الفيسيولوجيا المرضية للتصلب المتعدد (Arabic Wikipedia)

Analysis of information sources in references of the Wikipedia article "الفيسيولوجيا المرضية للتصلب المتعدد" in Arabic language version.

refsWebsite

Global rank Arabic rank

163nih.gov

4^th place

6^th place

110doi.org

2^nd place

5^th place

21web.archive.org

1^st place

8harvard.edu

18^th place

33^rd place

7archive.org

6^th place

3^rd place

2oxfordjournals.org

1,389^th place

656^th place

2ama-assn.org

3,257^th place

7,230^th place

1neurology.org

9,919^th place

7,764^th place

1cam.ac.uk

670^th place

1,191^st place

1neurologyreviews.com

low place

1direct-ms.org

low place

1cpnhelp.org

low place

1emedicine.com

3,595^th place

3,166^th place

1medicalnewstoday.com

3,011^th place

1,505^th place

1if-pan.krakow.pl

low place

1metapress.com

8,671^st place

3,277^th place

1neurosciencenews.com

low place

1uzh.ch

3,633^rd place

4,746^th place

1ajnr.org

low place

1bmj.com

1,226^th place

531^st place

1archive.today

14^th place

41^st place

1wiley.com

222^nd place

221^st place

1angeleshealth.com

low place

1bentham-direct.org

low place

1fondazionehilarescere.org

low place

1worldwidejournals.in

low place

ajnr.org

"Normal-appearing White and Grey Matter Damage in Multiple Sclerosis. Book review". AJNR. ج. 27 ع. 4: 945–946. 2004. مؤرشف من الأصل في 2011-09-27.

ama-assn.org

archneur.ama-assn.org

"Adhesion molecules in multiple sclerosis: relation to subtypes of disease and methylprednisolone therapy". Arch. Neurol. ج. 57 ع. 4: 546–51. أبريل 2000. DOI:10.1001/archneur.57.4.546. PMID:10768630. مؤرشف من الأصل في 2018-04-03.
"Imaging correlates of leukocyte accumulation and CXCR4/CXCR12 in multiple sclerosis". Arch. Neurol. ج. 66 ع. 1: 44–53. يناير 2009. DOI:10.1001/archneurol.2008.512. PMC:2792736. PMID:19139298. مؤرشف من الأصل في 2018-04-03.

angeleshealth.com

Bryce Weir (2010). J Neuro Sci 2010 - MS Vascular Etiology.pdf "MS, A vascular ethiology?" (PDF). Can. J. Neurol. Sci. ج. 37 ع. 6: 745–757. DOI:10.1017/s0317167100051404. PMID:21059535. مؤرشف من الأصل (PDF) في 2011-09-15. {{استشهاد بدورية محكمة}}: تحقق من قيمة |مسار أرشيف= (مساعدة)

archive.org

"Dysregulated Epstein-Barr virus infection in the multiple sclerosis brain". Journal of Experimental Medicine. ج. 204 ع. 12: 2899–2912. نوفمبر 2007. DOI:10.1084/jem.20071030. PMC:2118531. PMID:17984305.
"Homogeneity of active demyelinating lesions in established multiple sclerosis". Annals of Neurology. ج. 63 ع. 1: 16–25. 2008. DOI:10.1002/ana.21311. PMID:18232012.
"Neuromyelitis optica lesions may inform multiple sclerosis heterogeneity debate". Ann Neurol. ج. 72 ع. 3: 385–94. سبتمبر 2012. DOI:10.1002/ana.23621. PMID:23034911.
"Monoclonal antibody treatment exposes three mechanisms underlying the clinical course of multiple sclerosis". Ann Neurol. ج. 46 ع. 3: 296–304. 1999. DOI:10.1002/1531-8249(199909)46:3<296::AID-ANA4>3.0.CO;2-#. PMID:10482259.
"No cerebrocervical venous congestion in patients with multiple sclerosis". Annals of Neurology. ج. 68 ع. 2: 173–83. أغسطس 2010. DOI:10.1002/ana.22085. PMID:20695010.
"Venous and cerebrospinal fluid flow in multiple sclerosis: A case-control study". Annals of Neurology. ج. 68 ع. 2: 255–259. 2010. DOI:10.1002/ana.22132. PMID:20695018.
"Antibodies reactive to heat shock protein 90 induce oligodendrocyte precursor cell death in culture. Implications for demyelination in multiple sclerosis". FASEB J. ج. 18 ع. 2: 409–11. فبراير 2004. DOI:10.1096/fj.03-0606fje. PMID:14688203.{{استشهاد بدورية محكمة}}: صيانة الاستشهاد: دوي مجاني غير معلم (link)

archive.today

"Chronic cerebrospinal venous insufficiency and multiple sclerosis". Annals of Neurology. ج. 67 ع. 3: 286–90. مارس 2010. DOI:10.1002/ana.22001. PMID:20373339. مؤرشف من الأصل في 2010-11-23.

bentham-direct.org

"Intracranial venous haemodynamics in multiple sclerosis". Curr Neurovasc Res. ج. 4 ع. 4: 252–8. نوفمبر 2007. DOI:10.2174/156720207782446298. PMID:18045150. مؤرشف من الأصل في 2018-10-19.

bmj.com

jnnp.bmj.com

"Chronic cerebrospinal venous insufficiency in patients with multiple sclerosis". J. Neurol. Neurosurg. Psychiatry. ج. 80 ع. 4: 392–9. أبريل 2009. DOI:10.1136/jnnp.2008.157164. PMC:2647682. PMID:19060024. مؤرشف من الأصل في 2009-08-04.

cam.ac.uk

immserv1.path.cam.ac.uk

Holmes، Nick (15 نوفمبر 2001). "Part 1B Pathology: Lecture 11 - The Complement System". مؤرشف من الأصل في 2006-01-09. اطلع عليه بتاريخ 2006-05-10.

cpnhelp.org

Michael H. Barnett؛ John W. Prineas (2004). "Relapsing and Remitting Multiple Sclerosis: Pathology of the Newly Forming Lesion" (PDF). Annals of Neurology. ج. 55 ع. 1: 458–468. DOI:10.1002/ana.20016. PMID:15048884. مؤرشف من الأصل (PDF) في 2017-03-01.

direct-ms.org

Barnett، MH؛ Prineas، JW (أبريل 2004). "Relapsing and remitting multiple sclerosis: pathology of the newly forming lesion" (PDF). Annals of Neurology. ج. 55 ع. 4: 458–68. DOI:10.1002/ana.20016. PMID:15048884. مؤرشف من الأصل (PDF) في 2013-10-29. اطلع عليه بتاريخ 2009-10-28.

