تنكس واليرياني (Arabic Wikipedia)

Analysis of information sources in references of the Wikipedia article "تنكس واليرياني" in Arabic language version.

refsWebsite

Global rank Arabic rank

70nih.gov

4^th place

6^th place

46doi.org

2^nd place

5^th place

5harvard.edu

18^th place

33^rd place

4web.archive.org

1^st place

2archive.org

6^th place

3^rd place

1ucsf.edu

5,479^th place

7,280^th place

1jstor.org

26^th place

31^st place

1caltech.edu

887^th place

1,217^th place

1jbc.org

4,455^th place

4,178^th place

archive.org

"Schwann cell properties: 3. C-fos expression, bFGF production, phagocytosis and proliferation during Wallerian degeneration". Journal of Neuropathology and Experimental Neurology. ج. 54 ع. 4: 487–96. يوليو 1995. DOI:10.1097/00005072-199507000-00002. PMID:7602323.
Lundy-Ekman، Laurie (2007). Neuroscience: Fundamentals for Rehabilitation (ط. 3rd). Saunders. ISBN:978-1-4160-2578-8.

caltech.edu

authors.library.caltech.edu

"Changes of nerve growth factor synthesis in nonneuronal cells in response to sciatic nerve transection" (PDF). The Journal of Cell Biology. ج. 104 ع. 6: 1623–31. يونيو 1987. DOI:10.1083/jcb.104.6.1623. PMC:2114490. PMID:3034917. مؤرشف من الأصل (PDF) في 2017-08-14.

