
  • Hanukoglu A. Type I pseudohypoaldosteronism includes two clinically and genetically distinct entities with either renal or multiple target organ defects. The Journal of Clinical Endocrinology and Metabolism. Nov 1991, s. 936–44. Dostupné online. DOI 10.1210/jcem-73-5-936. PMID 1939532.