MeCP2 (Arabic Wikipedia)

Analysis of information sources in references of the Wikipedia article "MeCP2" in Arabic language version.

refsWebsite

Global rank Arabic rank

25nih.gov

4^th place

6^th place

16doi.org

2^nd place

5^th place

5semanticscholar.org

11^th place

86^th place

4harvard.edu

18^th place

33^rd place

2web.archive.org

1^st place

1cornell.edu

332^nd place

400^th place

cornell.edu

instruct1.cit.cornell.edu

Paul A. Wade (ديسمبر 2001). "Methyl CpG-binding proteins and transcriptional repression" (PDF). BioEssays. ج. 23 ع. 12: 1131–1137. DOI:10.1002/bies.10008. PMID:11746232. S2CID:37525856. مؤرشف من الأصل (PDF) في 2007-08-14.

doi.org

Amir RE، Van den Veyver IB، Wan M، Tran CQ، Francke U، Zoghbi HY (أكتوبر 1999). "Rett syndrome is caused by mutations in X-linked MECP2, encoding methyl-CpG-binding protein 2". Nat. Genet. ج. 23 ع. 2: 185–8. DOI:10.1038/13810. PMID:10508514. S2CID:3350350.
Lewis JD، Meehan RR، Henzel WJ، Maurer-Fogy I، Jeppesen P، Klein F، Bird A (يونيو 1992). "Purification, sequence, and cellular localization of a novel chromosomal protein that binds to methylated DNA". Cell. ج. 69 ع. 6: 905–14. DOI:10.1016/0092-8674(92)90610-O. PMID:1606614. S2CID:6825994.
Chahrour M، وآخرون (2008). "MECP2, a key contributor to neurological disease, activates and represses transcription". Science. ج. 320 ع. 5880: 1224–9. Bibcode:2008Sci...320.1224C. DOI:10.1126/science.1153252. PMC:2443785. PMID:18511691.
Šimčíková D، Heneberg P (ديسمبر 2019). "Refinement of evolutionary medicine predictions based on clinical evidence for the manifestations of Mendelian diseases". Scientific Reports. ج. 9 ع. 1: 18577. DOI:10.1038/s41598-019-54976-4. PMC:6901466. PMID:31819097.
Cohen S، Zhou Z، Greenberg ME (مايو 2008). "Medicine. Activating a repressor". Science. ج. 320 ع. 5880: 1172–3. DOI:10.1126/science.1159146. PMC:2857976. PMID:18511680.
Luikenhuis S، Giacometti E، Beard CF، Jaenisch R (أبريل 2004). "Expression of MeCP2 in postmitotic neurons rescues Rett syndrome in mice". Proc. Natl. Acad. Sci. U.S.A. ج. 101 ع. 16: 6033–8. Bibcode:2004PNAS..101.6033L. DOI:10.1073/pnas.0401626101. PMC:395918. PMID:15069197.
Yasui DH، Peddada S، Bieda MC، Vallero RO، Hogart A، Nagarajan RP، Thatcher KN، Farnham PJ، Lasalle JM (ديسمبر 2007). "Integrated epigenomic analyses of neuronal MeCP2 reveal a role for long-range interaction with active genes". Proc. Natl. Acad. Sci. U.S.A. ج. 104 ع. 49: 19416–21. Bibcode:2007PNAS..10419416Y. DOI:10.1073/pnas.0707442104. PMC:2148304. PMID:18042715.
