«Oral N-acetyl-L-cysteine is a safe and effective precursor of cysteine». Journal of Animal Science85 (7): 1712–8. Jul 2007. doi:10.2527/jas.2006-835. PMID17371789.
«Adverse reactions to N-acetylcysteine during treatment for paracetamol poisoning». The Medical Journal of Australia150 (6): 329–31. Mar 1989. doi:10.5694/j.1326-5377.1989.tb136496.x. PMID2716644.
«Anaphylactoid reactions to intravenous N-acetylcysteine: a prospective case controlled study». Accident and Emergency Nursing12 (1): 10–5. Jan 2004. doi:10.1016/j.aaen.2003.07.001. PMID14700565.
«Efficacy of oral long-term N-acetylcysteine in chronic bronchopulmonary disease: a meta-analysis of published double-blind, placebo-controlled clinical trials». Clinical Therapeutics22 (2): 209–21. Feb 2000. doi:10.1016/S0149-2918(00)88479-9. PMID10743980.
«The effect of oral N-acetylcysteine in chronic bronchitis: a quantitative systematic review». The European Respiratory Journal16 (2): 253–62. Aug 2000. doi:10.1034/j.1399-3003.2000.16b12.x. PMID10968500.
«Clinical trials of N-acetylcysteine in psychiatry and neurology: A systematic review». Neuroscience and Biobehavioral Reviews55: 294–321. Aug 2015. doi:10.1016/j.neubiorev.2015.04.015. PMID25957927. «In this systematic review we find favorable evidence for the use of NAC in several psychiatric and neurological disorders, particularly autism, Alzheimer's disease, cocaine, bipolar disorder, depression, trichotillomania, nail biting, skin picking, obsessive-compulsive disorder, schizophrenia, drug-induced neuropathy and progressive myoclonic epilepsy. Disorders such as nicotine addictions, anxiety, attention deficit hyperactivity disorder and mild traumatic brain injury have preliminary evidence and require larger confirmatory studies while current evidence does not support the use of NAC in gambling, methamphetamine and amyotrophic lateral sclerosis. Overall, NAC treatment appears to be safe and tolerable.».
«The chemistry and biological activities of N-acetylcysteine». Biochimica et Biophysica Acta (BBA) - General Subjects1830 (8): 4117–29. Aug 2013. doi:10.1016/j.bbagen.2013.04.016. PMID23618697.
Minarini, A; Ferrari, S; Galletti, M; Giambalvo, N; Perrone, D; Rioli, G; Galeazzi, GM (2 November 2016). «N-acetylcysteine in the treatment of psychiatric disorders: current status and future prospects.». Expert Opinion on Drug Metabolism & Toxicology13 (3): 279–292. doi:10.1080/17425255.2017.1251580. PMID27766914.
«Effect of N-acetyl-cysteine on the hypoxic ventilatory response and erythropoietin production: linkage between plasma thiol redox state and O(2) chemosensitivity». Blood99 (5): 1552–5. Mar 2002. doi:10.1182/blood.V99.5.1552. PMID11861267.
«A dual effect of N-acetylcysteine on acute ethanol-induced liver damage in mice». Hepatology Research34 (3): 199–206. Mar 2006. doi:10.1016/j.hepres.2005.12.005. PMID16439183.
Steullet, P.; Neijt, H.C.; Cuénod, M.; Do, K.Q. (2006). «Synaptic plasticity impairment and hypofunction of NMDA receptors induced by glutathione deficit: Relevance to schizophrenia». Neuroscience137 (3): 807–819. doi:10.1016/j.neuroscience.2005.10.014. ISSN0306-4522. PMID16330153.
Varga, V.; Jenei, Zs.; Janáky, R.; Saransaari, P.; Oja, S. S. (1997). «Glutathione Is an Endogenous Ligand of Rat Brain N-Methyl-D-Aspartate (NMDA) and 2-Amino-3-Hydroxy-5-Methyl-4-Isoxazolepropionate (AMPA) Receptors». Neurochemical Research22 (9): 1165–1171. doi:10.1023/A:1027377605054. ISSN0364-3190. PMID9251108.
«Glutathione precursor, N-acetyl-cysteine, improves mismatch negativity in schizophrenia patients». Neuropsychopharmacology33 (9): 2187–99. Aug 2008. doi:10.1038/sj.npp.1301624. PMID18004285.
«N-acetylcysteine for antioxidant therapy: pharmacology and clinical utility». Expert Opinion on Biological Therapy8 (12): 1955–62. Dec 2008. doi:10.1517/14728220802517901. PMID18990082.
«Acetylcysteine». The American Society of Health-System Pharmacists. Αρχειοθετήθηκε από το πρωτότυπο στις 23 Σεπτεμβρίου 2015. Ανακτήθηκε στις 22 Αυγούστου 2015.
«Oral N-acetyl-L-cysteine is a safe and effective precursor of cysteine». Journal of Animal Science85 (7): 1712–8. Jul 2007. doi:10.2527/jas.2006-835. PMID17371789.
«Adverse reactions to N-acetylcysteine during treatment for paracetamol poisoning». The Medical Journal of Australia150 (6): 329–31. Mar 1989. doi:10.5694/j.1326-5377.1989.tb136496.x. PMID2716644.
«Anaphylactoid reactions to intravenous N-acetylcysteine: a prospective case controlled study». Accident and Emergency Nursing12 (1): 10–5. Jan 2004. doi:10.1016/j.aaen.2003.07.001. PMID14700565.
«Efficacy of oral long-term N-acetylcysteine in chronic bronchopulmonary disease: a meta-analysis of published double-blind, placebo-controlled clinical trials». Clinical Therapeutics22 (2): 209–21. Feb 2000. doi:10.1016/S0149-2918(00)88479-9. PMID10743980.
