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فرط فيتامين أ (Arabic Wikipedia)

Analysis of information sources in references of the Wikipedia article "فرط فيتامين أ" in Arabic language version.

refsWebsite
Global rank Arabic rank
34nih.gov
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20doi.org
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8web.archive.org
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3archive.org
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2oup.com
485th place
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2mealographer.com
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1physiology.org
5,293rd place
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1europa.eu
68th place
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1bmj.com
1,226th place
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1mja.com.au
low place
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1harvard.edu
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archive.org

bmj.com

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  • Gorospe M، Fadare O (مايو 2007). "Gastric mucosal calcinosis: clinicopathologic considerations". Advances in Anatomic Pathology. ج. 14 ع. 3: 224–8. DOI:10.1097/PAP.0b013e31805048ea. PMID:17452819.
  • Wall M (مارس 2008). "Idiopathic intracranial hypertension (pseudotumor cerebri)". Current Neurology and Neuroscience Reports. ج. 8 ع. 2: 87–93. DOI:10.1007/s11910-008-0015-0. PMID:18460275.
  • Minuk GY، Kelly JK، Hwang WS (1988). "Vitamin A hepatotoxicity in multiple family members". Hepatology. ج. 8 ع. 2: 272–5. DOI:10.1002/hep.1840080214. PMID:3356407.
  • Levine PH، Delgado Y، Theise ND، West AB (فبراير 2003). "Stellate-cell lipidosis in liver biopsy specimens. Recognition and significance". American Journal of Clinical Pathology. ج. 119 ع. 2: 254–8. DOI:10.1309/6DKC-03C4-GAAE-N2DK. PMID:12579996.
  • Jorens PG، Michielsen PP، Pelckmans PA، Fevery J، Desmet VJ، Geubel AP، Rahier J، Van Maercke YM (ديسمبر 1992). "Vitamin A abuse: development of cirrhosis despite cessation of vitamin A. A six-year clinical and histopathologic follow-up". Liver. ج. 12 ع. 6: 381–6. DOI:10.1111/j.1600-0676.1992.tb00592.x. PMID:1470008.
  • Erickson JM، Mawson AR (سبتمبر 2000). "Possible role of endogenous retinoid (Vitamin A) toxicity in the pathophysiology of primary biliary cirrhosis". Journal of Theoretical Biology. ج. 206 ع. 1: 47–54. DOI:10.1006/jtbi.2000.2102. PMID:10968936.
  • Nollevaux MC، Guiot Y، Horsmans Y، Leclercq I، Rahier J، Geubel AP، Sempoux C (مارس 2006). "Hypervitaminosis A-induced liver fibrosis: stellate cell activation and daily dose consumption". Liver International. ج. 26 ع. 2: 182–6. DOI:10.1111/j.1478-3231.2005.01207.x. PMID:16448456.
  • Kodaka T، Takaki H، Soeta S، Mori R، Naito Y (يوليو 1998). "Local disappearance of epiphyseal growth plates in rats with hypervitaminosis A". The Journal of Veterinary Medical Science. ج. 60 ع. 7: 815–21. DOI:10.1292/jvms.60.815. PMID:9713809.
