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النحاس في الصحة (Arabic Wikipedia)

Analysis of information sources in references of the Wikipedia article "النحاس في الصحة" in Arabic language version.

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copperinfo.com

doi.org

  • Scheiber، Ivo؛ Dringen، Ralf؛ Mercer، Julian F. B. (2013). "Copper: Effects of Deficiency and Overload". في Sigel، Astrid؛ Sigel، Helmut؛ Sigel، Roland K.O. (المحررون). Interrelations between Essential Metal Ions and Human Diseases. Metal Ions in Life Sciences. Springer. ج. 13. ص. 359–87. DOI:10.1007/978-94-007-7500-8_11. ISBN:978-94-007-7500-8.
  • Vest، Katherine E.؛ Hashemi، Hayaa F.؛ Cobine، Paul A. (2013). "The Copper Metallome in Eukaryotic Cells". في Banci، Lucia (المحرر). Metallomics and the Cell. Metal Ions in Life Sciences. Springer. ج. 12. DOI:10.1007/978-94-007-5561-10_12. ISBN:978-94-007-5560-4.
  • Stern، Bonnie Ransom؛ Solioz، Marc؛ Krewski، Daniel؛ Aggett، Peter؛ Aw، Tar-Ching؛ Baker، Scott؛ Crump، Kenny؛ Dourson، Michael؛ Haber، Lynne؛ Hertzberg، Rick؛ Keen، Carl؛ Meek، Bette؛ Rudenko، Larisa؛ Schoeny، Rita؛ Slob، Wout؛ Starr، Tom (2007). "Copper and Human Health: Biochemistry, Genetics, and Strategies for Modeling Dose-response Relationships". Journal of Toxicology and Environmental Health, Part B. ج. 10 ع. 3: 157–222. DOI:10.1080/10937400600755911. PMID:17454552.
  • Stern، Bonnie Ransom (2010). "Essentiality and Toxicity in Copper Health Risk Assessment: Overview, Update and Regulatory Considerations". Journal of Toxicology and Environmental Health, Part A. ج. 73 ع. 2: 114–27. DOI:10.1080/15287390903337100. PMID:20077283.
  • Camakaris، J.؛ Voskoboinik، I.؛ Mercer، J.F. (1999). "Molecular Mechanisms of Copper Homeostasis". Biochemical and Biophysical Research Communications. ج. 261 ع. 2: 225–32. DOI:10.1006/bbrc.1999.1073. PMID:10425169.
  • Harris، Edward D. (2000). "Cellular copper transport and metabolism". Annual Review of Nutrition. ج. 20 ع. 1: 291–310. DOI:10.1146/annurev.nutr.20.1.291. PMID:10940336.
  • Harris، Edward D. (2001). "Copper Homeostasis: The Role of Cellular Transporters". Nutrition Reviews. ج. 59 ع. 9: 281–5. DOI:10.1111/j.1753-4887.2001.tb07017.x. PMID:11570430.
  • Bertinato، Jesse؛ L'Abbé، Mary R. (2004). "Maintaining copper homeostasis: regulation of copper-trafficking proteins in response to copper deficiency or overload". The Journal of Nutritional Biochemistry. ج. 15 ع. 6: 316–22. DOI:10.1016/j.jnutbio.2004.02.004. PMID:15157936.
  • Crampton، R. F.؛ Matthews، D. M.؛ Poisner، Roselle (1965). "Observations on the mechanism of absorption of copper by the small intestine". The Journal of Physiology. ج. 178 ع. 1: 111–26. DOI:10.1113/jphysiol.1965.sp007618. PMC:1357280. PMID:14298103.
  • Strickland، GT؛ Beckner، WM؛ Leu، ML (1972). "Absorption of copper in homozygotes and heterozygotes for Wilson's disease and controls: isotope tracer studies with 67 Cu and 64 Cu". Clinical Science. ج. 43 ع. 5: 617–25. DOI:10.1042/cs0430617. PMID:5083937.
  • Strickland، GT؛ Beckner، WM؛ Leu، ML؛ O'Reilly، S (1972). "Turnover studies of copper in homozygotes and heterozygotes for Wilson's disease and controls: isotope tracer studies with 67 Cu". Clinical Science. ج. 43 ع. 5: 605–15. DOI:10.1042/cs0430605. PMID:5083936.