Chahrour M، Jung SY، Shaw C، Zhou X، Wong ST، Qin J، Zoghbi HY (مايو 2008). "MeCP2, a key contributor to neurological disease, activates and represses transcription". Science. ج. 320 ع. 5880: 1224–9. Bibcode:2008Sci...320.1224C. DOI:10.1126/science.1153252. PMC:2443785. PMID:18511691.
Georgel PT، Horowitz-Scherer RA، Adkins N، Woodcock CL، Wade PA، Hansen JC (أغسطس 2003). "Chromatin compaction by human MeCP2. Assembly of novel secondary chromatin structures in the absence of DNA methylation". J. Biol. Chem. ج. 278 ع. 34: 32181–8. DOI:10.1074/jbc.M305308200. PMID:12788925.
LaSalle JM (2007). "The odyssey of MeCP2 and parental imprinting". Epigenetics. ج. 2 ع. 1: 5–10. DOI:10.4161/epi.2.1.3697. PMC:1866173. PMID:17486180.
Alessio N، Riccitiello F، Squillaro T، Capasso S، Del Gaudio S، Di Bernardo G، Cipollaro M، Melone MAB، Peluso G، Galderisi U (مارس 2018). "Neural stem cells from a mouse model of Rett syndrome are prone to senescence, show reduced capacity to cope with genotoxic stress, and are impaired in the differentiation process". Exp. Mol. Med. ج. 50 ع. 3: 1. DOI:10.1038/s12276-017-0005-x. PMC:6118406. PMID:29563495.
Wakefield RI، Smith BO، Nan X، Free A، Soteriou A، Uhrin D، Bird AP، Barlow PN (سبتمبر 1999). "The solution structure of the domain from MeCP2 that binds to methylated DNA". J. Mol. Biol. ج. 291 ع. 5: 1055–65. DOI:10.1006/jmbi.1999.3023. PMID:10518942.
Paul A. Wade (ديسمبر 2001). "Methyl CpG-binding proteins and transcriptional repression" (PDF). BioEssays. ج. 23 ع. 12: 1131–1137. DOI:10.1002/bies.10008. PMID:11746232. S2CID:37525856. مؤرشف من الأصل (PDF) في 2007-08-14.
Kokura K، Kaul SC، Wadhwa R، Nomura T، Khan MM، Shinagawa T، Yasukawa T، Colmenares C، Ishii S (سبتمبر 2001). "The Ski protein family is required for MeCP2-mediated transcriptional repression". J. Biol. Chem. ج. 276 ع. 36: 34115–21. DOI:10.1074/jbc.M105747200. PMID:11441023.
Klein ME، Lioy DT، Ma L، Impey S، Mandel G، Goodman RH (ديسمبر 2007). "Homeostatic regulation of MeCP2 expression by a CREB-induced microRNA". Nat. Neurosci. ج. 10 ع. 12: 1513–4. DOI:10.1038/nn2010. PMID:17994015. S2CID:29308441.
Murgatroyd C، Patchev AV، Wu Y، Micale V، Bockmühl Y، Fischer D، Holsboer F، Wotjak CT، Almeida OF، Spengler D (ديسمبر 2009). "Dynamic DNA methylation programs persistent adverse effects of early-life stress". Nat. Neurosci. ج. 12 ع. 12: 1559–66. DOI:10.1038/nn.2436. PMID:19898468. S2CID:3328884.

