Analysis of information sources in references of the Wikipedia article "Dementia with Lewy bodies" in English language version.
In 1976, Kenji Kosaka and colleagues described the first post-mortem case of presenile dementia with 'Lewylike-bodies' pathology and, in 1984, Kosaka introduced the term 'diffuse Lewy body disease'.
There is general agreement that there are three core EFs: inhibition [inhibitory control, including self-control (behavioral inhibition) and interference control (selective attention and cognitive inhibition)], working memory (WM), and cognitive flexibility (also called set shifting, mental flexibility, or mental set shifting and closely linked to creativity). From these, higher-order EFs are built such as reasoning, problem solving, and planning.
Parkinsonian syndromes are a group of movement disorders characterized by classical motor symptoms such as tremors, bradykinesia, and rigidity. They are most frequently due to primary neurodegenerative disease, resulting in the loss of dopaminergic nerve terminals along the nigrostriatal pathway, similar to idiopathic PD, MSA, PSP, CBD, and DLB.
Severe, and often fatal, neuroleptic sensitivity may occur [such as] ... exaggerated adverse responses to standard doses ... [and] development or worsening of extrapyramidal features ... or acute and severe physical deterioration"McKeith IG, Boeve BF, Dickson DW, et al. (July 2017). "Diagnosis and management of dementia with Lewy bodies: Fourth consensus report of the DLB Consortium". Neurology (Review). 89 (1): 88–100. doi:10.1212/WNL.0000000000004058. PMC 5496518. PMID 28592453.
In 1976, Kenji Kosaka and colleagues described the first post-mortem case of presenile dementia with 'Lewylike-bodies' pathology and, in 1984, Kosaka introduced the term 'diffuse Lewy body disease'.
There is general agreement that there are three core EFs: inhibition [inhibitory control, including self-control (behavioral inhibition) and interference control (selective attention and cognitive inhibition)], working memory (WM), and cognitive flexibility (also called set shifting, mental flexibility, or mental set shifting and closely linked to creativity). From these, higher-order EFs are built such as reasoning, problem solving, and planning.
Parkinsonian syndromes are a group of movement disorders characterized by classical motor symptoms such as tremors, bradykinesia, and rigidity. They are most frequently due to primary neurodegenerative disease, resulting in the loss of dopaminergic nerve terminals along the nigrostriatal pathway, similar to idiopathic PD, MSA, PSP, CBD, and DLB.
Severe, and often fatal, neuroleptic sensitivity may occur [such as] ... exaggerated adverse responses to standard doses ... [and] development or worsening of extrapyramidal features ... or acute and severe physical deterioration"McKeith IG, Boeve BF, Dickson DW, et al. (July 2017). "Diagnosis and management of dementia with Lewy bodies: Fourth consensus report of the DLB Consortium". Neurology (Review). 89 (1): 88–100. doi:10.1212/WNL.0000000000004058. PMC 5496518. PMID 28592453.
In 1976, Kenji Kosaka and colleagues described the first post-mortem case of presenile dementia with 'Lewylike-bodies' pathology and, in 1984, Kosaka introduced the term 'diffuse Lewy body disease'.
There is general agreement that there are three core EFs: inhibition [inhibitory control, including self-control (behavioral inhibition) and interference control (selective attention and cognitive inhibition)], working memory (WM), and cognitive flexibility (also called set shifting, mental flexibility, or mental set shifting and closely linked to creativity). From these, higher-order EFs are built such as reasoning, problem solving, and planning.
Parkinsonian syndromes are a group of movement disorders characterized by classical motor symptoms such as tremors, bradykinesia, and rigidity. They are most frequently due to primary neurodegenerative disease, resulting in the loss of dopaminergic nerve terminals along the nigrostriatal pathway, similar to idiopathic PD, MSA, PSP, CBD, and DLB.
Severe, and often fatal, neuroleptic sensitivity may occur [such as] ... exaggerated adverse responses to standard doses ... [and] development or worsening of extrapyramidal features ... or acute and severe physical deterioration"McKeith IG, Boeve BF, Dickson DW, et al. (July 2017). "Diagnosis and management of dementia with Lewy bodies: Fourth consensus report of the DLB Consortium". Neurology (Review). 89 (1): 88–100. doi:10.1212/WNL.0000000000004058. PMC 5496518. PMID 28592453.
In 1976, Kenji Kosaka and colleagues described the first post-mortem case of presenile dementia with 'Lewylike-bodies' pathology and, in 1984, Kosaka introduced the term 'diffuse Lewy body disease'.
In 1976, Kenji Kosaka and colleagues described the first post-mortem case of presenile dementia with 'Lewylike-bodies' pathology and, in 1984, Kosaka introduced the term 'diffuse Lewy body disease'.