doi.org

"Wallerian degeneration, wld(s), and nmnat". Annual Review of Neuroscience. ج. 33 ع. 1: 245–67. 1 يونيو 2010. DOI:10.1146/annurev-neuro-060909-153248. PMC:5223592. PMID:20345246.
"Endogenous Nmnat2 is an essential survival factor for maintenance of healthy axons". PLOS Biology. ج. 8 ع. 1: e1000300. يناير 2010. DOI:10.1371/journal.pbio.1000300. PMC:2811159. PMID:20126265.{{استشهاد بدورية محكمة}}: صيانة الاستشهاد: دوي مجاني غير معلم (link)
"NMNAT: It's an NAD + Synthase… It's a Chaperone… It's a Neuroprotector". Current Opinion in Genetics & Development. ج. 44: 156–162. 2017. DOI:10.1016/j.gde.2017.03.014. PMC:5515290. PMID:28445802.
"Experiments on the Section of the Glossopharyngeal and Hypoglossal Nerves of the Frog, and Observations of the Alterations Produced Thereby in the Structure of Their Primitive Fibres". Philosophical Transactions of the Royal Society of London. ج. 140: 423–429. 1 يناير 1850. DOI:10.1098/rstl.1850.0021. JSTOR:108444.
"An 85-kb tandem triplication in the slow Wallerian degeneration (Wlds) mouse". Proceedings of the National Academy of Sciences of the United States of America. ج. 95 ع. 17: 9985–90. أغسطس 1998. Bibcode:1998PNAS...95.9985C. DOI:10.1073/pnas.95.17.9985. PMC:21448. PMID:9707587.
"Nerve injury, axonal degeneration and neural regeneration: basic insights". Brain Pathology. ج. 9 ع. 2: 313–25. أبريل 1999. DOI:10.1111/j.1750-3639.1999.tb00229.x. PMID:10219748.
"In vivo imaging of axonal degeneration and regeneration in the injured spinal cord". Nature Medicine. ج. 11 ع. 5: 572–7. مايو 2005. DOI:10.1038/nm1229. PMID:15821747.
"Endocytotic formation of vesicles and other membranous structures induced by Ca2+ and axolemmal injury". The Journal of Neuroscience. ج. 18 ع. 11: 4029–41. يونيو 1998. DOI:10.1523/JNEUROSCI.18-11-04029.1998. PMC:6792792. PMID:9592084.
"Axon degeneration: molecular mechanisms of a self-destruction pathway". The Journal of Cell Biology. ج. 196 ع. 1: 7–18. يناير 2012. DOI:10.1083/jcb.201108111. PMC:3255986. PMID:22232700.
"Why is Wallerian degeneration in the CNS so slow?". Annual Review of Neuroscience. ج. 30 ع. 1: 153–79. 1 يوليو 2007. DOI:10.1146/annurev.neuro.30.051606.094354. PMID:17506644.
"Microanatomy of axon/glial signaling during Wallerian degeneration". The Journal of Neuroscience. ج. 25 ع. 13: 3478–87. مارس 2005. DOI:10.1523/JNEUROSCI.3766-04.2005. PMC:6724908. PMID:15800203.
"Complement depletion reduces macrophage infiltration and ctivation during Wallerian degeneration and axonal regeneration". The Journal of Neuroscience. ج. 18 ع. 17: 6713–22. سبتمبر 1998. DOI:10.1523/JNEUROSCI.18-17-06713.1998. PMC:6792968. PMID:9712643.
"Degeneration of myelinated efferent fibers prompts mitosis in Remak Schwann cells of uninjured C-fiber afferents". The Journal of Neuroscience. ج. 25 ع. 5: 1179–87. فبراير 2005. DOI:10.1523/JNEUROSCI.1372-04.2005. PMC:6725954. PMID:15689554.
"Schwann cell properties: 3. C-fos expression, bFGF production, phagocytosis and proliferation during Wallerian degeneration". Journal of Neuropathology and Experimental Neurology. ج. 54 ع. 4: 487–96. يوليو 1995. DOI:10.1097/00005072-199507000-00002. PMID:7602323.
"Does oligodendrocyte survival depend on axons?". Current Biology. ج. 3 ع. 8: 489–97. أغسطس 1993. DOI:10.1016/0960-9822(93)90039-Q. PMID:15335686.
"Oligodendrocyte survival in Wallerian degeneration". Acta Neuropathologica. ج. 80 ع. 2: 184–91. 31 مايو 1990. DOI:10.1007/BF00308922. PMID:1697140.
"The temporal and spatial activation of microglia in fiber tracts undergoing anterograde and retrograde degeneration following spinal cord lesion". Journal of Neurotrauma. ج. 12 ع. 2: 209–22. أبريل 1995. DOI:10.1089/neu.1995.12.209. PMID:7629867.
"Delayed macrophage responses and myelin clearance during Wallerian degeneration in the central nervous system: the dorsal radiculotomy model". Experimental Neurology. ج. 129 ع. 2: 225–36. أكتوبر 1994. DOI:10.1006/exnr.1994.1164. PMID:7957737.
"The Nogo signaling pathway for regeneration block". Annual Review of Neuroscience. ج. 27 ع. 1: 341–68. 21 يوليو 2004. DOI:10.1146/annurev.neuro.27.070203.144340. PMID:15217336.
"The early stages of Wallerian degeneration in the severed optic nerve of the newt (Triturus viridescens)". The Anatomical Record. ج. 187 ع. 3: 291–310. مارس 1977. DOI:10.1002/ar.1091870303. PMID:851236.
"Changes of nerve growth factor synthesis in nonneuronal cells in response to sciatic nerve transection" (PDF). The Journal of Cell Biology. ج. 104 ع. 6: 1623–31. يونيو 1987. DOI:10.1083/jcb.104.6.1623. PMC:2114490. PMID:3034917. مؤرشف من الأصل (PDF) في 2017-08-14.
"The degeneration of unmyelinated axons following nerve section: an ultrastructural study". Journal of Neurocytology. ج. 3 ع. 4: 497–512. أكتوبر 1974. DOI:10.1007/BF01098736. PMID:4436692.
"Ninjurin, a novel adhesion molecule, is induced by nerve injury and promotes axonal growth". Neuron. ج. 17 ع. 2: 353–61. أغسطس 1996. DOI:10.1016/S0896-6273(00)80166-X. PMID:8780658.
"The Effectiveness of the Gene Which Slows the Rate of Wallerian Degeneration in C57BL/Ola Mice Declines With Age". The European Journal of Neuroscience. ج. 4 ع. 10: 1000–2. 1 أكتوبر 1992. DOI:10.1111/j.1460-9568.1992.tb00126.x. PMID:12106435.
