علاج الشيخوخة (Arabic Wikipedia)

Analysis of information sources in references of the Wikipedia article "علاج الشيخوخة" in Arabic language version.

refsWebsite

Global rank Arabic rank

31nih.gov

4^th place

6^th place

17doi.org

2^nd place

5^th place

1web.archive.org

1^st place

1embopress.org

low place

1issn.org

57^th place

1,629^th place

doi.org (Global: 2^nd place; Arabic: 5^th place)

"Cellular senescence in aging and age-related disease: from mechanisms to therapy". Nature Medicine. ج. 21 ع. 12: 1424–35. 2015. DOI:10.1038/nm.4000. PMC:4748967. PMID:26646499.
Misra، Juhi؛ Mohanty، Sindhu T.؛ Madan، Sanjeev؛ Fernandes، James A.؛ Hal Ebetino، F.؛ Russell، R. Graham G.؛ Bellantuono، Ilaria (2015). "Zoledronate Attenuates Accumulation of DNA Damage in Mesenchymal Stem Cells and Protects Their Function". Stem Cells. ج. 34 ع. 3: 756–767. DOI:10.1002/stem.2255. PMC:4832316. PMID:26679354.
Xiong، Shiqin؛ Patrushev، Nikolay؛ Forouzandeh، Farshad؛ Hilenski، Lula؛ Alexander، R. Wayne (2015). "PGC-1α Modulates Telomere Function and DNA Damage in Protecting against Aging-Related Chronic Diseases". Cell Reports. ج. 12 ع. 9: 1391–9. DOI:10.1016/j.celrep.2015.07.047. PMC:4549794. PMID:26299964.
Eisenberg، Tobias؛ Knauer، Heide؛ Schauer، Alexandra؛ Büttner، Sabrina؛ Ruckenstuhl، Christoph؛ Carmona-Gutierrez، Didac؛ Ring، Julia؛ Schroeder، Sabrina؛ Magnes، Christoph (2009). "Induction of autophagy by spermidine promotes longevity". Nature Cell Biology. ج. 11 ع. 11: 1305–14. DOI:10.1038/ncb1975. PMID:19801973.
Pride، Harrison؛ Yu، Zhen؛ Sunchu، Bharath؛ Mochnick، Jillian؛ Coles، Alexander؛ Zhang، Yiqiang؛ Buffenstein، Rochelle؛ Hornsby، Peter J.؛ Austad، Steven N. (2015). "Long-lived species have improved proteostasis compared to phylogenetically-related shorter-lived species". Biochemical and Biophysical Research Communications. ج. 457 ع. 4: 669–75. DOI:10.1016/j.bbrc.2015.01.046. PMID:25615820.
Blackburn، E. H.؛ Epel، E. S.؛ Lin، J. (2015). "Human telomere biology: A contributory and interactive factor in aging, disease risks, and protection". Science. ج. 350 ع. 6265: 1193–8. DOI:10.1126/science.aab3389. PMID:26785477.
Byun، H. O.؛ Lee، Y. K.؛ Kim، J. M.؛ Yoon، G (2015). "From cell senescence to age-related diseases: Differential mechanisms of action of senescence-associated secretory phenotypes". BMB Reports. ج. 48 ع. 10: 549–58. DOI:10.5483/bmbrep.2015.48.10.122. PMC:4911181. PMID:26129674.
Young، Andrew R J؛ Narita، Masashi (2009). "SASP reflects senescence". EMBO Reports. ج. 10 ع. 3: 228–30. DOI:10.1038/embor.2009.22. PMC:2658552. PMID:19218920.
Laberge، Remi-Martin؛ Zhou، Lili؛ Sarantos، Melissa R.؛ Rodier، Francis؛ Freund، Adam؛ De Keizer، Peter L. J.؛ Liu، Su؛ Demaria، Marco؛ Cong، Yu-Sheng (2012). "Glucocorticoids suppress selected components of the senescence-associated secretory phenotype". Aging Cell. ج. 11 ع. 4: 569–78. DOI:10.1111/j.1474-9726.2012.00818.x. PMC:3387333. PMID:22404905.
