References analysis of the Wikipedia article "تسرطن" in the Arabic version

doi.org

Belikov، Aleksey V. (22 سبتمبر 2017). "The number of key carcinogenic events can be predicted from cancer incidence". Scientific Reports. ج. 7 ع. 1: 12170. Bibcode:2017NatSR...712170B. DOI:10.1038/s41598-017-12448-7. PMC:5610194. PMID:28939880.

Wood LD، Parsons DW، Jones S، Lin J، Sjöblom T، Leary RJ، وآخرون (نوفمبر 2007). "The genomic landscapes of human breast and colorectal cancers". Science. ج. 318 ع. 5853: 1108–13. Bibcode:2007Sci...318.1108W. CiteSeerX:10.1.1.218.5477. DOI:10.1126/science.1145720. PMID:17932254.

Knudson AG (نوفمبر 2001). "Two genetic hits (more or less) to cancer". Nature Reviews. Cancer. ج. 1 ع. 2: 157–62. DOI:10.1038/35101031. PMID:11905807.

Fearon ER، Vogelstein B (يونيو 1990). "A genetic model for colorectal tumorigenesis". Cell. ج. 61 ع. 5: 759–67. DOI:10.1016/0092-8674(90)90186-I. PMID:2188735.

Tomasetti C، Li L، Vogelstein B (23 مارس 2017). "Stem cell divisions, somatic mutations, cancer etiology, and cancer prevention". Science. ج. 355 ع. 6331: 1330–1334. Bibcode:2017Sci...355.1330T. DOI:10.1126/science.aaf9011. PMC:5852673. PMID:28336671.

Lim LP، Lau NC، Garrett-Engele P، Grimson A، Schelter JM، Castle J، Bartel DP، Linsley PS، Johnson JM (فبراير 2005). "Microarray analysis shows that some microRNAs downregulate large numbers of target mRNAs". Nature. ج. 433 ع. 7027: 769–73. Bibcode:2005Natur.433..769L. DOI:10.1038/nature03315. PMID:15685193.

Kastan MB (أبريل 2008). "DNA damage responses: mechanisms and roles in human disease: 2007 G.H.A. Clowes Memorial Award Lecture". Molecular Cancer Research. ج. 6 ع. 4: 517–24. DOI:10.1158/1541-7786.MCR-08-0020. PMID:18403632.

Handa O، Naito Y، Yoshikawa T (2011). "Redox biology and gastric carcinogenesis: the role of Helicobacter pylori". Redox Report. ج. 16 ع. 1: 1–7. DOI:10.1179/174329211X12968219310756. PMID:21605492.

Kanavy HE، Gerstenblith MR (ديسمبر 2011). "Ultraviolet radiation and melanoma". Seminars in Cutaneous Medicine and Surgery. ج. 30 ع. 4: 222–8. DOI:10.1016/j.sder.2011.08.003. PMID:22123420.

Bernstein C، Holubec H، Bhattacharyya AK، Nguyen H، Payne CM، Zaitlin B، Bernstein H (أغسطس 2011). "Carcinogenicity of deoxycholate, a secondary bile acid". Archives of Toxicology. ج. 85 ع. 8: 863–71. DOI:10.1007/s00204-011-0648-7. PMC:3149672. PMID:21267546.

Lichtenstein P، Holm NV، Verkasalo PK، Iliadou A، Kaprio J، Koskenvuo M، Pukkala E، Skytthe A، Hemminki K (يوليو 2000). "Environmental and heritable factors in the causation of cancer--analyses of cohorts of twins from Sweden, Denmark, and Finland". The New England Journal of Medicine. ج. 343 ع. 2: 78–85. DOI:10.1056/NEJM200007133430201. PMID:10891514.

Fearon ER (نوفمبر 1997). "Human cancer syndromes: clues to the origin and nature of cancer". Science. ج. 278 ع. 5340: 1043–50. Bibcode:1997Sci...278.1043F. DOI:10.1126/science.278.5340.1043. PMID:9353177.