Perry, V. H., Lunn, E. R., Brown, M. C., Cahusac, S. and Gordon, S. (1990), Evidence that the Rate of Wallerian Degeneration is Controlled by a Single Autosomal Dominant Gene. European Journal of Neuroscience, 2: 408-413. https://doi.org/10.1111/j.1460-9568.1990.tb00433.x نسخة محفوظة 4 يونيو 2018 على موقع واي باك مشين.
"A gene affecting Wallerian nerve degeneration maps distally on mouse chromosome 4". Proceedings of the National Academy of Sciences of the United States of America. ج. 90 ع. 20: 9717–20. أكتوبر 1993. Bibcode:1993PNAS...90.9717L. DOI:10.1073/pnas.90.20.9717. PMC:47641. PMID:8415768.
"Increased nuclear NAD biosynthesis and SIRT1 activation prevent axonal degeneration". Science. ج. 305 ع. 5686: 1010–3. أغسطس 2004. Bibcode:2004Sci...305.1010A. DOI:10.1126/science.1098014. PMID:15310905.
"Wallerian degeneration of injured axons and synapses is delayed by a Ube4b/Nmnat chimeric gene". Nature Neuroscience. ج. 4 ع. 12: 1199–206. ديسمبر 2001. DOI:10.1038/nn770. PMID:11770485.
"Non-nuclear Wld(S) determines its neuroprotective efficacy for axons and synapses in vivo". The Journal of Neuroscience. ج. 29 ع. 3: 653–68. يناير 2009. DOI:10.1523/JNEUROSCI.3814-08.2009. PMC:6665162. PMID:19158292.
"Prolonged survival of transected nerve fibres in C57BL/Ola mice is an intrinsic characteristic of the axon". Journal of Neurocytology. ج. 22 ع. 5: 311–21. مايو 1993. DOI:10.1007/BF01195555. PMID:8315413.
"The slow Wallerian degeneration gene in vivo protects motor axons but not their cell bodies after avulsion and neonatal axotomy". The European Journal of Neuroscience. ج. 24 ع. 8: 2163–8. أكتوبر 2006. DOI:10.1111/j.1460-9568.2006.05103.x. PMID:17074042.
"A local mechanism mediates NAD-dependent protection of axon degeneration". The Journal of Cell Biology. ج. 170 ع. 3: 349–55. أغسطس 2005. DOI:10.1083/jcb.200504028. PMC:2171458. PMID:16043516.
"NAD(+) and axon degeneration revisited: Nmnat1 cannot substitute for Wld(S) to delay Wallerian degeneration". Cell Death and Differentiation. ج. 14 ع. 1: 116–27. يناير 2007. DOI:10.1038/sj.cdd.4401944. PMID:16645633.
"Targeting NMNAT1 to axons and synapses transforms its neuroprotective potency in vivo". The Journal of Neuroscience. ج. 30 ع. 40: 13291–304. أكتوبر 2010. DOI:10.1523/JNEUROSCI.1189-10.2010. PMC:6634738. PMID:20926655.
"Genetic influences on cellular reactions to spinal cord injury: activation of macrophages/microglia and astrocytes is delayed in mice carrying a mutation (WldS) that causes delayed Wallerian degeneration". The Journal of Comparative Neurology. ج. 371 ع. 3: 469–84. يوليو 1996. DOI:10.1002/(SICI)1096-9861(19960729)371:3<469::AID-CNE9>3.0.CO;2-0. PMID:8842900.
"Further studies on motor and sensory nerve regeneration in mice with delayed Wallerian degeneration". The European Journal of Neuroscience. ج. 6 ع. 3: 420–8. مارس 1994. DOI:10.1111/j.1460-9568.1994.tb00285.x. PMID:8019679.
"dSarm/Sarm1 is required for activation of an injury-induced axon death pathway". Science. ج. 337 ع. 6093: 481–4. يوليو 2012. Bibcode:2012Sci...337..481O. DOI:10.1126/science.1223899. PMC:5225956. PMID:22678360.
"Sarm1-mediated axon degeneration requires both SAM and TIR interactions". The Journal of Neuroscience. ج. 33 ع. 33: 13569–80. أغسطس 2013. DOI:10.1523/JNEUROSCI.1197-13.2013. PMC:3742939. PMID:23946415.
"Resolving the topological enigma in Ca 2+ signaling by cyclic ADP-ribose and NAADP". Journal of Biological Chemistry. ج. 294 ع. 52: 19831–19843. 2019. DOI:10.1074/jbc.REV119.009635. PMC:6937575. PMID:31672920. مؤرشف من الأصل في 2020-10-20.{{استشهاد بدورية محكمة}}: صيانة الاستشهاد: دوي مجاني غير معلم (link)
"SARM1 activation triggers axon degeneration locally via NAD⁺ destruction". Science. ج. 348 ع. 6233: 453–7. أبريل 2015. Bibcode:2015Sci...348..453G. DOI:10.1126/science.1258366. PMC:4513950. PMID:25908823.
"+ Cleavage Activity that Promotes Pathological Axonal Degeneration". Neuron. ج. 93 ع. 6: 1334–1343.e5. مارس 2017. DOI:10.1016/j.neuron.2017.02.022. PMC:6284238. PMID:28334607.
"+ depletion". eLife. ج. 5. أكتوبر 2016. DOI:10.7554/eLife.19749. PMC:5063586. PMID:27735788.{{استشهاد بدورية محكمة}}: صيانة الاستشهاد: دوي مجاني غير معلم (link)
"S, Confers Lifelong Rescue in a Mouse Model of Severe Axonopathy". Cell Reports. ج. 21 ع. 1: 10–16. أكتوبر 2017. DOI:10.1016/j.celrep.2017.09.027. PMC:5640801. PMID:28978465.
"Pathological axonal death through a MAPK cascade that triggers a local energy deficit". Cell. ج. 160 ع. 1–2: 161–76. يناير 2015. DOI:10.1016/j.cell.2014.11.053. PMC:4306654. PMID:25594179.
"MAPK signaling promotes axonal degeneration by speeding the turnover of the axonal maintenance factor NMNAT2". eLife. ج. 6. يناير 2017. DOI:10.7554/eLife.22540. PMC:5241118. PMID:28095293.{{استشهاد بدورية محكمة}}: صيانة الاستشهاد: دوي مجاني غير معلم (link)
"Attenuated traumatic axonal injury and improved functional outcome after traumatic brain injury in mice lacking Sarm1". Brain. ج. 139 ع. 4: 1094–1105. 2016. DOI:10.1093/brain/aww001. PMC:5006226. PMID:26912636.

