Bernard C (1856). Leçons de physiologie expérimentale appliquée à la médecine (in French). Vol. 2. Paris, France: J.B. Baillière et fils. p. 430. From p. 430: "En effet, si l'on ouvre le duodenum sur un animal vivant et que l'on touche l'orifice du conduit cholédoque avec une baguette de verre imprégnée d'acide acétique faible, on voit immédiatement un flot de bile lancé dans l'intestin; ce qui ne se fait pas si, au lieu de toucher l'orifice du conduit cholédoque avec un liquide acide, on le touche avec un liquide lègérement alcalin, comme du carbonate de soude par example." (Indeed, if one opens the duodenum on a living animal and touches the orifice of the bile duct with a glass rod impregnated with weak acetic acid, one immediately sees a stream of bile squirted into the intestine; which is not done if, instead of touching the orifice of the bile duct with an acidic liquid, it is touched with a slightly alkaline liquid, such as sodium carbonate for example.)
colostate.edu
arbl.cvmbs.colostate.edu
Bowen R (28 January 2001). "Cholecystokinin". Colorado State University. Archived from the original on 17 March 2016. Retrieved 6 November 2015.
Jorpes E, Mutt V (January–February 1966). "Cholecystokinin and pancreozymin, one single hormone?". Acta Physiologica Scandinavica. 66 (1): 196–202. doi:10.1111/j.1748-1716.1966.tb03185.x. PMID5935672.
Reeve JR, Eysselein VE, Rosenquist G, Zeeh J, Regner U, Ho FJ, et al. (May 1996). "Evidence that CCK-58 has structure that influences its biological activity". The American Journal of Physiology. 270 (5 Pt 1): G860–G868. doi:10.1152/ajpgi.1996.270.5.G860. PMID8967499.
Agersnap M, Rehfeld JF (August 2014). "Measurement of nonsulfated cholecystokinins". Scandinavian Journal of Clinical and Laboratory Investigation. 74 (5): 424–431. doi:10.3109/00365513.2014.900695. PMID24734780. S2CID207421432.
Lenka A, Arumugham SS, Christopher R, Pal PK (May 2016). "Genetic substrates of psychosis in patients with Parkinson's disease: A critical review". Journal of the Neurological Sciences. 364: 33–41. doi:10.1016/j.jns.2016.03.005. PMID27084212. S2CID31298855.
Liddle RA (September 1995). "Regulation of cholecystokinin secretion by intraluminal releasing factors". The American Journal of Physiology. 269 (3 Pt 1): G319–G327. doi:10.1152/ajpgi.1995.269.3.G319. PMID7573441.
Shillabeer G, Davison JS (February 1987). "Proglumide, a cholecystokinin antagonist, increases gastric emptying in rats". The American Journal of Physiology. 252 (2 Pt 2): R353–R360. doi:10.1152/ajpregu.1987.252.2.R353. PMID3812772.
Holzer P (July 1998). "Neural injury, repair, and adaptation in the GI tract. II. The elusive action of capsaicin on the vagus nerve". The American Journal of Physiology. 275 (1): G8-13. doi:10.1152/ajpgi.1998.275.1.G8. PMID9655678.
Kobelt P, Tebbe JJ, Tjandra I, Stengel A, Bae HG, Andresen V, et al. (March 2005). "CCK inhibits the orexigenic effect of peripheral ghrelin". American Journal of Physiology. Regulatory, Integrative and Comparative Physiology. 288 (3): R751–R758. doi:10.1152/ajpregu.00094.2004. PMID15550621.
Fink H, Rex A, Voits M, Voigt JP (November 1998). "Major biological actions of CCK--a critical evaluation of research findings". Experimental Brain Research. 123 (1–2): 77–83. doi:10.1007/s002210050546. PMID9835394. S2CID11251325.
Galés C, Poirot M, Taillefer J, Maigret B, Martinez J, Moroder L, et al. (May 2003). "Identification of tyrosine 189 and asparagine 358 of the cholecystokinin 2 receptor in direct interaction with the crucial C-terminal amide of cholecystokinin by molecular modeling, site-directed mutagenesis, and structure/affinity studies". Molecular Pharmacology. 63 (5): 973–982. doi:10.1124/mol.63.5.973. PMID12695525. S2CID38395309.
Wertheimer E (1903). "De l'action des acides et du chloral sur la sécrétion biliaire (d'après les expériences de M. Ch. Dubois)" [On the action of acids and chloral on bile secretion (according to the experiments of Mr. Charles Dubois)]. Compte Rendus Hebdomadaires des Séances et Mémoires de la Société Biologie (in French). 55: 286–287. From p. 287: "Ces expériences furent ensuite répétées après section préalable des pneumogastriques au cou et des sympathiques dans le thorax: cinq sur douze ont encore donné des résultats positifs." (These experiments [namely, introducing dilute acid into the duodenum in order to determine whether the acid then stimulated the secretion of bile] were then repeated after prior section [i.e., cutting] of the pneumogastric [i.e., vagus nerves] in the neck and the sympathetic [nerves] in the thorax: five out of twelve [experiments] again gave positive results.)
