إدمان الجنس (Arabic Wikipedia)

Analysis of information sources in references of the Wikipedia article "إدمان الجنس" in Arabic language version.

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alquds.com

alriyadh.com

apa.org

doi.apa.org

bbc.co.uk

news.bbc.co.uk

bmj.com

jnnp.bmj.com

  • Trimble، M. (1 أغسطس 2002). "Molecular neuropharmacology, a foundation for clinical neuroscience: Edited by Eric J Nestler, Steven E Hyman, and Robert C Malenka (Pp 503, pound36.99). Published by McGraw-Hill, New York, 2001. ISBN 0-8385-6379-1". Journal of Neurology, Neurosurgery & Psychiatry. ج. 73 ع. 2: 210–a-210. DOI:10.1136/jnnp.73.2.210-a. ISSN:0022-3050. مؤرشف من الأصل في 2019-12-09.

doi.org

  • Trimble، M. (1 أغسطس 2002). "Molecular neuropharmacology, a foundation for clinical neuroscience: Edited by Eric J Nestler, Steven E Hyman, and Robert C Malenka (Pp 503, pound36.99). Published by McGraw-Hill, New York, 2001. ISBN 0-8385-6379-1". Journal of Neurology, Neurosurgery & Psychiatry. ج. 73 ع. 2: 210–a-210. DOI:10.1136/jnnp.73.2.210-a. ISSN:0022-3050. مؤرشف من الأصل في 2019-12-09.
  • Karila، Laurent؛ Wery، Aline؛ Weinstein، Aviv؛ Cottencin، Olivier؛ Petit، Aymeric؛ Reynaud، Michel؛ Billieux، Joel (2014-06). "Sexual Addiction or Hypersexual Disorder: Different Terms for the Same Problem? A Review of the Literature". Current Pharmaceutical Design. ج. 20 ع. 25: 4012–4020. DOI:10.2174/13816128113199990619. ISSN:1381-6128. مؤرشف من الأصل في 2019-12-09. {{استشهاد بدورية محكمة}}: تحقق من التاريخ في: |تاريخ= (مساعدة)
  • Coleman، Eli (18 سبتمبر 2012). "Impulsive/Compulsive Sexual Behavior: Assessment and Treatment". Oxford Handbooks Online. DOI:10.1093/oxfordhb/9780195389715.013.0108. مؤرشف من الأصل في 2020-04-07.
  • Hall، Paula (2 ديسمبر 2013). "Sex addiction – an extraordinarily contentious problem". Sexual and Relationship Therapy. ج. 29 ع. 1: 68–75. DOI:10.1080/14681994.2013.861898. ISSN:1468-1994. مؤرشف من الأصل في 2019-12-09.
  • Slavova، Angela؛ Zafirova، Zoya (2018). "Dynamic behavior of integro-differential CNN model". Author(s). DOI:10.1063/1.5082117. مؤرشف من الأصل في 2019-12-09. {{استشهاد بدورية محكمة}}: الاستشهاد بدورية محكمة يطلب |دورية محكمة= (مساعدة)
  • van Drimmelen-Krabbe، J.J. (1996-01). "WHO ICD-10 Evaluation and evolution: ICD-10 Training courses". European Psychiatry. ج. 11: 200s. DOI:10.1016/0924-9338(96)88574-9. ISSN:0924-9338. مؤرشف من الأصل في 2019-12-09. {{استشهاد بدورية محكمة}}: تحقق من التاريخ في: |تاريخ= (مساعدة)
  • Volkow ND، Koob GF، McLellan AT (يناير 2016). "Neurobiologic Advances from the Brain Disease Model of Addiction". N. Engl. J. Med. ج. 374 ع. 4: 363–371. DOI:10.1056/NEJMra1511480. PMID:26816013. Substance-use disorder: A diagnostic term in the fifth edition of the Diagnostic and Statistical Manual of Mental Disorders (DSM-5) referring to recurrent use of alcohol or other drugs that causes clinically and functionally significant impairment, such as health problems, disability, and failure to meet major responsibilities at work, school, or home. Depending on the level of severity, this disorder is classified as mild, moderate, or severe.
    Addiction: A term used to indicate the most severe, chronic stage of substance-use disorder, in which there is a substantial loss of self-control, as indicated by compulsive drug taking despite the desire to stop taking the drug. In the DSM-5, the term addiction is synonymous with the classification of severe substance-use disorder.