harvard.edu

ui.adsabs.harvard.edu

"An 85-kb tandem triplication in the slow Wallerian degeneration (Wlds) mouse". Proceedings of the National Academy of Sciences of the United States of America. ج. 95 ع. 17: 9985–90. أغسطس 1998. Bibcode:1998PNAS...95.9985C. DOI:10.1073/pnas.95.17.9985. PMC:21448. PMID:9707587.
"A gene affecting Wallerian nerve degeneration maps distally on mouse chromosome 4". Proceedings of the National Academy of Sciences of the United States of America. ج. 90 ع. 20: 9717–20. أكتوبر 1993. Bibcode:1993PNAS...90.9717L. DOI:10.1073/pnas.90.20.9717. PMC:47641. PMID:8415768.
"Increased nuclear NAD biosynthesis and SIRT1 activation prevent axonal degeneration". Science. ج. 305 ع. 5686: 1010–3. أغسطس 2004. Bibcode:2004Sci...305.1010A. DOI:10.1126/science.1098014. PMID:15310905.
"dSarm/Sarm1 is required for activation of an injury-induced axon death pathway". Science. ج. 337 ع. 6093: 481–4. يوليو 2012. Bibcode:2012Sci...337..481O. DOI:10.1126/science.1223899. PMC:5225956. PMID:22678360.
"SARM1 activation triggers axon degeneration locally via NAD⁺ destruction". Science. ج. 348 ع. 6233: 453–7. أبريل 2015. Bibcode:2015Sci...348..453G. DOI:10.1126/science.1258366. PMC:4513950. PMID:25908823.

jbc.org

"Resolving the topological enigma in Ca 2+ signaling by cyclic ADP-ribose and NAADP". Journal of Biological Chemistry. ج. 294 ع. 52: 19831–19843. 2019. DOI:10.1074/jbc.REV119.009635. PMC:6937575. PMID:31672920. مؤرشف من الأصل في 2020-10-20.{{استشهاد بدورية محكمة}}: صيانة الاستشهاد: دوي مجاني غير معلم (link)

jstor.org

"Experiments on the Section of the Glossopharyngeal and Hypoglossal Nerves of the Frog, and Observations of the Alterations Produced Thereby in the Structure of Their Primitive Fibres". Philosophical Transactions of the Royal Society of London. ج. 140: 423–429. 1 يناير 1850. DOI:10.1098/rstl.1850.0021. JSTOR:108444.