  • Soeta S، Mori R، Kodaka T، Naito Y، Taniguchi K (مارس 1999). "Immunohistochemical observations on the initial disorders of the epiphyseal growth plate in rats induced by high dose of vitamin A". The Journal of Veterinary Medical Science. ج. 61 ع. 3: 233–8. DOI:10.1292/jvms.61.233. PMID:10331194.
  • Soeta S، Mori R، Kodaka T، Naito Y، Taniguchi K (مارس 2000). "Histological disorders related to the focal disappearance of the epiphyseal growth plate in rats induced by high dose of vitamin A". The Journal of Veterinary Medical Science. ج. 62 ع. 3: 293–9. DOI:10.1292/jvms.62.293. PMID:10770602.
  • Rothenberg AB، Berdon WE، Woodard JC، Cowles RA (ديسمبر 2007). "Hypervitaminosis A-induced premature closure of epiphyses (physeal obliteration) in humans and calves (hyena disease): a historical review of the human and veterinary literature". Pediatric Radiology. ج. 37 ع. 12: 1264–7. DOI:10.1007/s00247-007-0604-0. PMID:17909784.
  • Wick JY (فبراير 2009). "Spontaneous fracture: multiple causes". The Consultant Pharmacist. ج. 24 ع. 2: 100–2, 105–8, 110–2. DOI:10.4140/TCP.n.2009.100. PMID:19275452.
  • Barker ME، Blumsohn A (نوفمبر 2003). "Is vitamin A consumption a risk factor for osteoporotic fracture?". The Proceedings of the Nutrition Society. ج. 62 ع. 4: 845–50. DOI:10.1079/PNS2003306. PMID:15018484.
  • Rodahl K، Moore T (يوليو 1943). "The vitamin A content and toxicity of bear and seal liver". The Biochemical Journal. ج. 37 ع. 2: 166–8. DOI:10.1042/bj0370166. PMC:1257872. PMID:16747610.
  • Rutkowski M، Grzegorczyk K (يونيو 2012). "Adverse effects of antioxidative vitamins". International Journal of Occupational Medicine and Environmental Health. ج. 25 ع. 2: 105–21. DOI:10.2478/S13382-012-0022-x. PMID:22528540.
  • Lips P (يناير 2003). "Hypervitaminosis A and fractures". The New England Journal of Medicine. ج. 348 ع. 4: 347–9. DOI:10.1056/NEJMe020167. PMID:12540650.
  • Weiser H، Probst HP، Bachmann H (سبتمبر 1992). "Vitamin E prevents side effects of high doses of vitamin A in chicks". Annals of the New York Academy of Sciences. ج. 669: 396–8. Bibcode:1992NYASA.669..396W. DOI:10.1111/j.1749-6632.1992.tb17134.x. PMID:1444058.
  • Yeh، Yen-Hung؛ Lee، Ya-Ting؛ Hsieh، Hung-Sheng؛ Hwang، Deng-Fwu (2008). "Effect of taurine on toxicity of vitamin a in rats". Food Chemistry. ج. 106: 260–8. DOI:10.1016/j.foodchem.2007.05.084. {{استشهاد بدورية محكمة}}: الوسيط غير المعروف |name-list-format= تم تجاهله يقترح استخدام |name-list-style= (مساعدة)
  • Skare KL، DeLuca HF (يوليو 1983). "Biliary metabolites of all-trans-retinoic acid in the rat". Archives of Biochemistry and Biophysics. ج. 224 ع. 1: 13–8. DOI:10.1016/0003-9861(83)90185-6. PMID:6870249.
  • Yeh YH، Lee YT، Hsieh YL (مايو 2012). "Effect of cholestin on toxicity of vitamin A in rats". Food Chemistry. ج. 132 ع. 1: 311–8. DOI:10.1016/j.foodchem.2011.10.082. PMID:26434295.