elsevier.com

linkinghub.elsevier.com

eurekaselect.com

maganin.com

mssm.edu

neuroscience.mssm.edu

  • "Glossary of Terms". Mount Sinai School of Medicine. Department of Neuroscience. اطلع عليه بتاريخ 2015-02-09.

nih.gov

pubmed.ncbi.nlm.nih.gov

  • Nestler EJ (ديسمبر 2013). "Cellular basis of memory for addiction". Dialogues Clin. Neurosci. ج. 15 ع. 4: 431–443. PMC:3898681. PMID:24459410. Despite the importance of numerous psychosocial factors, at its core, drug addiction involves a biological process: the ability of repeated exposure to a drug of abuse to induce changes in a vulnerable brain that drive the compulsive seeking and taking of drugs, and loss of control over drug use, that define a state of addiction. ... A large body of literature has demonstrated that such ΔFosB induction in D1-type [nucleus accumbens] neurons increases an animal's sensitivity to drug as well as natural rewards and promotes drug self-administration, presumably through a process of positive reinforcement ... Another ΔFosB target is cFos: as ΔFosB accumulates with repeated drug exposure it represses c-Fos and contributes to the molecular switch whereby ΔFosB is selectively induced in the chronic drug-treated state.41. ... Moreover, there is increasing evidence that, despite a range of genetic risks for addiction across the population, exposure to sufficiently high doses of a drug for long periods of time can transform someone who has relatively lower genetic loading into an addict.
  • Volkow ND، Koob GF، McLellan AT (يناير 2016). "Neurobiologic Advances from the Brain Disease Model of Addiction". N. Engl. J. Med. ج. 374 ع. 4: 363–371. DOI:10.1056/NEJMra1511480. PMID:26816013. Substance-use disorder: A diagnostic term in the fifth edition of the Diagnostic and Statistical Manual of Mental Disorders (DSM-5) referring to recurrent use of alcohol or other drugs that causes clinically and functionally significant impairment, such as health problems, disability, and failure to meet major responsibilities at work, school, or home. Depending on the level of severity, this disorder is classified as mild, moderate, or severe.
    Addiction: A term used to indicate the most severe, chronic stage of substance-use disorder, in which there is a substantial loss of self-control, as indicated by compulsive drug taking despite the desire to stop taking the drug. In the DSM-5, the term addiction is synonymous with the classification of severe substance-use disorder.

ncbi.nlm.nih.gov

  • Nestler EJ (ديسمبر 2013). "Cellular basis of memory for addiction". Dialogues Clin. Neurosci. ج. 15 ع. 4: 431–443. PMC:3898681. PMID:24459410. Despite the importance of numerous psychosocial factors, at its core, drug addiction involves a biological process: the ability of repeated exposure to a drug of abuse to induce changes in a vulnerable brain that drive the compulsive seeking and taking of drugs, and loss of control over drug use, that define a state of addiction. ... A large body of literature has demonstrated that such ΔFosB induction in D1-type [nucleus accumbens] neurons increases an animal's sensitivity to drug as well as natural rewards and promotes drug self-administration, presumably through a process of positive reinforcement ... Another ΔFosB target is cFos: as ΔFosB accumulates with repeated drug exposure it represses c-Fos and contributes to the molecular switch whereby ΔFosB is selectively induced in the chronic drug-treated state.41. ... Moreover, there is increasing evidence that, despite a range of genetic risks for addiction across the population, exposure to sufficiently high doses of a drug for long periods of time can transform someone who has relatively lower genetic loading into an addict.

oxfordhandbooks.com

sagepub.com

sk.sagepub.com

scitation.org

aip.scitation.org

tandfonline.com

taylorfrancis.com

web.archive.org

worldcat.org