Fas受容体 (Japanese Wikipedia)

Analysis of information sources in references of the Wikipedia article "Fas受容体" in Japanese language version.

refsWebsite

Global rank Japanese rank

39nih.gov

4^th place

24^th place

28doi.org

2^nd place

6^th place

6harvard.edu

18^th place

107^th place

2ensembl.org

1,626^th place

2,625^th place

2wikipedia.org

low place

1cnrs.fr

2,402^nd place

low place

1web.archive.org

1^st place

1broadinstitute.org

low place

1wiley.com

222^nd place

498^th place

1inserm.fr

9,568^th place

low place

1unil.ch

9,112^th place

low place

broadinstitute.org

“Tumorscape”. The Broad Institute. 2012年4月14日時点のオリジナルよりアーカイブ。2012年7月5日閲覧。

cnrs.fr

orthomam.mbb.cnrs.fr

“ENSG00000026103_FAS”. orthomam.mbb.cnrs.fr. 2021年11月13日閲覧。

doi.org

“The human APO-1 (APT) antigen maps to 10q23, a region that is syntenic with mouse chromosome 19”. Genomics 14 (1): 179–80. (September 1992). doi:10.1016/S0888-7543(05)80302-7. PMID 1385299.
“Assignment of the human Fas antigen gene (Fas) to 10q24.1”. Genomics 14 (3): 821–2. (November 1992). doi:10.1016/S0888-7543(05)80200-9. PMID 1385309.
“Early work on the function of CD95, an interview with Shige Nagata”. Cell Death and Differentiation 11 Suppl 1 (Suppl 1): S23-7. (July 2004). doi:10.1038/sj.cdd.4401453. PMID 15143352.
“The Fas signaling pathway: more than a paradigm”. Science 296 (5573): 1635–6. (May 2002). Bibcode: 2002Sci...296.1635W. doi:10.1126/science.1071553. PMID 12040174.
“Regulation of Fas alternative splicing by antagonistic effects of TIA-1 and PTB on exon definition”. Molecular Cell 19 (4): 475–84. (August 2005). doi:10.1016/j.molcel.2005.06.015. PMID 16109372.
“The Fas-FADD death domain complex structure reveals the basis of DISC assembly and disease mutations”. Nature Structural & Molecular Biology 17 (11): 1324–9. (November 2010). doi:10.1038/nsmb.1920. PMC 2988912. PMID 20935634.
“NMR structure and mutagenesis of the Fas (APO-1/CD95) death domain”. Nature 384 (6610): 638–41. (1996). Bibcode: 1996Natur.384..638H. doi:10.1038/384638a0. PMID 8967952.
“NMR structure and mutagenesis of the FADD (Mort1) death-effector domain”. Nature 392 (6679): 941–5. (April 1998). Bibcode: 1998Natur.392..941E. doi:10.1038/31972. PMID 9582077.
“CD95 promotes tumour growth”. Nature 465 (7297): 492–6. (May 2010). Bibcode: 2010Natur.465..492C. doi:10.1038/nature09075. PMC 2879093. PMID 20505730.
“NF-κB directly regulates Fas transcription to modulate Fas-mediated apoptosis and tumor suppression”. The Journal of Biological Chemistry 287 (30): 25530–40. (July 2012). doi:10.1074/jbc.M112.356279. PMC 3408167. PMID 22669972.
“Decitabine and vorinostat cooperate to sensitize colon carcinoma cells to Fas ligand-induced apoptosis in vitro and tumor suppression in vivo”. Journal of Immunology 188 (9): 4441–9. (May 2012). doi:10.4049/jimmunol.1103035. PMC 3398838. PMID 22461695.
“Lysis of bystander target cells after triggering of human cytotoxic T lymphocytes”. European Journal of Immunology 16 (8): 1021–4. (August 1986). doi:10.1002/eji.1830160826. PMID 3488908.
“Fas ligand-mediated lysis of self bystander targets by human papillomavirus-specific CD8+ cytotoxic T lymphocytes”. Journal of Virology 72 (7): 5948–54. (July 1998). doi:10.1128/JVI.72.7.5948-5954.1998. PMC 110399. PMID 9621057.
“Bispecific T cell engager (BiTE®) antibody constructs can mediate bystander tumor cell killing”. PLOS ONE 12 (8): e0183390. (2017-08-24). Bibcode: 2017PLoSO..1283390R. doi:10.1371/journal.pone.0183390. PMC 5570333. PMID 28837681.
“A critical role for fas-mediated off-target tumor killing in T cell immunotherapy”. Cancer Discovery 11 (3): 599–613. (December 2020). doi:10.1158/2159-8290.CD-20-0756. PMC 7933082. PMID 33334730.
“Cytoskeleton-mediated death receptor and ligand concentration in lipid rafts forms apoptosis-promoting clusters in cancer chemotherapy”. The Journal of Biological Chemistry 280 (12): 11641–7. (March 2005). doi:10.1074/jbc.M411781200. PMID 15659383.
“Identification and molecular cloning of two novel receptors for the cytotoxic ligand TRAIL”. The Journal of Biological Chemistry 272 (41): 25417–20. (October 1997). doi:10.1074/jbc.272.41.25417. PMID 9325248.
“Casper is a FADD- and caspase-related inducer of apoptosis”. Immunity 6 (6): 751–63. (June 1997). doi:10.1016/S1074-7613(00)80450-1. PMID 9208847.
“Fas-associated death domain protein interleukin-1beta-converting enzyme 2 (FLICE2), an ICE/Ced-3 homologue, is proximally involved in CD95- and p55-mediated death signaling”. The Journal of Biological Chemistry 272 (10): 6578–83. (March 1997). doi:10.1074/jbc.272.10.6578. PMID 9045686.
“The receptor for the cytotoxic ligand TRAIL”. Science 276 (5309): 111–3. (April 1997). doi:10.1126/science.276.5309.111. PMID 9082980.
“NMR structure and mutagenesis of the Fas (APO-1/CD95) death domain”. Nature 384 (6610): 638–41. (1996). Bibcode: 1996Natur.384..638H. doi:10.1038/384638a0. PMID 8967952.
“FADD, a novel death domain-containing protein, interacts with the death domain of Fas and initiates apoptosis”. Cell 81 (4): 505–12. (May 1995). doi:10.1016/0092-8674(95)90071-3. PMID 7538907.
“Regulation of Fas-associated death domain interactions by the death effector domain identified by a modified reverse two-hybrid screen”. The Journal of Biological Chemistry 277 (37): 34343–8. (September 2002). doi:10.1074/jbc.M204169200. PMID 12107169.
“Induction of TNF receptor I-mediated apoptosis via two sequential signaling complexes”. Cell 114 (2): 181–90. (July 2003). doi:10.1016/S0092-8674(03)00521-X. PMID 12887920.
“Identification of amino acid residues important for ligand binding to Fas”. The Journal of Experimental Medicine 185 (8): 1487–92. (April 1997). doi:10.1084/jem.185.8.1487. PMC 2196280. PMID 9126929.
“Characterization of Fas (Apo-1, CD95)-Fas ligand interaction”. The Journal of Biological Chemistry 272 (30): 18827–33. (July 1997). doi:10.1074/jbc.272.30.18827. PMID 9228058.
“Apoptosis-linked gene 2 binds to the death domain of Fas and dissociates from Fas during Fas-mediated apoptosis in Jurkat cells”. Biochemical and Biophysical Research Communications 288 (2): 420–6. (October 2001). doi:10.1006/bbrc.2001.5769. PMID 11606059.
“Interaction of Daxx, a Fas binding protein, with sentrin and Ubc9”. Biochemical and Biophysical Research Communications 279 (1): 6–10. (December 2000). doi:10.1006/bbrc.2000.3882. PMID 11112409.