europa.eu

eunis.eea.europa.eu

harvard.edu

ui.adsabs.harvard.edu

mealographer.com

mja.com.au

nih.gov

pubmed.ncbi.nlm.nih.gov

  • Gorospe M، Fadare O (مايو 2007). "Gastric mucosal calcinosis: clinicopathologic considerations". Advances in Anatomic Pathology. ج. 14 ع. 3: 224–8. DOI:10.1097/PAP.0b013e31805048ea. PMID:17452819.
  • Wall M (مارس 2008). "Idiopathic intracranial hypertension (pseudotumor cerebri)". Current Neurology and Neuroscience Reports. ج. 8 ع. 2: 87–93. DOI:10.1007/s11910-008-0015-0. PMID:18460275.
  • Castaño G، Etchart C، Sookoian S (2006). "Vitamin A toxicity in a physical culturist patient: a case report and review of the literature". Annals of Hepatology. ج. 5 ع. 4: 293–395. PMID:17151585.
  • Minuk GY، Kelly JK، Hwang WS (1988). "Vitamin A hepatotoxicity in multiple family members". Hepatology. ج. 8 ع. 2: 272–5. DOI:10.1002/hep.1840080214. PMID:3356407.
  • Levine PH، Delgado Y، Theise ND، West AB (فبراير 2003). "Stellate-cell lipidosis in liver biopsy specimens. Recognition and significance". American Journal of Clinical Pathology. ج. 119 ع. 2: 254–8. DOI:10.1309/6DKC-03C4-GAAE-N2DK. PMID:12579996.
  • Tholen W، Paquet KJ، Rohner HG، Albrecht M (أغسطس 1980). "[Cirrhosis of the liver and esophageal bleeding after chronic vitamin A intoxication (author's transl)]". Leber, Magen, Darm. ج. 10 ع. 4: 193–7. PMID:6969836.
  • Jorens PG، Michielsen PP، Pelckmans PA، Fevery J، Desmet VJ، Geubel AP، Rahier J، Van Maercke YM (ديسمبر 1992). "Vitamin A abuse: development of cirrhosis despite cessation of vitamin A. A six-year clinical and histopathologic follow-up". Liver. ج. 12 ع. 6: 381–6. DOI:10.1111/j.1600-0676.1992.tb00592.x. PMID:1470008.
  • Babb RR، Kieraldo JH (مارس 1978). "Cirrhosis due to hypervitaminosis A". The Western Journal of Medicine. ج. 128 ع. 3: 244–6. PMC:1238074. PMID:636413.
  • Erickson JM، Mawson AR (سبتمبر 2000). "Possible role of endogenous retinoid (Vitamin A) toxicity in the pathophysiology of primary biliary cirrhosis". Journal of Theoretical Biology. ج. 206 ع. 1: 47–54. DOI:10.1006/jtbi.2000.2102. PMID:10968936.
  • Singh M، Singh VN (مايو 1978). "Fatty liver in hypervitaminosis A: synthesis and release of hepatic triglycerides". The American Journal of Physiology. ج. 234 ع. 5: E511–4. PMID:645903. مؤرشف من الأصل في 2020-01-09.
  • Nollevaux MC، Guiot Y، Horsmans Y، Leclercq I، Rahier J، Geubel AP، Sempoux C (مارس 2006). "Hypervitaminosis A-induced liver fibrosis: stellate cell activation and daily dose consumption". Liver International. ج. 26 ع. 2: 182–6. DOI:10.1111/j.1478-3231.2005.01207.x. PMID:16448456.
  • Cho DY، Frey RA، Guffy MM، Leipold HW (نوفمبر 1975). "Hypervitaminosis A in the dog". American Journal of Veterinary Research. ج. 36 ع. 11: 1597–1603. PMID:1190603.
  • Kodaka T، Takaki H، Soeta S، Mori R، Naito Y (يوليو 1998). "Local disappearance of epiphyseal growth plates in rats with hypervitaminosis A". The Journal of Veterinary Medical Science. ج. 60 ع. 7: 815–21. DOI:10.1292/jvms.60.815. PMID:9713809.
  • Soeta S، Mori R، Kodaka T، Naito Y، Taniguchi K (مارس 1999). "Immunohistochemical observations on the initial disorders of the epiphyseal growth plate in rats induced by high dose of vitamin A". The Journal of Veterinary Medical Science. ج. 61 ع. 3: 233–8. DOI:10.1292/jvms.61.233. PMID:10331194.