doi.org

Golan، Daniel؛ Staun-Ram، Elsebeth؛ Miller، Ariel (2016). "Shifting paradigms in multiple sclerosis". Current Opinion in Neurology. ج. 29 ع. 3: 354–361. DOI:10.1097/WCO.0000000000000324. PMID:27070218.
"Imaging in multiple sclerosis: A new spin on lesions". Med. Imag. And Radiation Oncology. ج. 60 ع. 5: 577–586. 27 يوليو 2016. DOI:10.1111/1754-9485.12498. PMID:27464473.
"Perivenous demyelination: association with clinically defined acute disseminated encephalomyelitis and comparison with pathologically confirmed multiple sclerosis". Brain. ج. 133 ع. 2: 333–348. 2010. DOI:10.1093/brain/awp321. PMC:2822631. PMID:20129932.
Lassman H (مارس 2019). "The changing concepts in the neuropathology of acquired demyelinating central nervous system disorders". Curr Opin Neurol. ج. 32 ع. 3: 313–319. DOI:10.1097/WCO.0000000000000685. PMID:30893100.
Lassman H (2019). "Pathogenic Mechanisms Associated With Different Clinical Courses of Multiple Sclerosis". Front Immunol. ج. 9: 3116. DOI:10.3389/fimmu.2018.03116. PMC:6335289. PMID:30687321.{{استشهاد بدورية محكمة}}: صيانة الاستشهاد: دوي مجاني غير معلم (link)
"Preactive lesions in multiple sclerosis". Curr Opin Neurol. ج. 22 ع. 3: 207–13. 2009. DOI:10.1097/WCO.0b013e32832b4c76. PMID:19417567.
"Alpha-B-Crystallin Induces an Immune-Regulatory and Antiviral Microglial Response in Preactive Multiple Sclerosis Lesions". J Neuropathol Exp Neurol. ج. 72 ع. 10: 970–9. 2013. DOI:10.1097/NEN.0b013e3182a776bf. PMID:24042199.
Ontaneda؛ وآخرون (نوفمبر 2014). "Identifying the Start of Multiple Sclerosis Injury: A Serial DTI Study". J Neuroimaging. ج. 24 ع. 6: 569–76. DOI:10.1111/jon.12082. PMC:4221810. PMID:25370339.
"A serial study of new MS lesions and the white matter from which they arise". Neurology. ج. 51 ع. 6: 1689–97. ديسمبر 1998. DOI:10.1212/wnl.51.6.1689. PMID:9855524. مؤرشف من الأصل في 2008-05-22.
"Clinico-pathological evidence that axonal loss underlies disability in progressive multiple sclerosis". Mult Scler. ج. 16 ع. 4: 406–411. أبريل 2010. DOI:10.1177/1352458510364992. PMID:20215480.
Lassmann Hans (2014). "Multiple sclerosis: Lessons from molecular neuropathology". Experimental Neurology. ج. 262: 2–7. DOI:10.1016/j.expneurol.2013.12.003. PMID:24342027.
"The immunopathology of multiple sclerosis: an overview". Brain Pathol. ج. 17 ع. 2: 210–8. أبريل 2007. DOI:10.1111/j.1750-3639.2007.00064.x. PMID:17388952.
"Gray matter involvement in multiple sclerosis". Neurology. ج. 68 ع. 9: 634–42. فبراير 2007. DOI:10.1212/01.wnl.0000250267.85698.7a. PMID:17325269.
"Compartmentalization of inflammation in the CNS: A major mechanism driving progressive multiple sclerosis". J Neurol Sci. ج. 274 ع. 1–2: 42–4. نوفمبر 2008. DOI:10.1016/j.jns.2008.06.032. PMID:18715571.
"The astrocyte in multiple sclerosis revisited". Glia. ج. 61 ع. 4: 453–465. 2013. DOI:10.1002/glia.22443. PMID:23322421.
"MR Spectroscopy Indicates Diffuse Multiple Sclerosis Activity During Remission". J. Neurol. Neurosurg. Psychiatry. ج. 80 ع. 12: 1330–6. يونيو 2009. DOI:10.1136/jnnp.2009.176263. PMC:2900785. PMID:19546105.
Shinji Oki (مارس 2018). "Novel mechanisms of chronic inflammation in secondary progressive multiple sclerosis". Neuroimmunology. ج. 9 ع. S1: 13–19. DOI:10.1111/cen3.12437.
"Dysregulated Epstein-Barr virus infection in the multiple sclerosis brain". Journal of Experimental Medicine. ج. 204 ع. 12: 2899–2912. نوفمبر 2007. DOI:10.1084/jem.20071030. PMC:2118531. PMID:17984305.
"Cholesterol crystals impede nerve repair". ساينس. ج. 359 ع. 6376: 635–636. 2018. Bibcode:2018Sci...359..635C. DOI:10.1126/science.aar7369. PMID:29439228.
"Defective cholesterol clearance limits remyelination in the aged central nervous system". ساينس. ج. 359 ع. 6376: 684–688. 2018. Bibcode:2018Sci...359..684C. DOI:10.1126/science.aan4183. PMID:29301957.
"Distinct patterns of multiple sclerosis pathology indicates heterogeneity on pathogenesis". Brain Pathol. ج. 6 ع. 3: 259–74. يوليو 1996. DOI:10.1111/j.1750-3639.1996.tb00854.x. PMC:7161824. PMID:8864283.
"A quantitative analysis of oligodendrocytes in multiple sclerosis lesions - A study of 113 cases". Brain. ج. 122 ع. 12: 2279–2295. ديسمبر 1999. DOI:10.1093/brain/122.12.2279. PMID:10581222. مؤرشف من الأصل في 2008-08-29. اطلع عليه بتاريخ 2006-05-10.
"Humoral pattern II multiple sclerosis pathology not associated with neuromyelitis Optica IgG". Arch Neurol. ج. 66 ع. 10: 1298–9. أكتوبر 2009. DOI:10.1001/archneurol.2009.199. PMC:2767176. PMID:19822791.
Srivastava، Rajneesh؛ Aslam، Muhammad؛ Kalluri، Sudhakar Reddy؛ Schirmer، Lucas؛ Buck، Dorothea؛ Tackenberg، Björn؛ Rothhammer، Veit؛ Chan، Andrew؛ Gold، Ralf (2012). "Potassium Channel KIR4.1 as an Immune Target in Multiple Sclerosis". New England Journal of Medicine. ج. 367 ع. 2: 115–23. DOI:10.1056/NEJMoa1110740. PMC:5131800. PMID:22784115.
Ayoglu، Burcu؛ Mitsios، Nicholas؛ Kockum، Ingrid؛ Khademi، Mohsen؛ Zandian، Arash؛ Sjöberg، Ronald؛ Forsström، Björn؛ Bredenberg، Johan؛ Lima Bomfim، Izaura (2016). "Anoctamin 2 identified as an autoimmune target in multiple sclerosis". Proceedings of the National Academy of Sciences. ج. 113 ع. 8: 2188–2193. Bibcode:2016PNAS..113.2188A. DOI:10.1073/pnas.1518553113. PMC:4776531. PMID:26862169.
Spadaro Melania؛ وآخرون (2015). "Histopathology and clinical course of MOG-antibody-associated encephalomyelitis". Annals of Clinical and Translational Neurology. ج. 2 ع. 3: 295–301. DOI:10.1002/acn3.164. PMC:4369279. PMID:25815356.
"Screening for MOG-IgG and 27 other anti-glial and anti-neuronal autoantibodies in 'pattern II multiple sclerosis' and brain biopsy findings in a MOG-IgG-positive case". Mult Scler. ج. 22 ع. 12: 1541–1549. 11 فبراير 2016. DOI:10.1177/1352458515622986. PMID:26869529.
Planas Raquel؛ وآخرون (2015). "Central role of Th2/Tc2 lymphocytes in pattern II multiple sclerosis lesions". Journal. ج. 2 ع. 9: 875–893. DOI:10.1002/acn3.218. PMC:4574806. PMID:26401510.
"Sequencing the immunopathologic heterogeneity in multiple sclerosis". Annals of Clinical and Translational Neurology. ج. 2 ع. 9: 873–874. 2015. DOI:10.1002/acn3.230. PMC:4574805. PMID:26401509.
Barnett، MH؛ Prineas، JW (أبريل 2004). "Relapsing and remitting multiple sclerosis: pathology of the newly forming lesion" (PDF). Annals of Neurology. ج. 55 ع. 4: 458–68. DOI:10.1002/ana.20016. PMID:15048884. مؤرشف من الأصل (PDF) في 2013-10-29. اطلع عليه بتاريخ 2009-10-28.
Marik، C.؛ Felts، P. A.؛ Bauer، J.؛ Lassmann، H.؛ Smith، K. J. (2007). "Lesion genesis in a subset of patients with multiple sclerosis: A role for innate immunity?". Brain. ج. 130 ع. 11: 2800–2815. DOI:10.1093/brain/awm236. PMC:2981817. PMID:17956913.
Hardy TA, Tobin WO, Lucchinetti CF (2016). "Exploring the overlap between multiple sclerosis, tumefactive demyelination and Baló's concentric sclerosis". Multiple Sclerosis Journal. ج. 22 ع. 8: 986–992. DOI:10.1177/1352458516641776. PMID:27037180.{{استشهاد بدورية محكمة}}: صيانة الاستشهاد: أسماء متعددة: قائمة المؤلفين (link)
"Homogeneity of active demyelinating lesions in established multiple sclerosis". Annals of Neurology. ج. 63 ع. 1: 16–25. 2008. DOI:10.1002/ana.21311. PMID:18232012.
Michael H. Barnett؛ John W. Prineas (2004). "Relapsing and Remitting Multiple Sclerosis: Pathology of the Newly Forming Lesion" (PDF). Annals of Neurology. ج. 55 ع. 1: 458–468. DOI:10.1002/ana.20016. PMID:15048884. مؤرشف من الأصل (PDF) في 2017-03-01.
"Neuromyelitis optica lesions may inform multiple sclerosis heterogeneity debate". Ann Neurol. ج. 72 ع. 3: 385–94. سبتمبر 2012. DOI:10.1002/ana.23621. PMID:23034911.
"Pathophysiological processes in multiple sclerosis: focus on nuclear factor erythroid-2-related factor 2 and emerging pathways". Clin Pharmacol. ج. 6: 35–42. فبراير 2014. DOI:10.2147/CPAA.S35033. PMC:3938468. PMID:24591852.{{استشهاد بدورية محكمة}}: صيانة الاستشهاد: دوي مجاني غير معلم (link)
"Pattern II and pattern III MS are entities distinct from pattern I MS: evidence from cerebrospinal fluid analysis". J Neuroinflammation. ج. 14 ع. 1: 171. 29 أغسطس 2017. DOI:10.1186/s12974-017-0929-z. PMC:5576197. PMID:28851393.{{استشهاد بدورية محكمة}}: صيانة الاستشهاد: دوي مجاني غير معلم (link)
"Antigen microarrays identify unique serum autoantibody signatures in clinical and pathologic subtypes of multiple sclerosis". Proc Natl Acad Sci USA. ج. 105 ع. 48: 18889–94. ديسمبر 2008. Bibcode:2008PNAS..10518889Q. DOI:10.1073/pnas.0806310105. PMC:2596207. PMID:19028871.
"Mitochondrial defects in acute multiple sclerosis lesions". Brain. ج. 131 ع. Pt 7: 1722–35. 2008. DOI:10.1093/brain/awn105. PMC:2442422. PMID:18515320.
"Monoclonal antibody treatment exposes three mechanisms underlying the clinical course of multiple sclerosis". Ann Neurol. ج. 46 ع. 3: 296–304. 1999. DOI:10.1002/1531-8249(199909)46:3<296::AID-ANA4>3.0.CO;2-#. PMID:10482259.
"Mitochondrial dysfunction contributes to neurodegeneration in multiple sclerosis". Trends in Molecular Medicine. ج. 20 ع. 3: 179–187. 2014. DOI:10.1016/j.molmed.2013.11.007. PMID:24369898.
Wang Zhe؛ وآخرون (2016). "Nuclear Receptor NR1H3 in Familial Multiple Sclerosis". Neuron. ج. 90 ع. 5: 948–954. DOI:10.1016/j.neuron.2016.04.039. PMC:5092154. PMID:27253448.
"Primary Progressive Multiple Sclerosis: Putting Together the Puzzle". Front. Neurol. ج. 8: 8–234. 2017. DOI:10.3389/fneur.2017.00234. PMC:5449443. PMID:28620346.{{استشهاد بدورية محكمة}}: صيانة الاستشهاد: دوي مجاني غير معلم (link)
Dinesh K. Sivakolundu et al., Three‐Dimensional Lesion Phenotyping and Physiologic Characterization Inform Remyelination Ability in Multiple Sclerosis, 30 May 2019, https://doi.org/10.1111/jon.12633 نسخة محفوظة 21 يناير 2021 على موقع واي باك مشين.
"Pathological abnormalities in the normal-appearing white matter in multiple sclerosis". Neurological Sciences. ج. 22 ع. 2: 141–144. 2001. DOI:10.1007/s100720170012. PMID:11603615.
"Inside-Out versus Outside-In models for virus induced demyelination: axonal damage triggering demyelination". Springer Semin Immunopathol. ج. 24 ع. 2: 105–25. 2002. DOI:10.1007/s00281-002-0105-z. PMC:7079941. PMID:12503060.
"Loss of 'homeostatic' microglia and patterns of their activation in active multiple sclerosis". Brain. ج. 140 ع. 7: 1900–1913. 2017. DOI:10.1093/brain/awx113. PMC:6057548. PMID:28541408.
"The pathogenesis of lesions and normal-appearing white matter changes in multiple sclerosis: a serial diffusion MRI study". Brain. ج. 123 ع. 8: 1667–76. أغسطس 2000. DOI:10.1093/brain/123.8.1667. PMID:10908196. مؤرشف من الأصل في 2017-12-01.
Schmid، Andreas؛ Hochberg، Alexandra؛ Berghoff، Martin؛ Schlegel، Jutta؛ Karrasch، Thomas؛ Kaps، Manfred؛ Schäffler، Andreas (2016). "Quantification and regulation of adipsin in human cerebrospinal fluid (CSF)". Clinical Endocrinology. ج. 84 ع. 2: 194–202. DOI:10.1111/cen.12856. PMID:26186410.
Ireland، Sara J.؛ Guzman، Alyssa A.؛ Frohman، Elliot M.؛ Monson، Nancy L. (2016). "B cells from relapsing remitting multiple sclerosis patients support neuro-antigen-specific Th17 responses". Journal of Neuroimmunology. ج. 291: 46–53. DOI:10.1016/j.jneuroim.2015.11.022. PMID:26857494.
Correale، Jorge؛ Andrés Villa (24 يوليو 2006). "The blood–brain-barrier in multiple sclerosis: Functional roles and therapeutic targeting". Autoimmunity. ج. 40 ع. 2: 148–160. DOI:10.1080/08916930601183522. PMID:17453713.
"Identification of an essential endogenous regulator of blood–brain barrier integrity, and its pathological and therapeutic implications". Proceedings of the National Academy of Sciences of the United States of America. ج. 110 ع. 3: 832–841. 15 يناير 2013. Bibcode:2013PNAS..110..832C. DOI:10.1073/pnas.1209362110. PMC:3549094. PMID:23277546.
"Elevated matrix metalloproteinase-9 and degradation of perineuronal nets in cerebrocortical multiple sclerosis plaques". J Neuropathol Exp Neurol. ج. 67 ع. 9: 888–99. سبتمبر 2008. DOI:10.1097/NEN.0b013e318183d003. PMID:18716555.
"Quantification of subtle blood–brain barrier disruption in non-enhancing lesions in multiple sclerosis: a study of disease and lesion subtypes". Multiple Sclerosis. ج. 13 ع. 7: 884–94. 2007. DOI:10.1177/1352458507076970. PMID:17468443.
"Multiple sclerosis as a vascular disease". Neurol. Res. ج. 28 ع. 3: 230–5. 2006. DOI:10.1179/016164106X98080. PMID:16687046.
Allen؛ وآخرون (2001). "Pathological abnormalities in the normal-appearing white matter in multiple sclerosis". Neurol Sci. ج. 22 ع. 2: 141–4. DOI:10.1007/s100720170012. PMID:11603615.
"Population-Wide Principal Component-Based Quantification of Blood-Brain-Barrier Dynamics in Multiple Sclerosis". NeuroImage. ج. 57 ع. 4: 1430–46. مايو 2011. DOI:10.1016/j.neuroimage.2011.05.038. PMC:3138825. PMID:21635955.