harvard.edu

ui.adsabs.harvard.edu

Chahrour M، وآخرون (2008). "MECP2, a key contributor to neurological disease, activates and represses transcription". Science. ج. 320 ع. 5880: 1224–9. Bibcode:2008Sci...320.1224C. DOI:10.1126/science.1153252. PMC:2443785. PMID:18511691.
Luikenhuis S، Giacometti E، Beard CF، Jaenisch R (أبريل 2004). "Expression of MeCP2 in postmitotic neurons rescues Rett syndrome in mice". Proc. Natl. Acad. Sci. U.S.A. ج. 101 ع. 16: 6033–8. Bibcode:2004PNAS..101.6033L. DOI:10.1073/pnas.0401626101. PMC:395918. PMID:15069197.
Yasui DH، Peddada S، Bieda MC، Vallero RO، Hogart A، Nagarajan RP، Thatcher KN، Farnham PJ، Lasalle JM (ديسمبر 2007). "Integrated epigenomic analyses of neuronal MeCP2 reveal a role for long-range interaction with active genes". Proc. Natl. Acad. Sci. U.S.A. ج. 104 ع. 49: 19416–21. Bibcode:2007PNAS..10419416Y. DOI:10.1073/pnas.0707442104. PMC:2148304. PMID:18042715.
Chahrour M، Jung SY، Shaw C، Zhou X، Wong ST، Qin J، Zoghbi HY (مايو 2008). "MeCP2, a key contributor to neurological disease, activates and represses transcription". Science. ج. 320 ع. 5880: 1224–9. Bibcode:2008Sci...320.1224C. DOI:10.1126/science.1153252. PMC:2443785. PMID:18511691.