Liu، Su؛ Uppal، Harpreet؛ Demaria، Marco؛ Desprez، Pierre-Yves؛ Campisi، Judith؛ Kapahi، Pankaj (2015). "Simvastatin suppresses breast cancer cell proliferation induced by senescent cells". Scientific Reports. ج. 5: 17895. DOI:10.1038/srep17895. PMC:4677323. PMID:26658759.
Xu، Ming؛ Tchkonia، Tamara؛ Ding، Husheng؛ Ogrodnik، Mikolaj؛ Lubbers، Ellen R.؛ Pirtskhalava، Tamar؛ White، Thomas A.؛ Johnson، Kurt O.؛ Stout، Michael B. (2015). "JAK inhibition alleviates the cellular senescence-associated secretory phenotype and frailty in old age". Proceedings of the National Academy of Sciences. ج. 112 ع. 46: E6301–10. DOI:10.1073/pnas.1515386112. PMC:4655580. PMID:26578790.
Xu، Ming؛ Palmer، Allyson K؛ Ding، Husheng؛ Weivoda، Megan M؛ Pirtskhalava، Tamar؛ White، Thomas A؛ Sepe، Anna؛ Johnson، Kurt O؛ Stout، Michael B (2015). "Targeting senescent cells enhances adipogenesis and metabolic function in old age". eLife. ج. 4: e12997. DOI:10.7554/eLife.12997. PMC:4758946. PMID:26687007.{{استشهاد بدورية محكمة}}: صيانة الاستشهاد: دوي مجاني غير معلم (link)
"Mitochondria are required for pro-ageing features of the senescent phenotype". The EMBO Journal. ج. 35 ع. 7: 724–42. 2016. DOI:10.15252/embj.201592862. PMC:4818766. PMID:26848154. مؤرشف من الأصل في 2019-06-06. اطلع عليه بتاريخ 2016-02-06. 60% of the SASP genes which are significantly different between proliferating and senescent were reversed upon mitochondrial depletion, whereas only 5% were exacerbated
Zhu، Yi؛ Tchkonia، T؛ Fuhrmann-Stroissnigg، H؛ Dai، HM؛ Ling، YY؛ Stout، MB؛ Pirtskhalava، T؛ Giorgadze، N؛ Johnson، KO (2015). "Identification of a Novel Senolytic Agent, Navitoclax, Targeting the Bcl-2 Family of Anti-Apoptotic Factors". Aging Cell. ج. 15 ع. 3: 428–35. DOI:10.1111/acel.12445. PMC:4854923. PMID:26711051.
Zhu، Yi؛ Tchkonia، Tamara؛ Pirtskhalava، Tamar؛ Gower، Adam؛ Ding، Husheng؛ Giorgadze، Nino؛ Palmer، Allyson K.؛ Ikeno، Yuji؛ Borden، Gene (2015). "The Achilles' Heel of Senescent Cells: From Transcriptome to Senolytic Drugs". Aging Cell. ج. 14 ع. 4: 644–58. DOI:10.1111/acel.12344. PMC:4531078. PMID:25754370.
Fuhrmann-Stroissnigg, Heike; Ling, Yuan Yuan; Zhao, Jing; McGowan, Sara J.; Zhu, Yi; Brooks, Robert W.; Grassi, Diego; Gregg, Siobhan Q.; Stripay, Jennifer L. (4 Sep 2017). "Identification of HSP90 inhibitors as a novel class of senolytics". Nature Communications (بالإنجليزية). 8 (1): 422. DOI:10.1038/s41467-017-00314-z. ISSN:2041-1723. PMC:5583353. PMID:28871086.
Hofmann، Jeffrey W.؛ Zhao، Xiaoai؛ De Cecco، Marco؛ Peterson، Abigail L.؛ Pagliaroli، Luca؛ Manivannan، Jayameenakshi؛ Hubbard، Gene B.؛ Ikeno، Yuji؛ Zhang، Yongqing (2015). "Reduced Expression of MYC Increases Longevity and Enhances Healthspan". Cell. ج. 160 ع. 3: 477–88. DOI:10.1016/j.cell.2014.12.016. PMC:4624921. PMID:25619689.

