ستات3 (Arabic Wikipedia)

Analysis of information sources in references of the Wikipedia article "ستات3" in Arabic language version.

refsWebsite
Global rank Arabic rank
2nd place
5th place
4th place
6th place
6th place
3rd place

archive.org (Global: 6th place; Arabic: 3rd place)

doi.org (Global: 2nd place; Arabic: 5th place)

  • "Molecular cloning of APRF, a novel IFN-stimulated gene factor 3 p91-related transcription factor involved in the gp130-mediated signaling pathway". Cell. ج. 77 ع. 1: 63–71. أبريل 1994. DOI:10.1016/0092-8674(94)90235-6. PMID:7512451.
  • "p42/p44 MAPK-mediated Stat3Ser727 phosphorylation is required for progestin-induced full activation of Stat3 and breast cancer growth". Endocrine-Related Cancer. ج. 20 ع. 2: 197–212. أبريل 2013. DOI:10.1530/ERC-12-0194. PMID:23329648.
  • "Central role of the threonine residue within the p+1 loop of receptor tyrosine kinase in STAT3 constitutive phosphorylation in metastatic cancer cells". Molecular and Cellular Biology. ج. 24 ع. 21: 9390–400. نوفمبر 2004. DOI:10.1128/MCB.24.21.9390-9400.2004. PMID:15485908. 15485908.
  • "Targeted disruption of the mouse Stat3 gene leads to early embryonic lethality". Proceedings of the National Academy of Sciences of the United States of America. ج. 94 ع. 8: 3801–4. أبريل 1997. DOI:10.1073/pnas.94.8.3801. PMID:9108058.
  • "STAT3 regulates cytokine-mediated generation of inflammatory helper T cells". The Journal of Biological Chemistry. ج. 282 ع. 13: 9358–63. مارس 2007. DOI:10.1074/jbc.C600321200. PMID:17277312.{{استشهاد بدورية محكمة}}: صيانة الاستشهاد: دوي مجاني غير معلم (link)
  • "T-cell STAT3 is required for the maintenance of humoral immunity to LCMV". European Journal of Immunology. ج. 45 ع. 2: 418–27. فبراير 2015. DOI:10.1002/eji.201445060. PMID:25393615.
  • "STAT3 signaling and the hyper-IgE syndrome". The New England Journal of Medicine. ج. 357 ع. 16: 1655–8. أكتوبر 2007. DOI:10.1056/NEJMe078197. PMID:17881746.
  • "Identification of a PTEN-regulated STAT3 brain tumor suppressor pathway". Genes & Development. ج. 22 ع. 4: 449–62. فبراير 2008. DOI:10.1101/gad.1606508. PMID:18258752.
  • Vlahopoulos، SA (أغسطس 2017). "Aberrant control of NF-κB in cancer permits transcriptional and phenotypic plasticity, to curtail dependence on host tissue: molecular mode". Cancer biology & medicine. ج. 14: 254–270. DOI:10.20892/j.issn.2095-3941.2017.0029. PMID:28884042.

nih.gov (Global: 4th place; Arabic: 6th place)

pubmed.ncbi.nlm.nih.gov

  • "Molecular cloning of APRF, a novel IFN-stimulated gene factor 3 p91-related transcription factor involved in the gp130-mediated signaling pathway". Cell. ج. 77 ع. 1: 63–71. أبريل 1994. DOI:10.1016/0092-8674(94)90235-6. PMID:7512451.
  • "p42/p44 MAPK-mediated Stat3Ser727 phosphorylation is required for progestin-induced full activation of Stat3 and breast cancer growth". Endocrine-Related Cancer. ج. 20 ع. 2: 197–212. أبريل 2013. DOI:10.1530/ERC-12-0194. PMID:23329648.
  • "Central role of the threonine residue within the p+1 loop of receptor tyrosine kinase in STAT3 constitutive phosphorylation in metastatic cancer cells". Molecular and Cellular Biology. ج. 24 ع. 21: 9390–400. نوفمبر 2004. DOI:10.1128/MCB.24.21.9390-9400.2004. PMID:15485908. 15485908.
  • "Targeted disruption of the mouse Stat3 gene leads to early embryonic lethality". Proceedings of the National Academy of Sciences of the United States of America. ج. 94 ع. 8: 3801–4. أبريل 1997. DOI:10.1073/pnas.94.8.3801. PMID:9108058.
  • "STAT3 regulates cytokine-mediated generation of inflammatory helper T cells". The Journal of Biological Chemistry. ج. 282 ع. 13: 9358–63. مارس 2007. DOI:10.1074/jbc.C600321200. PMID:17277312.{{استشهاد بدورية محكمة}}: صيانة الاستشهاد: دوي مجاني غير معلم (link)
  • "T-cell STAT3 is required for the maintenance of humoral immunity to LCMV". European Journal of Immunology. ج. 45 ع. 2: 418–27. فبراير 2015. DOI:10.1002/eji.201445060. PMID:25393615.
  • "STAT3 signaling and the hyper-IgE syndrome". The New England Journal of Medicine. ج. 357 ع. 16: 1655–8. أكتوبر 2007. DOI:10.1056/NEJMe078197. PMID:17881746.
  • "Identification of a PTEN-regulated STAT3 brain tumor suppressor pathway". Genes & Development. ج. 22 ع. 4: 449–62. فبراير 2008. DOI:10.1101/gad.1606508. PMID:18258752.
  • Vlahopoulos، SA (أغسطس 2017). "Aberrant control of NF-κB in cancer permits transcriptional and phenotypic plasticity, to curtail dependence on host tissue: molecular mode". Cancer biology & medicine. ج. 14: 254–270. DOI:10.20892/j.issn.2095-3941.2017.0029. PMID:28884042.