nih.gov

pubmed.ncbi.nlm.nih.gov

"Wallerian degeneration, wld(s), and nmnat". Annual Review of Neuroscience. ج. 33 ع. 1: 245–67. 1 يونيو 2010. DOI:10.1146/annurev-neuro-060909-153248. PMC:5223592. PMID:20345246.
"Endogenous Nmnat2 is an essential survival factor for maintenance of healthy axons". PLOS Biology. ج. 8 ع. 1: e1000300. يناير 2010. DOI:10.1371/journal.pbio.1000300. PMC:2811159. PMID:20126265.{{استشهاد بدورية محكمة}}: صيانة الاستشهاد: دوي مجاني غير معلم (link)
"NMNAT: It's an NAD + Synthase… It's a Chaperone… It's a Neuroprotector". Current Opinion in Genetics & Development. ج. 44: 156–162. 2017. DOI:10.1016/j.gde.2017.03.014. PMC:5515290. PMID:28445802.
"An 85-kb tandem triplication in the slow Wallerian degeneration (Wlds) mouse". Proceedings of the National Academy of Sciences of the United States of America. ج. 95 ع. 17: 9985–90. أغسطس 1998. Bibcode:1998PNAS...95.9985C. DOI:10.1073/pnas.95.17.9985. PMC:21448. PMID:9707587.
"Nerve injury, axonal degeneration and neural regeneration: basic insights". Brain Pathology. ج. 9 ع. 2: 313–25. أبريل 1999. DOI:10.1111/j.1750-3639.1999.tb00229.x. PMID:10219748.
"In vivo imaging of axonal degeneration and regeneration in the injured spinal cord". Nature Medicine. ج. 11 ع. 5: 572–7. مايو 2005. DOI:10.1038/nm1229. PMID:15821747.
"Endocytotic formation of vesicles and other membranous structures induced by Ca2+ and axolemmal injury". The Journal of Neuroscience. ج. 18 ع. 11: 4029–41. يونيو 1998. DOI:10.1523/JNEUROSCI.18-11-04029.1998. PMC:6792792. PMID:9592084.
"Axon degeneration: molecular mechanisms of a self-destruction pathway". The Journal of Cell Biology. ج. 196 ع. 1: 7–18. يناير 2012. DOI:10.1083/jcb.201108111. PMC:3255986. PMID:22232700.
"Why is Wallerian degeneration in the CNS so slow?". Annual Review of Neuroscience. ج. 30 ع. 1: 153–79. 1 يوليو 2007. DOI:10.1146/annurev.neuro.30.051606.094354. PMID:17506644.
"Multiple forms of Ca-activated protease from rat brain and muscle". The Journal of Biological Chemistry. ج. 259 ع. 5: 3210–8. مارس 1984. PMID:6321500.
"Microanatomy of axon/glial signaling during Wallerian degeneration". The Journal of Neuroscience. ج. 25 ع. 13: 3478–87. مارس 2005. DOI:10.1523/JNEUROSCI.3766-04.2005. PMC:6724908. PMID:15800203.
"Complement depletion reduces macrophage infiltration and ctivation during Wallerian degeneration and axonal regeneration". The Journal of Neuroscience. ج. 18 ع. 17: 6713–22. سبتمبر 1998. DOI:10.1523/JNEUROSCI.18-17-06713.1998. PMC:6792968. PMID:9712643.
"Degeneration of myelinated efferent fibers prompts mitosis in Remak Schwann cells of uninjured C-fiber afferents". The Journal of Neuroscience. ج. 25 ع. 5: 1179–87. فبراير 2005. DOI:10.1523/JNEUROSCI.1372-04.2005. PMC:6725954. PMID:15689554.
"Schwann cell properties: 3. C-fos expression, bFGF production, phagocytosis and proliferation during Wallerian degeneration". Journal of Neuropathology and Experimental Neurology. ج. 54 ع. 4: 487–96. يوليو 1995. DOI:10.1097/00005072-199507000-00002. PMID:7602323.
"Does oligodendrocyte survival depend on axons?". Current Biology. ج. 3 ع. 8: 489–97. أغسطس 1993. DOI:10.1016/0960-9822(93)90039-Q. PMID:15335686.
"Oligodendrocyte survival in Wallerian degeneration". Acta Neuropathologica. ج. 80 ع. 2: 184–91. 31 مايو 1990. DOI:10.1007/BF00308922. PMID:1697140.
"The temporal and spatial activation of microglia in fiber tracts undergoing anterograde and retrograde degeneration following spinal cord lesion". Journal of Neurotrauma. ج. 12 ع. 2: 209–22. أبريل 1995. DOI:10.1089/neu.1995.12.209. PMID:7629867.
"Delayed macrophage responses and myelin clearance during Wallerian degeneration in the central nervous system: the dorsal radiculotomy model". Experimental Neurology. ج. 129 ع. 2: 225–36. أكتوبر 1994. DOI:10.1006/exnr.1994.1164. PMID:7957737.
"The Nogo signaling pathway for regeneration block". Annual Review of Neuroscience. ج. 27 ع. 1: 341–68. 21 يوليو 2004. DOI:10.1146/annurev.neuro.27.070203.144340. PMID:15217336.
"The early stages of Wallerian degeneration in the severed optic nerve of the newt (Triturus viridescens)". The Anatomical Record. ج. 187 ع. 3: 291–310. مارس 1977. DOI:10.1002/ar.1091870303. PMID:851236.
"Changes of nerve growth factor synthesis in nonneuronal cells in response to sciatic nerve transection" (PDF). The Journal of Cell Biology. ج. 104 ع. 6: 1623–31. يونيو 1987. DOI:10.1083/jcb.104.6.1623. PMC:2114490. PMID:3034917. مؤرشف من الأصل (PDF) في 2017-08-14.
"Interleukin 1 increases stability and transcription of mRNA encoding nerve growth factor in cultured rat fibroblasts". The Journal of Biological Chemistry. ج. 263 ع. 31: 16348–51. نوفمبر 1988. PMID:3263368.
"The degeneration of unmyelinated axons following nerve section: an ultrastructural study". Journal of Neurocytology. ج. 3 ع. 4: 497–512. أكتوبر 1974. DOI:10.1007/BF01098736. PMID:4436692.
"Ninjurin, a novel adhesion molecule, is induced by nerve injury and promotes axonal growth". Neuron. ج. 17 ع. 2: 353–61. أغسطس 1996. DOI:10.1016/S0896-6273(00)80166-X. PMID:8780658.