embopress.org (Global: low place; Arabic: low place)

emboj.embopress.org

"Mitochondria are required for pro-ageing features of the senescent phenotype". The EMBO Journal. ج. 35 ع. 7: 724–42. 2016. DOI:10.15252/embj.201592862. PMC:4818766. PMID:26848154. مؤرشف من الأصل في 2019-06-06. اطلع عليه بتاريخ 2016-02-06. 60% of the SASP genes which are significantly different between proliferating and senescent were reversed upon mitochondrial depletion, whereas only 5% were exacerbated

issn.org (Global: 57^th place; Arabic: 1,629^th place)

portal.issn.org

Fuhrmann-Stroissnigg, Heike; Ling, Yuan Yuan; Zhao, Jing; McGowan, Sara J.; Zhu, Yi; Brooks, Robert W.; Grassi, Diego; Gregg, Siobhan Q.; Stripay, Jennifer L. (4 Sep 2017). "Identification of HSP90 inhibitors as a novel class of senolytics". Nature Communications (بالإنجليزية). 8 (1): 422. DOI:10.1038/s41467-017-00314-z. ISSN:2041-1723. PMC:5583353. PMID:28871086.

nih.gov (Global: 4^th place; Arabic: 6^th place)

pubmed.ncbi.nlm.nih.gov

"Cellular senescence in aging and age-related disease: from mechanisms to therapy". Nature Medicine. ج. 21 ع. 12: 1424–35. 2015. DOI:10.1038/nm.4000. PMC:4748967. PMID:26646499.
Misra، Juhi؛ Mohanty، Sindhu T.؛ Madan، Sanjeev؛ Fernandes، James A.؛ Hal Ebetino، F.؛ Russell، R. Graham G.؛ Bellantuono، Ilaria (2015). "Zoledronate Attenuates Accumulation of DNA Damage in Mesenchymal Stem Cells and Protects Their Function". Stem Cells. ج. 34 ع. 3: 756–767. DOI:10.1002/stem.2255. PMC:4832316. PMID:26679354.
Xiong، Shiqin؛ Patrushev، Nikolay؛ Forouzandeh، Farshad؛ Hilenski، Lula؛ Alexander، R. Wayne (2015). "PGC-1α Modulates Telomere Function and DNA Damage in Protecting against Aging-Related Chronic Diseases". Cell Reports. ج. 12 ع. 9: 1391–9. DOI:10.1016/j.celrep.2015.07.047. PMC:4549794. PMID:26299964.
Eisenberg، Tobias؛ Knauer، Heide؛ Schauer، Alexandra؛ Büttner، Sabrina؛ Ruckenstuhl، Christoph؛ Carmona-Gutierrez، Didac؛ Ring، Julia؛ Schroeder، Sabrina؛ Magnes، Christoph (2009). "Induction of autophagy by spermidine promotes longevity". Nature Cell Biology. ج. 11 ع. 11: 1305–14. DOI:10.1038/ncb1975. PMID:19801973.
Pride، Harrison؛ Yu، Zhen؛ Sunchu، Bharath؛ Mochnick، Jillian؛ Coles، Alexander؛ Zhang، Yiqiang؛ Buffenstein، Rochelle؛ Hornsby، Peter J.؛ Austad، Steven N. (2015). "Long-lived species have improved proteostasis compared to phylogenetically-related shorter-lived species". Biochemical and Biophysical Research Communications. ج. 457 ع. 4: 669–75. DOI:10.1016/j.bbrc.2015.01.046. PMID:25615820.
Blackburn، E. H.؛ Epel، E. S.؛ Lin، J. (2015). "Human telomere biology: A contributory and interactive factor in aging, disease risks, and protection". Science. ج. 350 ع. 6265: 1193–8. DOI:10.1126/science.aab3389. PMID:26785477.
Byun، H. O.؛ Lee، Y. K.؛ Kim، J. M.؛ Yoon، G (2015). "From cell senescence to age-related diseases: Differential mechanisms of action of senescence-associated secretory phenotypes". BMB Reports. ج. 48 ع. 10: 549–58. DOI:10.5483/bmbrep.2015.48.10.122. PMC:4911181. PMID:26129674.
Young، Andrew R J؛ Narita، Masashi (2009). "SASP reflects senescence". EMBO Reports. ج. 10 ع. 3: 228–30. DOI:10.1038/embor.2009.22. PMC:2658552. PMID:19218920.
