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J. Huang et al.: Dehydroascorbic acid, a blood-brain barrier transportable form of vitamin C, mediates potent cerebroprotection in experimental stroke. In: Proceedings of the National Academy of Sciences of the United States of America. Band98, Nr.20, 25. September 2001, S.11720–11724, doi:10.1073/pnas.171325998, PMID 11573006.
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Y. Nishikawa et al.: Identification of 3,4-dihydroxy-2-oxo-butanal (L-threosone) as an intermediate compound in oxidative degradation of dehydro-L-ascorbic acid and 2,3-diketo-L-gulonic acid in a deuterium oxide phosphate buffer. In: Bioscience, Biotechnology, and Biochemistry. Band65, Nr.8, August 2001, S.1707–1712, doi:10.1271/bbb.65.1707, PMID 11577707.
J. D. Campbell et al.: Ascorbic acid is a potent inhibitor of various forms of T cell apoptosis. In: Cellular Immunology. Band194, Nr.1, 25. Mai 1999, S.1–5, doi:10.1006/cimm.1999.1485, PMID 10357874.
Eva S. Wintergerst et al.: Immune-enhancing role of vitamin C and zinc and effect on clinical conditions. In: Annals of Nutrition & Metabolism. Band50, Nr.2, 2006, S.85–94, doi:10.1159/000090495, PMID 16373990.
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Gwendolyn N.Y. van Gorkom et al.: The Effect of Vitamin C (Ascorbic Acid) in the Treatment of Patients with Cancer: A Systematic Review. In: Nutrients. Band11, Nr.5, 28. April 2019, doi:10.3390/nu11050977, PMID 31035414, PMC 6566697 (freier Volltext).
Pascal L. Langlois et al.: Vitamin C Administration to the Critically Ill: A Systematic Review and Meta-Analysis. In: JPEN. Journal of parenteral and enteral nutrition. Band43, Nr.3, März 2019, S.335–346, doi:10.1002/jpen.1471, PMID 30452091.
J. C. Boffi et al.: Positive modulation of the α9α10 nicotinic cholinergic receptor by ascorbic acid. In: British Journal of Pharmacology. Band168, Nr.4, Februar 2013, S.954–965, doi:10.1111/j.1476-5381.2012.02221.x, PMID 22994414, PMC 3631383 (freier Volltext).
M. Levine et al.: Vitamin C pharmacokinetics in healthy volunteers: evidence for a recommended dietary allowance. In: Proceedings of the National Academy of Sciences of the United States of America. Band93, Nr.8, 16. April 1996, S.3704–3709, doi:10.1073/pnas.93.8.3704, PMID 8623000.
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Cunningham P, Afzal-Ahmed I, Naftalin RJ: Docking studies show that D-glucose and quercetin slide through the transporter GLUT1. In: J. Biol. Chem. Vol. 281, Nr.9, März 2006, S.5797–803, doi:10.1074/jbc.M509422200, PMID 16407180 (englisch).
Montel-Hagen A, Kinet S, Manel N, et al: Erythrocyte Glut1 triggers dehydroascorbic acid uptake in mammals unable to synthesize vitamin C. In: Cell. Vol. 132, Nr.6, März 2008, S.1039–1048, doi:10.1016/j.cell.2008.01.042, PMID 18358815 (englisch).
Jeffrey S. Hampl, Christopher A. Taylor, and Carol S. Johnston: Vitamin C Deficiency and Depletion in the United States: The Third National Health and Nutrition Examination Survey, 1988 to 1994. In: American Journal of Public Health. Mai 2004, Vol. 94, No. 5, S. 870–875, doi:10.2105/AJPH.94.5.870, PMID 15117714, PMC 1448351 (freier Volltext).
S. C. Rumsey et al.: Glucose transporter isoforms GLUT1 and GLUT3 transport dehydroascorbic acid. In: The Journal of Biological Chemistry. Band272, Nr.30, 25. Juli 1997, S.18982–18989, doi:10.1074/jbc.272.30.18982, PMID 9228080.
W. J. Liang et al.: Vitamin C transport systems of mammalian cells. In: Molecular Membrane Biology. Band18, Nr.1, Januar 2001, S.87–95, doi:10.1080/09687680110033774, PMID 11396616.
H. Wang et al.: Human Na(+)-dependent vitamin C transporter 1 (hSVCT1): primary structure, functional characteristics and evidence for a non-functional splice variant. In: Biochimica Et Biophysica Acta. Band1461, Nr.1, 9. November 1999, S.1–9, doi:10.1016/s0005-2736(99)00182-0, PMID 10556483.
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profiles.nlm.nih.gov
J. L. Svirbely und A. Szent-Gyorgyi: The Chemical Nature Of Vitamin C. In: The Biochemical Journal. 1933, Nr. 27, S. 279–285; PDF (freier Volltextzugriff, engl.)
René Zipperlen: Wenig sinnvoll, aber viel gekauft: Der Hokuspokus ums Vitamin C. In: Die Tageszeitung: taz. 23. Oktober 2009 (taz.de [abgerufen am 2. Mai 2021]).