nih.gov

pubmed.ncbi.nlm.nih.gov

Amir RE، Van den Veyver IB، Wan M، Tran CQ، Francke U، Zoghbi HY (أكتوبر 1999). "Rett syndrome is caused by mutations in X-linked MECP2, encoding methyl-CpG-binding protein 2". Nat. Genet. ج. 23 ع. 2: 185–8. DOI:10.1038/13810. PMID:10508514. S2CID:3350350.
Lewis JD، Meehan RR، Henzel WJ، Maurer-Fogy I، Jeppesen P، Klein F، Bird A (يونيو 1992). "Purification, sequence, and cellular localization of a novel chromosomal protein that binds to methylated DNA". Cell. ج. 69 ع. 6: 905–14. DOI:10.1016/0092-8674(92)90610-O. PMID:1606614. S2CID:6825994.
Chahrour M، وآخرون (2008). "MECP2, a key contributor to neurological disease, activates and represses transcription". Science. ج. 320 ع. 5880: 1224–9. Bibcode:2008Sci...320.1224C. DOI:10.1126/science.1153252. PMC:2443785. PMID:18511691.
Šimčíková D، Heneberg P (ديسمبر 2019). "Refinement of evolutionary medicine predictions based on clinical evidence for the manifestations of Mendelian diseases". Scientific Reports. ج. 9 ع. 1: 18577. DOI:10.1038/s41598-019-54976-4. PMC:6901466. PMID:31819097.
Cohen S، Zhou Z، Greenberg ME (مايو 2008). "Medicine. Activating a repressor". Science. ج. 320 ع. 5880: 1172–3. DOI:10.1126/science.1159146. PMC:2857976. PMID:18511680.
Luikenhuis S، Giacometti E، Beard CF، Jaenisch R (أبريل 2004). "Expression of MeCP2 in postmitotic neurons rescues Rett syndrome in mice". Proc. Natl. Acad. Sci. U.S.A. ج. 101 ع. 16: 6033–8. Bibcode:2004PNAS..101.6033L. DOI:10.1073/pnas.0401626101. PMC:395918. PMID:15069197.
Yasui DH، Peddada S، Bieda MC، Vallero RO، Hogart A، Nagarajan RP، Thatcher KN، Farnham PJ، Lasalle JM (ديسمبر 2007). "Integrated epigenomic analyses of neuronal MeCP2 reveal a role for long-range interaction with active genes". Proc. Natl. Acad. Sci. U.S.A. ج. 104 ع. 49: 19416–21. Bibcode:2007PNAS..10419416Y. DOI:10.1073/pnas.0707442104. PMC:2148304. PMID:18042715.
Chahrour M، Jung SY، Shaw C، Zhou X، Wong ST، Qin J، Zoghbi HY (مايو 2008). "MeCP2, a key contributor to neurological disease, activates and represses transcription". Science. ج. 320 ع. 5880: 1224–9. Bibcode:2008Sci...320.1224C. DOI:10.1126/science.1153252. PMC:2443785. PMID:18511691.
Georgel PT، Horowitz-Scherer RA، Adkins N، Woodcock CL، Wade PA، Hansen JC (أغسطس 2003). "Chromatin compaction by human MeCP2. Assembly of novel secondary chromatin structures in the absence of DNA methylation". J. Biol. Chem. ج. 278 ع. 34: 32181–8. DOI:10.1074/jbc.M305308200. PMID:12788925.
LaSalle JM (2007). "The odyssey of MeCP2 and parental imprinting". Epigenetics. ج. 2 ع. 1: 5–10. DOI:10.4161/epi.2.1.3697. PMC:1866173. PMID:17486180.
Alessio N، Riccitiello F، Squillaro T، Capasso S، Del Gaudio S، Di Bernardo G، Cipollaro M، Melone MAB، Peluso G، Galderisi U (مارس 2018). "Neural stem cells from a mouse model of Rett syndrome are prone to senescence, show reduced capacity to cope with genotoxic stress, and are impaired in the differentiation process". Exp. Mol. Med. ج. 50 ع. 3: 1. DOI:10.1038/s12276-017-0005-x. PMC:6118406. PMID:29563495.
Wakefield RI، Smith BO، Nan X، Free A، Soteriou A، Uhrin D، Bird AP، Barlow PN (سبتمبر 1999). "The solution structure of the domain from MeCP2 that binds to methylated DNA". J. Mol. Biol. ج. 291 ع. 5: 1055–65. DOI:10.1006/jmbi.1999.3023. PMID:10518942.
Paul A. Wade (ديسمبر 2001). "Methyl CpG-binding proteins and transcriptional repression" (PDF). BioEssays. ج. 23 ع. 12: 1131–1137. DOI:10.1002/bies.10008. PMID:11746232. S2CID:37525856. مؤرشف من الأصل (PDF) في 2007-08-14.
Kokura K، Kaul SC، Wadhwa R، Nomura T، Khan MM، Shinagawa T، Yasukawa T، Colmenares C، Ishii S (سبتمبر 2001). "The Ski protein family is required for MeCP2-mediated transcriptional repression". J. Biol. Chem. ج. 276 ع. 36: 34115–21. DOI:10.1074/jbc.M105747200. PMID:11441023.
Klein ME، Lioy DT، Ma L، Impey S، Mandel G، Goodman RH (ديسمبر 2007). "Homeostatic regulation of MeCP2 expression by a CREB-induced microRNA". Nat. Neurosci. ج. 10 ع. 12: 1513–4. DOI:10.1038/nn2010. PMID:17994015. S2CID:29308441.
Murgatroyd C، Patchev AV، Wu Y، Micale V، Bockmühl Y، Fischer D، Holsboer F، Wotjak CT، Almeida OF، Spengler D (ديسمبر 2009). "Dynamic DNA methylation programs persistent adverse effects of early-life stress". Nat. Neurosci. ج. 12 ع. 12: 1559–66. DOI:10.1038/nn.2436. PMID:19898468. S2CID:3328884.

