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غيبوبة مستحثة (Arabic Wikipedia)

Analysis of information sources in references of the Wikipedia article "غيبوبة مستحثة" in Arabic language version.

refsWebsite
Global rank Arabic rank
7doi.org
2nd place
5th place
7nih.gov
4th place
6th place
3web.archive.org
1st place
1st place
1scahq.org
low place
low place
1neuroanesthesia.info
low place
low place
1trauma.org
low place
low place
1archive.org
6th place
3rd place

archive.org

doi.org

  • "Human Rabies: a 2016 Update". Curr Infect Dis Rep (Review). ج. 18 ع. 11: 38. 2016. DOI:10.1007/s11908-016-0540-y. PMID:27730539.
  • "Critical Appraisal of the Milwaukee Protocol for Rabies: This Failed Approach Should Be Abandoned". Can J Neurol Sci (Review). ج. 43 ع. 1: 44–51. 2016. DOI:10.1017/cjn.2015.331. PMID:26639059.
  • "Use of barbiturates in the control of intracranial hypertension". Mary Ann Liebert, Inc. ج. 17 ع. 6–7: 527–30. 2000. DOI:10.1089/neu.2000.17.527. PMID:10937896.
  • "The efficacy of barbiturate coma in the management of uncontrolled intracranial hypertension following neurosurgical trauma". J. Neurotrauma. ج. 11 ع. 3: 325–31. 1994. DOI:10.1089/neu.1994.11.325. PMID:7996586.
  • "The effect of high dose barbiturate decompression after severe head injury. A controlled clinical trial". Acta Neurochir (Wien). ج. 72 ع. 3–4: 157–66. 1984. DOI:10.1007/BF01406868. PMID:6382945.
  • "The University of Toronto head injury treatment study: a prospective, randomized comparison of pentobarbital and mannitol". Can J Neurol Sci. ج. 11 ع. 4: 434–40. 1984. DOI:10.1017/s0317167100045960. PMID:6440704.
  • "Clinical outcome and cognitive impairment in patients with severe head injuries treated with barbiturate coma". Acta Neurochir (Wien). ج. 117 ع. 3–4: 153–9. 1992. DOI:10.1007/BF01400613. PMID:1414516.

neuroanesthesia.info

  • "Cerebral protection and resuscitation". CNS Clinic – Jordan – Amman. مؤرشف من الأصل في 2020-11-04. اطلع عليه بتاريخ 2016-04-16. The primary mechanism of protection involves a reduction in CMRo2 of up to 55% to 60% at which point the EEG becomes isoelectric.

nih.gov

pubmed.ncbi.nlm.nih.gov

  • "Human Rabies: a 2016 Update". Curr Infect Dis Rep (Review). ج. 18 ع. 11: 38. 2016. DOI:10.1007/s11908-016-0540-y. PMID:27730539.
  • "Critical Appraisal of the Milwaukee Protocol for Rabies: This Failed Approach Should Be Abandoned". Can J Neurol Sci (Review). ج. 43 ع. 1: 44–51. 2016. DOI:10.1017/cjn.2015.331. PMID:26639059.
  • "Use of barbiturates in the control of intracranial hypertension". Mary Ann Liebert, Inc. ج. 17 ع. 6–7: 527–30. 2000. DOI:10.1089/neu.2000.17.527. PMID:10937896.
  • "The efficacy of barbiturate coma in the management of uncontrolled intracranial hypertension following neurosurgical trauma". J. Neurotrauma. ج. 11 ع. 3: 325–31. 1994. DOI:10.1089/neu.1994.11.325. PMID:7996586.
  • "The effect of high dose barbiturate decompression after severe head injury. A controlled clinical trial". Acta Neurochir (Wien). ج. 72 ع. 3–4: 157–66. 1984. DOI:10.1007/BF01406868. PMID:6382945.
  • "The University of Toronto head injury treatment study: a prospective, randomized comparison of pentobarbital and mannitol". Can J Neurol Sci. ج. 11 ع. 4: 434–40. 1984. DOI:10.1017/s0317167100045960. PMID:6440704.
  • "Clinical outcome and cognitive impairment in patients with severe head injuries treated with barbiturate coma". Acta Neurochir (Wien). ج. 117 ع. 3–4: 153–9. 1992. DOI:10.1007/BF01400613. PMID:1414516.

scahq.org

trauma.org

  • "Barbiturate Coma". Trauma.org. مؤرشف من الأصل في 2016-08-19. اطلع عليه بتاريخ 2016-04-16. Therapeutic EEG response: burst suppression or cortical electrical silence (with preservation of SSEP and BAEF).

web.archive.org

  • Grocott HP. "Update on Techniques for Neuroprotection during Hypothermic Arrest" (PDF). Society of Cardiovascular Anesthesiologists. مؤرشف من الأصل (PDF) في 2016-04-23. اطلع عليه بتاريخ 2016-04-14. approximately 60% of CMRO2 is utilized for neuronal function (with the remainder being required for cellular integrity)
  • "Cerebral protection and resuscitation". CNS Clinic – Jordan – Amman. مؤرشف من الأصل في 2020-11-04. اطلع عليه بتاريخ 2016-04-16. The primary mechanism of protection involves a reduction in CMRo2 of up to 55% to 60% at which point the EEG becomes isoelectric.
  • "Barbiturate Coma". Trauma.org. مؤرشف من الأصل في 2016-08-19. اطلع عليه بتاريخ 2016-04-16. Therapeutic EEG response: burst suppression or cortical electrical silence (with preservation of SSEP and BAEF).