  • Soeta S، Mori R، Kodaka T، Naito Y، Taniguchi K (مارس 2000). "Histological disorders related to the focal disappearance of the epiphyseal growth plate in rats induced by high dose of vitamin A". The Journal of Veterinary Medical Science. ج. 62 ع. 3: 293–9. DOI:10.1292/jvms.62.293. PMID:10770602.
  • Rothenberg AB، Berdon WE، Woodard JC، Cowles RA (ديسمبر 2007). "Hypervitaminosis A-induced premature closure of epiphyses (physeal obliteration) in humans and calves (hyena disease): a historical review of the human and veterinary literature". Pediatric Radiology. ج. 37 ع. 12: 1264–7. DOI:10.1007/s00247-007-0604-0. PMID:17909784.
  • Wick JY (فبراير 2009). "Spontaneous fracture: multiple causes". The Consultant Pharmacist. ج. 24 ع. 2: 100–2, 105–8, 110–2. DOI:10.4140/TCP.n.2009.100. PMID:19275452.
  • Penniston KL، Tanumihardjo SA (فبراير 2006). "The acute and chronic toxic effects of vitamin A". The American Journal of Clinical Nutrition. ج. 83 ع. 2: 191–201. PMID:16469975. مؤرشف من الأصل في 2020-03-13.
  • Corić-Martinović V، Basić-Jukić N (2008). "[Uremic pruritus]". Acta Medica Croatica. 62 Suppl 1: 32–6. PMID:18578330.
  • Barker ME، Blumsohn A (نوفمبر 2003). "Is vitamin A consumption a risk factor for osteoporotic fracture?". The Proceedings of the Nutrition Society. ج. 62 ع. 4: 845–50. DOI:10.1079/PNS2003306. PMID:15018484.
  • Rodahl K، Moore T (يوليو 1943). "The vitamin A content and toxicity of bear and seal liver". The Biochemical Journal. ج. 37 ع. 2: 166–8. DOI:10.1042/bj0370166. PMC:1257872. PMID:16747610.
  • Rutkowski M، Grzegorczyk K (يونيو 2012). "Adverse effects of antioxidative vitamins". International Journal of Occupational Medicine and Environmental Health. ج. 25 ع. 2: 105–21. DOI:10.2478/S13382-012-0022-x. PMID:22528540.
  • Lips P (يناير 2003). "Hypervitaminosis A and fractures". The New England Journal of Medicine. ج. 348 ع. 4: 347–9. DOI:10.1056/NEJMe020167. PMID:12540650.
  • Carrington-Smith D (2005). "Mawson and Mertz: a re-evaluation of their ill-fated mapping journey during the 1911-1914 Australasian Antarctic Expedition". The Medical Journal of Australia. ج. 183 ع. 11–12: 638–41. PMID:16336159. مؤرشف من الأصل في 2019-12-05.
  • St Claire MB، Kennett MJ، Besch-Williford CL (يوليو 2004). "Vitamin A toxicity and vitamin E deficiency in a rabbit colony". Contemporary Topics in Laboratory Animal Science. ج. 43 ع. 4: 26–30. PMID:15264766.
  • Weiser H، Probst HP، Bachmann H (سبتمبر 1992). "Vitamin E prevents side effects of high doses of vitamin A in chicks". Annals of the New York Academy of Sciences. ج. 669: 396–8. Bibcode:1992NYASA.669..396W. DOI:10.1111/j.1749-6632.1992.tb17134.x. PMID:1444058.
  • Skare KL، DeLuca HF (يوليو 1983). "Biliary metabolites of all-trans-retinoic acid in the rat". Archives of Biochemistry and Biophysics. ج. 224 ع. 1: 13–8. DOI:10.1016/0003-9861(83)90185-6. PMID:6870249.
  • Skare KL، Sietsema WK، DeLuca HF (أغسطس 1982). "The biological activity of retinotaurine". The Journal of Nutrition. ج. 112 ع. 8: 1626–30. PMID:7097369. مؤرشف من الأصل في 2020-05-17.
  • Yeh YH، Lee YT، Hsieh YL (مايو 2012). "Effect of cholestin on toxicity of vitamin A in rats". Food Chemistry. ج. 132 ع. 1: 311–8. DOI:10.1016/j.foodchem.2011.10.082. PMID:26434295.
  • Walker SE، Eylenburg E، Moore T (1947). "The action of vitamin K in hypervitaminosis A". The Biochemical Journal. ج. 41 ع. 4: 575–80. PMC:1258540. PMID:16748217.

ncbi.nlm.nih.gov

oup.com

academic.oup.com

physiology.org

journals.physiology.org

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