Laberge، Remi-Martin؛ Zhou، Lili؛ Sarantos، Melissa R.؛ Rodier، Francis؛ Freund، Adam؛ De Keizer، Peter L. J.؛ Liu، Su؛ Demaria، Marco؛ Cong، Yu-Sheng (2012). "Glucocorticoids suppress selected components of the senescence-associated secretory phenotype". Aging Cell. ج. 11 ع. 4: 569–78. DOI:10.1111/j.1474-9726.2012.00818.x. PMC:3387333. PMID:22404905.
Liu، Su؛ Uppal، Harpreet؛ Demaria، Marco؛ Desprez، Pierre-Yves؛ Campisi، Judith؛ Kapahi، Pankaj (2015). "Simvastatin suppresses breast cancer cell proliferation induced by senescent cells". Scientific Reports. ج. 5: 17895. DOI:10.1038/srep17895. PMC:4677323. PMID:26658759.
Xu، Ming؛ Tchkonia، Tamara؛ Ding، Husheng؛ Ogrodnik، Mikolaj؛ Lubbers، Ellen R.؛ Pirtskhalava، Tamar؛ White، Thomas A.؛ Johnson، Kurt O.؛ Stout، Michael B. (2015). "JAK inhibition alleviates the cellular senescence-associated secretory phenotype and frailty in old age". Proceedings of the National Academy of Sciences. ج. 112 ع. 46: E6301–10. DOI:10.1073/pnas.1515386112. PMC:4655580. PMID:26578790.
Xu، Ming؛ Palmer، Allyson K؛ Ding، Husheng؛ Weivoda، Megan M؛ Pirtskhalava، Tamar؛ White، Thomas A؛ Sepe، Anna؛ Johnson، Kurt O؛ Stout، Michael B (2015). "Targeting senescent cells enhances adipogenesis and metabolic function in old age". eLife. ج. 4: e12997. DOI:10.7554/eLife.12997. PMC:4758946. PMID:26687007.{{استشهاد بدورية محكمة}}: صيانة الاستشهاد: دوي مجاني غير معلم (link)
"Mitochondria are required for pro-ageing features of the senescent phenotype". The EMBO Journal. ج. 35 ع. 7: 724–42. 2016. DOI:10.15252/embj.201592862. PMC:4818766. PMID:26848154. مؤرشف من الأصل في 2019-06-06. اطلع عليه بتاريخ 2016-02-06. 60% of the SASP genes which are significantly different between proliferating and senescent were reversed upon mitochondrial depletion, whereas only 5% were exacerbated
Zhu، Yi؛ Tchkonia، T؛ Fuhrmann-Stroissnigg، H؛ Dai، HM؛ Ling، YY؛ Stout، MB؛ Pirtskhalava، T؛ Giorgadze، N؛ Johnson، KO (2015). "Identification of a Novel Senolytic Agent, Navitoclax, Targeting the Bcl-2 Family of Anti-Apoptotic Factors". Aging Cell. ج. 15 ع. 3: 428–35. DOI:10.1111/acel.12445. PMC:4854923. PMID:26711051.
Zhu، Yi؛ Tchkonia، Tamara؛ Pirtskhalava، Tamar؛ Gower، Adam؛ Ding، Husheng؛ Giorgadze، Nino؛ Palmer، Allyson K.؛ Ikeno، Yuji؛ Borden، Gene (2015). "The Achilles' Heel of Senescent Cells: From Transcriptome to Senolytic Drugs". Aging Cell. ج. 14 ع. 4: 644–58. DOI:10.1111/acel.12344. PMC:4531078. PMID:25754370.
Fuhrmann-Stroissnigg, Heike; Ling, Yuan Yuan; Zhao, Jing; McGowan, Sara J.; Zhu, Yi; Brooks, Robert W.; Grassi, Diego; Gregg, Siobhan Q.; Stripay, Jennifer L. (4 Sep 2017). "Identification of HSP90 inhibitors as a novel class of senolytics". Nature Communications (بالإنجليزية). 8 (1): 422. DOI:10.1038/s41467-017-00314-z. ISSN:2041-1723. PMC:5583353. PMID:28871086.
Hofmann، Jeffrey W.؛ Zhao، Xiaoai؛ De Cecco، Marco؛ Peterson، Abigail L.؛ Pagliaroli، Luca؛ Manivannan، Jayameenakshi؛ Hubbard، Gene B.؛ Ikeno، Yuji؛ Zhang، Yongqing (2015). "Reduced Expression of MYC Increases Longevity and Enhances Healthspan". Cell. ج. 160 ع. 3: 477–88. DOI:10.1016/j.cell.2014.12.016. PMC:4624921. PMID:25619689.