  • Ehrenkranz، Richard A.؛ Gettner، Patricia A.؛ Nelli، Catherine M. (1989). "Nutrient Balance Studies in Premature Infants Fed Premature Formula or Fortified Preterm Human Milk". Journal of Pediatric Gastroenterology and Nutrition. ج. 8 ع. 1: 58–67. DOI:10.1097/00005176-198901000-00012. PMID:2499673.
  • Werman، Moshe J.؛ Bhathena، Sam J. (1995). "Fructose metabolizing enzymes in the rat liver and metabolic parameters: Interactions between dietary copper, type of carbohydrates, and gender". The Journal of Nutritional Biochemistry. ج. 6 ع. 7: 373–379. DOI:10.1016/0955-2863(95)80005-W. PMID:12049998.
  • Lutsenko، Svetlana؛ Kaplan، Jack H. (1995). "Organization of P-type ATPases: significance of structural diversity". Biochemistry. ج. 34 ع. 48: 15607–13. DOI:10.1021/bi00048a001. PMID:7495787.
  • Solioz، Marc؛ Vulpe، Christopher (1996). "CPx-type ATPases: a class of P-type ATPases that pump heavy metals". Trends in Biochemical Sciences. ج. 21 ع. 7: 237–41. DOI:10.1016/S0968-0004(96)20016-7. PMID:8755241.
  • Lee، J.؛ Petris، M. J.؛ Thiele، D. J. (2002). "Characterization of mouse embryonic cells deficient in the ctr1 high affinity copper transporter: Identification of a Ctr1-independent copper transport system". The Journal of Biological Chemistry. ج. 277 ع. 43: 40253–9. DOI:10.1074/jbc.M208002200. PMID:12177073.{{استشهاد بدورية محكمة}}: صيانة الاستشهاد: دوي مجاني غير معلم (link)
  • Bligh، S.W.Annie؛ Boyle، Helena A.؛ Mcewen، Andrew B.؛ Sadler، Peter J.؛ Woodham، Robert H. (1992). "1H NMR studies of reactions of copper complexes with human blood plasma and urine". Biochemical Pharmacology. ج. 43 ع. 2: 137–45. DOI:10.1016/0006-2952(92)90270-S. PMID:1739401.
  • Winge، Dennis R.؛ Mehra، Rajesh K. (1990). "Host Defenses against Copper Toxicity". International Review of Experimental Pathology. ج. 31: 47–83. DOI:10.1016/b978-0-12-364931-7.50007-0. PMID:2292474.
  • Georgopoulos، PG؛ Roy، A؛ Yonone-Lioy، MJ؛ Opiekun، RE؛ Lioy، PJ (2001). "Environmental copper: its dynamics and human exposure issues". Journal of Toxicology and Environmental Health. Part B, Critical Reviews. ج. 4 ع. 4: 341–94. DOI:10.1080/109374001753146207. PMID:11695043.
  • Sadhra، Steven S.؛ Wheatley، Andrew D.؛ Cross، Hilary J. (2007). "Dietary exposure to copper in the European Union and its assessment for EU regulatory risk assessment". Science of The Total Environment. ج. 374 ع. 2–3: 223–34. DOI:10.1016/j.scitotenv.2006.12.041. PMID:17270248.
  • Georgopoulos، Panos G؛ Wang، Sheng Wei؛ Georgopoulos، Ioannis G؛ Yonone-Lioy، Mary Jean؛ Lioy، Paul J (2006). "Assessment of human exposure to copper: A case study using the NHEXAS database". Journal of Exposure Analysis and Environmental Epidemiology. ج. 16 ع. 5: 397–409. DOI:10.1038/sj.jea.7500462. PMID:16249795.
  • Spinazzi، Marco؛ De Lazzari، Franca؛ Tavolato، Bruno؛ Angelini، Corrado؛ Manara، Renzo؛ Armani، Mario (2007). "Myelo-optico-neuropathy in copper deficiency occurring after partial gastrectomy". Journal of Neurology. ج. 254 ع. 8: 1012–7. DOI:10.1007/s00415-006-0479-2. PMID:17415508.