"Permeation of blood-borne IL15 across the blood–brain barrier and the effect of LPS". J. Neurochem. ج. 106 ع. 1: 313–9. 2008. DOI:10.1111/j.1471-4159.2008.05390.x. PMC:3939609. PMID:18384647.
"Tissue-type plasminogen activator is a regulator of monocyte diapedesis through the brain endothelial barrier". Journal of Immunology. ج. 181 ع. 5: 3567–74. 2008. DOI:10.4049/jimmunol.181.5.3567. PMID:18714030.
"Monocyte migration and LFA-1 mediated attachment to brain microvascular endothelia is regulated by SDF-1α through Lyn kinase". Journal of Immunology. ج. 181 ع. 7: 4632–7. أكتوبر 2008. DOI:10.4049/jimmunol.181.7.4632. PMC:2721474. PMID:18802065.
"Magnetic resonance imaging of human brain macrophage infiltration". Neurotherapeutics. ج. 4 ع. 3: 434–42. 2007. DOI:10.1016/j.nurt.2007.05.005. PMC:7479730. PMID:17599709.
"Matrix metalloproteinase-9 (MMP-9) and tissue inhibitor of matrix metalloproteinase (TIMP-1) in patients with relapsing-remitting multiple sclerosis treated with interferon beta". Clin Neurol Neurosurg. ج. 108 ع. 2: 124–8. فبراير 2006. DOI:10.1016/j.clineuro.2005.01.005. PMID:16412833.
"Biomarkers indicative of blood–brain barrier disruption in multiple sclerosis". Dis. Markers. ج. 22 ع. 4: 235–44. 2006. DOI:10.1155/2006/709869. PMC:3850823. PMID:17124345.{{استشهاد بدورية محكمة}}: صيانة الاستشهاد: دوي مجاني غير معلم (link)
"Adhesion molecules in multiple sclerosis: relation to subtypes of disease and methylprednisolone therapy". Arch. Neurol. ج. 57 ع. 4: 546–51. أبريل 2000. DOI:10.1001/archneur.57.4.546. PMID:10768630. مؤرشف من الأصل في 2018-04-03.
"Pathological Expression of CXCL12 at the Blood-Brain Barrier Correlates with Severity of Multiple Sclerosis". Am J Pathol. ج. 172 ع. 3: 799–808. مارس 2008. DOI:10.2353/ajpath.2008.070918. PMC:2258272. PMID:18276777.
"Imaging correlates of leukocyte accumulation and CXCR4/CXCR12 in multiple sclerosis". Arch. Neurol. ج. 66 ع. 1: 44–53. يناير 2009. DOI:10.1001/archneurol.2008.512. PMC:2792736. PMID:19139298. مؤرشف من الأصل في 2018-04-03.
"A molecular trio in relapse and remission in multiple sclerosis". Nature Reviews Immunology. ج. 9 ع. 6: 440–7. مايو 2009. DOI:10.1038/nri2548. PMID:19444308.
"Biomarkers indicative of blood–brain barrier disruption in multiple sclerosis". Disease Markers. ج. 22 ع. 4: 235–44. 2006. DOI:10.1155/2006/709869. PMC:3850823. PMID:17124345. مؤرشف من الأصل في 2020-08-01.{{استشهاد بدورية محكمة}}: صيانة الاستشهاد: دوي مجاني غير معلم (link)
"Persistent endothelial abnormalities and blood–brain barrier leak in primary and secondary progressive multiple sclerosis". Neuropathol. Appl. Neurobiol. ج. 33 ع. 1: 86–98. 2007. DOI:10.1111/j.1365-2990.2006.00781.x. PMID:17239011.
"The peroxynitrite scavenger uric acid prevents inflammatory cell invasion into the central nervous system in experimental allergic encephalomyelitis through maintenance of blood-central nervous system barrier integrity". Journal of Immunology. ج. 165 ع. 11: 6511–8. 2000. DOI:10.4049/jimmunol.165.11.6511. PMID:11086092.
"Serum uric acid and multiple sclerosis". Clinical Neurology and Neurosurgery. ج. 108 ع. 6: 527–31. 2006. DOI:10.1016/j.clineuro.2005.08.004. PMID:16202511.
"The blood–brain barrier in cortical multiple sclerosis lesions". J Neuropathol Exp Neurol. ج. 66 ع. 4: 321–8. أبريل 2007. DOI:10.1097/nen.0b013e318040b2de. PMID:17413323.
"Variation of serum uric acid levels in multiple sclerosis during relapses and immunomodulatory treatment". Eur. J. Neurol. ج. 15 ع. 4: 394–7. أبريل 2008. DOI:10.1111/j.1468-1331.2008.02087.x. PMID:18312403.
"White Matter Hemodynamic Abnormalities precede Sub-cortical Gray Matter Changes in Multiple Sclerosis". J. Neurol. Sci. ج. 282 ع. 1–2: 28–33. يوليو 2009. DOI:10.1016/j.jns.2008.12.036. PMC:2737614. PMID:19181347.
Planas R, Santos R, Tomas-Ojer P, Cruciani C, Lutterotti A, Faigle W, Schaeren-Wiemers N, Espejo C, Eixarch H, Pinilla C, Martin R, Sospedra M (2018). "GDP-l-fucose synthase is a CD4+ T cell–specific autoantigen in DRB3*02:02 patients with multiple sclerosis" (PDF). Science Translational Medicine. ج. 10 ع. 462: eaat4301. DOI:10.1126/scitranslmed.aat4301. PMID:30305453. مؤرشف من الأصل (PDF) في 2020-02-10.{{استشهاد بدورية محكمة}}: صيانة الاستشهاد: أسماء متعددة: قائمة المؤلفين (link)
Kremer؛ وآخرون (2019). "pHERV-W envelope protein fuels microglial cell-dependent damage of myelinated axons in multiple sclerosis". PNAS. ج. 116 ع. 30: 15216–15225. DOI:10.1073/pnas.1901283116. PMC:6660731. PMID:31213545.
Lisak RP (2019). "Human retrovirus pHEV-W envelope protein and the pathogenesis of multiple sclerosis". PNAS. ج. 116 ع. 30: 14791–14793. DOI:10.1073/pnas.1909786116. PMC:6660775. PMID:31289223.
"Multiple sclerosis pathology: evolution of pathogenetic concepts". Brain Pathology. ج. 15 ع. 3: 217–22. يوليو 2005. DOI:10.1111/j.1750-3639.2005.tb00523.x. PMID:16196388.
"Transcranial brain sonography findings predict disease progression in multiple sclerosis". Neurology. ج. 73 ع. 13: 1010–7. سبتمبر 2009. DOI:10.1212/WNL.0b013e3181b8a9f8. PMID:19657105.
"Persistent endothelial abnormalities and blood–brain barrier leak in primary and secondary progressive multiple sclerosis". Neuropathol. Appl. Neurobiol. ج. 33 ع. 1: 86–98. فبراير 2007. DOI:10.1111/j.1365-2990.2006.00781.x. PMID:17239011.
"7T MRI: New Vision of Microvascular Abnormalities in Multiple Sclerosis". Archives of Neurology. ج. 65 ع. 6: 812–6. 2008. DOI:10.1001/archneur.65.6.812. PMC:2579786. PMID:18541803.
Qiu، W؛ Raven، S؛ Wu، JS؛ Carroll، WM؛ Mastaglia، FL؛ Kermode، AG (مارس 2010). "Wedge-shaped medullary lesions in multiple sclerosis". Journal of the Neurological Sciences. ج. 290 ع. 1–2: 190–3. DOI:10.1016/j.jns.2009.12.017. PMID:20056253.
"Chlamydia pneumoniae DNA in the Arterial Wall of Patients with Peripheral Vascular Disease". Infection. ج. 29 ع. 4: 196–200. 2001. DOI:10.1007/s15010-001-1180-0. PMID:11545479.
"Chronic cerebrospinal venous insufficiency in patients with multiple sclerosis". J. Neurol. Neurosurg. Psychiatry. ج. 80 ع. 4: 392–9. أبريل 2009. DOI:10.1136/jnnp.2008.157164. PMC:2647682. PMID:19060024. مؤرشف من الأصل في 2009-08-04.
"Chronic cerebrospinal venous insufficiency and multiple sclerosis". Annals of Neurology. ج. 67 ع. 3: 286–90. مارس 2010. DOI:10.1002/ana.22001. PMID:20373339. مؤرشف من الأصل في 2010-11-23.
Bryce Weir (2010). J Neuro Sci 2010 - MS Vascular Etiology.pdf "MS, A vascular ethiology?" (PDF). Can. J. Neurol. Sci. ج. 37 ع. 6: 745–757. DOI:10.1017/s0317167100051404. PMID:21059535. مؤرشف من الأصل (PDF) في 2011-09-15. {{استشهاد بدورية محكمة}}: تحقق من قيمة |مسار أرشيف= (مساعدة)
"Intracranial venous haemodynamics in multiple sclerosis". Curr Neurovasc Res. ج. 4 ع. 4: 252–8. نوفمبر 2007. DOI:10.2174/156720207782446298. PMID:18045150. مؤرشف من الأصل في 2018-10-19.
"Chronic cerebrospinal venous insufficiency in patients with multiple sclerosis". J. Neurol. Neurosurg. Psychiatry. ج. 80 ع. 4: 392–9. أبريل 2009. DOI:10.1136/jnnp.2008.157164. PMC:2647682. PMID:19060024.
"["Chronic cerebrospinal venous insufficiency" and multiple sclerosis : Critical analysis and first observation in an unselected cohort of MS patients.]". Nervenarzt. ج. 81 ع. 6: 740–6. أبريل 2010. DOI:10.1007/s00115-010-2972-1. PMID:20386873.
"No cerebrocervical venous congestion in patients with multiple sclerosis". Annals of Neurology. ج. 68 ع. 2: 173–83. أغسطس 2010. DOI:10.1002/ana.22085. PMID:20695010.
"Venous and cerebrospinal fluid flow in multiple sclerosis: A case-control study". Annals of Neurology. ج. 68 ع. 2: 255–259. 2010. DOI:10.1002/ana.22132. PMID:20695018.
"Secretory products of multiple sclerosis B cells are cytotoxic to oligodendroglia in vitro". Journal of Neuroimmunology. ج. 246 ع. 1–2: 85–95. 2012. DOI:10.1016/j.jneuroim.2012.02.015. PMID:22458983.
Lassmann H (2019). "Pathogenic Mechanisms Associated With Different Clinical Courses of Multiple Sclerosis". Front Immunol. ج. 9: 3116. DOI:10.3389/fimmu.2018.03116. PMC:6335289. PMID:30687321.{{استشهاد بدورية محكمة}}: صيانة الاستشهاد: دوي مجاني غير معلم (link)
"Axonal damage induced by cerebrospinal fluid from patients with relapsing-remitting multiple sclerosis". J Neuroimmunol. ج. 104 ع. 1: 58–67. أبريل 2000. DOI:10.1016/s0165-5728(99)00225-8. PMID:10683515.
"Antibodies reactive to heat shock protein 90 induce oligodendrocyte precursor cell death in culture. Implications for demyelination in multiple sclerosis". FASEB J. ج. 18 ع. 2: 409–11. فبراير 2004. DOI:10.1096/fj.03-0606fje. PMID:14688203.{{استشهاد بدورية محكمة}}: صيانة الاستشهاد: دوي مجاني غير معلم (link)
Tiwari-Woodruff SK, Myers LW, Bronstein JM (أغسطس 2004). "Cerebrospinal fluid immunoglobulin G promotes oligodendrocyte progenitor cell migration". J. Neurosci. Res. ج. 77 ع. 3: 363–6. DOI:10.1002/jnr.20178. PMID:15248292.{{استشهاد بدورية محكمة}}: صيانة الاستشهاد: أسماء متعددة: قائمة المؤلفين (link)
"Cerebrospinal fluid derived from progressive multiple sclerosis patients promotes neuronal and oligodendroglial differentiation of human neural precursor cells in vitro". Neuroscience. ج. 250: 614–21. أكتوبر 2013. DOI:10.1016/j.neuroscience.2013.07.022. PMID:23876320.
Cristofanilli، Massimiliano؛ Rosenthal، Hannah؛ Cymring، Barbara؛ Gratch، Daniel؛ Pagano، Benjamin؛ Xie، Boxun؛ Sadiq، Saud A. (2014). "Progressive multiple sclerosis cerebrospinal fluid induces inflammatory demyelination, axonal loss, and astrogliosis in mice". Experimental Neurology. ج. 261: 620–32. DOI:10.1016/j.expneurol.2014.07.020. PMID:25111532.
"Transfer of multiple sclerosis into severe combined immunodeficiency mice by mononuclear cells from cerebrospinal fluid of the patients". PNAS. ج. 89 ع. 13: 6157–6161. 1992. Bibcode:1992PNAS...89.6157S. DOI:10.1073/pnas.89.13.6157. PMC:402141. PMID:1631103.
"Cerebrospinal fluid ceramides from patients with multiple sclerosis impair neuronal bioenergetics". Brain. ج. 137 ع. 8: 2271–86. أغسطس 2014. DOI:10.1093/brain/awu139. PMC:4164163. PMID:24893707.
Burgal, Mathur (يوليو 2014). "Molecular Shots". Ann Neurosci. ج. 21 ع. 3: 123. DOI:10.5214/ans.0972.7531.210311. PMC:4158786. PMID:25206080.
"Potassium channel KIR4.1 as an immune target in multiple sclerosis". N Engl J Med. ج. 367 ع. 2: 115–23. يوليو 2012. DOI:10.1056/NEJMoa1110740. PMC:5131800. PMID:22784115.
Schneider، Raphael (2013). "Autoantibodies to Potassium Channel KIR4.1 in Multiple Sclerosis". Frontiers in Neurology. ج. 4: 125. DOI:10.3389/fneur.2013.00125. PMC:3759297. PMID:24032025.{{استشهاد بدورية محكمة}}: صيانة الاستشهاد: دوي مجاني غير معلم (link)
Marnetto، Fabiana (2017). "Detection of potassium channel KIR4.1 antibodies in Multiple Sclerosis patients". Journal of Immunological Methods. ج. 445: 53–58. DOI:10.1016/j.jim.2017.03.008. PMID:28300540.
Ayoglua Burcu؛ وآخرون (2016). "Anoctamin 2 identified as an autoimmune target in multiple sclerosis". PNAS. ج. 113 ع. 8: 2188–2193. Bibcode:2016PNAS..113.2188A. DOI:10.1073/pnas.1518553113. PMC:4776531. PMID:26862169.
Matthews Lucy؛ وآخرون (2015). "Imaging Surrogates of Disease Activity in Neuromyelitis Optica Allow Distinction from Multiple Sclerosis". PLOS ONE. ج. 10 ع. 9: e0137715. Bibcode:2015PLoSO..1037715M. DOI:10.1371/journal.pone.0137715. PMC:4575169. PMID:26381510.{{استشهاد بدورية محكمة}}: صيانة الاستشهاد: دوي مجاني غير معلم (link)
"Axonal damage induced by cerebrospinal fluid from patients with relapsing-remitting multiple sclerosis". Journal of Neuroimmunology. ج. 104 ع. 1: 58–67. 2000. DOI:10.1016/S0165-5728(99)00225-8. PMID:10683515.
"Clusters of activated microglia in normal-appearing white matter show signs of innate immune activation". Journal of Neuroinflammation. ج. 9: 156. 2012. DOI:10.1186/1742-2094-9-156. PMC:3411485. PMID:22747960.{{استشهاد بدورية محكمة}}: صيانة الاستشهاد: دوي مجاني غير معلم (link)
"A gradient in cortical pathology in multiple sclerosis by in vivo quantitative 7 T imaging". Brain. ج. 138 ع. Pt 4: 932–45. 2015. DOI:10.1093/brain/awv011. PMC:4677339. PMID:25681411.
Hottenrott T, Dersch R, Berger B, Rauer S, Eckenweiler M, Huzly D, Stich O (2015). "The intrathecal, polyspecific antiviral immune response in neurosarcoidosis, acute disseminated encephalomyelitis and autoimmune encephalitis compared to multiple sclerosis in a tertiary hospital cohort". Fluids Barriers CNS. ج. 12: 27. DOI:10.1186/s12987-015-0024-8. PMC:4677451. PMID:26652013.{{استشهاد بدورية محكمة}}: صيانة الاستشهاد: أسماء متعددة: قائمة المؤلفين (link) صيانة الاستشهاد: دوي مجاني غير معلم (link)

