H3K9me2 (Arabic Wikipedia)

Analysis of information sources in references of the Wikipedia article "H3K9me2" in Arabic language version.

refsWebsite

Global rank Arabic rank

33nih.gov

4^th place

6^th place

18doi.org

2^nd place

5^th place

2web.archive.org

1^st place

2actrec.gov.in

low place

1harvard.edu

18^th place

33^rd place

actrec.gov.in

"H3K9me2". HIstome: The Histone Infobase. مؤرشف من الأصل في 2019-12-26. اطلع عليه بتاريخ 2018-06-08.
"Histone-lysine N-methyltransferase, H3 lysine-9 specific 3". HIstome: The Histone Infobase. مؤرشف من الأصل في 2020-01-14. اطلع عليه بتاريخ 2018-06-08.

doi.org

Robison AJ، Nestler EJ (أكتوبر 2011). "Transcriptional and epigenetic mechanisms of addiction". Nature Reviews. Neuroscience. ج. 12 ع. 11: 623–37. DOI:10.1038/nrn3111. PMC:3272277. PMID:21989194.
Nestler EJ (أغسطس 2015). "Role of the Brain's Reward Circuitry in Depression: Transcriptional Mechanisms". International Review of Neurobiology. ج. 124: 151–70. DOI:10.1016/bs.irn.2015.07.003. PMC:4690450. PMID:26472529. Chronic social defeat stress decreases expression of G9a and GLP (G9a-like protein), two histone methyltransferases that catalyze the dimethylation of Lys9 of histone H3 (H3K9me2) (Covington et al., 2011), a mark associated with gene repression.
Barski A، Cuddapah S، Cui K، Roh TY، Schones DE، Wang Z، وآخرون (مايو 2007). "High-resolution profiling of histone methylations in the human genome". Cell. ج. 129 ع. 4: 823–37. DOI:10.1016/j.cell.2007.05.009. PMID:17512414.
Wang Z، Zang C، Rosenfeld JA، Schones DE، Barski A، Cuddapah S، وآخرون (يوليو 2008). "Combinatorial patterns of histone acetylations and methylations in the human genome". Nature Genetics. ج. 40 ع. 7: 897–903. DOI:10.1038/ng.154. PMC:2769248. PMID:18552846.
Shinkai Y، Tachibana M (أبريل 2011). "H3K9 methyltransferase G9a and the related molecule GLP". Genes & Development. ج. 25 ع. 8: 781–8. DOI:10.1101/gad.2027411. PMC:3078703. PMID:21498567.
Zhang T، Termanis A، Özkan B، Bao XX، Culley J، de Lima Alves F، وآخرون (أبريل 2016). "G9a/GLP Complex Maintains Imprinted DNA Methylation in Embryonic Stem Cells". Cell Reports. ج. 15 ع. 1: 77–85. DOI:10.1016/j.celrep.2016.03.007. PMC:4826439. PMID:27052169.
Filion GJ، van Steensel B (يناير 2010). "Reassessing the abundance of H3K9me2 chromatin domains in embryonic stem cells". Nature Genetics. ج. 42 ع. 1: 4, author reply 5–6. DOI:10.1038/ng0110-4. PMID:20037608.
McDonald OG، Wu H، Timp W، Doi A، Feinberg AP (يوليو 2011). "Genome-scale epigenetic reprogramming during epithelial-to-mesenchymal transition". Nature Structural & Molecular Biology. ج. 18 ع. 8: 867–74. DOI:10.1038/nsmb.2084. PMC:3150339. PMID:21725293.
