KarolinaK.Kulik-KupkaKarolinaK. i inni, Wpływ dysruptorów endokrynnych na funkcje organizmu, „Postępy Higieny i Medycyny Doświadczalnej”, 71, 2017, s. 1231–1238, DOI: 10.5604/01.3001.0010.7748.
Frederick S. vomF.S.SaalFrederick S. vomF.S. i inni, Chapel Hill bisphenol A expert panel consensus statement: integration of mechanisms, effects in animals and potential to impact human health at current levels of exposure, „Reproductive Toxicology”, 24 (2), 2007, s. 131–138, DOI: 10.1016/j.reprotox.2007.07.005, PMID: 17768031, PMCID: PMC2967230.
EleniE.KandarakiEleniE. i inni, Endocrine disruptors and polycystic ovary syndrome (PCOS): elevated serum levels of bisphenol A in women with PCOS, „The Journal of Clinical Endocrinology and Metabolism”, 96 (3), 2011, E480–E484, DOI: 10.1210/jc.2010-1658, PMID: 21193545.
Mikhail Y.M.Y.KochukovMikhail Y.M.Y., Yow-JiunY.J.JengYow-JiunY.J., Cheryl S.Ch.S.WatsonCheryl S.Ch.S., Alkylphenol xenoestrogens with varying carbon chain lengths differentially and potently activate signaling and functional responses in GH3/B6/F10 somatomammotropes, „Environmental Health Perspectives”, 117 (5), 2009, s. 723–730, DOI: 10.1289/ehp.0800182, PMID: 19479013, PMCID: PMC2685833.
Johanna R.J.R.RochesterJohanna R.J.R., Ashley L.A.L.BoldenAshley L.A.L., Bisphenol S and F: A Systematic Review and Comparison of the Hormonal Activity of Bisphenol A Substitutes, „Environmental Health Perspectives”, 123 (7), 2015, s. 643–650, DOI: 10.1289/ehp.1408989, PMID: 25775505, PMCID: PMC4492270(ang.).
Stijn L.S.L.VerhulstStijn L.S.L. i inni, Intrauterine Exposure to Environmental Pollutants and Body Mass Index during the First 3 Years of Life, „Environmental Health Perspectives”, 117 (1), 2009, s. 122–126, DOI: 10.1289/ehp.0800003, PMID: 19165398, PMCID: PMC2627855(ang.).
James S.J.S.BrownJames S.J.S., jr., Effects of Bisphenol-A and Other Endocrine Disruptors Compared With Abnormalities of Schizophrenia: An Endocrine-Disruption Theory of Schizophrenia, „Schizophr Bull”, 35(1), 2009, s. 256–278, DOI: 10.1093/schbul/sbm147, PMCID: PMC2643957(ang.).
N.N.SzczepańskaN.N., B.B.KudłakB.B., J.J.NamieśnikJ.J., Assessing ecotoxicity and the endocrine potential of selected phthalates, BADGE and BFDGE derivatives in relation to environmentally detectable levels, „Sci. Tot. Environ.”, 2018, 610-611:854-866, DOI: 10.1016/j.scitotenv.2017.08.160, PMID: 28826123(ang.).
Michael J.M.J.WilliamsMichael J.M.J. i inni, Dibutyl Phthalate Exposure Disrupts Evolutionarily Conserved Insulin and Glucagon-Like Signaling in Drosophila Males, „Endocrinology”, 157 (6), 2016, s. 2309–2321, DOI: 10.1210/en.2015-2006, PMID: 27100621(ang.).
DD.MullerovaDD. i inni, Negative association between plasma levels of adiponectin and polychlorinated biphenyl 153 in obese women under non-energy-restrictive regime, „International Journal of Obesity”, 32 (12), 2008, s. 1875–1878, DOI: 10.1038/ijo.2008.169, PMID: 18825156(ang.).
Andrea AA.A.HoppeAndrea AA.A., Gale BG.B.CareyGale BG.B., Polybrominated diphenyl ethers as endocrine disruptors of adipocyte metabolism, „Obesity”, 15, 2007, s. 2942–50, DOI: 10.1038/oby.2007.351, PMID: 18198302(ang.).