emedicine.com

Multiple Sclerosis في موقع إي ميديسين

fondazionehilarescere.org

"Embryological background of truncular venous malformation in the extracranial venous pathways as the cause of chronic cerebro spinal venous insufficiency" (PDF). Int Angiol. ج. 29 ع. 2: 95–108. أبريل 2010. PMID:20351665. مؤرشف من الأصل (PDF) في 2010-07-04.

harvard.edu

ui.adsabs.harvard.edu

"Cholesterol crystals impede nerve repair". ساينس. ج. 359 ع. 6376: 635–636. 2018. Bibcode:2018Sci...359..635C. DOI:10.1126/science.aar7369. PMID:29439228.
"Defective cholesterol clearance limits remyelination in the aged central nervous system". ساينس. ج. 359 ع. 6376: 684–688. 2018. Bibcode:2018Sci...359..684C. DOI:10.1126/science.aan4183. PMID:29301957.
Ayoglu، Burcu؛ Mitsios، Nicholas؛ Kockum، Ingrid؛ Khademi، Mohsen؛ Zandian، Arash؛ Sjöberg، Ronald؛ Forsström، Björn؛ Bredenberg، Johan؛ Lima Bomfim، Izaura (2016). "Anoctamin 2 identified as an autoimmune target in multiple sclerosis". Proceedings of the National Academy of Sciences. ج. 113 ع. 8: 2188–2193. Bibcode:2016PNAS..113.2188A. DOI:10.1073/pnas.1518553113. PMC:4776531. PMID:26862169.
"Antigen microarrays identify unique serum autoantibody signatures in clinical and pathologic subtypes of multiple sclerosis". Proc Natl Acad Sci USA. ج. 105 ع. 48: 18889–94. ديسمبر 2008. Bibcode:2008PNAS..10518889Q. DOI:10.1073/pnas.0806310105. PMC:2596207. PMID:19028871.
"Identification of an essential endogenous regulator of blood–brain barrier integrity, and its pathological and therapeutic implications". Proceedings of the National Academy of Sciences of the United States of America. ج. 110 ع. 3: 832–841. 15 يناير 2013. Bibcode:2013PNAS..110..832C. DOI:10.1073/pnas.1209362110. PMC:3549094. PMID:23277546.
"Transfer of multiple sclerosis into severe combined immunodeficiency mice by mononuclear cells from cerebrospinal fluid of the patients". PNAS. ج. 89 ع. 13: 6157–6161. 1992. Bibcode:1992PNAS...89.6157S. DOI:10.1073/pnas.89.13.6157. PMC:402141. PMID:1631103.
Ayoglua Burcu؛ وآخرون (2016). "Anoctamin 2 identified as an autoimmune target in multiple sclerosis". PNAS. ج. 113 ع. 8: 2188–2193. Bibcode:2016PNAS..113.2188A. DOI:10.1073/pnas.1518553113. PMC:4776531. PMID:26862169.
Matthews Lucy؛ وآخرون (2015). "Imaging Surrogates of Disease Activity in Neuromyelitis Optica Allow Distinction from Multiple Sclerosis". PLOS ONE. ج. 10 ع. 9: e0137715. Bibcode:2015PLoSO..1037715M. DOI:10.1371/journal.pone.0137715. PMC:4575169. PMID:26381510.{{استشهاد بدورية محكمة}}: صيانة الاستشهاد: دوي مجاني غير معلم (link)

if-pan.krakow.pl

"Impact of methylprednisolone treatment on the expression of macrophage inflammatory protein 3alpha and B lymphocyte chemoattractant in serum of multiple sclerosis patients" (PDF). Pharmacol Rep. ج. 60 ع. 4: 549–54. 2008. PMID:18799824. مؤرشف من الأصل (PDF) في 2016-03-04.

medicalnewstoday.com

Alexandre Prat, Nicole Beaulieu, Sylvain-Jacques Desjardins, New Therapeutic Target For Treatment Of Multiple Sclerosis, Jan. 2008 نسخة محفوظة 2011-05-16 على موقع واي باك مشين.

metapress.com

iospress.metapress.com

"Biomarkers indicative of blood–brain barrier disruption in multiple sclerosis". Disease Markers. ج. 22 ع. 4: 235–44. 2006. DOI:10.1155/2006/709869. PMC:3850823. PMID:17124345. مؤرشف من الأصل في 2020-08-01.{{استشهاد بدورية محكمة}}: صيانة الاستشهاد: دوي مجاني غير معلم (link)

neurology.org

"A serial study of new MS lesions and the white matter from which they arise". Neurology. ج. 51 ع. 6: 1689–97. ديسمبر 1998. DOI:10.1212/wnl.51.6.1689. PMID:9855524. مؤرشف من الأصل في 2008-05-22.

neurologyreviews.com

"Some MS patients have "Dramatic" responses to Plasma Exchange". Neurology Reviews. ج. 8 ع. 3. مارس 2000. مؤرشف من الأصل في 2001-02-23. اطلع عليه بتاريخ 2006-05-05.

neurosciencenews.com

University of Zurich(2018, October 11). Link Between Gut Flora and Multiple Sclerosis Discovered. NeuroscienceNews. Retrieved October 11, 2018 from http://neurosciencenews.com/multiple-sclerosis-gut-flora-10003/ نسخة محفوظة 2020-11-09 على موقع واي باك مشين.

