كيمياء الزرنيخ الحيوية (Arabic Wikipedia)

Analysis of information sources in references of the Wikipedia article "كيمياء الزرنيخ الحيوية" in Arabic language version.

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archive.org

Pearce، Fred (2006). When the Rivers Run Dry: Journeys Into the Heart of the World's Water Crisis. Toronto: Key Porter. ISBN:978-1-55263-741-8.
Grollman، A.P؛ Moriya، M (1993). "Mutagenesis by 8-oxoguanine: An enemy within". Trends in Genetics. ج. 9: 246–249. DOI:10.1016/0168-9525(93)90089-Z.
Hartwig، A؛ Groblinghoff، U.D (1997). "Interaction of arsenic(III) with nucleotide excision repair in UV-irradiated human fibroblasts". Carcinogenesis. ج. 18: 399–405. DOI:10.1093/carcin/18.2.399. PMID:9054635.
Schwerdtle، T؛ Walter، I (2003). "Induction of oxidative DNA damage by arsenite and its trivalent and pentavalent methylated metabolites in cultured human cells and isolated DNA". Carcinogenesis. ج. 24: 967–974. DOI:10.1093/carcin/bgg018.

doi.org

Elke Dopp, Andrew D. Kligerman and Roland A. Diaz-Bone Organoarsenicals. Uptake, Metabolism, and Toxicity 2010, Royal Society of Chemistry. (ردمك 978-1-84973-082-2). دُوِي:10.1039/9781849730822-00231
Rossman، T.G (2003). "Mechanism of arsenic carcinogenesis: An integrated approach". Mutation Research/Fundamental and Molecular Mechanisms of Mutagenesis. ج. 533: 37–65. DOI:10.1016/j.mrfmmm.2003.07.009.
Pierce، B.L؛ Kibriya، M.G (2012). "Genome-wide association study identifies chromosome 10q24.32 variants associated with arsenic metabolism and toxicity phenotypes in Bangladesh". PLoS Genetics. ج. 8: e1002522. DOI:10.1371/journal.pgen.1002522. PMC:3285587. PMID:22383894.{{استشهاد بدورية محكمة}}: صيانة الاستشهاد: دوي مجاني غير معلم (link)
Li، J.H؛ Rossman، T.G (1991). "Comutagenesis of sodium arsenite with ultraviolet radiation in Chinese hamster V79 cells". Biology of Metals. ج. 4: 197–200. DOI:10.1007/BF01141180.
Lee، T.C؛ Oshimura، M (1985). "Comparison of arsenic-induced cell transformation, cytotoxicity, mutation and cytogenetic effects in Syrian hamster embryo cells in culture". Carcinogenesis. ج. 6: 1421–1426. DOI:10.1093/carcin/6.10.1421.
Kessel، M؛ Liu، S.X (2002). "Arsenic induces oxidative DNA damage in mammalian cells". Molecular and Cellular Biochemistry. 234/235: 234–235:301–308. DOI:10.1023/A:1015927406142.
Nesnow، S؛ Roop، B.C (2002). "DNA damage induced by methylated trivalent arsenicals is mediated by reactive oxygen species". Chemical Research in Toxicology. ج. 15: 1627–1634. DOI:10.1021/tx025598y.
Jomova، K؛ Jenisova، Z (2011). "Arsenic: Toxicity, oxidative stress and human disease". Journal of Applied Toxicology. ج. 31: 95–107. DOI:10.1002/jat.1649.
Kitchin، K.T؛ Wallace، K (2008). "Evidence against the nuclear in situ binding of arsenicals—Oxidative stress theory of arsenic carcinogenesis". Toxicology and Applied Pharmacology. ج. 232: 252–257. DOI:10.1016/j.taap.2008.06.021.
Bau، D.T؛ Wang، T.S (2002). "Oxidative DNA adducts and DNA-protein cross-links are the major DNA lesions induced by arsenite". Environmental Health Perspectives. ج. 110: 753–756. DOI:10.1289/ehp.02110s5753. PMC:1241239. PMID:12426126.
Hwang، E.S؛ Kim، G.H (2007). "Biomarkers for oxidative stress status of DNA, lipids, and proteins in vitro and in vivo cancer research". Toxicology. ج. 229: 1–10. DOI:10.1016/j.tox.2006.10.013.
Liu, Su X. (ديسمبر 2000). "Induction of oxyradicals by arsenic: Implication for mechanism of genotoxicity". Proceedings of the National Academy of Sciences of the United States of America. ج. 98: 1643–1648. Bibcode:2001PNAS...98.1643L. DOI:10.1073/pnas.98.4.1643. PMC:29310. مؤرشف من الأصل في 2019-12-06. اطلع عليه بتاريخ 2013-04-04.
Grollman، A.P؛ Moriya، M (1993). "Mutagenesis by 8-oxoguanine: An enemy within". Trends in Genetics. ج. 9: 246–249. DOI:10.1016/0168-9525(93)90089-Z.
Martinez، V.D؛ Vucic، E.A (2011). "Arsenic biotransformation as a cancer promoting factor by inducing DNA damage and disruption of repair mechanisms". Molecular Biology International. ج. 2011: 1–11. DOI:10.4061/2011/718974.{{استشهاد بدورية محكمة}}: صيانة الاستشهاد: دوي مجاني غير معلم (link)
Lai، Y؛ Zhao، W (2011). "Role of DNA polymerase beta in the genotoxicity of arsenic". Environmental and Molecular Mutagenesis. ج. 52: 460–468. DOI:10.1002/em.20643.
Hartwig، A؛ Groblinghoff، U.D (1997). "Interaction of arsenic(III) with nucleotide excision repair in UV-irradiated human fibroblasts". Carcinogenesis. ج. 18: 399–405. DOI:10.1093/carcin/18.2.399. PMID:9054635.
Curnow، A؛ Salter، L (2001). "A preliminary investigation of the effects of arsenate on irradiation-induced DNA damage in cultured human lung fibroblasts". Journal of Toxicology and Environmental Health, Part A. ج. 63: 605–616. DOI:10.1080/152873901316857789.
Schwerdtle، T؛ Walter، I (2003). "Induction of oxidative DNA damage by arsenite and its trivalent and pentavalent methylated metabolites in cultured human cells and isolated DNA". Carcinogenesis. ج. 24: 967–974. DOI:10.1093/carcin/bgg018.
Ebert، F؛ Weiss، A (2011). "Arsenicals affect base excision repair by several mechanisms". Mutat. Res. ج. 715: 32–41. DOI:10.1016/j.mrfmmm.2011.07.004. PMID:21782832.
Sykora، P؛ Snow، E.T (2008). "Modulation of DNA polymerase beta-dependent base excision repair in cultured human cells after low dose exposure to arsenite". Toxicology and Applied Pharmacology. ج. 228: 385–394. DOI:10.1016/j.taap.2007.12.019.