Sally S.S.S.WhiteSally S.S.S., Suzanne E.S.E.FentonSuzanne E.S.E., Erin P.E.P.HinesErin P.E.P., Endocrine disrupting properties of perfluorooctanoic acid, „The Journal of Steroid Biochemistry and Molecular Biology”, 127 (1–2), 2011, s. 16–26, DOI: 10.1016/j.jsbmb.2011.03.011, PMID: 21397692, PMCID: PMC3335904(ang.).
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AleksandraA.RutkowskaAleksandraA. i inni, Polish Society of Endocrinology Position statement on endocrine disrupting chemicals (EDCs), „Endokrynologia Polska”, 66 (3), 2015, s. 276–281, DOI: 10.5603/EP.2015.0035, PMID: 26136137.
L.L.CobellisL.L. i inni, High plasma concentrations of di-(2-ethylhexyl)-phthalate in women with endometriosis, „Human Reproduction”, 18 (7), 2003, s. 1512–1515, DOI: 10.1093/humrep/deg254, PMID: 12832380.
Laura N.L.N.VandenbergLaura N.L.N. i inni, Human exposure to bisphenol A (BPA), „Reproductive Toxicology (Elmsford, N.Y.)”, 24 (2), 2007, s. 139–177, DOI: 10.1016/j.reprotox.2007.07.010, PMID: 17825522.
YumikoY.IkezukiYumikoY. i inni, Determination of bisphenol A concentrations in human biological fluids reveals significant early prenatal exposure, „Human Reproduction (Oxford, England)”, 17 (11), 2002, s. 2839–2841, DOI: 10.1093/humrep/17.11.2839, PMID: 12407035.
Laura N.L.N.VandenbergLaura N.L.N. i inni, Urinary, circulating, and tissue biomonitoring studies indicate widespread exposure to bisphenol A, „Environmental Health Perspectives”, 118 (8), 2010, s. 1055–1070, DOI: 10.1289/ehp.0901716, PMID: 20338858, PMCID: PMC2920080.
M.F.M.F.FernandezM.F.M.F. i inni, Bisphenol-A and chlorinated derivatives in adipose tissue of women, „Reproductive Toxicology”, 24 (2), 2007, s. 259–264, DOI: 10.1016/j.reprotox.2007.06.007, PMID: 17689919(ang.).
M. deM.CockM. deM., Y.G.Y.G.MaasY.G.Y.G., M. van deM.BorM. van deM., Does perinatal exposure to endocrine disruptors induce autism spectrum and attention deficit hyperactivity disorders? Review, „Acta Paediatr.”, 101, 2012, s. 811–818, DOI: 10.1111/j.1651-2227.2012.02693.x., PMID: 22458970(ang.).
Linda SL.S.BirnbaumLinda SL.S., Suzanne ES.E.FentonSuzanne ES.E., Cancer and developmental exposure to endocrine disruptors, „Environ Health Perspectives”, 111, 2003, s. 389–394, DOI: 10.1111/j.1749-6632.2001.tb03994.x, PMID: 12676588, PMCID: PMC1241417(ang.).
M.M.BoasM.M., U.U.Feldt-RasmussenU.U., K.M.K.M.MainK.M.K.M., Thyroid effects of endocrine disrupting chemicals, „Molecular and Cellular Endocrinology”, 355, 2012, s. 240–248, DOI: 10.1016/j.mce.2011.09.005., PMID: 21939731, PMCID: PMC1241417(ang.).
Heather B.H.B.PatisaulHeather B.H.B., Heather B.H.B.AdewaleHeather B.H.B., Long-Term Effects of Environmental Endocrine Disruptors on Reproductive Physiology and Behavior, „Front Behav Neurosci”, DOI: 10.3389/neuro.08.010.2009, PMID: 19587848, PMCID: PMC2706654(ang.).
TolgaT.ÜnüvarTolgaT., AtillaA.BüyükgebizAtillaA., Fetal and Neonatal Endocrine Disruptors, „J Clin Res Pediatr Endocrinol.”, 4, 2012, s. 51–60, DOI: 10.4274/jcrpe.569, PMID: 22672860, PMCID: PMC3386773.