nih.gov

pubmed.ncbi.nlm.nih.gov

Golan، Daniel؛ Staun-Ram، Elsebeth؛ Miller، Ariel (2016). "Shifting paradigms in multiple sclerosis". Current Opinion in Neurology. ج. 29 ع. 3: 354–361. DOI:10.1097/WCO.0000000000000324. PMID:27070218.
"Pathology and definition of multiple sclerosis". Rev Prat. ج. 56 ع. 12: 1293–8. يونيو 2006. PMID:16948216.
"Imaging in multiple sclerosis: A new spin on lesions". Med. Imag. And Radiation Oncology. ج. 60 ع. 5: 577–586. 27 يوليو 2016. DOI:10.1111/1754-9485.12498. PMID:27464473.
"Perivenous demyelination: association with clinically defined acute disseminated encephalomyelitis and comparison with pathologically confirmed multiple sclerosis". Brain. ج. 133 ع. 2: 333–348. 2010. DOI:10.1093/brain/awp321. PMC:2822631. PMID:20129932.
Lassman H (مارس 2019). "The changing concepts in the neuropathology of acquired demyelinating central nervous system disorders". Curr Opin Neurol. ج. 32 ع. 3: 313–319. DOI:10.1097/WCO.0000000000000685. PMID:30893100.
Lassman H (2019). "Pathogenic Mechanisms Associated With Different Clinical Courses of Multiple Sclerosis". Front Immunol. ج. 9: 3116. DOI:10.3389/fimmu.2018.03116. PMC:6335289. PMID:30687321.{{استشهاد بدورية محكمة}}: صيانة الاستشهاد: دوي مجاني غير معلم (link)
"Preactive lesions in multiple sclerosis". Curr Opin Neurol. ج. 22 ع. 3: 207–13. 2009. DOI:10.1097/WCO.0b013e32832b4c76. PMID:19417567.
"Alpha-B-Crystallin Induces an Immune-Regulatory and Antiviral Microglial Response in Preactive Multiple Sclerosis Lesions". J Neuropathol Exp Neurol. ج. 72 ع. 10: 970–9. 2013. DOI:10.1097/NEN.0b013e3182a776bf. PMID:24042199.
Ontaneda؛ وآخرون (نوفمبر 2014). "Identifying the Start of Multiple Sclerosis Injury: A Serial DTI Study". J Neuroimaging. ج. 24 ع. 6: 569–76. DOI:10.1111/jon.12082. PMC:4221810. PMID:25370339.
"A serial study of new MS lesions and the white matter from which they arise". Neurology. ج. 51 ع. 6: 1689–97. ديسمبر 1998. DOI:10.1212/wnl.51.6.1689. PMID:9855524. مؤرشف من الأصل في 2008-05-22.
"Clinico-pathological evidence that axonal loss underlies disability in progressive multiple sclerosis". Mult Scler. ج. 16 ع. 4: 406–411. أبريل 2010. DOI:10.1177/1352458510364992. PMID:20215480.
Lassmann Hans (2014). "Multiple sclerosis: Lessons from molecular neuropathology". Experimental Neurology. ج. 262: 2–7. DOI:10.1016/j.expneurol.2013.12.003. PMID:24342027.
"The immunopathology of multiple sclerosis: an overview". Brain Pathol. ج. 17 ع. 2: 210–8. أبريل 2007. DOI:10.1111/j.1750-3639.2007.00064.x. PMID:17388952.
"Gray matter involvement in multiple sclerosis". Neurology. ج. 68 ع. 9: 634–42. فبراير 2007. DOI:10.1212/01.wnl.0000250267.85698.7a. PMID:17325269.
"Compartmentalization of inflammation in the CNS: A major mechanism driving progressive multiple sclerosis". J Neurol Sci. ج. 274 ع. 1–2: 42–4. نوفمبر 2008. DOI:10.1016/j.jns.2008.06.032. PMID:18715571.
"The astrocyte in multiple sclerosis revisited". Glia. ج. 61 ع. 4: 453–465. 2013. DOI:10.1002/glia.22443. PMID:23322421.
"MR Spectroscopy Indicates Diffuse Multiple Sclerosis Activity During Remission". J. Neurol. Neurosurg. Psychiatry. ج. 80 ع. 12: 1330–6. يونيو 2009. DOI:10.1136/jnnp.2009.176263. PMC:2900785. PMID:19546105.
"Dysregulated Epstein-Barr virus infection in the multiple sclerosis brain". Journal of Experimental Medicine. ج. 204 ع. 12: 2899–2912. نوفمبر 2007. DOI:10.1084/jem.20071030. PMC:2118531. PMID:17984305.
"Cholesterol crystals impede nerve repair". ساينس. ج. 359 ع. 6376: 635–636. 2018. Bibcode:2018Sci...359..635C. DOI:10.1126/science.aar7369. PMID:29439228.
"Defective cholesterol clearance limits remyelination in the aged central nervous system". ساينس. ج. 359 ع. 6376: 684–688. 2018. Bibcode:2018Sci...359..684C. DOI:10.1126/science.aan4183. PMID:29301957.
"Distinct patterns of multiple sclerosis pathology indicates heterogeneity on pathogenesis". Brain Pathol. ج. 6 ع. 3: 259–74. يوليو 1996. DOI:10.1111/j.1750-3639.1996.tb00854.x. PMC:7161824. PMID:8864283.
"A quantitative analysis of oligodendrocytes in multiple sclerosis lesions - A study of 113 cases". Brain. ج. 122 ع. 12: 2279–2295. ديسمبر 1999. DOI:10.1093/brain/122.12.2279. PMID:10581222. مؤرشف من الأصل في 2008-08-29. اطلع عليه بتاريخ 2006-05-10.
"Humoral pattern II multiple sclerosis pathology not associated with neuromyelitis Optica IgG". Arch Neurol. ج. 66 ع. 10: 1298–9. أكتوبر 2009. DOI:10.1001/archneurol.2009.199. PMC:2767176. PMID:19822791.
Srivastava، Rajneesh؛ Aslam، Muhammad؛ Kalluri، Sudhakar Reddy؛ Schirmer، Lucas؛ Buck، Dorothea؛ Tackenberg، Björn؛ Rothhammer، Veit؛ Chan، Andrew؛ Gold، Ralf (2012). "Potassium Channel KIR4.1 as an Immune Target in Multiple Sclerosis". New England Journal of Medicine. ج. 367 ع. 2: 115–23. DOI:10.1056/NEJMoa1110740. PMC:5131800. PMID:22784115.
Ayoglu، Burcu؛ Mitsios، Nicholas؛ Kockum، Ingrid؛ Khademi، Mohsen؛ Zandian، Arash؛ Sjöberg، Ronald؛ Forsström، Björn؛ Bredenberg، Johan؛ Lima Bomfim، Izaura (2016). "Anoctamin 2 identified as an autoimmune target in multiple sclerosis". Proceedings of the National Academy of Sciences. ج. 113 ع. 8: 2188–2193. Bibcode:2016PNAS..113.2188A. DOI:10.1073/pnas.1518553113. PMC:4776531. PMID:26862169.
Spadaro Melania؛ وآخرون (2015). "Histopathology and clinical course of MOG-antibody-associated encephalomyelitis". Annals of Clinical and Translational Neurology. ج. 2 ع. 3: 295–301. DOI:10.1002/acn3.164. PMC:4369279. PMID:25815356.
"Screening for MOG-IgG and 27 other anti-glial and anti-neuronal autoantibodies in 'pattern II multiple sclerosis' and brain biopsy findings in a MOG-IgG-positive case". Mult Scler. ج. 22 ع. 12: 1541–1549. 11 فبراير 2016. DOI:10.1177/1352458515622986. PMID:26869529.
Planas Raquel؛ وآخرون (2015). "Central role of Th2/Tc2 lymphocytes in pattern II multiple sclerosis lesions". Journal. ج. 2 ع. 9: 875–893. DOI:10.1002/acn3.218. PMC:4574806. PMID:26401510.
"Sequencing the immunopathologic heterogeneity in multiple sclerosis". Annals of Clinical and Translational Neurology. ج. 2 ع. 9: 873–874. 2015. DOI:10.1002/acn3.230. PMC:4574805. PMID:26401509.
Barnett، MH؛ Prineas، JW (أبريل 2004). "Relapsing and remitting multiple sclerosis: pathology of the newly forming lesion" (PDF). Annals of Neurology. ج. 55 ع. 4: 458–68. DOI:10.1002/ana.20016. PMID:15048884. مؤرشف من الأصل (PDF) في 2013-10-29. اطلع عليه بتاريخ 2009-10-28.
Marik، C.؛ Felts، P. A.؛ Bauer، J.؛ Lassmann، H.؛ Smith، K. J. (2007). "Lesion genesis in a subset of patients with multiple sclerosis: A role for innate immunity?". Brain. ج. 130 ع. 11: 2800–2815. DOI:10.1093/brain/awm236. PMC:2981817. PMID:17956913.
Hardy TA, Tobin WO, Lucchinetti CF (2016). "Exploring the overlap between multiple sclerosis, tumefactive demyelination and Baló's concentric sclerosis". Multiple Sclerosis Journal. ج. 22 ع. 8: 986–992. DOI:10.1177/1352458516641776. PMID:27037180.{{استشهاد بدورية محكمة}}: صيانة الاستشهاد: أسماء متعددة: قائمة المؤلفين (link)
"Homogeneity of active demyelinating lesions in established multiple sclerosis". Annals of Neurology. ج. 63 ع. 1: 16–25. 2008. DOI:10.1002/ana.21311. PMID:18232012.
Michael H. Barnett؛ John W. Prineas (2004). "Relapsing and Remitting Multiple Sclerosis: Pathology of the Newly Forming Lesion" (PDF). Annals of Neurology. ج. 55 ع. 1: 458–468. DOI:10.1002/ana.20016. PMID:15048884. مؤرشف من الأصل (PDF) في 2017-03-01.
"Neuromyelitis optica lesions may inform multiple sclerosis heterogeneity debate". Ann Neurol. ج. 72 ع. 3: 385–94. سبتمبر 2012. DOI:10.1002/ana.23621. PMID:23034911.
"Pathophysiological processes in multiple sclerosis: focus on nuclear factor erythroid-2-related factor 2 and emerging pathways". Clin Pharmacol. ج. 6: 35–42. فبراير 2014. DOI:10.2147/CPAA.S35033. PMC:3938468. PMID:24591852.{{استشهاد بدورية محكمة}}: صيانة الاستشهاد: دوي مجاني غير معلم (link)
"Pattern II and pattern III MS are entities distinct from pattern I MS: evidence from cerebrospinal fluid analysis". J Neuroinflammation. ج. 14 ع. 1: 171. 29 أغسطس 2017. DOI:10.1186/s12974-017-0929-z. PMC:5576197. PMID:28851393.{{استشهاد بدورية محكمة}}: صيانة الاستشهاد: دوي مجاني غير معلم (link)
"Antigen microarrays identify unique serum autoantibody signatures in clinical and pathologic subtypes of multiple sclerosis". Proc Natl Acad Sci USA. ج. 105 ع. 48: 18889–94. ديسمبر 2008. Bibcode:2008PNAS..10518889Q. DOI:10.1073/pnas.0806310105. PMC:2596207. PMID:19028871.
"Mitochondrial defects in acute multiple sclerosis lesions". Brain. ج. 131 ع. Pt 7: 1722–35. 2008. DOI:10.1093/brain/awn105. PMC:2442422. PMID:18515320.
"Monoclonal antibody treatment exposes three mechanisms underlying the clinical course of multiple sclerosis". Ann Neurol. ج. 46 ع. 3: 296–304. 1999. DOI:10.1002/1531-8249(199909)46:3<296::AID-ANA4>3.0.CO;2-#. PMID:10482259.
"Mitochondrial dysfunction contributes to neurodegeneration in multiple sclerosis". Trends in Molecular Medicine. ج. 20 ع. 3: 179–187. 2014. DOI:10.1016/j.molmed.2013.11.007. PMID:24369898.
Wang Zhe؛ وآخرون (2016). "Nuclear Receptor NR1H3 in Familial Multiple Sclerosis". Neuron. ج. 90 ع. 5: 948–954. DOI:10.1016/j.neuron.2016.04.039. PMC:5092154. PMID:27253448.
"Primary Progressive Multiple Sclerosis: Putting Together the Puzzle". Front. Neurol. ج. 8: 8–234. 2017. DOI:10.3389/fneur.2017.00234. PMC:5449443. PMID:28620346.{{استشهاد بدورية محكمة}}: صيانة الاستشهاد: دوي مجاني غير معلم (link)
"Pathological abnormalities in the normal-appearing white matter in multiple sclerosis". Neurological Sciences. ج. 22 ع. 2: 141–144. 2001. DOI:10.1007/s100720170012. PMID:11603615.
"Inside-Out versus Outside-In models for virus induced demyelination: axonal damage triggering demyelination". Springer Semin Immunopathol. ج. 24 ع. 2: 105–25. 2002. DOI:10.1007/s00281-002-0105-z. PMC:7079941. PMID:12503060.
"Loss of 'homeostatic' microglia and patterns of their activation in active multiple sclerosis". Brain. ج. 140 ع. 7: 1900–1913. 2017. DOI:10.1093/brain/awx113. PMC:6057548. PMID:28541408.
"The pathogenesis of lesions and normal-appearing white matter changes in multiple sclerosis: a serial diffusion MRI study". Brain. ج. 123 ع. 8: 1667–76. أغسطس 2000. DOI:10.1093/brain/123.8.1667. PMID:10908196. مؤرشف من الأصل في 2017-12-01.
Schmid، Andreas؛ Hochberg، Alexandra؛ Berghoff، Martin؛ Schlegel، Jutta؛ Karrasch، Thomas؛ Kaps، Manfred؛ Schäffler، Andreas (2016). "Quantification and regulation of adipsin in human cerebrospinal fluid (CSF)". Clinical Endocrinology. ج. 84 ع. 2: 194–202. DOI:10.1111/cen.12856. PMID:26186410.
Ireland، Sara J.؛ Guzman، Alyssa A.؛ Frohman، Elliot M.؛ Monson، Nancy L. (2016). "B cells from relapsing remitting multiple sclerosis patients support neuro-antigen-specific Th17 responses". Journal of Neuroimmunology. ج. 291: 46–53. DOI:10.1016/j.jneuroim.2015.11.022. PMID:26857494.
Correale، Jorge؛ Andrés Villa (24 يوليو 2006). "The blood–brain-barrier in multiple sclerosis: Functional roles and therapeutic targeting". Autoimmunity. ج. 40 ع. 2: 148–160. DOI:10.1080/08916930601183522. PMID:17453713.
"Identification of an essential endogenous regulator of blood–brain barrier integrity, and its pathological and therapeutic implications". Proceedings of the National Academy of Sciences of the United States of America. ج. 110 ع. 3: 832–841. 15 يناير 2013. Bibcode:2013PNAS..110..832C. DOI:10.1073/pnas.1209362110. PMC:3549094. PMID:23277546.
Prat، Elisabetta؛ Roland Martin (مارس–أبريل 2002). "The immunopathogenesis of multiple sclerosis". Journal of Rehabilitation Research and Development. ج. 39 ع. 2: 187–99. PMID:12051463.
"Elevated matrix metalloproteinase-9 and degradation of perineuronal nets in cerebrocortical multiple sclerosis plaques". J Neuropathol Exp Neurol. ج. 67 ع. 9: 888–99. سبتمبر 2008. DOI:10.1097/NEN.0b013e318183d003. PMID:18716555.
"Quantification of subtle blood–brain barrier disruption in non-enhancing lesions in multiple sclerosis: a study of disease and lesion subtypes". Multiple Sclerosis. ج. 13 ع. 7: 884–94. 2007. DOI:10.1177/1352458507076970. PMID:17468443.
"Multiple sclerosis as a vascular disease". Neurol. Res. ج. 28 ع. 3: 230–5. 2006. DOI:10.1179/016164106X98080. PMID:16687046.
Allen؛ وآخرون (2001). "Pathological abnormalities in the normal-appearing white matter in multiple sclerosis". Neurol Sci. ج. 22 ع. 2: 141–4. DOI:10.1007/s100720170012. PMID:11603615.
"Population-Wide Principal Component-Based Quantification of Blood-Brain-Barrier Dynamics in Multiple Sclerosis". NeuroImage. ج. 57 ع. 4: 1430–46. مايو 2011. DOI:10.1016/j.neuroimage.2011.05.038. PMC:3138825. PMID:21635955.
"Permeation of blood-borne IL15 across the blood–brain barrier and the effect of LPS". J. Neurochem. ج. 106 ع. 1: 313–9. 2008. DOI:10.1111/j.1471-4159.2008.05390.x. PMC:3939609. PMID:18384647.
"Tissue-type plasminogen activator is a regulator of monocyte diapedesis through the brain endothelial barrier". Journal of Immunology. ج. 181 ع. 5: 3567–74. 2008. DOI:10.4049/jimmunol.181.5.3567. PMID:18714030.
"Monocyte migration and LFA-1 mediated attachment to brain microvascular endothelia is regulated by SDF-1α through Lyn kinase". Journal of Immunology. ج. 181 ع. 7: 4632–7. أكتوبر 2008. DOI:10.4049/jimmunol.181.7.4632. PMC:2721474. PMID:18802065.