"The Effectiveness of the Gene Which Slows the Rate of Wallerian Degeneration in C57BL/Ola Mice Declines With Age". The European Journal of Neuroscience. ج. 4 ع. 10: 1000–2. 1 أكتوبر 1992. DOI:10.1111/j.1460-9568.1992.tb00126.x. PMID:12106435.
"A gene affecting Wallerian nerve degeneration maps distally on mouse chromosome 4". Proceedings of the National Academy of Sciences of the United States of America. ج. 90 ع. 20: 9717–20. أكتوبر 1993. Bibcode:1993PNAS...90.9717L. DOI:10.1073/pnas.90.20.9717. PMC:47641. PMID:8415768.
"Increased nuclear NAD biosynthesis and SIRT1 activation prevent axonal degeneration". Science. ج. 305 ع. 5686: 1010–3. أغسطس 2004. Bibcode:2004Sci...305.1010A. DOI:10.1126/science.1098014. PMID:15310905.
"Wallerian degeneration of injured axons and synapses is delayed by a Ube4b/Nmnat chimeric gene". Nature Neuroscience. ج. 4 ع. 12: 1199–206. ديسمبر 2001. DOI:10.1038/nn770. PMID:11770485.
"Non-nuclear Wld(S) determines its neuroprotective efficacy for axons and synapses in vivo". The Journal of Neuroscience. ج. 29 ع. 3: 653–68. يناير 2009. DOI:10.1523/JNEUROSCI.3814-08.2009. PMC:6665162. PMID:19158292.
"Prolonged survival of transected nerve fibres in C57BL/Ola mice is an intrinsic characteristic of the axon". Journal of Neurocytology. ج. 22 ع. 5: 311–21. مايو 1993. DOI:10.1007/BF01195555. PMID:8315413.
"The slow Wallerian degeneration gene in vivo protects motor axons but not their cell bodies after avulsion and neonatal axotomy". The European Journal of Neuroscience. ج. 24 ع. 8: 2163–8. أكتوبر 2006. DOI:10.1111/j.1460-9568.2006.05103.x. PMID:17074042.
"A local mechanism mediates NAD-dependent protection of axon degeneration". The Journal of Cell Biology. ج. 170 ع. 3: 349–55. أغسطس 2005. DOI:10.1083/jcb.200504028. PMC:2171458. PMID:16043516.
"NAD(+) and axon degeneration revisited: Nmnat1 cannot substitute for Wld(S) to delay Wallerian degeneration". Cell Death and Differentiation. ج. 14 ع. 1: 116–27. يناير 2007. DOI:10.1038/sj.cdd.4401944. PMID:16645633.
"Targeting NMNAT1 to axons and synapses transforms its neuroprotective potency in vivo". The Journal of Neuroscience. ج. 30 ع. 40: 13291–304. أكتوبر 2010. DOI:10.1523/JNEUROSCI.1189-10.2010. PMC:6634738. PMID:20926655.
"Genetic influences on cellular reactions to spinal cord injury: activation of macrophages/microglia and astrocytes is delayed in mice carrying a mutation (WldS) that causes delayed Wallerian degeneration". The Journal of Comparative Neurology. ج. 371 ع. 3: 469–84. يوليو 1996. DOI:10.1002/(SICI)1096-9861(19960729)371:3<469::AID-CNE9>3.0.CO;2-0. PMID:8842900.
"Further studies on motor and sensory nerve regeneration in mice with delayed Wallerian degeneration". The European Journal of Neuroscience. ج. 6 ع. 3: 420–8. مارس 1994. DOI:10.1111/j.1460-9568.1994.tb00285.x. PMID:8019679.
"dSarm/Sarm1 is required for activation of an injury-induced axon death pathway". Science. ج. 337 ع. 6093: 481–4. يوليو 2012. Bibcode:2012Sci...337..481O. DOI:10.1126/science.1223899. PMC:5225956. PMID:22678360.
"Sarm1-mediated axon degeneration requires both SAM and TIR interactions". The Journal of Neuroscience. ج. 33 ع. 33: 13569–80. أغسطس 2013. DOI:10.1523/JNEUROSCI.1197-13.2013. PMC:3742939. PMID:23946415.
"Resolving the topological enigma in Ca 2+ signaling by cyclic ADP-ribose and NAADP". Journal of Biological Chemistry. ج. 294 ع. 52: 19831–19843. 2019. DOI:10.1074/jbc.REV119.009635. PMC:6937575. PMID:31672920. مؤرشف من الأصل في 2020-10-20.{{استشهاد بدورية محكمة}}: صيانة الاستشهاد: دوي مجاني غير معلم (link)
"SARM1 activation triggers axon degeneration locally via NAD⁺ destruction". Science. ج. 348 ع. 6233: 453–7. أبريل 2015. Bibcode:2015Sci...348..453G. DOI:10.1126/science.1258366. PMC:4513950. PMID:25908823.
"+ Cleavage Activity that Promotes Pathological Axonal Degeneration". Neuron. ج. 93 ع. 6: 1334–1343.e5. مارس 2017. DOI:10.1016/j.neuron.2017.02.022. PMC:6284238. PMID:28334607.
"+ depletion". eLife. ج. 5. أكتوبر 2016. DOI:10.7554/eLife.19749. PMC:5063586. PMID:27735788.{{استشهاد بدورية محكمة}}: صيانة الاستشهاد: دوي مجاني غير معلم (link)
"S, Confers Lifelong Rescue in a Mouse Model of Severe Axonopathy". Cell Reports. ج. 21 ع. 1: 10–16. أكتوبر 2017. DOI:10.1016/j.celrep.2017.09.027. PMC:5640801. PMID:28978465.
"Pathological axonal death through a MAPK cascade that triggers a local energy deficit". Cell. ج. 160 ع. 1–2: 161–76. يناير 2015. DOI:10.1016/j.cell.2014.11.053. PMC:4306654. PMID:25594179.
"MAPK signaling promotes axonal degeneration by speeding the turnover of the axonal maintenance factor NMNAT2". eLife. ج. 6. يناير 2017. DOI:10.7554/eLife.22540. PMC:5241118. PMID:28095293.{{استشهاد بدورية محكمة}}: صيانة الاستشهاد: دوي مجاني غير معلم (link)
"Attenuated traumatic axonal injury and improved functional outcome after traumatic brain injury in mice lacking Sarm1". Brain. ج. 139 ع. 4: 1094–1105. 2016. DOI:10.1093/brain/aww001. PMC:5006226. PMID:26912636.