O'Hagan HM، Mohammad HP، Baylin SB (أغسطس 2008). Lee JT (المحرر). "Double strand breaks can initiate gene silencing and SIRT1-dependent onset of DNA methylation in an exogenous promoter CpG island". PLOS Genetics. ج. 4 ع. 8: e1000155. DOI:10.1371/journal.pgen.1000155. PMC:2491723. PMID:18704159.{{استشهاد بدورية محكمة}}: صيانة الاستشهاد: دوي مجاني غير معلم (link)

Cuozzo C، Porcellini A، Angrisano T، Morano A، Lee B، Di Pardo A، Messina S، Iuliano R، Fusco A، Santillo MR، Muller MT، Chiariotti L، Gottesman ME، Avvedimento EV (يوليو 2007). "DNA damage, homology-directed repair, and DNA methylation". PLOS Genetics. ج. 3 ع. 7: e110. DOI:10.1371/journal.pgen.0030110. PMC:1913100. PMID:17616978.{{استشهاد بدورية محكمة}}: صيانة الاستشهاد: دوي مجاني غير معلم (link)

Cunningham FH، Fiebelkorn S، Johnson M، Meredith C (نوفمبر 2011). "A novel application of the Margin of Exposure approach: segregation of tobacco smoke toxicants". Food and Chemical Toxicology. ج. 49 ع. 11: 2921–33. DOI:10.1016/j.fct.2011.07.019. PMID:21802474.

Gerlinger M، Rowan AJ، Horswell S، Larkin J، Endesfelder D، Gronroos E، وآخرون (مارس 2012). "Intratumor heterogeneity and branched evolution revealed by multiregion sequencing". The New England Journal of Medicine. ج. 366 ع. 10: 883–92. DOI:10.1056/NEJMoa1113205. PMC:4878653. PMID:22397650.

López-Lázaro M (مايو 2015). "The migration ability of stem cells can explain the existence of cancer of unknown primary site. Rethinking metastasis". Oncoscience. ج. 2 ع. 5: 467–75. DOI:10.18632/oncoscience.159. PMC:4468332. PMID:26097879.

Tomasetti C، Vogelstein B (يناير 2015). "Cancer etiology. Variation in cancer risk among tissues can be explained by the number of stem cell divisions". Science. ج. 347 ع. 6217: 78–81. DOI:10.1126/science.1260825. PMC:4446723. PMID:25554788.

Rubin H (مارس 2011). "Fields and field cancerization: the preneoplastic origins of cancer: asymptomatic hyperplastic fields are precursors of neoplasia, and their progression to tumors can be tracked by saturation density in culture". BioEssays. ج. 33 ع. 3: 224–31. DOI:10.1002/bies.201000067. PMID:21254148.

Vogelstein B، Papadopoulos N، Velculescu VE، Zhou S، Diaz LA، Kinzler KW (مارس 2013). "Cancer genome landscapes". review. Science. ج. 339 ع. 6127: 1546–58. Bibcode:2013Sci...339.1546V. DOI:10.1126/science.1235122. PMC:3749880. PMID:23539594.

Schmitt MW، Prindle MJ، Loeb LA (سبتمبر 2012). "Implications of genetic heterogeneity in cancer". Annals of the New York Academy of Sciences. ج. 1267 ع. 1: 110–6. Bibcode:2012NYASA1267..110S. DOI:10.1111/j.1749-6632.2012.06590.x. PMC:3674777. PMID:22954224.

Tutt AN، van Oostrom CT، Ross GM، van Steeg H، Ashworth A (مارس 2002). "Disruption of Brca2 increases the spontaneous mutation rate in vivo: synergism with ionizing radiation". EMBO Reports. ج. 3 ع. 3: 255–60. DOI:10.1093/embo-reports/kvf037. PMC:1084010. PMID:11850397.

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