ensembl.org

May2017.archive.ensembl.org

GRCh38: Ensembl release 89: ENSG00000026103 - Ensembl, May 2017
GRCm38: Ensembl release 89: ENSMUSG00000024778 - Ensembl, May 2017

harvard.edu

ui.adsabs.harvard.edu

“The Fas signaling pathway: more than a paradigm”. Science 296 (5573): 1635–6. (May 2002). Bibcode: 2002Sci...296.1635W. doi:10.1126/science.1071553. PMID 12040174.
“NMR structure and mutagenesis of the Fas (APO-1/CD95) death domain”. Nature 384 (6610): 638–41. (1996). Bibcode: 1996Natur.384..638H. doi:10.1038/384638a0. PMID 8967952.
“NMR structure and mutagenesis of the FADD (Mort1) death-effector domain”. Nature 392 (6679): 941–5. (April 1998). Bibcode: 1998Natur.392..941E. doi:10.1038/31972. PMID 9582077.
“CD95 promotes tumour growth”. Nature 465 (7297): 492–6. (May 2010). Bibcode: 2010Natur.465..492C. doi:10.1038/nature09075. PMC 2879093. PMID 20505730.
“Bispecific T cell engager (BiTE®) antibody constructs can mediate bystander tumor cell killing”. PLOS ONE 12 (8): e0183390. (2017-08-24). Bibcode: 2017PLoSO..1283390R. doi:10.1371/journal.pone.0183390. PMC 5570333. PMID 28837681.
“NMR structure and mutagenesis of the Fas (APO-1/CD95) death domain”. Nature 384 (6610): 638–41. (1996). Bibcode: 1996Natur.384..638H. doi:10.1038/384638a0. PMID 8967952.