ncbi.nlm.nih.gov

"Wallerian degeneration, wld(s), and nmnat". Annual Review of Neuroscience. ج. 33 ع. 1: 245–67. 1 يونيو 2010. DOI:10.1146/annurev-neuro-060909-153248. PMC:5223592. PMID:20345246.
"Endogenous Nmnat2 is an essential survival factor for maintenance of healthy axons". PLOS Biology. ج. 8 ع. 1: e1000300. يناير 2010. DOI:10.1371/journal.pbio.1000300. PMC:2811159. PMID:20126265.{{استشهاد بدورية محكمة}}: صيانة الاستشهاد: دوي مجاني غير معلم (link)
"NMNAT: It's an NAD + Synthase… It's a Chaperone… It's a Neuroprotector". Current Opinion in Genetics & Development. ج. 44: 156–162. 2017. DOI:10.1016/j.gde.2017.03.014. PMC:5515290. PMID:28445802.
"An 85-kb tandem triplication in the slow Wallerian degeneration (Wlds) mouse". Proceedings of the National Academy of Sciences of the United States of America. ج. 95 ع. 17: 9985–90. أغسطس 1998. Bibcode:1998PNAS...95.9985C. DOI:10.1073/pnas.95.17.9985. PMC:21448. PMID:9707587.
"Endocytotic formation of vesicles and other membranous structures induced by Ca2+ and axolemmal injury". The Journal of Neuroscience. ج. 18 ع. 11: 4029–41. يونيو 1998. DOI:10.1523/JNEUROSCI.18-11-04029.1998. PMC:6792792. PMID:9592084.
"Axon degeneration: molecular mechanisms of a self-destruction pathway". The Journal of Cell Biology. ج. 196 ع. 1: 7–18. يناير 2012. DOI:10.1083/jcb.201108111. PMC:3255986. PMID:22232700.
"Microanatomy of axon/glial signaling during Wallerian degeneration". The Journal of Neuroscience. ج. 25 ع. 13: 3478–87. مارس 2005. DOI:10.1523/JNEUROSCI.3766-04.2005. PMC:6724908. PMID:15800203.
"Complement depletion reduces macrophage infiltration and ctivation during Wallerian degeneration and axonal regeneration". The Journal of Neuroscience. ج. 18 ع. 17: 6713–22. سبتمبر 1998. DOI:10.1523/JNEUROSCI.18-17-06713.1998. PMC:6792968. PMID:9712643.
"Degeneration of myelinated efferent fibers prompts mitosis in Remak Schwann cells of uninjured C-fiber afferents". The Journal of Neuroscience. ج. 25 ع. 5: 1179–87. فبراير 2005. DOI:10.1523/JNEUROSCI.1372-04.2005. PMC:6725954. PMID:15689554.
"Changes of nerve growth factor synthesis in nonneuronal cells in response to sciatic nerve transection" (PDF). The Journal of Cell Biology. ج. 104 ع. 6: 1623–31. يونيو 1987. DOI:10.1083/jcb.104.6.1623. PMC:2114490. PMID:3034917. مؤرشف من الأصل (PDF) في 2017-08-14.
"A gene affecting Wallerian nerve degeneration maps distally on mouse chromosome 4". Proceedings of the National Academy of Sciences of the United States of America. ج. 90 ع. 20: 9717–20. أكتوبر 1993. Bibcode:1993PNAS...90.9717L. DOI:10.1073/pnas.90.20.9717. PMC:47641. PMID:8415768.
"Non-nuclear Wld(S) determines its neuroprotective efficacy for axons and synapses in vivo". The Journal of Neuroscience. ج. 29 ع. 3: 653–68. يناير 2009. DOI:10.1523/JNEUROSCI.3814-08.2009. PMC:6665162. PMID:19158292.
"A local mechanism mediates NAD-dependent protection of axon degeneration". The Journal of Cell Biology. ج. 170 ع. 3: 349–55. أغسطس 2005. DOI:10.1083/jcb.200504028. PMC:2171458. PMID:16043516.
"Targeting NMNAT1 to axons and synapses transforms its neuroprotective potency in vivo". The Journal of Neuroscience. ج. 30 ع. 40: 13291–304. أكتوبر 2010. DOI:10.1523/JNEUROSCI.1189-10.2010. PMC:6634738. PMID:20926655.
"dSarm/Sarm1 is required for activation of an injury-induced axon death pathway". Science. ج. 337 ع. 6093: 481–4. يوليو 2012. Bibcode:2012Sci...337..481O. DOI:10.1126/science.1223899. PMC:5225956. PMID:22678360.
"Sarm1-mediated axon degeneration requires both SAM and TIR interactions". The Journal of Neuroscience. ج. 33 ع. 33: 13569–80. أغسطس 2013. DOI:10.1523/JNEUROSCI.1197-13.2013. PMC:3742939. PMID:23946415.
"Resolving the topological enigma in Ca 2+ signaling by cyclic ADP-ribose and NAADP". Journal of Biological Chemistry. ج. 294 ع. 52: 19831–19843. 2019. DOI:10.1074/jbc.REV119.009635. PMC:6937575. PMID:31672920. مؤرشف من الأصل في 2020-10-20.{{استشهاد بدورية محكمة}}: صيانة الاستشهاد: دوي مجاني غير معلم (link)
"SARM1 activation triggers axon degeneration locally via NAD⁺ destruction". Science. ج. 348 ع. 6233: 453–7. أبريل 2015. Bibcode:2015Sci...348..453G. DOI:10.1126/science.1258366. PMC:4513950. PMID:25908823.
"+ Cleavage Activity that Promotes Pathological Axonal Degeneration". Neuron. ج. 93 ع. 6: 1334–1343.e5. مارس 2017. DOI:10.1016/j.neuron.2017.02.022. PMC:6284238. PMID:28334607.
"+ depletion". eLife. ج. 5. أكتوبر 2016. DOI:10.7554/eLife.19749. PMC:5063586. PMID:27735788.{{استشهاد بدورية محكمة}}: صيانة الاستشهاد: دوي مجاني غير معلم (link)
"S, Confers Lifelong Rescue in a Mouse Model of Severe Axonopathy". Cell Reports. ج. 21 ع. 1: 10–16. أكتوبر 2017. DOI:10.1016/j.celrep.2017.09.027. PMC:5640801. PMID:28978465.
"Pathological axonal death through a MAPK cascade that triggers a local energy deficit". Cell. ج. 160 ع. 1–2: 161–76. يناير 2015. DOI:10.1016/j.cell.2014.11.053. PMC:4306654. PMID:25594179.
"MAPK signaling promotes axonal degeneration by speeding the turnover of the axonal maintenance factor NMNAT2". eLife. ج. 6. يناير 2017. DOI:10.7554/eLife.22540. PMC:5241118. PMID:28095293.{{استشهاد بدورية محكمة}}: صيانة الاستشهاد: دوي مجاني غير معلم (link)
"Attenuated traumatic axonal injury and improved functional outcome after traumatic brain injury in mice lacking Sarm1". Brain. ج. 139 ع. 4: 1094–1105. 2016. DOI:10.1093/brain/aww001. PMC:5006226. PMID:26912636.

