“Arabidopsis DNA ligase IV is induced by gamma-irradiation and interacts with an Arabidopsis homologue of the double strand break repair protein XRCC4”. Plant J.24 (1): 67–78. (October 2000). doi:10.1046/j.1365-313x.2000.00856.x. PMID11029705.
“An updated meta-analysis of XRCC4 polymorphisms and cancer risk based on 31 case-control studies”. Cancer Biomark12 (1): 37–47. (2012). doi:10.3233/CBM-120292. PMID23321468.
“Endogenous DNA damage in humans: a review of quantitative data”. Mutagenesis19 (3): 169–85. (May 2004). doi:10.1093/mutage/geh025. PMID15123782.
“Flexibility in the order of action and in the enzymology of the nuclease, polymerases, and ligase of vertebrate non-homologous DNA end joining: relevance to cancer, aging, and the immune system”. Cell Res.18 (1): 125–33. (January 2008). doi:10.1038/cr.2007.108. PMID18087292.
“Gene variants of XRCC4 and XRCC3 and their association with risk for urothelial bladder cancer”. Mol. Biol. Rep.39 (2): 1667–75. (February 2012). doi:10.1007/s11033-011-0906-z. PMID21617942.
“Association of functional polymorphisms of the XRCC4 gene with the risk of breast cancer: a meta-analysis”. Asian Pac. J. Cancer Prev.13 (7): 3431–6. (2012). doi:10.7314/APJCP.2012.13.7.3431. PMID22994773.
“Do polymorphisms in XRCC4 influence prostate cancer susceptibility in North Indian population?”. Biomarkers16 (3): 236–42. (May 2011). doi:10.3109/1354750X.2010.547599. PMID21506695.
“DNA repair genes polymorphisms in multiple myeloma: no association with XRCC1 (Arg399Gln) polymorphism, but the XRCC4 (VNTR in intron 3 and G-1394T) and XPD (Lys751Gln) polymorphisms is associated with the disease in Turkish patients”. Hematology16 (6): 361–7. (November 2011). doi:10.1179/102453311X13127324303399. PMID22183071.
“The role of XRCC4 in carcinogenesis and anticancer drug discovery”. Recent Pat Anti-Cancer Drug Discov3 (3): 209–19. (November 2008). doi:10.2174/157489208786242304. PMID18991789.
“Identification of human autoantibodies to the DNA ligase IV/XRCC4 complex and mapping of an autoimmune epitope to a potential regulatory region”. J. Immunol.169 (6): 3413–21. (September 2002). doi:10.4049/jimmunol.169.6.3413. PMID12218164.
“Autoantibodies against DNA double-strand break repair proteins”. Front. Biosci.6: D1412–22. (November 2001). doi:10.2741/Takeda. PMID11689355.
“XRCC4 codon 247*A and XRCC4 promoter -1394*T related genotypes but not XRCC4 intron 3 gene polymorphism are associated with higher susceptibility for endometriosis”. Mol. Reprod. Dev.75 (5): 946–51. (May 2008). doi:10.1002/mrd.20829. PMID18246529.
“RNAi-mediated targeting of noncoding and coding sequences in DNA repair gene messages efficiently radiosensitizes human tumor cells”. Cancer Res.72 (5): 1221–8. (March 2012). doi:10.1158/0008-5472.CAN-11-2785. PMID22237628.
“Cell cycle-dependent repair of double-strand DNA breaks in a gamma-ray-sensitive Chinese hamster cell”. Somat. Cell Mol. Genet.11 (5): 485–91. (September 1985). doi:10.1007/BF01534842. PMID3862244.
“Genetic analysis of XR-1 mutation in hamster and human hybrids”. Somat. Cell Mol. Genet.15 (1): 71–7. (January 1989). doi:10.1007/BF01534671. PMID2916163.