ncbi.nlm.nih.gov

"Cellular senescence in aging and age-related disease: from mechanisms to therapy". Nature Medicine. ج. 21 ع. 12: 1424–35. 2015. DOI:10.1038/nm.4000. PMC:4748967. PMID:26646499.
Misra، Juhi؛ Mohanty، Sindhu T.؛ Madan، Sanjeev؛ Fernandes، James A.؛ Hal Ebetino، F.؛ Russell، R. Graham G.؛ Bellantuono، Ilaria (2015). "Zoledronate Attenuates Accumulation of DNA Damage in Mesenchymal Stem Cells and Protects Their Function". Stem Cells. ج. 34 ع. 3: 756–767. DOI:10.1002/stem.2255. PMC:4832316. PMID:26679354.
Xiong، Shiqin؛ Patrushev، Nikolay؛ Forouzandeh، Farshad؛ Hilenski، Lula؛ Alexander، R. Wayne (2015). "PGC-1α Modulates Telomere Function and DNA Damage in Protecting against Aging-Related Chronic Diseases". Cell Reports. ج. 12 ع. 9: 1391–9. DOI:10.1016/j.celrep.2015.07.047. PMC:4549794. PMID:26299964.
Byun، H. O.؛ Lee، Y. K.؛ Kim، J. M.؛ Yoon، G (2015). "From cell senescence to age-related diseases: Differential mechanisms of action of senescence-associated secretory phenotypes". BMB Reports. ج. 48 ع. 10: 549–58. DOI:10.5483/bmbrep.2015.48.10.122. PMC:4911181. PMID:26129674.
Young، Andrew R J؛ Narita، Masashi (2009). "SASP reflects senescence". EMBO Reports. ج. 10 ع. 3: 228–30. DOI:10.1038/embor.2009.22. PMC:2658552. PMID:19218920.
Laberge، Remi-Martin؛ Zhou، Lili؛ Sarantos، Melissa R.؛ Rodier، Francis؛ Freund، Adam؛ De Keizer، Peter L. J.؛ Liu، Su؛ Demaria، Marco؛ Cong، Yu-Sheng (2012). "Glucocorticoids suppress selected components of the senescence-associated secretory phenotype". Aging Cell. ج. 11 ع. 4: 569–78. DOI:10.1111/j.1474-9726.2012.00818.x. PMC:3387333. PMID:22404905.
Liu، Su؛ Uppal، Harpreet؛ Demaria، Marco؛ Desprez، Pierre-Yves؛ Campisi، Judith؛ Kapahi، Pankaj (2015). "Simvastatin suppresses breast cancer cell proliferation induced by senescent cells". Scientific Reports. ج. 5: 17895. DOI:10.1038/srep17895. PMC:4677323. PMID:26658759.
Xu، Ming؛ Tchkonia، Tamara؛ Ding، Husheng؛ Ogrodnik، Mikolaj؛ Lubbers، Ellen R.؛ Pirtskhalava، Tamar؛ White، Thomas A.؛ Johnson، Kurt O.؛ Stout، Michael B. (2015). "JAK inhibition alleviates the cellular senescence-associated secretory phenotype and frailty in old age". Proceedings of the National Academy of Sciences. ج. 112 ع. 46: E6301–10. DOI:10.1073/pnas.1515386112. PMC:4655580. PMID:26578790.