Fleig C (1904). "Du mode d'action des excitants chimiques des glandes digestives" [On the mode of action of the chemical stimulants of the digestive glands]. Archives Internationales de Physiologie et de Biochimie (in French). 1: 286–346. From pp. 316-317: "Expérience. — Chien 13 k. chloralosé. On isole une anse de duodéno-jejunum, … C'est là pour le moment une question non résolue et qui ne m'a donné aucun résultat." (Experiment: A dog of 13 kg. was anaesthetized with chloral hydrate. One isolates a section of the duodenum-jejunum; one introduces into it a solution of 0.5% HCl, and one collects the venous blood from the section as usual. Infusion of the [venous] blood which is administered during 30 minutes (about 100 cc.) to a dog of 7 kg. having a canula in the bile duct and [having] the cystic duct bound. The flow of bile is increased to double ([see] fig. 44). But is this humoral action due to the same secretin that acts on the pancrease or [is it due to the action of] a special "crinine" [i.e., a hypothetical hormone that's involved in digestion, like Fleig's "sapocrinine" (see p. 293)] on the liver? For the moment it's an unresolved question and [one] that has given me no result.)
Jorpes E, Mutt V (January–February 1966). "Cholecystokinin and pancreozymin, one single hormone?". Acta Physiologica Scandinavica. 66 (1): 196–202. doi:10.1111/j.1748-1716.1966.tb03185.x. PMID5935672.
Reeve JR, Eysselein VE, Rosenquist G, Zeeh J, Regner U, Ho FJ, et al. (May 1996). "Evidence that CCK-58 has structure that influences its biological activity". The American Journal of Physiology. 270 (5 Pt 1): G860–G868. doi:10.1152/ajpgi.1996.270.5.G860. PMID8967499.
Agersnap M, Rehfeld JF (August 2014). "Measurement of nonsulfated cholecystokinins". Scandinavian Journal of Clinical and Laboratory Investigation. 74 (5): 424–431. doi:10.3109/00365513.2014.900695. PMID24734780. S2CID207421432.
Lenka A, Arumugham SS, Christopher R, Pal PK (May 2016). "Genetic substrates of psychosis in patients with Parkinson's disease: A critical review". Journal of the Neurological Sciences. 364: 33–41. doi:10.1016/j.jns.2016.03.005. PMID27084212. S2CID31298855.
Liddle RA (September 1995). "Regulation of cholecystokinin secretion by intraluminal releasing factors". The American Journal of Physiology. 269 (3 Pt 1): G319–G327. doi:10.1152/ajpgi.1995.269.3.G319. PMID7573441.
Shillabeer G, Davison JS (February 1987). "Proglumide, a cholecystokinin antagonist, increases gastric emptying in rats". The American Journal of Physiology. 252 (2 Pt 2): R353–R360. doi:10.1152/ajpregu.1987.252.2.R353. PMID3812772.
Holzer P (July 1998). "Neural injury, repair, and adaptation in the GI tract. II. The elusive action of capsaicin on the vagus nerve". The American Journal of Physiology. 275 (1): G8-13. doi:10.1152/ajpgi.1998.275.1.G8. PMID9655678.
Kobelt P, Tebbe JJ, Tjandra I, Stengel A, Bae HG, Andresen V, et al. (March 2005). "CCK inhibits the orexigenic effect of peripheral ghrelin". American Journal of Physiology. Regulatory, Integrative and Comparative Physiology. 288 (3): R751–R758. doi:10.1152/ajpregu.00094.2004. PMID15550621.
Fink H, Rex A, Voits M, Voigt JP (November 1998). "Major biological actions of CCK--a critical evaluation of research findings". Experimental Brain Research. 123 (1–2): 77–83. doi:10.1007/s002210050546. PMID9835394. S2CID11251325.
Galés C, Poirot M, Taillefer J, Maigret B, Martinez J, Moroder L, et al. (May 2003). "Identification of tyrosine 189 and asparagine 358 of the cholecystokinin 2 receptor in direct interaction with the crucial C-terminal amide of cholecystokinin by molecular modeling, site-directed mutagenesis, and structure/affinity studies". Molecular Pharmacology. 63 (5): 973–982. doi:10.1124/mol.63.5.973. PMID12695525. S2CID38395309.
Agersnap M, Rehfeld JF (August 2014). "Measurement of nonsulfated cholecystokinins". Scandinavian Journal of Clinical and Laboratory Investigation. 74 (5): 424–431. doi:10.3109/00365513.2014.900695. PMID24734780. S2CID207421432.
Lenka A, Arumugham SS, Christopher R, Pal PK (May 2016). "Genetic substrates of psychosis in patients with Parkinson's disease: A critical review". Journal of the Neurological Sciences. 364: 33–41. doi:10.1016/j.jns.2016.03.005. PMID27084212. S2CID31298855.
Chey WY, Chang T (1 January 2001). "Neural hormonal regulation of exocrine pancreatic secretion". Pancreatology. 1 (4): 320–335. doi:10.1159/000055831. PMID12120211. S2CID22629842.
Fink H, Rex A, Voits M, Voigt JP (November 1998). "Major biological actions of CCK--a critical evaluation of research findings". Experimental Brain Research. 123 (1–2): 77–83. doi:10.1007/s002210050546. PMID9835394. S2CID11251325.
Galés C, Poirot M, Taillefer J, Maigret B, Martinez J, Moroder L, et al. (May 2003). "Identification of tyrosine 189 and asparagine 358 of the cholecystokinin 2 receptor in direct interaction with the crucial C-terminal amide of cholecystokinin by molecular modeling, site-directed mutagenesis, and structure/affinity studies". Molecular Pharmacology. 63 (5): 973–982. doi:10.1124/mol.63.5.973. PMID12695525. S2CID38395309.
