Analysis of information sources in references of the Wikipedia article "Delayed sleep phase disorder" in English language version.
Last modified:
Individuals with delayed sleep phase tend to be more alert in the evening and early nighttime, stay up later, and are more tired in the morning.
Early onset of CRSD, the ease of diagnosis, the high frequency of misdiagnosis and erroneous treatment, the potentially harmful psychological and adjustment consequences, and the availability of promising treatments, all indicate the importance of greater awareness of these disorders.
Our findings therefore suggest that evening light restriction is important for preventing patients with DSPS from developing a sleep phase delay.
{{cite journal}}: Cite uses deprecated parameter |citeseerx= (help)We have used APZ to treat DSPS. One reason it was effective may be that the insomnia induced by daytime APZ was effective in treating the patient's daytime sleepiness. Another reason may be APZ increases histamine release which controls sleep-wake cycles. Thus, APZ may be therapeutic for DSPS.
Sleep onset, midpoint of sleep, and sleep offset were significantly advanced by 1.1, 1.8, and 2.5 hours, respectively. Unexpectedly, sleep duration became significantly shorter by 1.3 hours after treatment. Their depressive moods showed an unremarkable change.
Using exogenous melatonin as a sleep aid at night has minimal phase shifting effects
Implicit in the diagnosis of circadian rhythm disorder is a desire to conform to traditionally accepted sleep–wake patterns.
Delayed sleep phase syndrome: Patients consistently go to sleep and awaken late (eg, 3 AM and 10 AM). This pattern is more common during adolescence than in adulthood. If required to awaken earlier for work or school, excessive daytime sleepiness results; patients often present because school performance is poor or they miss morning classes. They can be distinguished from people who stay up late by choice because they cannot fall asleep earlier even if they try. Mild phase delay (< 3 hours) is treated by progressive earlier arising plus morning bright light therapy, perhaps with melatonin 4 to 5 hours before the desired bedtime. An alternative method is to progressively delay bedtime and awakening time by 1 to 3 hours/day until the correct sleep and wake times are reached.
Early onset of CRSD, the ease of diagnosis, the high frequency of misdiagnosis and erroneous treatment, the potentially harmful psychological and adjustment consequences, and the availability of promising treatments, all indicate the importance of greater awareness of these disorders.
Early onset of CRSD, the ease of diagnosis, the high frequency of misdiagnosis and erroneous treatment, the potentially harmful psychological and adjustment consequences, and the availability of promising treatments, all indicate the importance of greater awareness of these disorders.
Our findings therefore suggest that evening light restriction is important for preventing patients with DSPS from developing a sleep phase delay.
{{cite journal}}: Cite uses deprecated parameter |citeseerx= (help)Non-photic stimuli such as scheduled voluntary exercise, food, exogenous melatonin or serotonergic activation are also capable of shifting the endogenous circadian rhythms.
We have used APZ to treat DSPS. One reason it was effective may be that the insomnia induced by daytime APZ was effective in treating the patient's daytime sleepiness. Another reason may be APZ increases histamine release which controls sleep-wake cycles. Thus, APZ may be therapeutic for DSPS.
Sleep onset, midpoint of sleep, and sleep offset were significantly advanced by 1.1, 1.8, and 2.5 hours, respectively. Unexpectedly, sleep duration became significantly shorter by 1.3 hours after treatment. Their depressive moods showed an unremarkable change.
Using exogenous melatonin as a sleep aid at night has minimal phase shifting effects
Non-photic stimuli such as scheduled voluntary exercise, food, exogenous melatonin or serotonergic activation are also capable of shifting the endogenous circadian rhythms.
Sleep onset, midpoint of sleep, and sleep offset were significantly advanced by 1.1, 1.8, and 2.5 hours, respectively. Unexpectedly, sleep duration became significantly shorter by 1.3 hours after treatment. Their depressive moods showed an unremarkable change.
Using exogenous melatonin as a sleep aid at night has minimal phase shifting effects
{{cite journal}}: Cite uses deprecated parameter |citeseerx= (help)Our findings therefore suggest that evening light restriction is important for preventing patients with DSPS from developing a sleep phase delay.
{{cite journal}}: Cite uses deprecated parameter |citeseerx= (help)Early onset of CRSD, the ease of diagnosis, the high frequency of misdiagnosis and erroneous treatment, the potentially harmful psychological and adjustment consequences, and the availability of promising treatments, all indicate the importance of greater awareness of these disorders.