inserm.fr

hal.inserm.fr

“Induction of TNF receptor I-mediated apoptosis via two sequential signaling complexes”. Cell 114 (2): 181–90. (July 2003). doi:10.1016/S0092-8674(03)00521-X. PMID 12887920.

nih.gov

pubmed.ncbi.nlm.nih.gov

“The human APO-1 (APT) antigen maps to 10q23, a region that is syntenic with mouse chromosome 19”. Genomics 14 (1): 179–80. (September 1992). doi:10.1016/S0888-7543(05)80302-7. PMID 1385299.
“Assignment of the human Fas antigen gene (Fas) to 10q24.1”. Genomics 14 (3): 821–2. (November 1992). doi:10.1016/S0888-7543(05)80200-9. PMID 1385309.
“Early work on the function of CD95, an interview with Shige Nagata”. Cell Death and Differentiation 11 Suppl 1 (Suppl 1): S23-7. (July 2004). doi:10.1038/sj.cdd.4401453. PMID 15143352.
“The Fas signaling pathway: more than a paradigm”. Science 296 (5573): 1635–6. (May 2002). Bibcode: 2002Sci...296.1635W. doi:10.1126/science.1071553. PMID 12040174.
“Regulation of Fas alternative splicing by antagonistic effects of TIA-1 and PTB on exon definition”. Molecular Cell 19 (4): 475–84. (August 2005). doi:10.1016/j.molcel.2005.06.015. PMID 16109372.
“The Fas-FADD death domain complex structure reveals the basis of DISC assembly and disease mutations”. Nature Structural & Molecular Biology 17 (11): 1324–9. (November 2010). doi:10.1038/nsmb.1920. PMC 2988912. PMID 20935634.
“NMR structure and mutagenesis of the Fas (APO-1/CD95) death domain”. Nature 384 (6610): 638–41. (1996). Bibcode: 1996Natur.384..638H. doi:10.1038/384638a0. PMID 8967952.
“NMR structure and mutagenesis of the FADD (Mort1) death-effector domain”. Nature 392 (6679): 941–5. (April 1998). Bibcode: 1998Natur.392..941E. doi:10.1038/31972. PMID 9582077.
“CD95 promotes tumour growth”. Nature 465 (7297): 492–6. (May 2010). Bibcode: 2010Natur.465..492C. doi:10.1038/nature09075. PMC 2879093. PMID 20505730.
“NF-κB directly regulates Fas transcription to modulate Fas-mediated apoptosis and tumor suppression”. The Journal of Biological Chemistry 287 (30): 25530–40. (July 2012). doi:10.1074/jbc.M112.356279. PMC 3408167. PMID 22669972.
“Decitabine and vorinostat cooperate to sensitize colon carcinoma cells to Fas ligand-induced apoptosis in vitro and tumor suppression in vivo”. Journal of Immunology 188 (9): 4441–9. (May 2012). doi:10.4049/jimmunol.1103035. PMC 3398838. PMID 22461695.
“Lysis of bystander target cells after triggering of human cytotoxic T lymphocytes”. European Journal of Immunology 16 (8): 1021–4. (August 1986). doi:10.1002/eji.1830160826. PMID 3488908.
“Fas ligand-mediated lysis of self bystander targets by human papillomavirus-specific CD8+ cytotoxic T lymphocytes”. Journal of Virology 72 (7): 5948–54. (July 1998). doi:10.1128/JVI.72.7.5948-5954.1998. PMC 110399. PMID 9621057.
“Bispecific T cell engager (BiTE®) antibody constructs can mediate bystander tumor cell killing”. PLOS ONE 12 (8): e0183390. (2017-08-24). Bibcode: 2017PLoSO..1283390R. doi:10.1371/journal.pone.0183390. PMC 5570333. PMID 28837681.
“A critical role for fas-mediated off-target tumor killing in T cell immunotherapy”. Cancer Discovery 11 (3): 599–613. (December 2020). doi:10.1158/2159-8290.CD-20-0756. PMC 7933082. PMID 33334730.
“Cytoskeleton-mediated death receptor and ligand concentration in lipid rafts forms apoptosis-promoting clusters in cancer chemotherapy”. The Journal of Biological Chemistry 280 (12): 11641–7. (March 2005). doi:10.1074/jbc.M411781200. PMID 15659383.
