Slokdarmkanker (Dutch Wikipedia)

Analysis of information sources in references of the Wikipedia article "Slokdarmkanker" in Dutch language version.

refsWebsite

Global rank Dutch rank

11nih.gov

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2doi.org

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2cancer.gov

1,684^th place

7,668^th place

1bmj.com

1,226^th place

337^th place

1oxfordjournals.org

1,389^th place

539^th place

1web.archive.org

1^st place

bmj.com

jcp.bmj.com

Syrjänen KJ (2002). HPV infections and oesophageal cancer. J. Clin. Pathol. 55 (10): 721–8. PMID 12354793.

cancer.gov

Corley DA, Kerlikowske K, Verma R, Buffler P. Protective association of aspirin/NSAIDs and esophageal cancer: a systematic review and meta-analysis. Gastroenterology 2003;124:47-56. PMID 12512029. See also NCI - "Esophageal Cancer (PDQ®): Prevention"
NCI Prevention: Dietary Factors, based on Chainani-Wu N. Diet and oral, pharyngeal, and esophageal cancer. Nutr Cancer 2002;44:104-26. PMID 12734057.

doi.org

Merry AH, Schouten LJ, Goldbohm RA, van den Brandt PA (2007). Body Mass Index, height and risk of adenocarcinoma of the oesophagus and gastric cardia: a prospective cohort study. PMID 17337464. DOI: 10.1136/gut.2006.116665.
It is postulated that H. pylori prevents chronic gastritis, which is a risk factor for reflux, which in turn is a risk factor for esophageal adenocarcinoma: Nakajima S, Hattori T (2004). Oesophageal adenocarcinoma or gastric cancer with or without eradication of Helicobacter pylori infection in chronic atrophic gastritis patients: a hypothetical opinion from a systematic review. Aliment. Pharmacol. Ther. 20 Suppl 1: 54–61. PMID 15298606. DOI: 10.1111/j.1365-2036.2004.01975.x. Gearchiveerd van origineel op 29 mei 2021. Geraadpleegd op 11 april 2008.

nih.gov

ncbi.nlm.nih.gov

Lagergren J, Bergström R, Lindgren A, Nyrén O (1999). Symptomatic gastroesophageal reflux as a risk factor for esophageal adenocarcinoma. N. Engl. J. Med. 340 (11): 825-31. PMID 10080844.
Syrjänen KJ (2002). HPV infections and oesophageal cancer. J. Clin. Pathol. 55 (10): 721–8. PMID 12354793.
Green PH, Fleischauer AT, Bhagat G, Goyal R, Jabri B, Neugut AI (2003). Risk of malignancy in patients with celiac disease. Am. J. Med. 115 (3): 191-5. PMID 12935825.
Merry AH, Schouten LJ, Goldbohm RA, van den Brandt PA (2007). Body Mass Index, height and risk of adenocarcinoma of the oesophagus and gastric cardia: a prospective cohort study. PMID 17337464. DOI: 10.1136/gut.2006.116665.
Layke JC, Lopez PP (2006). Esophageal cancer: a review and update. American family physician 73 (12): 2187-94. PMID 16836035.
Corley DA, Kerlikowske K, Verma R, Buffler P. Protective association of aspirin/NSAIDs and esophageal cancer: a systematic review and meta-analysis. Gastroenterology 2003;124:47-56. PMID 12512029. See also NCI - "Esophageal Cancer (PDQ®): Prevention"
Wong A, Fitzgerald RC (2005). Epidemiologic risk factors for Barrett's esophagus and associated adenocarcinoma. Clin. Gastroenterol. Hepatol. 3 (1): 1–10. PMID 15645398.
Ye W, Held M, Lagergren J, et al (2004). Helicobacter pylori infection and gastric atrophy: risk of adenocarcinoma and squamous-cell carcinoma of the esophagus and adenocarcinoma of the gastric cardia. J. Natl. Cancer Inst. 96 (5): 388–96. PMID 14996860.
It is postulated that H. pylori prevents chronic gastritis, which is a risk factor for reflux, which in turn is a risk factor for esophageal adenocarcinoma: Nakajima S, Hattori T (2004). Oesophageal adenocarcinoma or gastric cancer with or without eradication of Helicobacter pylori infection in chronic atrophic gastritis patients: a hypothetical opinion from a systematic review. Aliment. Pharmacol. Ther. 20 Suppl 1: 54–61. PMID 15298606. DOI: 10.1111/j.1365-2036.2004.01975.x. Gearchiveerd van origineel op 29 mei 2021. Geraadpleegd op 11 april 2008.
NCI Prevention: Dietary Factors, based on Chainani-Wu N. Diet and oral, pharyngeal, and esophageal cancer. Nutr Cancer 2002;44:104-26. PMID 12734057.
Tavani, A, Bertuzzi M, Talamini R, Gallus S, Parpinel M, Franceschi S, Levi F, La Vecchia C. (10 2003). Coffee and tea intake and risk of oral, pharyngeal and esophageal cancer. Oral Oncol. 39 (7): 695-700. PMID 12907209.

oxfordjournals.org

jnci.oxfordjournals.org

Ye W, Held M, Lagergren J, et al (2004). Helicobacter pylori infection and gastric atrophy: risk of adenocarcinoma and squamous-cell carcinoma of the esophagus and adenocarcinoma of the gastric cardia. J. Natl. Cancer Inst. 96 (5): 388–96. PMID 14996860.

web.archive.org

It is postulated that H. pylori prevents chronic gastritis, which is a risk factor for reflux, which in turn is a risk factor for esophageal adenocarcinoma: Nakajima S, Hattori T (2004). Oesophageal adenocarcinoma or gastric cancer with or without eradication of Helicobacter pylori infection in chronic atrophic gastritis patients: a hypothetical opinion from a systematic review. Aliment. Pharmacol. Ther. 20 Suppl 1: 54–61. PMID 15298606. DOI: 10.1111/j.1365-2036.2004.01975.x. Gearchiveerd van origineel op 29 mei 2021. Geraadpleegd op 11 april 2008.