اعتلال تاو (Arabic Wikipedia)

Analysis of information sources in references of the Wikipedia article "اعتلال تاو" in Arabic language version.

refsWebsite

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29nih.gov

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22doi.org

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5archive.org

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2web.archive.org

1^st place

1medicalxpress.com

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1gla.ac.uk

2,948^th place

2,339^th place

archive.org

"Neuropathological stageing of Alzheimer-related changes". Acta Neuropathologica. ج. 82 ع. 4: 239–59. 1991. DOI:10.1007/BF00308809. PMID:1759558.
"Amyotrophic lateral sclerosis/parkinsonism-dementia complex of Guam: quantitative neuropathology, immunohistochemical analysis of neuronal vulnerability, and comparison with related neurodegenerative disorders". Acta Neuropathologica. ج. 88 ع. 5: 397–404. 1994. DOI:10.1007/BF00389490. PMID:7847067.
"Meningio-angiomatosis: a report of six cases with special reference to the occurrence of neurofibrillary tangles". Journal of Neuropathology and Experimental Neurology. ج. 45 ع. 4: 426–46. يوليو 1986. DOI:10.1097/00005072-198607000-00005. PMID:3088216.
"Alzheimer neurofibrillary tangles in diseases other than senile and presenile dementia". Annals of Neurology. ج. 5 ع. 3: 288–94. مارس 1979. DOI:10.1002/ana.410050311. PMID:156000.
"Argyrophilic grains: a distinct disease or an additive pathology?". Neurobiology of Aging. ج. 29 ع. 4: 566–73. أبريل 2008. DOI:10.1016/j.neurobiolaging.2006.10.032. PMC:2727715. PMID:17188783.