harvard.edu

ui.adsabs.harvard.edu

Liu, Su X. (ديسمبر 2000). "Induction of oxyradicals by arsenic: Implication for mechanism of genotoxicity". Proceedings of the National Academy of Sciences of the United States of America. ج. 98: 1643–1648. Bibcode:2001PNAS...98.1643L. DOI:10.1073/pnas.98.4.1643. PMC:29310. مؤرشف من الأصل في 2019-12-06. اطلع عليه بتاريخ 2013-04-04.

nih.gov

pubmed.ncbi.nlm.nih.gov

Pierce، B.L؛ Kibriya، M.G (2012). "Genome-wide association study identifies chromosome 10q24.32 variants associated with arsenic metabolism and toxicity phenotypes in Bangladesh". PLoS Genetics. ج. 8: e1002522. DOI:10.1371/journal.pgen.1002522. PMC:3285587. PMID:22383894.{{استشهاد بدورية محكمة}}: صيانة الاستشهاد: دوي مجاني غير معلم (link)
Bau، D.T؛ Wang، T.S (2002). "Oxidative DNA adducts and DNA-protein cross-links are the major DNA lesions induced by arsenite". Environmental Health Perspectives. ج. 110: 753–756. DOI:10.1289/ehp.02110s5753. PMC:1241239. PMID:12426126.
Hartwig، A؛ Groblinghoff، U.D (1997). "Interaction of arsenic(III) with nucleotide excision repair in UV-irradiated human fibroblasts". Carcinogenesis. ج. 18: 399–405. DOI:10.1093/carcin/18.2.399. PMID:9054635.
Ebert، F؛ Weiss، A (2011). "Arsenicals affect base excision repair by several mechanisms". Mutat. Res. ج. 715: 32–41. DOI:10.1016/j.mrfmmm.2011.07.004. PMID:21782832.

ncbi.nlm.nih.gov

Pierce، B.L؛ Kibriya، M.G (2012). "Genome-wide association study identifies chromosome 10q24.32 variants associated with arsenic metabolism and toxicity phenotypes in Bangladesh". PLoS Genetics. ج. 8: e1002522. DOI:10.1371/journal.pgen.1002522. PMC:3285587. PMID:22383894.{{استشهاد بدورية محكمة}}: صيانة الاستشهاد: دوي مجاني غير معلم (link)
Bau، D.T؛ Wang، T.S (2002). "Oxidative DNA adducts and DNA-protein cross-links are the major DNA lesions induced by arsenite". Environmental Health Perspectives. ج. 110: 753–756. DOI:10.1289/ehp.02110s5753. PMC:1241239. PMID:12426126.
Liu, Su X. (ديسمبر 2000). "Induction of oxyradicals by arsenic: Implication for mechanism of genotoxicity". Proceedings of the National Academy of Sciences of the United States of America. ج. 98: 1643–1648. Bibcode:2001PNAS...98.1643L. DOI:10.1073/pnas.98.4.1643. PMC:29310. مؤرشف من الأصل في 2019-12-06. اطلع عليه بتاريخ 2013-04-04.

pnas.org

Liu, Su X. (ديسمبر 2000). "Induction of oxyradicals by arsenic: Implication for mechanism of genotoxicity". Proceedings of the National Academy of Sciences of the United States of America. ج. 98: 1643–1648. Bibcode:2001PNAS...98.1643L. DOI:10.1073/pnas.98.4.1643. PMC:29310. مؤرشف من الأصل في 2019-12-06. اطلع عليه بتاريخ 2013-04-04.

web.archive.org

Liu, Su X. (ديسمبر 2000). "Induction of oxyradicals by arsenic: Implication for mechanism of genotoxicity". Proceedings of the National Academy of Sciences of the United States of America. ج. 98: 1643–1648. Bibcode:2001PNAS...98.1643L. DOI:10.1073/pnas.98.4.1643. PMC:29310. مؤرشف من الأصل في 2019-12-06. اطلع عليه بتاريخ 2013-04-04.