“Arabidopsis DNA ligase IV is induced by gamma-irradiation and interacts with an Arabidopsis homologue of the double strand break repair protein XRCC4”. Plant J.24 (1): 67–78. (October 2000). doi:10.1046/j.1365-313x.2000.00856.x. PMID11029705.
“An updated meta-analysis of XRCC4 polymorphisms and cancer risk based on 31 case-control studies”. Cancer Biomark12 (1): 37–47. (2012). doi:10.3233/CBM-120292. PMID23321468.
“Endogenous DNA damage in humans: a review of quantitative data”. Mutagenesis19 (3): 169–85. (May 2004). doi:10.1093/mutage/geh025. PMID15123782.
“Flexibility in the order of action and in the enzymology of the nuclease, polymerases, and ligase of vertebrate non-homologous DNA end joining: relevance to cancer, aging, and the immune system”. Cell Res.18 (1): 125–33. (January 2008). doi:10.1038/cr.2007.108. PMID18087292.
“[Non-homologous DNA end joining--new proteins, new functions, new mechanisms]” (ポーランド語). Postepy Biochem.55 (1): 36–45. (2009). PMID19514464.
“Gene variants of XRCC4 and XRCC3 and their association with risk for urothelial bladder cancer”. Mol. Biol. Rep.39 (2): 1667–75. (February 2012). doi:10.1007/s11033-011-0906-z. PMID21617942.
“Association of functional polymorphisms of the XRCC4 gene with the risk of breast cancer: a meta-analysis”. Asian Pac. J. Cancer Prev.13 (7): 3431–6. (2012). doi:10.7314/APJCP.2012.13.7.3431. PMID22994773.
“Do polymorphisms in XRCC4 influence prostate cancer susceptibility in North Indian population?”. Biomarkers16 (3): 236–42. (May 2011). doi:10.3109/1354750X.2010.547599. PMID21506695.
“DNA repair genes polymorphisms in multiple myeloma: no association with XRCC1 (Arg399Gln) polymorphism, but the XRCC4 (VNTR in intron 3 and G-1394T) and XPD (Lys751Gln) polymorphisms is associated with the disease in Turkish patients”. Hematology16 (6): 361–7. (November 2011). doi:10.1179/102453311X13127324303399. PMID22183071.
“The role of XRCC4 in carcinogenesis and anticancer drug discovery”. Recent Pat Anti-Cancer Drug Discov3 (3): 209–19. (November 2008). doi:10.2174/157489208786242304. PMID18991789.
“Identification of human autoantibodies to the DNA ligase IV/XRCC4 complex and mapping of an autoimmune epitope to a potential regulatory region”. J. Immunol.169 (6): 3413–21. (September 2002). doi:10.4049/jimmunol.169.6.3413. PMID12218164.
“Autoantibodies against DNA double-strand break repair proteins”. Front. Biosci.6: D1412–22. (November 2001). doi:10.2741/Takeda. PMID11689355.
“XRCC4 codon 247*A and XRCC4 promoter -1394*T related genotypes but not XRCC4 intron 3 gene polymorphism are associated with higher susceptibility for endometriosis”. Mol. Reprod. Dev.75 (5): 946–51. (May 2008). doi:10.1002/mrd.20829. PMID18246529.
“RNAi-mediated targeting of noncoding and coding sequences in DNA repair gene messages efficiently radiosensitizes human tumor cells”. Cancer Res.72 (5): 1221–8. (March 2012). doi:10.1158/0008-5472.CAN-11-2785. PMID22237628.
“Cell cycle-dependent repair of double-strand DNA breaks in a gamma-ray-sensitive Chinese hamster cell”. Somat. Cell Mol. Genet.11 (5): 485–91. (September 1985). doi:10.1007/BF01534842. PMID3862244.
“Genetic analysis of XR-1 mutation in hamster and human hybrids”. Somat. Cell Mol. Genet.15 (1): 71–7. (January 1989). doi:10.1007/BF01534671. PMID2916163.