doi.org

"Propagation of Tau aggregates". Molecular Brain. ج. 10 ع. 1: 18. مايو 2017. DOI:10.1186/s13041-017-0298-7. PMC:5450399. PMID:28558799.{{استشهاد بدورية محكمة}}: صيانة الاستشهاد: دوي مجاني غير معلم (link)
"Abnormal hyperphosphorylation of tau: sites, regulation, and molecular mechanism of neurofibrillary degeneration". Journal of Alzheimer's Disease. 33 Suppl 1: S123-39. 2013. DOI:10.3233/JAD-2012-129031. PMID:22710920.
"Tau in physiology and pathology". Nature Reviews. Neuroscience. ج. 17 ع. 1: 5–21. يناير 2016. DOI:10.1038/nrn.2015.1. PMID:26631930.
"Neuropathological stageing of Alzheimer-related changes". Acta Neuropathologica. ج. 82 ع. 4: 239–59. 1991. DOI:10.1007/BF00308809. PMID:1759558.
"The MAPT H1 haplotype is associated with tangle-predominant dementia". Acta Neuropathologica. ج. 124 ع. 5: 693–704. نوفمبر 2012. DOI:10.1007/s00401-012-1017-1. PMC:3608475. PMID:22802095.
"Neurofibrillary tangle-predominant dementia: comparison with classical Alzheimer disease". Acta Neuropathologica. ج. 113 ع. 2: 107–17. فبراير 2007. DOI:10.1007/s00401-006-0156-7. PMID:17089134.
"The first NINDS/NIBIB consensus meeting to define neuropathological criteria for the diagnosis of chronic traumatic encephalopathy" (PDF). Acta Neuropathologica. ج. 131 ع. 1: 75–86. يناير 2016. DOI:10.1007/s00401-015-1515-z. PMC:4698281. PMID:26667418. مؤرشف من الأصل (PDF) في 2017-09-23.
"Immunocytochemistry of neurofibrillary tangles in dementia pugilistica and Alzheimer's disease: evidence for common genesis". Lancet. ج. 2 ع. 8626–8627: 1456–8. 1988. DOI:10.1016/S0140-6736(88)90934-8. PMID:2904573.
"Progressive supranuclear palsy: clinicopathological concepts and diagnostic challenges". The Lancet. Neurology. ج. 8 ع. 3: 270–9. مارس 2009. DOI:10.1016/S1474-4422(09)70042-0. PMID:19233037.
"Deciphering the genetic basis of Alzheimer's disease". Annual Review of Genomics and Human Genetics. ج. 3: 67–99. 2002. DOI:10.1146/annurev.genom.3.022502.103022. PMID:12142353.
"Amyotrophic lateral sclerosis/parkinsonism-dementia complex of Guam: quantitative neuropathology, immunohistochemical analysis of neuronal vulnerability, and comparison with related neurodegenerative disorders". Acta Neuropathologica. ج. 88 ع. 5: 397–404. 1994. DOI:10.1007/BF00389490. PMID:7847067.
"Tau-associated neuropathology in ganglion cell tumours increases with patient age but appears unrelated to ApoE genotype". Neuropathology and Applied Neurobiology. ج. 27 ع. 3: 197–205. يونيو 2001. DOI:10.1046/j.1365-2990.2001.00311.x. PMID:11489139.
"Meningio-angiomatosis: a report of six cases with special reference to the occurrence of neurofibrillary tangles". Journal of Neuropathology and Experimental Neurology. ج. 45 ع. 4: 426–46. يوليو 1986. DOI:10.1097/00005072-198607000-00005. PMID:3088216.
"Neurofibrillary changes in the cerebral cortex of a patient with subacute sclerosing panencephalitis (SSPE)". Acta Neuropathologica. ج. 48 ع. 2: 157–60. نوفمبر 1979. DOI:10.1007/BF00691159. PMID:506699.
"Alzheimer neurofibrillary tangles in diseases other than senile and presenile dementia". Annals of Neurology. ج. 5 ع. 3: 288–94. مارس 1979. DOI:10.1002/ana.410050311. PMID:156000.
"Argyrophilic grain disease". Brain. ج. 131 ع. Pt 6: 1416–32. يونيو 2008. DOI:10.1093/brain/awm305. PMID:18234698.
"Argyrophilic grains: a distinct disease or an additive pathology?". Neurobiology of Aging. ج. 29 ع. 4: 566–73. أبريل 2008. DOI:10.1016/j.neurobiolaging.2006.10.032. PMC:2727715. PMID:17188783.
"[Argyrophilic grain disease: synergistic component of dementia?]" [Argyrophilic grain disease: synergistic component of dementia?]. Revue Neurologique (بالفرنسية). 166 (4): 428–32. Apr 2010. DOI:10.1016/j.neurol.2009.10.012. PMID:19963233.
"[Argyrophilic grain disease: differentiation from Alzheimer disease]". Der Pathologe. ج. 20 ع. 3: 159–68. مايو 1999. DOI:10.1007/s002920050339. PMID:10412175.
"Dementia with grains (argyrophilic grain disease)". Brain Pathology. ج. 8 ع. 2: 377–86. أبريل 1998. DOI:10.1111/j.1750-3639.1998.tb00161.x. PMID:9546294.
"Alpha-synuclein and tau: teammates in neurodegeneration?". Molecular Neurodegeneration. ج. 9: 43. أكتوبر 2014. DOI:10.1186/1750-1326-9-43. PMC:4230508. PMID:25352339. {{استشهاد بدورية محكمة}}: الوسيط غير المعروف |last-author-amp= تم تجاهله يقترح استخدام |name-list-style= (مساعدة)صيانة الاستشهاد: دوي مجاني غير معلم (link)
"Extrapyramidal syndromes in frontotemporal degeneration". Journal of Molecular Neuroscience. ج. 45 ع. 3: 336–42. نوفمبر 2011. DOI:10.1007/s12031-011-9616-1. PMID:21887521.

gla.ac.uk

eprints.gla.ac.uk

"The first NINDS/NIBIB consensus meeting to define neuropathological criteria for the diagnosis of chronic traumatic encephalopathy" (PDF). Acta Neuropathologica. ج. 131 ع. 1: 75–86. يناير 2016. DOI:10.1007/s00401-015-1515-z. PMC:4698281. PMID:26667418. مؤرشف من الأصل (PDF) في 2017-09-23.

medicalxpress.com

Alzheimer 'tau' protein far surpasses amyloid in predicting toll on brain tissue نسخة محفوظة 19 فبراير 2020 على موقع واي باك مشين.