“Identification and molecular cloning of two novel receptors for the cytotoxic ligand TRAIL”. The Journal of Biological Chemistry 272 (41): 25417–20. (October 1997). doi:10.1074/jbc.272.41.25417. PMID 9325248.
“Casper is a FADD- and caspase-related inducer of apoptosis”. Immunity 6 (6): 751–63. (June 1997). doi:10.1016/S1074-7613(00)80450-1. PMID 9208847.
“Fas-associated death domain protein interleukin-1beta-converting enzyme 2 (FLICE2), an ICE/Ced-3 homologue, is proximally involved in CD95- and p55-mediated death signaling”. The Journal of Biological Chemistry 272 (10): 6578–83. (March 1997). doi:10.1074/jbc.272.10.6578. PMID 9045686.
“The receptor for the cytotoxic ligand TRAIL”. Science 276 (5309): 111–3. (April 1997). doi:10.1126/science.276.5309.111. PMID 9082980.
“NMR structure and mutagenesis of the Fas (APO-1/CD95) death domain”. Nature 384 (6610): 638–41. (1996). Bibcode: 1996Natur.384..638H. doi:10.1038/384638a0. PMID 8967952.
“FADD, a novel death domain-containing protein, interacts with the death domain of Fas and initiates apoptosis”. Cell 81 (4): 505–12. (May 1995). doi:10.1016/0092-8674(95)90071-3. PMID 7538907.
“Regulation of Fas-associated death domain interactions by the death effector domain identified by a modified reverse two-hybrid screen”. The Journal of Biological Chemistry 277 (37): 34343–8. (September 2002). doi:10.1074/jbc.M204169200. PMID 12107169.
“Induction of TNF receptor I-mediated apoptosis via two sequential signaling complexes”. Cell 114 (2): 181–90. (July 2003). doi:10.1016/S0092-8674(03)00521-X. PMID 12887920.
“Identification of amino acid residues important for ligand binding to Fas”. The Journal of Experimental Medicine 185 (8): 1487–92. (April 1997). doi:10.1084/jem.185.8.1487. PMC 2196280. PMID 9126929.
“Characterization of Fas (Apo-1, CD95)-Fas ligand interaction”. The Journal of Biological Chemistry 272 (30): 18827–33. (July 1997). doi:10.1074/jbc.272.30.18827. PMID 9228058.
“Apoptosis-linked gene 2 binds to the death domain of Fas and dissociates from Fas during Fas-mediated apoptosis in Jurkat cells”. Biochemical and Biophysical Research Communications 288 (2): 420–6. (October 2001). doi:10.1006/bbrc.2001.5769. PMID 11606059.
“Protection against Fas/APO-1- and tumor necrosis factor-mediated cell death by a novel protein, sentrin”. Journal of Immunology 157 (10): 4277–81. (November 1996). PMID 8906799.
“Interaction of Daxx, a Fas binding protein, with sentrin and Ubc9”. Biochemical and Biophysical Research Communications 279 (1): 6–10. (December 2000). doi:10.1006/bbrc.2000.3882. PMID 11112409.

ncbi.nlm.nih.gov

unil.ch

serval.unil.ch

“Characterization of Fas (Apo-1, CD95)-Fas ligand interaction”. The Journal of Biological Chemistry 272 (30): 18827–33. (July 1997). doi:10.1074/jbc.272.30.18827. PMID 9228058.

web.archive.org

“Tumorscape”. The Broad Institute. 2012年4月14日時点のオリジナルよりアーカイブ。2012年7月5日閲覧。

wikipedia.org

en.wikipedia.org

GRCh38: Ensembl release 89: ENSG00000026103 - Ensembl, May 2017
GRCm38: Ensembl release 89: ENSMUSG00000024778 - Ensembl, May 2017

wiley.com

onlinelibrary.wiley.com

“Lysis of bystander target cells after triggering of human cytotoxic T lymphocytes”. European Journal of Immunology 16 (8): 1021–4. (August 1986). doi:10.1002/eji.1830160826. PMID 3488908.