ncbi.nlm.nih.gov

Chahrour M، وآخرون (2008). "MECP2, a key contributor to neurological disease, activates and represses transcription". Science. ج. 320 ع. 5880: 1224–9. Bibcode:2008Sci...320.1224C. DOI:10.1126/science.1153252. PMC:2443785. PMID:18511691.
"Entrez Gene: MECP2 methyl CpG binding protein 2 (Rett syndrome)". مؤرشف من الأصل في 2010-12-05.
Šimčíková D، Heneberg P (ديسمبر 2019). "Refinement of evolutionary medicine predictions based on clinical evidence for the manifestations of Mendelian diseases". Scientific Reports. ج. 9 ع. 1: 18577. DOI:10.1038/s41598-019-54976-4. PMC:6901466. PMID:31819097.
Cohen S، Zhou Z، Greenberg ME (مايو 2008). "Medicine. Activating a repressor". Science. ج. 320 ع. 5880: 1172–3. DOI:10.1126/science.1159146. PMC:2857976. PMID:18511680.
Luikenhuis S، Giacometti E، Beard CF، Jaenisch R (أبريل 2004). "Expression of MeCP2 in postmitotic neurons rescues Rett syndrome in mice". Proc. Natl. Acad. Sci. U.S.A. ج. 101 ع. 16: 6033–8. Bibcode:2004PNAS..101.6033L. DOI:10.1073/pnas.0401626101. PMC:395918. PMID:15069197.
Yasui DH، Peddada S، Bieda MC، Vallero RO، Hogart A، Nagarajan RP، Thatcher KN، Farnham PJ، Lasalle JM (ديسمبر 2007). "Integrated epigenomic analyses of neuronal MeCP2 reveal a role for long-range interaction with active genes". Proc. Natl. Acad. Sci. U.S.A. ج. 104 ع. 49: 19416–21. Bibcode:2007PNAS..10419416Y. DOI:10.1073/pnas.0707442104. PMC:2148304. PMID:18042715.
Chahrour M، Jung SY، Shaw C، Zhou X، Wong ST، Qin J، Zoghbi HY (مايو 2008). "MeCP2, a key contributor to neurological disease, activates and represses transcription". Science. ج. 320 ع. 5880: 1224–9. Bibcode:2008Sci...320.1224C. DOI:10.1126/science.1153252. PMC:2443785. PMID:18511691.
LaSalle JM (2007). "The odyssey of MeCP2 and parental imprinting". Epigenetics. ج. 2 ع. 1: 5–10. DOI:10.4161/epi.2.1.3697. PMC:1866173. PMID:17486180.
Alessio N، Riccitiello F، Squillaro T، Capasso S، Del Gaudio S، Di Bernardo G، Cipollaro M، Melone MAB، Peluso G، Galderisi U (مارس 2018). "Neural stem cells from a mouse model of Rett syndrome are prone to senescence, show reduced capacity to cope with genotoxic stress, and are impaired in the differentiation process". Exp. Mol. Med. ج. 50 ع. 3: 1. DOI:10.1038/s12276-017-0005-x. PMC:6118406. PMID:29563495.

semanticscholar.org

api.semanticscholar.org

Amir RE، Van den Veyver IB، Wan M، Tran CQ، Francke U، Zoghbi HY (أكتوبر 1999). "Rett syndrome is caused by mutations in X-linked MECP2, encoding methyl-CpG-binding protein 2". Nat. Genet. ج. 23 ع. 2: 185–8. DOI:10.1038/13810. PMID:10508514. S2CID:3350350.
Lewis JD، Meehan RR، Henzel WJ، Maurer-Fogy I، Jeppesen P، Klein F، Bird A (يونيو 1992). "Purification, sequence, and cellular localization of a novel chromosomal protein that binds to methylated DNA". Cell. ج. 69 ع. 6: 905–14. DOI:10.1016/0092-8674(92)90610-O. PMID:1606614. S2CID:6825994.
Paul A. Wade (ديسمبر 2001). "Methyl CpG-binding proteins and transcriptional repression" (PDF). BioEssays. ج. 23 ع. 12: 1131–1137. DOI:10.1002/bies.10008. PMID:11746232. S2CID:37525856. مؤرشف من الأصل (PDF) في 2007-08-14.
Klein ME، Lioy DT، Ma L، Impey S، Mandel G، Goodman RH (ديسمبر 2007). "Homeostatic regulation of MeCP2 expression by a CREB-induced microRNA". Nat. Neurosci. ج. 10 ع. 12: 1513–4. DOI:10.1038/nn2010. PMID:17994015. S2CID:29308441.
Murgatroyd C، Patchev AV، Wu Y، Micale V، Bockmühl Y، Fischer D، Holsboer F، Wotjak CT، Almeida OF، Spengler D (ديسمبر 2009). "Dynamic DNA methylation programs persistent adverse effects of early-life stress". Nat. Neurosci. ج. 12 ع. 12: 1559–66. DOI:10.1038/nn.2436. PMID:19898468. S2CID:3328884.

web.archive.org

"Entrez Gene: MECP2 methyl CpG binding protein 2 (Rett syndrome)". مؤرشف من الأصل في 2010-12-05.
Paul A. Wade (ديسمبر 2001). "Methyl CpG-binding proteins and transcriptional repression" (PDF). BioEssays. ج. 23 ع. 12: 1131–1137. DOI:10.1002/bies.10008. PMID:11746232. S2CID:37525856. مؤرشف من الأصل (PDF) في 2007-08-14.