ucsf.edu

missinglink.ucsf.edu

Trauma and Wallerian Degeneration, جامعة كاليفورنيا (سان فرانسيسكو) نسخة محفوظة 2 مايو 2006 على موقع واي باك مشين.

web.archive.org

Trauma and Wallerian Degeneration, جامعة كاليفورنيا (سان فرانسيسكو) نسخة محفوظة 2 مايو 2006 على موقع واي باك مشين.
"Changes of nerve growth factor synthesis in nonneuronal cells in response to sciatic nerve transection" (PDF). The Journal of Cell Biology. ج. 104 ع. 6: 1623–31. يونيو 1987. DOI:10.1083/jcb.104.6.1623. PMC:2114490. PMID:3034917. مؤرشف من الأصل (PDF) في 2017-08-14.
Perry, V. H., Lunn, E. R., Brown, M. C., Cahusac, S. and Gordon, S. (1990), Evidence that the Rate of Wallerian Degeneration is Controlled by a Single Autosomal Dominant Gene. European Journal of Neuroscience, 2: 408-413. https://doi.org/10.1111/j.1460-9568.1990.tb00433.x نسخة محفوظة 4 يونيو 2018 على موقع واي باك مشين.
"Resolving the topological enigma in Ca 2+ signaling by cyclic ADP-ribose and NAADP". Journal of Biological Chemistry. ج. 294 ع. 52: 19831–19843. 2019. DOI:10.1074/jbc.REV119.009635. PMC:6937575. PMID:31672920. مؤرشف من الأصل في 2020-10-20.{{استشهاد بدورية محكمة}}: صيانة الاستشهاد: دوي مجاني غير معلم (link)