  • Goodman، B P؛ Bosch، E P؛ Ross، M A؛ Hoffman-Snyder، C؛ Dodick، D D؛ Smith، B E (2009). "Clinical and electrodiagnostic findings in copper deficiency myeloneuropathy". Journal of Neurology, Neurosurgery & Psychiatry. ج. 80 ع. 5: 524–7. DOI:10.1136/jnnp.2008.144683. PMID:18495738.
  • Kumar، Neeraj؛ Elliott، Michelle A.؛ Hoyer، James D.؛ Harper، Charles M.؛ Ahlskog، J. Eric؛ Phyliky، Robert L. (2005). "'Myelodysplasia,' Myeloneuropathy, and Copper Deficiency". Mayo Clinic Proceedings. ج. 80 ع. 7: 943–6. DOI:10.4065/80.7.943. PMID:16007901.
  • Danks، D M (1988). "Copper Deficiency in Humans". Annual Review of Nutrition. ج. 8: 235–57. DOI:10.1146/annurev.nu.08.070188.001315. PMID:3060166.
  • Strain، J. J. (1994). "Newer aspects of micronutrients in chronic disease: copper". Proceedings of the Nutrition Society. ج. 53 ع. 3: 583–98. DOI:10.1079/PNS19940067. PMID:7886057.
  • Salem، Harry؛ Green، Sidney؛ Bigelow، Sanford؛ Borzelleca، Joseph؛ Baskin، Steven (1992). "Preface". Critical Reviews in Food Science and Nutrition. ج. 32 ع. 1: 1–31. DOI:10.1080/10408399209527583. PMID:1290583.
  • Kaegi، Jeremias H. R.؛ Schaeffer، Andreas (1988). "Biochemistry of metallothionein". Biochemistry. ج. 27 ع. 23: 8509–15. DOI:10.1021/bi00423a001. PMID:3064814.
  • Bartzokis، George؛ Sultzer، David؛ Cummings، Jeffrey؛ Holt، Lori E.؛ Hance، Darwood B.؛ Henderson، Victor W.؛ Mintz، Jim (2000). "In Vivo Evaluation of Brain Iron in Alzheimer Disease Using Magnetic Resonance Imaging". Archives of General Psychiatry. ج. 57 ع. 1: 47–53. DOI:10.1001/archpsyc.57.1.47. PMID:10632232. {{استشهاد بدورية محكمة}}: الوسيط غير المعروف |laysummary= تم تجاهله (مساعدة)
  • Duce، James A.؛ Tsatsanis، Andrew؛ Cater، Michael A.؛ James، Simon A.؛ Robb، Elysia؛ Wikhe، Krutika؛ Leong، Su Ling؛ Perez، Keyla؛ Johanssen، Timothy؛ Greenough، Mark A.؛ Cho، Hyun-Hee؛ Galatis، Denise؛ Moir، Robert D.؛ Masters، Colin L.؛ McLean، Catriona؛ Tanzi، Rudolph E.؛ Cappai، Roberto؛ Barnham، Kevin J.؛ Ciccotosto، Giuseppe D.؛ Rogers، Jack T.؛ Bush، Ashley I. (2010). "Iron-Export Ferroxidase Activity of β-Amyloid Precursor Protein Is Inhibited by Zinc in Alzheimer's Disease". Cell. ج. 142 ع. 6: 857–67. DOI:10.1016/j.cell.2010.08.014. PMC:2943017. PMID:20817278. {{استشهاد بدورية محكمة}}: الوسيط غير المعروف |laysummary= تم تجاهله (مساعدة)
  • Brewer، George J. (2010). "Copper toxicity in the general population". Clinical Neurophysiology. ج. 121 ع. 4: 459–60. DOI:10.1016/j.clinph.2009.12.015. PMID:20071223.
  • Llanos، Roxana M.؛ Mercer، Julian F.B. (2002). "The Molecular Basis of Copper Homeostasis Copper-Related Disorders". DNA and Cell Biology. ج. 21 ع. 4: 259–70. DOI:10.1089/104454902753759681. PMID:12042066.
