Willis, G. L.; Armstrong, S. M. (1999). "A therapeutic role for melatonin antagonism in experimental models of Parkinson's disease"". Physiology & Behavior. 66 (5): 785–795. doi:10.1016/s0031-9384(99)00023-2. PMID10405106.
Willis, G. L. (2008). "Parkinson's disease as a neuroendocrine disorder of circadian function: dopamine-melatonin imbalance and the visual system in the genesis and progression of the degenerative process". Reviews in the Neurosciences. 19 (4–5): 245–316. doi:10.1515/REVNEURO.2008.19.4-5.245. PMID19145986.
Willis, G. L.; Turner, E. J. D. (2007). "Primary and secondary features of Parkinson's disease improve with strategic exposure to bright light: a case series study". Chronobiology International. 24 (3): 521–537. doi:10.1080/07420520701420717. PMID17612949.
Willis, G. L.; Armstrong, S. M. (1999). "A therapeutic role for melatonin antagonism in experimental models of Parkinson's disease"". Physiology & Behavior. 66 (5): 785–795. doi:10.1016/s0031-9384(99)00023-2. PMID10405106.
Willis, G. L. (2008). "Parkinson's disease as a neuroendocrine disorder of circadian function: dopamine-melatonin imbalance and the visual system in the genesis and progression of the degenerative process". Reviews in the Neurosciences. 19 (4–5): 245–316. doi:10.1515/REVNEURO.2008.19.4-5.245. PMID19145986.
Willis, G. L.; Turner, E. J. D. (2007). "Primary and secondary features of Parkinson's disease improve with strategic exposure to bright light: a case series study". Chronobiology International. 24 (3): 521–537. doi:10.1080/07420520701420717. PMID17612949.