Wen B، Wu H، Shinkai Y، Irizarry RA، Feinberg AP (فبراير 2009). "Large histone H3 lysine 9 dimethylated chromatin blocks distinguish differentiated from embryonic stem cells". Nature Genetics. ج. 41 ع. 2: 246–50. DOI:10.1038/ng.297. PMC:2632725. PMID:19151716.
Jørgensen HF، Fisher AG (مارس 2009). "LOCKing in Cellular Potential". Cell Stem Cell. ج. 4 ع. 3: 192–4. DOI:10.1016/j.stem.2009.02.007. PMID:19265653.
Cloos PA، Christensen J، Agger K، Helin K (مايو 2008). "Erasing the methyl mark: histone demethylases at the center of cellular differentiation and disease". Genes & Development. ج. 22 ع. 9: 1115–40. DOI:10.1101/gad.1652908. PMC:2732404. PMID:18451103.
Chen X، Skutt-Kakaria K، Davison J، Ou YL، Choi E، Malik P، وآخرون (نوفمبر 2012). "G9a/GLP-dependent histone H3K9me2 patterning during human hematopoietic stem cell lineage commitment". Genes & Development. ج. 26 ع. 22: 2499–511. DOI:10.1101/gad.200329.112. PMC:3505820. PMID:23105005.
Rodriguez-Madoz JR، San Jose-Eneriz E، Rabal O، Zapata-Linares N، Miranda E، Rodriguez S، وآخرون (2017). "Reversible dual inhibitor against G9a and DNMT1 improves human iPSC derivation enhancing MET and facilitating transcription factor engagement to the genome". PLOS ONE. ج. 12 ع. 12: e0190275. Bibcode:2017PLoSO..1290275R. DOI:10.1371/journal.pone.0190275. PMC:5744984. PMID:29281720.
Harman JL، Dobnikar L، Chappell J، Stokell BG، Dalby A، Foote K، وآخرون (نوفمبر 2019). "Epigenetic Regulation of Vascular Smooth Muscle Cells by Histone H3 Lysine 9 Dimethylation Attenuates Target Gene-Induction by Inflammatory Signaling". Arteriosclerosis, Thrombosis, and Vascular Biology. ج. 39 ع. 11: 2289–2302. DOI:10.1161/ATVBAHA.119.312765. PMC:6818986. PMID:31434493.
Fang TC، Schaefer U، Mecklenbrauker I، Stienen A، Dewell S، Chen MS، وآخرون (أبريل 2012). "Histone H3 lysine 9 di-methylation as an epigenetic signature of the interferon response". The Journal of Experimental Medicine. ج. 209 ع. 4: 661–9. DOI:10.1084/jem.20112343. PMC:3328357. PMID:22412156.
Fang TC، Schaefer U، Mecklenbrauker I، Stienen A، Dewell S، Chen MS، وآخرون (أبريل 2012). "Histone H3 lysine 9 di-methylation as an epigenetic signature of the interferon response". The Journal of Experimental Medicine. ج. 209 ع. 4: 661–9. DOI:10.1084/jem.20112343. PMC:3328357. PMID:22412156.
Fang TC، Schaefer U، Mecklenbrauker I، Stienen A، Dewell S، Chen MS، وآخرون (أبريل 2012). "Histone H3 lysine 9 di-methylation as an epigenetic signature of the interferon response". The Journal of Experimental Medicine. ج. 209 ع. 4: 661–9. DOI:10.1084/jem.20112343. PMC:3328357. PMID:22412156.
Fang TC، Schaefer U، Mecklenbrauker I، Stienen A، Dewell S، Chen MS، وآخرون (أبريل 2012). "Histone H3 lysine 9 di-methylation as an epigenetic signature of the interferon response". The Journal of Experimental Medicine. ج. 209 ع. 4: 661–9. DOI:10.1084/jem.20112343. PMC:3328357. PMID:22412156.