"Magnetic resonance imaging of human brain macrophage infiltration". Neurotherapeutics. ج. 4 ع. 3: 434–42. 2007. DOI:10.1016/j.nurt.2007.05.005. PMC:7479730. PMID:17599709.
"Matrix metalloproteinase-9 (MMP-9) and tissue inhibitor of matrix metalloproteinase (TIMP-1) in patients with relapsing-remitting multiple sclerosis treated with interferon beta". Clin Neurol Neurosurg. ج. 108 ع. 2: 124–8. فبراير 2006. DOI:10.1016/j.clineuro.2005.01.005. PMID:16412833.
"Biomarkers indicative of blood–brain barrier disruption in multiple sclerosis". Dis. Markers. ج. 22 ع. 4: 235–44. 2006. DOI:10.1155/2006/709869. PMC:3850823. PMID:17124345.{{استشهاد بدورية محكمة}}: صيانة الاستشهاد: دوي مجاني غير معلم (link)
"Adhesion molecules in multiple sclerosis: relation to subtypes of disease and methylprednisolone therapy". Arch. Neurol. ج. 57 ع. 4: 546–51. أبريل 2000. DOI:10.1001/archneur.57.4.546. PMID:10768630. مؤرشف من الأصل في 2018-04-03.
"Pathological Expression of CXCL12 at the Blood-Brain Barrier Correlates with Severity of Multiple Sclerosis". Am J Pathol. ج. 172 ع. 3: 799–808. مارس 2008. DOI:10.2353/ajpath.2008.070918. PMC:2258272. PMID:18276777.
"Imaging correlates of leukocyte accumulation and CXCR4/CXCR12 in multiple sclerosis". Arch. Neurol. ج. 66 ع. 1: 44–53. يناير 2009. DOI:10.1001/archneurol.2008.512. PMC:2792736. PMID:19139298. مؤرشف من الأصل في 2018-04-03.
"Impact of methylprednisolone treatment on the expression of macrophage inflammatory protein 3alpha and B lymphocyte chemoattractant in serum of multiple sclerosis patients" (PDF). Pharmacol Rep. ج. 60 ع. 4: 549–54. 2008. PMID:18799824. مؤرشف من الأصل (PDF) في 2016-03-04.
"A molecular trio in relapse and remission in multiple sclerosis". Nature Reviews Immunology. ج. 9 ع. 6: 440–7. مايو 2009. DOI:10.1038/nri2548. PMID:19444308.
"Biomarkers indicative of blood–brain barrier disruption in multiple sclerosis". Disease Markers. ج. 22 ع. 4: 235–44. 2006. DOI:10.1155/2006/709869. PMC:3850823. PMID:17124345. مؤرشف من الأصل في 2020-08-01.{{استشهاد بدورية محكمة}}: صيانة الاستشهاد: دوي مجاني غير معلم (link)
"Persistent endothelial abnormalities and blood–brain barrier leak in primary and secondary progressive multiple sclerosis". Neuropathol. Appl. Neurobiol. ج. 33 ع. 1: 86–98. 2007. DOI:10.1111/j.1365-2990.2006.00781.x. PMID:17239011.
"The peroxynitrite scavenger uric acid prevents inflammatory cell invasion into the central nervous system in experimental allergic encephalomyelitis through maintenance of blood-central nervous system barrier integrity". Journal of Immunology. ج. 165 ع. 11: 6511–8. 2000. DOI:10.4049/jimmunol.165.11.6511. PMID:11086092.
"Serum uric acid and multiple sclerosis". Clinical Neurology and Neurosurgery. ج. 108 ع. 6: 527–31. 2006. DOI:10.1016/j.clineuro.2005.08.004. PMID:16202511.
"The blood–brain barrier in cortical multiple sclerosis lesions". J Neuropathol Exp Neurol. ج. 66 ع. 4: 321–8. أبريل 2007. DOI:10.1097/nen.0b013e318040b2de. PMID:17413323.
"Variation of serum uric acid levels in multiple sclerosis during relapses and immunomodulatory treatment". Eur. J. Neurol. ج. 15 ع. 4: 394–7. أبريل 2008. DOI:10.1111/j.1468-1331.2008.02087.x. PMID:18312403.
"White Matter Hemodynamic Abnormalities precede Sub-cortical Gray Matter Changes in Multiple Sclerosis". J. Neurol. Sci. ج. 282 ع. 1–2: 28–33. يوليو 2009. DOI:10.1016/j.jns.2008.12.036. PMC:2737614. PMID:19181347.
Planas R, Santos R, Tomas-Ojer P, Cruciani C, Lutterotti A, Faigle W, Schaeren-Wiemers N, Espejo C, Eixarch H, Pinilla C, Martin R, Sospedra M (2018). "GDP-l-fucose synthase is a CD4+ T cell–specific autoantigen in DRB3*02:02 patients with multiple sclerosis" (PDF). Science Translational Medicine. ج. 10 ع. 462: eaat4301. DOI:10.1126/scitranslmed.aat4301. PMID:30305453. مؤرشف من الأصل (PDF) في 2020-02-10.{{استشهاد بدورية محكمة}}: صيانة الاستشهاد: أسماء متعددة: قائمة المؤلفين (link)
Kremer؛ وآخرون (2019). "pHERV-W envelope protein fuels microglial cell-dependent damage of myelinated axons in multiple sclerosis". PNAS. ج. 116 ع. 30: 15216–15225. DOI:10.1073/pnas.1901283116. PMC:6660731. PMID:31213545.
Lisak RP (2019). "Human retrovirus pHEV-W envelope protein and the pathogenesis of multiple sclerosis". PNAS. ج. 116 ع. 30: 14791–14793. DOI:10.1073/pnas.1909786116. PMC:6660775. PMID:31289223.
"Multiple sclerosis pathology: evolution of pathogenetic concepts". Brain Pathology. ج. 15 ع. 3: 217–22. يوليو 2005. DOI:10.1111/j.1750-3639.2005.tb00523.x. PMID:16196388.
"Transcranial brain sonography findings predict disease progression in multiple sclerosis". Neurology. ج. 73 ع. 13: 1010–7. سبتمبر 2009. DOI:10.1212/WNL.0b013e3181b8a9f8. PMID:19657105.
"Persistent endothelial abnormalities and blood–brain barrier leak in primary and secondary progressive multiple sclerosis". Neuropathol. Appl. Neurobiol. ج. 33 ع. 1: 86–98. فبراير 2007. DOI:10.1111/j.1365-2990.2006.00781.x. PMID:17239011.
"7T MRI: New Vision of Microvascular Abnormalities in Multiple Sclerosis". Archives of Neurology. ج. 65 ع. 6: 812–6. 2008. DOI:10.1001/archneur.65.6.812. PMC:2579786. PMID:18541803.
Qiu، W؛ Raven، S؛ Wu، JS؛ Carroll، WM؛ Mastaglia، FL؛ Kermode، AG (مارس 2010). "Wedge-shaped medullary lesions in multiple sclerosis". Journal of the Neurological Sciences. ج. 290 ع. 1–2: 190–3. DOI:10.1016/j.jns.2009.12.017. PMID:20056253.
"Chlamydia pneumoniae DNA in the Arterial Wall of Patients with Peripheral Vascular Disease". Infection. ج. 29 ع. 4: 196–200. 2001. DOI:10.1007/s15010-001-1180-0. PMID:11545479.
"Chronic cerebrospinal venous insufficiency in patients with multiple sclerosis". J. Neurol. Neurosurg. Psychiatry. ج. 80 ع. 4: 392–9. أبريل 2009. DOI:10.1136/jnnp.2008.157164. PMC:2647682. PMID:19060024. مؤرشف من الأصل في 2009-08-04.
"Chronic cerebrospinal venous insufficiency and multiple sclerosis". Annals of Neurology. ج. 67 ع. 3: 286–90. مارس 2010. DOI:10.1002/ana.22001. PMID:20373339. مؤرشف من الأصل في 2010-11-23.
Bryce Weir (2010). J Neuro Sci 2010 - MS Vascular Etiology.pdf "MS, A vascular ethiology?" (PDF). Can. J. Neurol. Sci. ج. 37 ع. 6: 745–757. DOI:10.1017/s0317167100051404. PMID:21059535. مؤرشف من الأصل (PDF) في 2011-09-15. {{استشهاد بدورية محكمة}}: تحقق من قيمة |مسار أرشيف= (مساعدة)
"Haemodynamic patterns in chronic cereblrospinal venous insufficiency in multiple sclerosis. Correlation of symptoms at onset and clinical course". Int Angiol. ج. 29 ع. 2: 183–8. أبريل 2010. PMID:20351667.
"Intracranial venous haemodynamics in multiple sclerosis". Curr Neurovasc Res. ج. 4 ع. 4: 252–8. نوفمبر 2007. DOI:10.2174/156720207782446298. PMID:18045150. مؤرشف من الأصل في 2018-10-19.
"Chronic cerebrospinal venous insufficiency in patients with multiple sclerosis". J. Neurol. Neurosurg. Psychiatry. ج. 80 ع. 4: 392–9. أبريل 2009. DOI:10.1136/jnnp.2008.157164. PMC:2647682. PMID:19060024.
"Embryological background of truncular venous malformation in the extracranial venous pathways as the cause of chronic cerebro spinal venous insufficiency" (PDF). Int Angiol. ج. 29 ع. 2: 95–108. أبريل 2010. PMID:20351665. مؤرشف من الأصل (PDF) في 2010-07-04.
"Internal jugular vein morphology and hemodynamics in patients with multiple sclerosis". Int Angiol. ج. 29 ع. 2: 115–20. أبريل 2010. PMID:20351667.
"["Chronic cerebrospinal venous insufficiency" and multiple sclerosis : Critical analysis and first observation in an unselected cohort of MS patients.]". Nervenarzt. ج. 81 ع. 6: 740–6. أبريل 2010. DOI:10.1007/s00115-010-2972-1. PMID:20386873.
"No cerebrocervical venous congestion in patients with multiple sclerosis". Annals of Neurology. ج. 68 ع. 2: 173–83. أغسطس 2010. DOI:10.1002/ana.22085. PMID:20695010.
"Venous and cerebrospinal fluid flow in multiple sclerosis: A case-control study". Annals of Neurology. ج. 68 ع. 2: 255–259. 2010. DOI:10.1002/ana.22132. PMID:20695018.
Zamboni؛ وآخرون (2010). "CSF dynamics and brain volume in multiple sclerosis are associated with extracranial venous flow anomalies". Int Angiol. ج. 29 ع. 2: 140–8. PMID:20351670.
"Secretory products of multiple sclerosis B cells are cytotoxic to oligodendroglia in vitro". Journal of Neuroimmunology. ج. 246 ع. 1–2: 85–95. 2012. DOI:10.1016/j.jneuroim.2012.02.015. PMID:22458983.
Lassmann H (2019). "Pathogenic Mechanisms Associated With Different Clinical Courses of Multiple Sclerosis". Front Immunol. ج. 9: 3116. DOI:10.3389/fimmu.2018.03116. PMC:6335289. PMID:30687321.{{استشهاد بدورية محكمة}}: صيانة الاستشهاد: دوي مجاني غير معلم (link)
"Axonal damage induced by cerebrospinal fluid from patients with relapsing-remitting multiple sclerosis". J Neuroimmunol. ج. 104 ع. 1: 58–67. أبريل 2000. DOI:10.1016/s0165-5728(99)00225-8. PMID:10683515.
"The effect of cerebrospinal fluid on neurone culture: implications in the pathogenesis of multiple sclerosis". Rev Neurol. ج. 35 ع. 10: 994–7. 2002. PMID:12436405.
"Antibodies reactive to heat shock protein 90 induce oligodendrocyte precursor cell death in culture. Implications for demyelination in multiple sclerosis". FASEB J. ج. 18 ع. 2: 409–11. فبراير 2004. DOI:10.1096/fj.03-0606fje. PMID:14688203.{{استشهاد بدورية محكمة}}: صيانة الاستشهاد: دوي مجاني غير معلم (link)
Tiwari-Woodruff SK, Myers LW, Bronstein JM (أغسطس 2004). "Cerebrospinal fluid immunoglobulin G promotes oligodendrocyte progenitor cell migration". J. Neurosci. Res. ج. 77 ع. 3: 363–6. DOI:10.1002/jnr.20178. PMID:15248292.{{استشهاد بدورية محكمة}}: صيانة الاستشهاد: أسماء متعددة: قائمة المؤلفين (link)
"Cerebrospinal fluid derived from progressive multiple sclerosis patients promotes neuronal and oligodendroglial differentiation of human neural precursor cells in vitro". Neuroscience. ج. 250: 614–21. أكتوبر 2013. DOI:10.1016/j.neuroscience.2013.07.022. PMID:23876320.
Cristofanilli، Massimiliano؛ Rosenthal، Hannah؛ Cymring، Barbara؛ Gratch، Daniel؛ Pagano، Benjamin؛ Xie، Boxun؛ Sadiq، Saud A. (2014). "Progressive multiple sclerosis cerebrospinal fluid induces inflammatory demyelination, axonal loss, and astrogliosis in mice". Experimental Neurology. ج. 261: 620–32. DOI:10.1016/j.expneurol.2014.07.020. PMID:25111532.
"Transfer of multiple sclerosis into severe combined immunodeficiency mice by mononuclear cells from cerebrospinal fluid of the patients". PNAS. ج. 89 ع. 13: 6157–6161. 1992. Bibcode:1992PNAS...89.6157S. DOI:10.1073/pnas.89.13.6157. PMC:402141. PMID:1631103.
"Cerebrospinal fluid ceramides from patients with multiple sclerosis impair neuronal bioenergetics". Brain. ج. 137 ع. 8: 2271–86. أغسطس 2014. DOI:10.1093/brain/awu139. PMC:4164163. PMID:24893707.
Burgal, Mathur (يوليو 2014). "Molecular Shots". Ann Neurosci. ج. 21 ع. 3: 123. DOI:10.5214/ans.0972.7531.210311. PMC:4158786. PMID:25206080.
"Potassium channel KIR4.1 as an immune target in multiple sclerosis". N Engl J Med. ج. 367 ع. 2: 115–23. يوليو 2012. DOI:10.1056/NEJMoa1110740. PMC:5131800. PMID:22784115.
Schneider، Raphael (2013). "Autoantibodies to Potassium Channel KIR4.1 in Multiple Sclerosis". Frontiers in Neurology. ج. 4: 125. DOI:10.3389/fneur.2013.00125. PMC:3759297. PMID:24032025.{{استشهاد بدورية محكمة}}: صيانة الاستشهاد: دوي مجاني غير معلم (link)
Marnetto، Fabiana (2017). "Detection of potassium channel KIR4.1 antibodies in Multiple Sclerosis patients". Journal of Immunological Methods. ج. 445: 53–58. DOI:10.1016/j.jim.2017.03.008. PMID:28300540.
Ayoglua Burcu؛ وآخرون (2016). "Anoctamin 2 identified as an autoimmune target in multiple sclerosis". PNAS. ج. 113 ع. 8: 2188–2193. Bibcode:2016PNAS..113.2188A. DOI:10.1073/pnas.1518553113. PMC:4776531. PMID:26862169.
Matthews Lucy؛ وآخرون (2015). "Imaging Surrogates of Disease Activity in Neuromyelitis Optica Allow Distinction from Multiple Sclerosis". PLOS ONE. ج. 10 ع. 9: e0137715. Bibcode:2015PLoSO..1037715M. DOI:10.1371/journal.pone.0137715. PMC:4575169. PMID:26381510.{{استشهاد بدورية محكمة}}: صيانة الاستشهاد: دوي مجاني غير معلم (link)
"Axonal damage induced by cerebrospinal fluid from patients with relapsing-remitting multiple sclerosis". Journal of Neuroimmunology. ج. 104 ع. 1: 58–67. 2000. DOI:10.1016/S0165-5728(99)00225-8. PMID:10683515.
"Clusters of activated microglia in normal-appearing white matter show signs of innate immune activation". Journal of Neuroinflammation. ج. 9: 156. 2012. DOI:10.1186/1742-2094-9-156. PMC:3411485. PMID:22747960.{{استشهاد بدورية محكمة}}: صيانة الاستشهاد: دوي مجاني غير معلم (link)
"A gradient in cortical pathology in multiple sclerosis by in vivo quantitative 7 T imaging". Brain. ج. 138 ع. Pt 4: 932–45. 2015. DOI:10.1093/brain/awv011. PMC:4677339. PMID:25681411.
Hottenrott T, Dersch R, Berger B, Rauer S, Eckenweiler M, Huzly D, Stich O (2015). "The intrathecal, polyspecific antiviral immune response in neurosarcoidosis, acute disseminated encephalomyelitis and autoimmune encephalitis compared to multiple sclerosis in a tertiary hospital cohort". Fluids Barriers CNS. ج. 12: 27. DOI:10.1186/s12987-015-0024-8. PMC:4677451. PMID:26652013.{{استشهاد بدورية محكمة}}: صيانة الاستشهاد: أسماء متعددة: قائمة المؤلفين (link) صيانة الاستشهاد: دوي مجاني غير معلم (link)