  • Singh، Itender؛ Sagare، Abhay P.؛ Coma، Mireia؛ Perlmutter، David؛ Gelein، Robert؛ Bell، Robert D.؛ Deane، Richard J.؛ Zhong، Elaine؛ Parisi، Margaret؛ Ciszewski، Joseph؛ Kasper، R. Tristan؛ Deane، Rashid (2013). "Low levels of copper disrupt brain amyloid-β homeostasis by altering its production and clearance". Proceedings of the National Academy of Sciences. ج. 110 ع. 36: 14771–6. Bibcode:2013PNAS..11014771S. DOI:10.1073/pnas.1302212110. PMC:3767519. PMID:23959870. {{استشهاد بدورية محكمة}}: الوسيط غير المعروف |laysummary= تم تجاهله (مساعدة)
  • Loef، Martin؛ Walach، Harald (1 يناير 2012). "Copper and iron in Alzheimer's disease: a systematic review and its dietary implications". The British Journal of Nutrition. ج. 107 ع. 1: 7–19. DOI:10.1017/S000711451100376X. ISSN:1475-2662. PMID:21767446.
  • Kaden، Daniela؛ Bush، Ashley I.؛ Danzeisen، Ruth؛ Bayer، Thomas A.؛ Multhaup، Gerd (2011). "Disturbed Copper Bioavailability in Alzheimer's Disease". International Journal of Alzheimer's Disease. ج. 2011: 345614. DOI:10.4061/2011/345614. PMC:3227474. PMID:22145082.{{استشهاد بدورية محكمة}}: صيانة الاستشهاد: دوي مجاني غير معلم (link)
  • Kessler، Holger؛ Bayer، Thomas A.؛ Bach، Daniela؛ Schneider-Axmann، Thomas؛ Supprian، Tillmann؛ Herrmann، Wolfgang؛ Haber، Manfred؛ Multhaup، Gerd؛ Falkai، Peter؛ Pajonk، Frank-Gerald (2008). "Intake of copper has no effect on cognition in patients with mild Alzheimer's disease: a pilot phase 2 clinical trial". Journal of Neural Transmission. ج. 115 ع. 8: 1181–7. DOI:10.1007/s00702-008-0080-1. PMC:2516533. PMID:18587525.
  • Kessler، Holger؛ Pajonk، Frank-Gerald؛ Bach، Daniela؛ Schneider-Axmann، Thomas؛ Falkai، Peter؛ Herrmann، Wolfgang؛ Multhaup، Gerd؛ Wiltfang، Jens؛ Schäfer، Stephanie؛ Wirths، Oliver؛ Bayer، Thomas A. (2008). "Effect of copper intake on CSF parameters in patients with mild Alzheimer's disease: a pilot phase 2 clinical trial". Journal of Neural Transmission. ج. 115 ع. 12: 1651–9. DOI:10.1007/s00702-008-0136-2. PMID:18972062.
  • Brewer، George J. (2012). "Copper toxicity in Alzheimer's disease: Cognitive loss from ingestion of inorganic copper". Journal of Trace Elements in Medicine and Biology. ج. 26 ع. 2–3: 89–92. DOI:10.1016/j.jtemb.2012.04.019. PMID:22673823.
  • Brewer، George J. (2000). "Editorial: Is heterozygosity for a Wilson's disease gene defect an important underlying cause of infantile and childhood copper toxicosis syndromes?". The Journal of Trace Elements in Experimental Medicine. ج. 13 ع. 3: 249–54. DOI:10.1002/1520-670X(2000)13:3<249::AID-JTRA1>3.0.CO;2-L.
  • Araya، Magdalena؛ McGoldrick، Marie Clare؛ Klevay، Leslie M.؛ Strain، J.J.؛ Robson، Paula؛ Nielsen، Forrest؛ Olivares، Manuel؛ Pizarro، Fernando؛ Johnson، LuAnn؛ Poirier، Kenneth A. (2001). "Determination of an Acute No-Observed-Adverse-Effect Level (NOAEL) for Copper in Water". Regulatory Toxicology and Pharmacology. ج. 34 ع. 2: 137–45. DOI:10.1006/rtph.2001.1492. PMID:11603956.
  • Araya، Magdalena؛ Chen، Bingheng؛ Klevay، Leslie M؛ Strain، J.J؛ Johnson، LuAnn؛ Robson، Paula؛ Shi، Wei؛ Nielsen، Forrest؛ Zhu، Huigang؛ Olivares، Manuel؛ Pizarro، Fernando؛ Haber، Lynne T (2003). "Confirmation of an acute no-observed-adverse-effect and low-observed-adverse-effect level for copper in bottled drinking water in a multi-site international study". Regulatory Toxicology and Pharmacology. ج. 38 ع. 3: 389–99. DOI:10.1016/j.yrtph.2003.08.001. PMID:14623488.