TolgaT.ÜnüvarTolgaT., AtillaA.BüyükgebizAtillaA., Developmental programming: impact of excess prenatal testosterone on intrauterine fetal endocrine milieu and growth in sheep, „Biol Reprod”, 87–96, 2011, s. 1, DOI: 10.1095/biolreprod.110.086686, PMID: 22672860, PMCID: PMC3012564.
YukimasaY.TakedaYukimasaY. i inni, Placenta expressing the greatest quantity of bisphenol A receptor ERR{gamma} among the human reproductive tissues: Predominant expression of type-1 ERRgamma isoform, „J Biochem.”, 146 (1), 2009, s. 113–22, DOI: 10.1093/jb/mvp049, PMID: 19304792.
MeghanM.HewlettMeghanM. i inni, Prenatal Exposure to Endocrine Disruptors: A Developmental Etiology for Polycystic Ovary Syndrome, „Human Reproduction Update”, 24 (1), 2016, DOI: 10.1177/1933719116654992, PMID: 27342273, PMCID: PMC6344820.
GilbertG.SchönfelderGilbertG. i inni, Developmental effects of prenatal exposure to bisphenol a on the uterus of rat offspring, „Neoplasia”, 6 (5), 2004, s. 584–94, DOI: 10.1593/neo.04217, PMID: 15548368, PMCID: PMC1531663.
M.F.M.F.FernandezM.F.M.F. i inni, Bisphenol-A and chlorinated derivatives in adipose tissue of women, „Reproductive Toxicology”, 24 (2), 2007, s. 259–264, DOI: 10.1016/j.reprotox.2007.06.007, PMID: 17689919.
Frederick S. vomF.S.SaalFrederick S. vomF.S. i inni, Chapel Hill bisphenol A expert panel consensus statement: integration of mechanisms, effects in animals and potential to impact human health at current levels of exposure, „Reproductive Toxicology”, 24 (2), 2007, s. 131–138, DOI: 10.1016/j.reprotox.2007.07.005, PMID: 17768031, PMCID: PMC2967230.
EleniE.KandarakiEleniE. i inni, Endocrine disruptors and polycystic ovary syndrome (PCOS): elevated serum levels of bisphenol A in women with PCOS, „The Journal of Clinical Endocrinology and Metabolism”, 96 (3), 2011, E480–E484, DOI: 10.1210/jc.2010-1658, PMID: 21193545.
Mikhail Y.M.Y.KochukovMikhail Y.M.Y., Yow-JiunY.J.JengYow-JiunY.J., Cheryl S.Ch.S.WatsonCheryl S.Ch.S., Alkylphenol xenoestrogens with varying carbon chain lengths differentially and potently activate signaling and functional responses in GH3/B6/F10 somatomammotropes, „Environmental Health Perspectives”, 117 (5), 2009, s. 723–730, DOI: 10.1289/ehp.0800182, PMID: 19479013, PMCID: PMC2685833.
Johanna R.J.R.RochesterJohanna R.J.R., Ashley L.A.L.BoldenAshley L.A.L., Bisphenol S and F: A Systematic Review and Comparison of the Hormonal Activity of Bisphenol A Substitutes, „Environmental Health Perspectives”, 123 (7), 2015, s. 643–650, DOI: 10.1289/ehp.1408989, PMID: 25775505, PMCID: PMC4492270(ang.).
Stijn L.S.L.VerhulstStijn L.S.L. i inni, Intrauterine Exposure to Environmental Pollutants and Body Mass Index during the First 3 Years of Life, „Environmental Health Perspectives”, 117 (1), 2009, s. 122–126, DOI: 10.1289/ehp.0800003, PMID: 19165398, PMCID: PMC2627855(ang.).
James S.J.S.BrownJames S.J.S., jr., Effects of Bisphenol-A and Other Endocrine Disruptors Compared With Abnormalities of Schizophrenia: An Endocrine-Disruption Theory of Schizophrenia, „Schizophr Bull”, 35(1), 2009, s. 256–278, DOI: 10.1093/schbul/sbm147, PMCID: PMC2643957(ang.).
N.N.SzczepańskaN.N., B.B.KudłakB.B., J.J.NamieśnikJ.J., Assessing ecotoxicity and the endocrine potential of selected phthalates, BADGE and BFDGE derivatives in relation to environmentally detectable levels, „Sci. Tot. Environ.”, 2018, 610-611:854-866, DOI: 10.1016/j.scitotenv.2017.08.160, PMID: 28826123(ang.).