harvard.edu

ui.adsabs.harvard.edu

Rodriguez-Madoz JR، San Jose-Eneriz E، Rabal O، Zapata-Linares N، Miranda E، Rodriguez S، وآخرون (2017). "Reversible dual inhibitor against G9a and DNMT1 improves human iPSC derivation enhancing MET and facilitating transcription factor engagement to the genome". PLOS ONE. ج. 12 ع. 12: e0190275. Bibcode:2017PLoSO..1290275R. DOI:10.1371/journal.pone.0190275. PMC:5744984. PMID:29281720.

nih.gov

pubmed.ncbi.nlm.nih.gov

Robison AJ، Nestler EJ (أكتوبر 2011). "Transcriptional and epigenetic mechanisms of addiction". Nature Reviews. Neuroscience. ج. 12 ع. 11: 623–37. DOI:10.1038/nrn3111. PMC:3272277. PMID:21989194.
Nestler EJ (أغسطس 2015). "Role of the Brain's Reward Circuitry in Depression: Transcriptional Mechanisms". International Review of Neurobiology. ج. 124: 151–70. DOI:10.1016/bs.irn.2015.07.003. PMC:4690450. PMID:26472529. Chronic social defeat stress decreases expression of G9a and GLP (G9a-like protein), two histone methyltransferases that catalyze the dimethylation of Lys9 of histone H3 (H3K9me2) (Covington et al., 2011), a mark associated with gene repression.
Barski A، Cuddapah S، Cui K، Roh TY، Schones DE، Wang Z، وآخرون (مايو 2007). "High-resolution profiling of histone methylations in the human genome". Cell. ج. 129 ع. 4: 823–37. DOI:10.1016/j.cell.2007.05.009. PMID:17512414.
Wang Z، Zang C، Rosenfeld JA، Schones DE، Barski A، Cuddapah S، وآخرون (يوليو 2008). "Combinatorial patterns of histone acetylations and methylations in the human genome". Nature Genetics. ج. 40 ع. 7: 897–903. DOI:10.1038/ng.154. PMC:2769248. PMID:18552846.
Shinkai Y، Tachibana M (أبريل 2011). "H3K9 methyltransferase G9a and the related molecule GLP". Genes & Development. ج. 25 ع. 8: 781–8. DOI:10.1101/gad.2027411. PMC:3078703. PMID:21498567.
Zhang T، Termanis A، Özkan B، Bao XX، Culley J، de Lima Alves F، وآخرون (أبريل 2016). "G9a/GLP Complex Maintains Imprinted DNA Methylation in Embryonic Stem Cells". Cell Reports. ج. 15 ع. 1: 77–85. DOI:10.1016/j.celrep.2016.03.007. PMC:4826439. PMID:27052169.
Filion GJ، van Steensel B (يناير 2010). "Reassessing the abundance of H3K9me2 chromatin domains in embryonic stem cells". Nature Genetics. ج. 42 ع. 1: 4, author reply 5–6. DOI:10.1038/ng0110-4. PMID:20037608.
McDonald OG، Wu H، Timp W، Doi A، Feinberg AP (يوليو 2011). "Genome-scale epigenetic reprogramming during epithelial-to-mesenchymal transition". Nature Structural & Molecular Biology. ج. 18 ع. 8: 867–74. DOI:10.1038/nsmb.2084. PMC:3150339. PMID:21725293.
Wen B، Wu H، Shinkai Y، Irizarry RA، Feinberg AP (فبراير 2009). "Large histone H3 lysine 9 dimethylated chromatin blocks distinguish differentiated from embryonic stem cells". Nature Genetics. ج. 41 ع. 2: 246–50. DOI:10.1038/ng.297. PMC:2632725. PMID:19151716.