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  • Buchanan، Sharunda D.؛ Diseker، Robert A.؛ Sinks، Thomas؛ Olson، David R.؛ Daniel، Jack؛ Flodman، Thomas (1999). "Copper in Drinking Water, Nebraska, 1994". International Journal of Occupational and Environmental Health. ج. 5 ع. 4: 256–61. DOI:10.1179/oeh.1999.5.4.256. PMID:10633241.
  • O’Connor، J.M.؛ Bonham، M.P.؛ Turley، E.؛ McKeown، A.؛ McKelvey-Martin، V.J.؛ Gilmore، W.S.؛ Strain، J.J. (2003). "Copper Supplementation Has No Effect on Markers of DNA Damage and Liver Function in Healthy Adults (FOODCUE Project)". Annals of Nutrition and Metabolism. ج. 47 ع. 5: 201–6. DOI:10.1159/000070486. PMID:12748413.
  • Turley، Eithne؛ McKeown، Andrea؛ Bonham، Maxine P.؛ O’Connor، Jacqueline M.؛ Chopra، Mridula؛ Harvey، Linda J.؛ Majsak-Newman، Gosia؛ Fairweather-Tait، Susan J.؛ Bügel، Susanne؛ Sandström، Brittmarie؛ Rock، Edmond؛ Mazur، Andrzej؛ Rayssiguier، Yves؛ Strain، J.J. (2000). "Copper supplementation in humans does not affect the susceptibility of low density lipoprotein to in vitro induced oxidation (FOODCUE project)". Free Radical Biology and Medicine. ج. 29 ع. 11: 1129–34. DOI:10.1016/S0891-5849(00)00409-3. PMID:11121720.
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  • Goodman، Vicki؛ Brewer، George؛ Merajver، Sofia (2005). "Control of Copper Status for Cancer Therapy". Current Cancer Drug Targets. ج. 5 ع. 7: 543–9. DOI:10.2174/156800905774574066. PMID:16305350.
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  • Pass، Harvey I.؛ Brewer، George J.؛ Dick، Robert؛ Carbone، Michele؛ Merajver، Sofia (2008). "A Phase II Trial of Tetrathiomolybdate After Surgery for Malignant Mesothelioma: Final Results". The Annals of Thoracic Surgery. ج. 86 ع. 2: 383–9, discussion 390. DOI:10.1016/j.athoracsur.2008.03.016. PMID:18640301.
  • Brewer، G. J.؛ Merajver، S. D. (2002). "Cancer Therapy With Tetrathiomolybdate: Antiangiogenesis by Lowering Body Copper—A Review". Integrative Cancer Therapies. ج. 1 ع. 4: 327–37. DOI:10.1177/1534735402238185. PMID:14664727.
  • Fan، Chuandong؛ Zhao، Jing؛ Zhao، Baoxiang؛ Zhang، Shangli؛ Miao، Junying (2009). "Novel Complex of Copper and a Salicylaldehyde Pyrazole Hydrazone Derivative Induces Apoptosis through Up-Regulating Integrin β4 in Vascular Endothelial Cells". Chemical Research in Toxicology. ج. 22 ع. 9: 1517–25. DOI:10.1021/tx900111y. PMID:19621939.
  • Lowndes، Sarah A.؛ Harris، Adrian L. (2005). "The Role of Copper in Tumour Angiogenesis". Journal of Mammary Gland Biology and Neoplasia. ج. 10 ع. 4: 299–310. DOI:10.1007/s10911-006-9003-7. PMID:16924372.
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    Conclusions
    Active substances released from the IUD or IUS, together with products derived from the inflammatory reaction present in the luminal fluids of the genital tract, are toxic for spermatozoa and oocytes, preventing the encounter of healthy gametes and the formation of viable embryos. The current data do not indicate that embryos are formed in IUD users at a rate comparable to that of nonusers. The common belief that the usual mechanism of action of IUDs in women is destruction of embryos in the uterus is not supported by empirical evidence. The bulk of the data indicate that interference with the reproductive process after fertilization has taken place is exceptional in the presence of a T-Cu or LNG-IUD and that the usual mechanism by which they prevent pregnancy in women is by preventing fertilization.

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