Michael J.M.J.WilliamsMichael J.M.J. i inni, Dibutyl Phthalate Exposure Disrupts Evolutionarily Conserved Insulin and Glucagon-Like Signaling in Drosophila Males, „Endocrinology”, 157 (6), 2016, s. 2309–2321, DOI: 10.1210/en.2015-2006, PMID: 27100621(ang.).
DD.MullerovaDD. i inni, Negative association between plasma levels of adiponectin and polychlorinated biphenyl 153 in obese women under non-energy-restrictive regime, „International Journal of Obesity”, 32 (12), 2008, s. 1875–1878, DOI: 10.1038/ijo.2008.169, PMID: 18825156(ang.).
Andrea AA.A.HoppeAndrea AA.A., Gale BG.B.CareyGale BG.B., Polybrominated diphenyl ethers as endocrine disruptors of adipocyte metabolism, „Obesity”, 15, 2007, s. 2942–50, DOI: 10.1038/oby.2007.351, PMID: 18198302(ang.).
Sally S.S.S.WhiteSally S.S.S., Suzanne E.S.E.FentonSuzanne E.S.E., Erin P.E.P.HinesErin P.E.P., Endocrine disrupting properties of perfluorooctanoic acid, „The Journal of Steroid Biochemistry and Molecular Biology”, 127 (1–2), 2011, s. 16–26, DOI: 10.1016/j.jsbmb.2011.03.011, PMID: 21397692, PMCID: PMC3335904(ang.).
T.M.T.M.CrispT.M.T.M. i inni, Environmental endocrine disruption: an effects assessment and analysis, „Environmental Health Perspectives”, 106 (Suppl 1), 1998, s. 11–56, PMID: 9539004, PMCID: PMC1533291.
AleksandraA.RutkowskaAleksandraA. i inni, Polish Society of Endocrinology Position statement on endocrine disrupting chemicals (EDCs), „Endokrynologia Polska”, 66 (3), 2015, s. 276–281, DOI: 10.5603/EP.2015.0035, PMID: 26136137.
L.L.CobellisL.L. i inni, High plasma concentrations of di-(2-ethylhexyl)-phthalate in women with endometriosis, „Human Reproduction”, 18 (7), 2003, s. 1512–1515, DOI: 10.1093/humrep/deg254, PMID: 12832380.
Laura N.L.N.VandenbergLaura N.L.N. i inni, Human exposure to bisphenol A (BPA), „Reproductive Toxicology (Elmsford, N.Y.)”, 24 (2), 2007, s. 139–177, DOI: 10.1016/j.reprotox.2007.07.010, PMID: 17825522.
YumikoY.IkezukiYumikoY. i inni, Determination of bisphenol A concentrations in human biological fluids reveals significant early prenatal exposure, „Human Reproduction (Oxford, England)”, 17 (11), 2002, s. 2839–2841, DOI: 10.1093/humrep/17.11.2839, PMID: 12407035.
Laura N.L.N.VandenbergLaura N.L.N. i inni, Urinary, circulating, and tissue biomonitoring studies indicate widespread exposure to bisphenol A, „Environmental Health Perspectives”, 118 (8), 2010, s. 1055–1070, DOI: 10.1289/ehp.0901716, PMID: 20338858, PMCID: PMC2920080.
M.F.M.F.FernandezM.F.M.F. i inni, Bisphenol-A and chlorinated derivatives in adipose tissue of women, „Reproductive Toxicology”, 24 (2), 2007, s. 259–264, DOI: 10.1016/j.reprotox.2007.06.007, PMID: 17689919(ang.).
D.D.MüllerováD.D., J.J.KopeckýJ.J., White adipose tissue: storage and effector site for environmental pollutants, „Physiological Research”, 56 (4), 2007, s. 375–381, PMID: 16925464.
M. deM.CockM. deM., Y.G.Y.G.MaasY.G.Y.G., M. van deM.BorM. van deM., Does perinatal exposure to endocrine disruptors induce autism spectrum and attention deficit hyperactivity disorders? Review, „Acta Paediatr.”, 101, 2012, s. 811–818, DOI: 10.1111/j.1651-2227.2012.02693.x., PMID: 22458970(ang.).