ncbi.nlm.nih.gov

"Perivenous demyelination: association with clinically defined acute disseminated encephalomyelitis and comparison with pathologically confirmed multiple sclerosis". Brain. ج. 133 ع. 2: 333–348. 2010. DOI:10.1093/brain/awp321. PMC:2822631. PMID:20129932.
Lassman H (2019). "Pathogenic Mechanisms Associated With Different Clinical Courses of Multiple Sclerosis". Front Immunol. ج. 9: 3116. DOI:10.3389/fimmu.2018.03116. PMC:6335289. PMID:30687321.{{استشهاد بدورية محكمة}}: صيانة الاستشهاد: دوي مجاني غير معلم (link)
Ontaneda؛ وآخرون (نوفمبر 2014). "Identifying the Start of Multiple Sclerosis Injury: A Serial DTI Study". J Neuroimaging. ج. 24 ع. 6: 569–76. DOI:10.1111/jon.12082. PMC:4221810. PMID:25370339.
"MR Spectroscopy Indicates Diffuse Multiple Sclerosis Activity During Remission". J. Neurol. Neurosurg. Psychiatry. ج. 80 ع. 12: 1330–6. يونيو 2009. DOI:10.1136/jnnp.2009.176263. PMC:2900785. PMID:19546105.
"Dysregulated Epstein-Barr virus infection in the multiple sclerosis brain". Journal of Experimental Medicine. ج. 204 ع. 12: 2899–2912. نوفمبر 2007. DOI:10.1084/jem.20071030. PMC:2118531. PMID:17984305.
"Distinct patterns of multiple sclerosis pathology indicates heterogeneity on pathogenesis". Brain Pathol. ج. 6 ع. 3: 259–74. يوليو 1996. DOI:10.1111/j.1750-3639.1996.tb00854.x. PMC:7161824. PMID:8864283.
"Humoral pattern II multiple sclerosis pathology not associated with neuromyelitis Optica IgG". Arch Neurol. ج. 66 ع. 10: 1298–9. أكتوبر 2009. DOI:10.1001/archneurol.2009.199. PMC:2767176. PMID:19822791.
Srivastava، Rajneesh؛ Aslam، Muhammad؛ Kalluri، Sudhakar Reddy؛ Schirmer، Lucas؛ Buck، Dorothea؛ Tackenberg، Björn؛ Rothhammer، Veit؛ Chan، Andrew؛ Gold، Ralf (2012). "Potassium Channel KIR4.1 as an Immune Target in Multiple Sclerosis". New England Journal of Medicine. ج. 367 ع. 2: 115–23. DOI:10.1056/NEJMoa1110740. PMC:5131800. PMID:22784115.
Ayoglu، Burcu؛ Mitsios، Nicholas؛ Kockum، Ingrid؛ Khademi، Mohsen؛ Zandian، Arash؛ Sjöberg، Ronald؛ Forsström، Björn؛ Bredenberg، Johan؛ Lima Bomfim، Izaura (2016). "Anoctamin 2 identified as an autoimmune target in multiple sclerosis". Proceedings of the National Academy of Sciences. ج. 113 ع. 8: 2188–2193. Bibcode:2016PNAS..113.2188A. DOI:10.1073/pnas.1518553113. PMC:4776531. PMID:26862169.
Spadaro Melania؛ وآخرون (2015). "Histopathology and clinical course of MOG-antibody-associated encephalomyelitis". Annals of Clinical and Translational Neurology. ج. 2 ع. 3: 295–301. DOI:10.1002/acn3.164. PMC:4369279. PMID:25815356.
Planas Raquel؛ وآخرون (2015). "Central role of Th2/Tc2 lymphocytes in pattern II multiple sclerosis lesions". Journal. ج. 2 ع. 9: 875–893. DOI:10.1002/acn3.218. PMC:4574806. PMID:26401510.
"Sequencing the immunopathologic heterogeneity in multiple sclerosis". Annals of Clinical and Translational Neurology. ج. 2 ع. 9: 873–874. 2015. DOI:10.1002/acn3.230. PMC:4574805. PMID:26401509.
Marik، C.؛ Felts، P. A.؛ Bauer، J.؛ Lassmann، H.؛ Smith، K. J. (2007). "Lesion genesis in a subset of patients with multiple sclerosis: A role for innate immunity?". Brain. ج. 130 ع. 11: 2800–2815. DOI:10.1093/brain/awm236. PMC:2981817. PMID:17956913.
"Pathophysiological processes in multiple sclerosis: focus on nuclear factor erythroid-2-related factor 2 and emerging pathways". Clin Pharmacol. ج. 6: 35–42. فبراير 2014. DOI:10.2147/CPAA.S35033. PMC:3938468. PMID:24591852.{{استشهاد بدورية محكمة}}: صيانة الاستشهاد: دوي مجاني غير معلم (link)
"Pattern II and pattern III MS are entities distinct from pattern I MS: evidence from cerebrospinal fluid analysis". J Neuroinflammation. ج. 14 ع. 1: 171. 29 أغسطس 2017. DOI:10.1186/s12974-017-0929-z. PMC:5576197. PMID:28851393.{{استشهاد بدورية محكمة}}: صيانة الاستشهاد: دوي مجاني غير معلم (link)
"Antigen microarrays identify unique serum autoantibody signatures in clinical and pathologic subtypes of multiple sclerosis". Proc Natl Acad Sci USA. ج. 105 ع. 48: 18889–94. ديسمبر 2008. Bibcode:2008PNAS..10518889Q. DOI:10.1073/pnas.0806310105. PMC:2596207. PMID:19028871.
"Mitochondrial defects in acute multiple sclerosis lesions". Brain. ج. 131 ع. Pt 7: 1722–35. 2008. DOI:10.1093/brain/awn105. PMC:2442422. PMID:18515320.
Wang Zhe؛ وآخرون (2016). "Nuclear Receptor NR1H3 in Familial Multiple Sclerosis". Neuron. ج. 90 ع. 5: 948–954. DOI:10.1016/j.neuron.2016.04.039. PMC:5092154. PMID:27253448.
"Primary Progressive Multiple Sclerosis: Putting Together the Puzzle". Front. Neurol. ج. 8: 8–234. 2017. DOI:10.3389/fneur.2017.00234. PMC:5449443. PMID:28620346.{{استشهاد بدورية محكمة}}: صيانة الاستشهاد: دوي مجاني غير معلم (link)
"Inside-Out versus Outside-In models for virus induced demyelination: axonal damage triggering demyelination". Springer Semin Immunopathol. ج. 24 ع. 2: 105–25. 2002. DOI:10.1007/s00281-002-0105-z. PMC:7079941. PMID:12503060.
"Loss of 'homeostatic' microglia and patterns of their activation in active multiple sclerosis". Brain. ج. 140 ع. 7: 1900–1913. 2017. DOI:10.1093/brain/awx113. PMC:6057548. PMID:28541408.
"Identification of an essential endogenous regulator of blood–brain barrier integrity, and its pathological and therapeutic implications". Proceedings of the National Academy of Sciences of the United States of America. ج. 110 ع. 3: 832–841. 15 يناير 2013. Bibcode:2013PNAS..110..832C. DOI:10.1073/pnas.1209362110. PMC:3549094. PMID:23277546.
"Population-Wide Principal Component-Based Quantification of Blood-Brain-Barrier Dynamics in Multiple Sclerosis". NeuroImage. ج. 57 ع. 4: 1430–46. مايو 2011. DOI:10.1016/j.neuroimage.2011.05.038. PMC:3138825. PMID:21635955.
"Permeation of blood-borne IL15 across the blood–brain barrier and the effect of LPS". J. Neurochem. ج. 106 ع. 1: 313–9. 2008. DOI:10.1111/j.1471-4159.2008.05390.x. PMC:3939609. PMID:18384647.
"Monocyte migration and LFA-1 mediated attachment to brain microvascular endothelia is regulated by SDF-1α through Lyn kinase". Journal of Immunology. ج. 181 ع. 7: 4632–7. أكتوبر 2008. DOI:10.4049/jimmunol.181.7.4632. PMC:2721474. PMID:18802065.
"Magnetic resonance imaging of human brain macrophage infiltration". Neurotherapeutics. ج. 4 ع. 3: 434–42. 2007. DOI:10.1016/j.nurt.2007.05.005. PMC:7479730. PMID:17599709.
"Biomarkers indicative of blood–brain barrier disruption in multiple sclerosis". Dis. Markers. ج. 22 ع. 4: 235–44. 2006. DOI:10.1155/2006/709869. PMC:3850823. PMID:17124345.{{استشهاد بدورية محكمة}}: صيانة الاستشهاد: دوي مجاني غير معلم (link)
"Pathological Expression of CXCL12 at the Blood-Brain Barrier Correlates with Severity of Multiple Sclerosis". Am J Pathol. ج. 172 ع. 3: 799–808. مارس 2008. DOI:10.2353/ajpath.2008.070918. PMC:2258272. PMID:18276777.
"Imaging correlates of leukocyte accumulation and CXCR4/CXCR12 in multiple sclerosis". Arch. Neurol. ج. 66 ع. 1: 44–53. يناير 2009. DOI:10.1001/archneurol.2008.512. PMC:2792736. PMID:19139298. مؤرشف من الأصل في 2018-04-03.
"Biomarkers indicative of blood–brain barrier disruption in multiple sclerosis". Disease Markers. ج. 22 ع. 4: 235–44. 2006. DOI:10.1155/2006/709869. PMC:3850823. PMID:17124345. مؤرشف من الأصل في 2020-08-01.{{استشهاد بدورية محكمة}}: صيانة الاستشهاد: دوي مجاني غير معلم (link)
"White Matter Hemodynamic Abnormalities precede Sub-cortical Gray Matter Changes in Multiple Sclerosis". J. Neurol. Sci. ج. 282 ع. 1–2: 28–33. يوليو 2009. DOI:10.1016/j.jns.2008.12.036. PMC:2737614. PMID:19181347.
Kremer؛ وآخرون (2019). "pHERV-W envelope protein fuels microglial cell-dependent damage of myelinated axons in multiple sclerosis". PNAS. ج. 116 ع. 30: 15216–15225. DOI:10.1073/pnas.1901283116. PMC:6660731. PMID:31213545.
Lisak RP (2019). "Human retrovirus pHEV-W envelope protein and the pathogenesis of multiple sclerosis". PNAS. ج. 116 ع. 30: 14791–14793. DOI:10.1073/pnas.1909786116. PMC:6660775. PMID:31289223.
"7T MRI: New Vision of Microvascular Abnormalities in Multiple Sclerosis". Archives of Neurology. ج. 65 ع. 6: 812–6. 2008. DOI:10.1001/archneur.65.6.812. PMC:2579786. PMID:18541803.
"Chronic cerebrospinal venous insufficiency in patients with multiple sclerosis". J. Neurol. Neurosurg. Psychiatry. ج. 80 ع. 4: 392–9. أبريل 2009. DOI:10.1136/jnnp.2008.157164. PMC:2647682. PMID:19060024. مؤرشف من الأصل في 2009-08-04.
"Chronic cerebrospinal venous insufficiency in patients with multiple sclerosis". J. Neurol. Neurosurg. Psychiatry. ج. 80 ع. 4: 392–9. أبريل 2009. DOI:10.1136/jnnp.2008.157164. PMC:2647682. PMID:19060024.
Lassmann H (2019). "Pathogenic Mechanisms Associated With Different Clinical Courses of Multiple Sclerosis". Front Immunol. ج. 9: 3116. DOI:10.3389/fimmu.2018.03116. PMC:6335289. PMID:30687321.{{استشهاد بدورية محكمة}}: صيانة الاستشهاد: دوي مجاني غير معلم (link)
"Transfer of multiple sclerosis into severe combined immunodeficiency mice by mononuclear cells from cerebrospinal fluid of the patients". PNAS. ج. 89 ع. 13: 6157–6161. 1992. Bibcode:1992PNAS...89.6157S. DOI:10.1073/pnas.89.13.6157. PMC:402141. PMID:1631103.
"Cerebrospinal fluid ceramides from patients with multiple sclerosis impair neuronal bioenergetics". Brain. ج. 137 ع. 8: 2271–86. أغسطس 2014. DOI:10.1093/brain/awu139. PMC:4164163. PMID:24893707.
Burgal, Mathur (يوليو 2014). "Molecular Shots". Ann Neurosci. ج. 21 ع. 3: 123. DOI:10.5214/ans.0972.7531.210311. PMC:4158786. PMID:25206080.
"Potassium channel KIR4.1 as an immune target in multiple sclerosis". N Engl J Med. ج. 367 ع. 2: 115–23. يوليو 2012. DOI:10.1056/NEJMoa1110740. PMC:5131800. PMID:22784115.
Schneider، Raphael (2013). "Autoantibodies to Potassium Channel KIR4.1 in Multiple Sclerosis". Frontiers in Neurology. ج. 4: 125. DOI:10.3389/fneur.2013.00125. PMC:3759297. PMID:24032025.{{استشهاد بدورية محكمة}}: صيانة الاستشهاد: دوي مجاني غير معلم (link)
Ayoglua Burcu؛ وآخرون (2016). "Anoctamin 2 identified as an autoimmune target in multiple sclerosis". PNAS. ج. 113 ع. 8: 2188–2193. Bibcode:2016PNAS..113.2188A. DOI:10.1073/pnas.1518553113. PMC:4776531. PMID:26862169.
Matthews Lucy؛ وآخرون (2015). "Imaging Surrogates of Disease Activity in Neuromyelitis Optica Allow Distinction from Multiple Sclerosis". PLOS ONE. ج. 10 ع. 9: e0137715. Bibcode:2015PLoSO..1037715M. DOI:10.1371/journal.pone.0137715. PMC:4575169. PMID:26381510.{{استشهاد بدورية محكمة}}: صيانة الاستشهاد: دوي مجاني غير معلم (link)
"Clusters of activated microglia in normal-appearing white matter show signs of innate immune activation". Journal of Neuroinflammation. ج. 9: 156. 2012. DOI:10.1186/1742-2094-9-156. PMC:3411485. PMID:22747960.{{استشهاد بدورية محكمة}}: صيانة الاستشهاد: دوي مجاني غير معلم (link)
"A gradient in cortical pathology in multiple sclerosis by in vivo quantitative 7 T imaging". Brain. ج. 138 ع. Pt 4: 932–45. 2015. DOI:10.1093/brain/awv011. PMC:4677339. PMID:25681411.
Hottenrott T, Dersch R, Berger B, Rauer S, Eckenweiler M, Huzly D, Stich O (2015). "The intrathecal, polyspecific antiviral immune response in neurosarcoidosis, acute disseminated encephalomyelitis and autoimmune encephalitis compared to multiple sclerosis in a tertiary hospital cohort". Fluids Barriers CNS. ج. 12: 27. DOI:10.1186/s12987-015-0024-8. PMC:4677451. PMID:26652013.{{استشهاد بدورية محكمة}}: صيانة الاستشهاد: أسماء متعددة: قائمة المؤلفين (link) صيانة الاستشهاد: دوي مجاني غير معلم (link)