«The effect of oral N-acetylcysteine in chronic bronchitis: a quantitative systematic review». The European Respiratory Journal16 (2): 253–62. Aug 2000. doi:10.1034/j.1399-3003.2000.16b12.x. PMID10968500.
«Clinical trials of N-acetylcysteine in psychiatry and neurology: A systematic review». Neuroscience and Biobehavioral Reviews55: 294–321. Aug 2015. doi:10.1016/j.neubiorev.2015.04.015. PMID25957927. «In this systematic review we find favorable evidence for the use of NAC in several psychiatric and neurological disorders, particularly autism, Alzheimer's disease, cocaine, bipolar disorder, depression, trichotillomania, nail biting, skin picking, obsessive-compulsive disorder, schizophrenia, drug-induced neuropathy and progressive myoclonic epilepsy. Disorders such as nicotine addictions, anxiety, attention deficit hyperactivity disorder and mild traumatic brain injury have preliminary evidence and require larger confirmatory studies while current evidence does not support the use of NAC in gambling, methamphetamine and amyotrophic lateral sclerosis. Overall, NAC treatment appears to be safe and tolerable.».
«The chemistry and biological activities of N-acetylcysteine». Biochimica et Biophysica Acta (BBA) - General Subjects1830 (8): 4117–29. Aug 2013. doi:10.1016/j.bbagen.2013.04.016. PMID23618697.
Minarini, A; Ferrari, S; Galletti, M; Giambalvo, N; Perrone, D; Rioli, G; Galeazzi, GM (2 November 2016). «N-acetylcysteine in the treatment of psychiatric disorders: current status and future prospects.». Expert Opinion on Drug Metabolism & Toxicology13 (3): 279–292. doi:10.1080/17425255.2017.1251580. PMID27766914.
«Effect of N-acetyl-cysteine on the hypoxic ventilatory response and erythropoietin production: linkage between plasma thiol redox state and O(2) chemosensitivity». Blood99 (5): 1552–5. Mar 2002. doi:10.1182/blood.V99.5.1552. PMID11861267.
«A dual effect of N-acetylcysteine on acute ethanol-induced liver damage in mice». Hepatology Research34 (3): 199–206. Mar 2006. doi:10.1016/j.hepres.2005.12.005. PMID16439183.
Steullet, P.; Neijt, H.C.; Cuénod, M.; Do, K.Q. (2006). «Synaptic plasticity impairment and hypofunction of NMDA receptors induced by glutathione deficit: Relevance to schizophrenia». Neuroscience137 (3): 807–819. doi:10.1016/j.neuroscience.2005.10.014. ISSN0306-4522. PMID16330153.
Varga, V.; Jenei, Zs.; Janáky, R.; Saransaari, P.; Oja, S. S. (1997). «Glutathione Is an Endogenous Ligand of Rat Brain N-Methyl-D-Aspartate (NMDA) and 2-Amino-3-Hydroxy-5-Methyl-4-Isoxazolepropionate (AMPA) Receptors». Neurochemical Research22 (9): 1165–1171. doi:10.1023/A:1027377605054. ISSN0364-3190. PMID9251108.
«Glutathione precursor, N-acetyl-cysteine, improves mismatch negativity in schizophrenia patients». Neuropsychopharmacology33 (9): 2187–99. Aug 2008. doi:10.1038/sj.npp.1301624. PMID18004285.
«N-acetylcysteine for antioxidant therapy: pharmacology and clinical utility». Expert Opinion on Biological Therapy8 (12): 1955–62. Dec 2008. doi:10.1517/14728220802517901. PMID18990082.
«L-Cysteine, N-acetyl- - Compound Summary». PubChem Compound. USA: National Center for Biotechnology Information. 25 Μαρτίου 2005. Identification. Αρχειοθετήθηκε από το πρωτότυπο στις 12 Ιανουαρίου 2014. Ανακτήθηκε στις 9 Ιανουαρίου 2012.
«Cysteine». University of Maryland Medical Center (στα Αγγλικά). Αρχειοθετήθηκε από το πρωτότυπο στις 1 Ιουλίου 2017. Ανακτήθηκε στις 23 Ιουνίου 2017.
web.archive.org
«L-Cysteine, N-acetyl- - Compound Summary». PubChem Compound. USA: National Center for Biotechnology Information. 25 Μαρτίου 2005. Identification. Αρχειοθετήθηκε από το πρωτότυπο στις 12 Ιανουαρίου 2014. Ανακτήθηκε στις 9 Ιανουαρίου 2012.
«Acetylcysteine». The American Society of Health-System Pharmacists. Αρχειοθετήθηκε από το πρωτότυπο στις 23 Σεπτεμβρίου 2015. Ανακτήθηκε στις 22 Αυγούστου 2015.
Steullet, P.; Neijt, H.C.; Cuénod, M.; Do, K.Q. (2006). «Synaptic plasticity impairment and hypofunction of NMDA receptors induced by glutathione deficit: Relevance to schizophrenia». Neuroscience137 (3): 807–819. doi:10.1016/j.neuroscience.2005.10.014. ISSN0306-4522. PMID16330153.
Varga, V.; Jenei, Zs.; Janáky, R.; Saransaari, P.; Oja, S. S. (1997). «Glutathione Is an Endogenous Ligand of Rat Brain N-Methyl-D-Aspartate (NMDA) and 2-Amino-3-Hydroxy-5-Methyl-4-Isoxazolepropionate (AMPA) Receptors». Neurochemical Research22 (9): 1165–1171. doi:10.1023/A:1027377605054. ISSN0364-3190. PMID9251108.