Xu، Ming؛ Palmer، Allyson K؛ Ding، Husheng؛ Weivoda، Megan M؛ Pirtskhalava، Tamar؛ White، Thomas A؛ Sepe، Anna؛ Johnson، Kurt O؛ Stout، Michael B (2015). "Targeting senescent cells enhances adipogenesis and metabolic function in old age". eLife. ج. 4: e12997. DOI:10.7554/eLife.12997. PMC:4758946. PMID:26687007.{{استشهاد بدورية محكمة}}: صيانة الاستشهاد: دوي مجاني غير معلم (link)
"Mitochondria are required for pro-ageing features of the senescent phenotype". The EMBO Journal. ج. 35 ع. 7: 724–42. 2016. DOI:10.15252/embj.201592862. PMC:4818766. PMID:26848154. مؤرشف من الأصل في 2019-06-06. اطلع عليه بتاريخ 2016-02-06. 60% of the SASP genes which are significantly different between proliferating and senescent were reversed upon mitochondrial depletion, whereas only 5% were exacerbated
Zhu، Yi؛ Tchkonia، T؛ Fuhrmann-Stroissnigg، H؛ Dai، HM؛ Ling، YY؛ Stout، MB؛ Pirtskhalava، T؛ Giorgadze، N؛ Johnson، KO (2015). "Identification of a Novel Senolytic Agent, Navitoclax, Targeting the Bcl-2 Family of Anti-Apoptotic Factors". Aging Cell. ج. 15 ع. 3: 428–35. DOI:10.1111/acel.12445. PMC:4854923. PMID:26711051.
Zhu، Yi؛ Tchkonia، Tamara؛ Pirtskhalava، Tamar؛ Gower، Adam؛ Ding، Husheng؛ Giorgadze، Nino؛ Palmer، Allyson K.؛ Ikeno، Yuji؛ Borden، Gene (2015). "The Achilles' Heel of Senescent Cells: From Transcriptome to Senolytic Drugs". Aging Cell. ج. 14 ع. 4: 644–58. DOI:10.1111/acel.12344. PMC:4531078. PMID:25754370.
Fuhrmann-Stroissnigg, Heike; Ling, Yuan Yuan; Zhao, Jing; McGowan, Sara J.; Zhu, Yi; Brooks, Robert W.; Grassi, Diego; Gregg, Siobhan Q.; Stripay, Jennifer L. (4 Sep 2017). "Identification of HSP90 inhibitors as a novel class of senolytics". Nature Communications (بالإنجليزية). 8 (1): 422. DOI:10.1038/s41467-017-00314-z. ISSN:2041-1723. PMC:5583353. PMID:28871086.
Hofmann، Jeffrey W.؛ Zhao، Xiaoai؛ De Cecco، Marco؛ Peterson، Abigail L.؛ Pagliaroli، Luca؛ Manivannan، Jayameenakshi؛ Hubbard، Gene B.؛ Ikeno، Yuji؛ Zhang، Yongqing (2015). "Reduced Expression of MYC Increases Longevity and Enhances Healthspan". Cell. ج. 160 ع. 3: 477–88. DOI:10.1016/j.cell.2014.12.016. PMC:4624921. PMID:25619689.

web.archive.org (Global: 1^st place; Arabic: 1^st place)

"Mitochondria are required for pro-ageing features of the senescent phenotype". The EMBO Journal. ج. 35 ع. 7: 724–42. 2016. DOI:10.15252/embj.201592862. PMC:4818766. PMID:26848154. مؤرشف من الأصل في 2019-06-06. اطلع عليه بتاريخ 2016-02-06. 60% of the SASP genes which are significantly different between proliferating and senescent were reversed upon mitochondrial depletion, whereas only 5% were exacerbated