oxfordjournals.org

brain.oxfordjournals.org

"A quantitative analysis of oligodendrocytes in multiple sclerosis lesions - A study of 113 cases". Brain. ج. 122 ع. 12: 2279–2295. ديسمبر 1999. DOI:10.1093/brain/122.12.2279. PMID:10581222. مؤرشف من الأصل في 2008-08-29. اطلع عليه بتاريخ 2006-05-10.
"The pathogenesis of lesions and normal-appearing white matter changes in multiple sclerosis: a serial diffusion MRI study". Brain. ج. 123 ع. 8: 1667–76. أغسطس 2000. DOI:10.1093/brain/123.8.1667. PMID:10908196. مؤرشف من الأصل في 2017-12-01.

uzh.ch

zora.uzh.ch

Planas R, Santos R, Tomas-Ojer P, Cruciani C, Lutterotti A, Faigle W, Schaeren-Wiemers N, Espejo C, Eixarch H, Pinilla C, Martin R, Sospedra M (2018). "GDP-l-fucose synthase is a CD4+ T cell–specific autoantigen in DRB3*02:02 patients with multiple sclerosis" (PDF). Science Translational Medicine. ج. 10 ع. 462: eaat4301. DOI:10.1126/scitranslmed.aat4301. PMID:30305453. مؤرشف من الأصل (PDF) في 2020-02-10.{{استشهاد بدورية محكمة}}: صيانة الاستشهاد: أسماء متعددة: قائمة المؤلفين (link)

web.archive.org

"A serial study of new MS lesions and the white matter from which they arise". Neurology. ج. 51 ع. 6: 1689–97. ديسمبر 1998. DOI:10.1212/wnl.51.6.1689. PMID:9855524. مؤرشف من الأصل في 2008-05-22.
Holmes، Nick (15 نوفمبر 2001). "Part 1B Pathology: Lecture 11 - The Complement System". مؤرشف من الأصل في 2006-01-09. اطلع عليه بتاريخ 2006-05-10.
"A quantitative analysis of oligodendrocytes in multiple sclerosis lesions - A study of 113 cases". Brain. ج. 122 ع. 12: 2279–2295. ديسمبر 1999. DOI:10.1093/brain/122.12.2279. PMID:10581222. مؤرشف من الأصل في 2008-08-29. اطلع عليه بتاريخ 2006-05-10.
"Some MS patients have "Dramatic" responses to Plasma Exchange". Neurology Reviews. ج. 8 ع. 3. مارس 2000. مؤرشف من الأصل في 2001-02-23. اطلع عليه بتاريخ 2006-05-05.
Barnett، MH؛ Prineas، JW (أبريل 2004). "Relapsing and remitting multiple sclerosis: pathology of the newly forming lesion" (PDF). Annals of Neurology. ج. 55 ع. 4: 458–68. DOI:10.1002/ana.20016. PMID:15048884. مؤرشف من الأصل (PDF) في 2013-10-29. اطلع عليه بتاريخ 2009-10-28.
Michael H. Barnett؛ John W. Prineas (2004). "Relapsing and Remitting Multiple Sclerosis: Pathology of the Newly Forming Lesion" (PDF). Annals of Neurology. ج. 55 ع. 1: 458–468. DOI:10.1002/ana.20016. PMID:15048884. مؤرشف من الأصل (PDF) في 2017-03-01.
Dinesh K. Sivakolundu et al., Three‐Dimensional Lesion Phenotyping and Physiologic Characterization Inform Remyelination Ability in Multiple Sclerosis, 30 May 2019, https://doi.org/10.1111/jon.12633 نسخة محفوظة 21 يناير 2021 على موقع واي باك مشين.
"The pathogenesis of lesions and normal-appearing white matter changes in multiple sclerosis: a serial diffusion MRI study". Brain. ج. 123 ع. 8: 1667–76. أغسطس 2000. DOI:10.1093/brain/123.8.1667. PMID:10908196. مؤرشف من الأصل في 2017-12-01.
"Adhesion molecules in multiple sclerosis: relation to subtypes of disease and methylprednisolone therapy". Arch. Neurol. ج. 57 ع. 4: 546–51. أبريل 2000. DOI:10.1001/archneur.57.4.546. PMID:10768630. مؤرشف من الأصل في 2018-04-03.
Alexandre Prat, Nicole Beaulieu, Sylvain-Jacques Desjardins, New Therapeutic Target For Treatment Of Multiple Sclerosis, Jan. 2008 نسخة محفوظة 2011-05-16 على موقع واي باك مشين.
"Imaging correlates of leukocyte accumulation and CXCR4/CXCR12 in multiple sclerosis". Arch. Neurol. ج. 66 ع. 1: 44–53. يناير 2009. DOI:10.1001/archneurol.2008.512. PMC:2792736. PMID:19139298. مؤرشف من الأصل في 2018-04-03.
"Impact of methylprednisolone treatment on the expression of macrophage inflammatory protein 3alpha and B lymphocyte chemoattractant in serum of multiple sclerosis patients" (PDF). Pharmacol Rep. ج. 60 ع. 4: 549–54. 2008. PMID:18799824. مؤرشف من الأصل (PDF) في 2016-03-04.
"Biomarkers indicative of blood–brain barrier disruption in multiple sclerosis". Disease Markers. ج. 22 ع. 4: 235–44. 2006. DOI:10.1155/2006/709869. PMC:3850823. PMID:17124345. مؤرشف من الأصل في 2020-08-01.{{استشهاد بدورية محكمة}}: صيانة الاستشهاد: دوي مجاني غير معلم (link)
University of Zurich(2018, October 11). Link Between Gut Flora and Multiple Sclerosis Discovered. NeuroscienceNews. Retrieved October 11, 2018 from http://neurosciencenews.com/multiple-sclerosis-gut-flora-10003/ نسخة محفوظة 2020-11-09 على موقع واي باك مشين.
Planas R, Santos R, Tomas-Ojer P, Cruciani C, Lutterotti A, Faigle W, Schaeren-Wiemers N, Espejo C, Eixarch H, Pinilla C, Martin R, Sospedra M (2018). "GDP-l-fucose synthase is a CD4+ T cell–specific autoantigen in DRB3*02:02 patients with multiple sclerosis" (PDF). Science Translational Medicine. ج. 10 ع. 462: eaat4301. DOI:10.1126/scitranslmed.aat4301. PMID:30305453. مؤرشف من الأصل (PDF) في 2020-02-10.{{استشهاد بدورية محكمة}}: صيانة الاستشهاد: أسماء متعددة: قائمة المؤلفين (link)
"Normal-appearing White and Grey Matter Damage in Multiple Sclerosis. Book review". AJNR. ج. 27 ع. 4: 945–946. 2004. مؤرشف من الأصل في 2011-09-27.
"Chronic cerebrospinal venous insufficiency in patients with multiple sclerosis". J. Neurol. Neurosurg. Psychiatry. ج. 80 ع. 4: 392–9. أبريل 2009. DOI:10.1136/jnnp.2008.157164. PMC:2647682. PMID:19060024. مؤرشف من الأصل في 2009-08-04.
Bryce Weir (2010). J Neuro Sci 2010 - MS Vascular Etiology.pdf "MS, A vascular ethiology?" (PDF). Can. J. Neurol. Sci. ج. 37 ع. 6: 745–757. DOI:10.1017/s0317167100051404. PMID:21059535. مؤرشف من الأصل (PDF) في 2011-09-15. {{استشهاد بدورية محكمة}}: تحقق من قيمة |مسار أرشيف= (مساعدة)
"Intracranial venous haemodynamics in multiple sclerosis". Curr Neurovasc Res. ج. 4 ع. 4: 252–8. نوفمبر 2007. DOI:10.2174/156720207782446298. PMID:18045150. مؤرشف من الأصل في 2018-10-19.
"Embryological background of truncular venous malformation in the extracranial venous pathways as the cause of chronic cerebro spinal venous insufficiency" (PDF). Int Angiol. ج. 29 ع. 2: 95–108. أبريل 2010. PMID:20351665. مؤرشف من الأصل (PDF) في 2010-07-04.
Fabio Duranti؛ Massimo Pieri؛ Rossella Zenobi؛ Diego Centonze؛ Fabio Buttari؛ Sergio Bernardini؛ Mariarita Dessi. "kFLC Index: a novel approach in early diagnosis of Multiple Sclerosis". International Journal of Scientific Research. ج. 4 ع. 8. مؤرشف من الأصل في 2016-08-28. اطلع عليه بتاريخ 2018-08-27.

wiley.com

www3.interscience.wiley.com

"Chronic cerebrospinal venous insufficiency and multiple sclerosis". Annals of Neurology. ج. 67 ع. 3: 286–90. مارس 2010. DOI:10.1002/ana.22001. PMID:20373339. مؤرشف من الأصل في 2010-11-23.

worldwidejournals.in

Fabio Duranti؛ Massimo Pieri؛ Rossella Zenobi؛ Diego Centonze؛ Fabio Buttari؛ Sergio Bernardini؛ Mariarita Dessi. "kFLC Index: a novel approach in early diagnosis of Multiple Sclerosis". International Journal of Scientific Research. ج. 4 ع. 8. مؤرشف من الأصل في 2016-08-28. اطلع عليه بتاريخ 2018-08-27.