Linda SL.S.BirnbaumLinda SL.S., Suzanne ES.E.FentonSuzanne ES.E., Cancer and developmental exposure to endocrine disruptors, „Environ Health Perspectives”, 111, 2003, s. 389–394, DOI: 10.1111/j.1749-6632.2001.tb03994.x, PMID: 12676588, PMCID: PMC1241417(ang.).
M.M.BoasM.M., U.U.Feldt-RasmussenU.U., K.M.K.M.MainK.M.K.M., Thyroid effects of endocrine disrupting chemicals, „Molecular and Cellular Endocrinology”, 355, 2012, s. 240–248, DOI: 10.1016/j.mce.2011.09.005., PMID: 21939731, PMCID: PMC1241417(ang.).
Heather B.H.B.PatisaulHeather B.H.B., Heather B.H.B.AdewaleHeather B.H.B., Long-Term Effects of Environmental Endocrine Disruptors on Reproductive Physiology and Behavior, „Front Behav Neurosci”, DOI: 10.3389/neuro.08.010.2009, PMID: 19587848, PMCID: PMC2706654(ang.).
TolgaT.ÜnüvarTolgaT., AtillaA.BüyükgebizAtillaA., Fetal and Neonatal Endocrine Disruptors, „J Clin Res Pediatr Endocrinol.”, 4, 2012, s. 51–60, DOI: 10.4274/jcrpe.569, PMID: 22672860, PMCID: PMC3386773.
TolgaT.ÜnüvarTolgaT., AtillaA.BüyükgebizAtillaA., Developmental programming: impact of excess prenatal testosterone on intrauterine fetal endocrine milieu and growth in sheep, „Biol Reprod”, 87–96, 2011, s. 1, DOI: 10.1095/biolreprod.110.086686, PMID: 22672860, PMCID: PMC3012564.
YukimasaY.TakedaYukimasaY. i inni, Placenta expressing the greatest quantity of bisphenol A receptor ERR{gamma} among the human reproductive tissues: Predominant expression of type-1 ERRgamma isoform, „J Biochem.”, 146 (1), 2009, s. 113–22, DOI: 10.1093/jb/mvp049, PMID: 19304792.
MeghanM.HewlettMeghanM. i inni, Prenatal Exposure to Endocrine Disruptors: A Developmental Etiology for Polycystic Ovary Syndrome, „Human Reproduction Update”, 24 (1), 2016, DOI: 10.1177/1933719116654992, PMID: 27342273, PMCID: PMC6344820.
B SB.S.RubinB SB.S. i inni, Perinatal exposure to low doses of bisphenol A affects body weight, patterns of estrous cyclicity, and plasma LH levels, „Environmental Health Perspectives”, 109 (7), 2001, s. 675–680, PMID: PMC1240370.
GilbertG.SchönfelderGilbertG. i inni, Developmental effects of prenatal exposure to bisphenol a on the uterus of rat offspring, „Neoplasia”, 6 (5), 2004, s. 584–94, DOI: 10.1593/neo.04217, PMID: 15548368, PMCID: PMC1531663.
M.F.M.F.FernandezM.F.M.F. i inni, Bisphenol-A and chlorinated derivatives in adipose tissue of women, „Reproductive Toxicology”, 24 (2), 2007, s. 259–264, DOI: 10.1016/j.reprotox.2007.06.007, PMID: 17689919.
KazimieraK.Ćwiek-LudwickaKazimieraK., Bisphenol A (BPA) in food contact materials – new scientific opinion from EFSA regarding public health risk, „Roczniki Państwowego Zakładu Higieny”, 66 (4), 2015, s. 299–307, ISSN0035-7715, PMID: 26656411.
niehs.nih.gov
Endocrine Disruptors [online], National Institute of Environmental Health Sciences [dostęp 2018-02-22](ang.).
KazimieraK.Ćwiek-LudwickaKazimieraK., Bisphenol A (BPA) in food contact materials – new scientific opinion from EFSA regarding public health risk, „Roczniki Państwowego Zakładu Higieny”, 66 (4), 2015, s. 299–307, ISSN0035-7715, PMID: 26656411.