nih.gov

pubmed.ncbi.nlm.nih.gov

"Propagation of Tau aggregates". Molecular Brain. ج. 10 ع. 1: 18. مايو 2017. DOI:10.1186/s13041-017-0298-7. PMC:5450399. PMID:28558799.{{استشهاد بدورية محكمة}}: صيانة الاستشهاد: دوي مجاني غير معلم (link)
"Neuropathology of non-Alzheimer degenerative disorders". International Journal of Clinical and Experimental Pathology. ج. 3 ع. 1: 1–23. أغسطس 2009. PMC:2776269. PMID:19918325.
"Abnormal hyperphosphorylation of tau: sites, regulation, and molecular mechanism of neurofibrillary degeneration". Journal of Alzheimer's Disease. 33 Suppl 1: S123-39. 2013. DOI:10.3233/JAD-2012-129031. PMID:22710920.
"Tau in physiology and pathology". Nature Reviews. Neuroscience. ج. 17 ع. 1: 5–21. يناير 2016. DOI:10.1038/nrn.2015.1. PMID:26631930.
"Neuropathological stageing of Alzheimer-related changes". Acta Neuropathologica. ج. 82 ع. 4: 239–59. 1991. DOI:10.1007/BF00308809. PMID:1759558.
"The MAPT H1 haplotype is associated with tangle-predominant dementia". Acta Neuropathologica. ج. 124 ع. 5: 693–704. نوفمبر 2012. DOI:10.1007/s00401-012-1017-1. PMC:3608475. PMID:22802095.
"Neurofibrillary tangle-predominant dementia: comparison with classical Alzheimer disease". Acta Neuropathologica. ج. 113 ع. 2: 107–17. فبراير 2007. DOI:10.1007/s00401-006-0156-7. PMID:17089134.
"The first NINDS/NIBIB consensus meeting to define neuropathological criteria for the diagnosis of chronic traumatic encephalopathy" (PDF). Acta Neuropathologica. ج. 131 ع. 1: 75–86. يناير 2016. DOI:10.1007/s00401-015-1515-z. PMC:4698281. PMID:26667418. مؤرشف من الأصل (PDF) في 2017-09-23.
"Immunocytochemistry of neurofibrillary tangles in dementia pugilistica and Alzheimer's disease: evidence for common genesis". Lancet. ج. 2 ع. 8626–8627: 1456–8. 1988. DOI:10.1016/S0140-6736(88)90934-8. PMID:2904573.
"Progressive supranuclear palsy: clinicopathological concepts and diagnostic challenges". The Lancet. Neurology. ج. 8 ع. 3: 270–9. مارس 2009. DOI:10.1016/S1474-4422(09)70042-0. PMID:19233037.
"Deciphering the genetic basis of Alzheimer's disease". Annual Review of Genomics and Human Genetics. ج. 3: 67–99. 2002. DOI:10.1146/annurev.genom.3.022502.103022. PMID:12142353.
"Amyotrophic lateral sclerosis/parkinsonism-dementia complex of Guam: quantitative neuropathology, immunohistochemical analysis of neuronal vulnerability, and comparison with related neurodegenerative disorders". Acta Neuropathologica. ج. 88 ع. 5: 397–404. 1994. DOI:10.1007/BF00389490. PMID:7847067.
"Tau-associated neuropathology in ganglion cell tumours increases with patient age but appears unrelated to ApoE genotype". Neuropathology and Applied Neurobiology. ج. 27 ع. 3: 197–205. يونيو 2001. DOI:10.1046/j.1365-2990.2001.00311.x. PMID:11489139.
"Meningio-angiomatosis: a report of six cases with special reference to the occurrence of neurofibrillary tangles". Journal of Neuropathology and Experimental Neurology. ج. 45 ع. 4: 426–46. يوليو 1986. DOI:10.1097/00005072-198607000-00005. PMID:3088216.
"Neurofibrillary changes in the cerebral cortex of a patient with subacute sclerosing panencephalitis (SSPE)". Acta Neuropathologica. ج. 48 ع. 2: 157–60. نوفمبر 1979. DOI:10.1007/BF00691159. PMID:506699.
"Alzheimer neurofibrillary tangles in diseases other than senile and presenile dementia". Annals of Neurology. ج. 5 ع. 3: 288–94. مارس 1979. DOI:10.1002/ana.410050311. PMID:156000.
"Argyrophilic grain disease". Brain. ج. 131 ع. Pt 6: 1416–32. يونيو 2008. DOI:10.1093/brain/awm305. PMID:18234698.
"Argyrophilic grains: a distinct disease or an additive pathology?". Neurobiology of Aging. ج. 29 ع. 4: 566–73. أبريل 2008. DOI:10.1016/j.neurobiolaging.2006.10.032. PMC:2727715. PMID:17188783.
"[Argyrophilic grain disease: synergistic component of dementia?]" [Argyrophilic grain disease: synergistic component of dementia?]. Revue Neurologique (بالفرنسية). 166 (4): 428–32. Apr 2010. DOI:10.1016/j.neurol.2009.10.012. PMID:19963233.
"[Argyrophilic grain disease: differentiation from Alzheimer disease]". Der Pathologe. ج. 20 ع. 3: 159–68. مايو 1999. DOI:10.1007/s002920050339. PMID:10412175.
"Dementia with grains (argyrophilic grain disease)". Brain Pathology. ج. 8 ع. 2: 377–86. أبريل 1998. DOI:10.1111/j.1750-3639.1998.tb00161.x. PMID:9546294.
"Alpha-synuclein and tau: teammates in neurodegeneration?". Molecular Neurodegeneration. ج. 9: 43. أكتوبر 2014. DOI:10.1186/1750-1326-9-43. PMC:4230508. PMID:25352339. {{استشهاد بدورية محكمة}}: الوسيط غير المعروف |last-author-amp= تم تجاهله يقترح استخدام |name-list-style= (مساعدة)صيانة الاستشهاد: دوي مجاني غير معلم (link)
"Extrapyramidal syndromes in frontotemporal degeneration". Journal of Molecular Neuroscience. ج. 45 ع. 3: 336–42. نوفمبر 2011. DOI:10.1007/s12031-011-9616-1. PMID:21887521.