Jørgensen HF، Fisher AG (مارس 2009). "LOCKing in Cellular Potential". Cell Stem Cell. ج. 4 ع. 3: 192–4. DOI:10.1016/j.stem.2009.02.007. PMID:19265653.
Cloos PA، Christensen J، Agger K، Helin K (مايو 2008). "Erasing the methyl mark: histone demethylases at the center of cellular differentiation and disease". Genes & Development. ج. 22 ع. 9: 1115–40. DOI:10.1101/gad.1652908. PMC:2732404. PMID:18451103.
Chen X، Skutt-Kakaria K، Davison J، Ou YL، Choi E، Malik P، وآخرون (نوفمبر 2012). "G9a/GLP-dependent histone H3K9me2 patterning during human hematopoietic stem cell lineage commitment". Genes & Development. ج. 26 ع. 22: 2499–511. DOI:10.1101/gad.200329.112. PMC:3505820. PMID:23105005.
Rodriguez-Madoz JR، San Jose-Eneriz E، Rabal O، Zapata-Linares N، Miranda E، Rodriguez S، وآخرون (2017). "Reversible dual inhibitor against G9a and DNMT1 improves human iPSC derivation enhancing MET and facilitating transcription factor engagement to the genome". PLOS ONE. ج. 12 ع. 12: e0190275. Bibcode:2017PLoSO..1290275R. DOI:10.1371/journal.pone.0190275. PMC:5744984. PMID:29281720.
Harman JL، Dobnikar L، Chappell J، Stokell BG، Dalby A، Foote K، وآخرون (نوفمبر 2019). "Epigenetic Regulation of Vascular Smooth Muscle Cells by Histone H3 Lysine 9 Dimethylation Attenuates Target Gene-Induction by Inflammatory Signaling". Arteriosclerosis, Thrombosis, and Vascular Biology. ج. 39 ع. 11: 2289–2302. DOI:10.1161/ATVBAHA.119.312765. PMC:6818986. PMID:31434493.
Fang TC، Schaefer U، Mecklenbrauker I، Stienen A، Dewell S، Chen MS، وآخرون (أبريل 2012). "Histone H3 lysine 9 di-methylation as an epigenetic signature of the interferon response". The Journal of Experimental Medicine. ج. 209 ع. 4: 661–9. DOI:10.1084/jem.20112343. PMC:3328357. PMID:22412156.
Fang TC، Schaefer U، Mecklenbrauker I، Stienen A، Dewell S، Chen MS، وآخرون (أبريل 2012). "Histone H3 lysine 9 di-methylation as an epigenetic signature of the interferon response". The Journal of Experimental Medicine. ج. 209 ع. 4: 661–9. DOI:10.1084/jem.20112343. PMC:3328357. PMID:22412156.
Fang TC، Schaefer U، Mecklenbrauker I، Stienen A، Dewell S، Chen MS، وآخرون (أبريل 2012). "Histone H3 lysine 9 di-methylation as an epigenetic signature of the interferon response". The Journal of Experimental Medicine. ج. 209 ع. 4: 661–9. DOI:10.1084/jem.20112343. PMC:3328357. PMID:22412156.
Fang TC، Schaefer U، Mecklenbrauker I، Stienen A، Dewell S، Chen MS، وآخرون (أبريل 2012). "Histone H3 lysine 9 di-methylation as an epigenetic signature of the interferon response". The Journal of Experimental Medicine. ج. 209 ع. 4: 661–9. DOI:10.1084/jem.20112343. PMC:3328357. PMID:22412156.

ncbi.nlm.nih.gov

Robison AJ، Nestler EJ (أكتوبر 2011). "Transcriptional and epigenetic mechanisms of addiction". Nature Reviews. Neuroscience. ج. 12 ع. 11: 623–37. DOI:10.1038/nrn3111. PMC:3272277. PMID:21989194.
Nestler EJ (أغسطس 2015). "Role of the Brain's Reward Circuitry in Depression: Transcriptional Mechanisms". International Review of Neurobiology. ج. 124: 151–70. DOI:10.1016/bs.irn.2015.07.003. PMC:4690450. PMID:26472529. Chronic social defeat stress decreases expression of G9a and GLP (G9a-like protein), two histone methyltransferases that catalyze the dimethylation of Lys9 of histone H3 (H3K9me2) (Covington et al., 2011), a mark associated with gene repression.
Wang Z، Zang C، Rosenfeld JA، Schones DE، Barski A، Cuddapah S، وآخرون (يوليو 2008). "Combinatorial patterns of histone acetylations and methylations in the human genome". Nature Genetics. ج. 40 ع. 7: 897–903. DOI:10.1038/ng.154. PMC:2769248. PMID:18552846.
Shinkai Y، Tachibana M (أبريل 2011). "H3K9 methyltransferase G9a and the related molecule GLP". Genes & Development. ج. 25 ع. 8: 781–8. DOI:10.1101/gad.2027411. PMC:3078703. PMID:21498567.
Zhang T، Termanis A، Özkan B، Bao XX، Culley J، de Lima Alves F، وآخرون (أبريل 2016). "G9a/GLP Complex Maintains Imprinted DNA Methylation in Embryonic Stem Cells". Cell Reports. ج. 15 ع. 1: 77–85. DOI:10.1016/j.celrep.2016.03.007. PMC:4826439. PMID:27052169.
McDonald OG، Wu H، Timp W، Doi A، Feinberg AP (يوليو 2011). "Genome-scale epigenetic reprogramming during epithelial-to-mesenchymal transition". Nature Structural & Molecular Biology. ج. 18 ع. 8: 867–74. DOI:10.1038/nsmb.2084. PMC:3150339. PMID:21725293.
Wen B، Wu H، Shinkai Y، Irizarry RA، Feinberg AP (فبراير 2009). "Large histone H3 lysine 9 dimethylated chromatin blocks distinguish differentiated from embryonic stem cells". Nature Genetics. ج. 41 ع. 2: 246–50. DOI:10.1038/ng.297. PMC:2632725. PMID:19151716.
Cloos PA، Christensen J، Agger K، Helin K (مايو 2008). "Erasing the methyl mark: histone demethylases at the center of cellular differentiation and disease". Genes & Development. ج. 22 ع. 9: 1115–40. DOI:10.1101/gad.1652908. PMC:2732404. PMID:18451103.
Chen X، Skutt-Kakaria K، Davison J، Ou YL، Choi E، Malik P، وآخرون (نوفمبر 2012). "G9a/GLP-dependent histone H3K9me2 patterning during human hematopoietic stem cell lineage commitment". Genes & Development. ج. 26 ع. 22: 2499–511. DOI:10.1101/gad.200329.112. PMC:3505820. PMID:23105005.
Rodriguez-Madoz JR، San Jose-Eneriz E، Rabal O، Zapata-Linares N، Miranda E، Rodriguez S، وآخرون (2017). "Reversible dual inhibitor against G9a and DNMT1 improves human iPSC derivation enhancing MET and facilitating transcription factor engagement to the genome". PLOS ONE. ج. 12 ع. 12: e0190275. Bibcode:2017PLoSO..1290275R. DOI:10.1371/journal.pone.0190275. PMC:5744984. PMID:29281720.
Harman JL، Dobnikar L، Chappell J، Stokell BG، Dalby A، Foote K، وآخرون (نوفمبر 2019). "Epigenetic Regulation of Vascular Smooth Muscle Cells by Histone H3 Lysine 9 Dimethylation Attenuates Target Gene-Induction by Inflammatory Signaling". Arteriosclerosis, Thrombosis, and Vascular Biology. ج. 39 ع. 11: 2289–2302. DOI:10.1161/ATVBAHA.119.312765. PMC:6818986. PMID:31434493.
Fang TC، Schaefer U، Mecklenbrauker I، Stienen A، Dewell S، Chen MS، وآخرون (أبريل 2012). "Histone H3 lysine 9 di-methylation as an epigenetic signature of the interferon response". The Journal of Experimental Medicine. ج. 209 ع. 4: 661–9. DOI:10.1084/jem.20112343. PMC:3328357. PMID:22412156.
Fang TC، Schaefer U، Mecklenbrauker I، Stienen A، Dewell S، Chen MS، وآخرون (أبريل 2012). "Histone H3 lysine 9 di-methylation as an epigenetic signature of the interferon response". The Journal of Experimental Medicine. ج. 209 ع. 4: 661–9. DOI:10.1084/jem.20112343. PMC:3328357. PMID:22412156.
Fang TC، Schaefer U، Mecklenbrauker I، Stienen A، Dewell S، Chen MS، وآخرون (أبريل 2012). "Histone H3 lysine 9 di-methylation as an epigenetic signature of the interferon response". The Journal of Experimental Medicine. ج. 209 ع. 4: 661–9. DOI:10.1084/jem.20112343. PMC:3328357. PMID:22412156.
Fang TC، Schaefer U، Mecklenbrauker I، Stienen A، Dewell S، Chen MS، وآخرون (أبريل 2012). "Histone H3 lysine 9 di-methylation as an epigenetic signature of the interferon response". The Journal of Experimental Medicine. ج. 209 ع. 4: 661–9. DOI:10.1084/jem.20112343. PMC:3328357. PMID:22412156.

web.archive.org

"H3K9me2". HIstome: The Histone Infobase. مؤرشف من الأصل في 2019-12-26. اطلع عليه بتاريخ 2018-06-08.
"Histone-lysine N-methyltransferase, H3 lysine-9 specific 3". HIstome: The Histone Infobase. مؤرشف من الأصل في 2020-01-14. اطلع عليه بتاريخ 2018-06-08.