ncbi.nlm.nih.gov

"Propagation of Tau aggregates". Molecular Brain. ج. 10 ع. 1: 18. مايو 2017. DOI:10.1186/s13041-017-0298-7. PMC:5450399. PMID:28558799.{{استشهاد بدورية محكمة}}: صيانة الاستشهاد: دوي مجاني غير معلم (link)
"Neuropathology of non-Alzheimer degenerative disorders". International Journal of Clinical and Experimental Pathology. ج. 3 ع. 1: 1–23. أغسطس 2009. PMC:2776269. PMID:19918325.
"The MAPT H1 haplotype is associated with tangle-predominant dementia". Acta Neuropathologica. ج. 124 ع. 5: 693–704. نوفمبر 2012. DOI:10.1007/s00401-012-1017-1. PMC:3608475. PMID:22802095.
"The first NINDS/NIBIB consensus meeting to define neuropathological criteria for the diagnosis of chronic traumatic encephalopathy" (PDF). Acta Neuropathologica. ج. 131 ع. 1: 75–86. يناير 2016. DOI:10.1007/s00401-015-1515-z. PMC:4698281. PMID:26667418. مؤرشف من الأصل (PDF) في 2017-09-23.
"Argyrophilic grains: a distinct disease or an additive pathology?". Neurobiology of Aging. ج. 29 ع. 4: 566–73. أبريل 2008. DOI:10.1016/j.neurobiolaging.2006.10.032. PMC:2727715. PMID:17188783.
"Alpha-synuclein and tau: teammates in neurodegeneration?". Molecular Neurodegeneration. ج. 9: 43. أكتوبر 2014. DOI:10.1186/1750-1326-9-43. PMC:4230508. PMID:25352339. {{استشهاد بدورية محكمة}}: الوسيط غير المعروف |last-author-amp= تم تجاهله يقترح استخدام |name-list-style= (مساعدة)صيانة الاستشهاد: دوي مجاني غير معلم (link)

web.archive.org

Alzheimer 'tau' protein far surpasses amyloid in predicting toll on brain tissue نسخة محفوظة 19 فبراير 2020 على موقع واي باك مشين.
"The first NINDS/NIBIB consensus meeting to define neuropathological criteria for the diagnosis of chronic traumatic encephalopathy" (PDF). Acta Neuropathologica. ج. 131 ع. 1: 75–86. يناير 2016. DOI:10.1007/s00401-015-1515-z. PMC:4698281. PMID:26667418. مؤرشف من الأصل (PDF) في 2017-09-23.