Camacho A, Massieu L (January 2006). "Role of glutamate transporters in the clearance and release of glutamate during ischemia and its relation to neuronal death". Archives of Medical Research. 37 (1): 11–18. doi:10.1016/j.arcmed.2005.05.014. PMID16314180.
Lucas DR, Newhouse JP (August 1957). "The toxic effect of sodium L-glutamate on the inner layers of the retina". A.M.A. Archives of Ophthalmology. 58 (2): 193–201. doi:10.1001/archopht.1957.00940010205006. PMID13443577.
Yang DD, Kuan CY, Whitmarsh AJ, Rincón M, Zheng TS, Davis RJ, et al. (October 1997). "Absence of excitotoxicity-induced apoptosis in the hippocampus of mice lacking the Jnk3 gene". Nature. 389 (6653): 865–870. Bibcode:1997Natur.389..865Y. doi:10.1038/39899. PMID9349820. S2CID4430535.
Stavrovskaya IG, Kristal BS (March 2005). "The powerhouse takes control of the cell: is the mitochondrial permeability transition a viable therapeutic target against neuronal dysfunction and death?". Free Radical Biology & Medicine. 38 (6): 687–697. doi:10.1016/j.freeradbiomed.2004.11.032. PMID15721979.
Li S, Stys PK (2001). "Na(+)-K(+)-ATPase inhibition and depolarization induce glutamate release via reverse Na(+)-dependent transport in spinal cord white matter". Neuroscience. 107 (4): 675–683. doi:10.1016/s0306-4522(01)00385-2. PMID11720790. S2CID25693141.
Hardingham GE, Fukunaga Y, Bading H (May 2002). "Extrasynaptic NMDARs oppose synaptic NMDARs by triggering CREB shut-off and cell death pathways". Nature Neuroscience. 5 (5): 405–414. doi:10.1038/nn835. PMID11953750. S2CID659716.
Chiu AS, Gehringer MM, Braidy N, Guillemin GJ, Welch JH, Neilan BA (November 2012). "Excitotoxic potential of the cyanotoxin β-methyl-amino-L-alanine (BMAA) in primary human neurons". Toxicon. 60 (6): 1159–1165. Bibcode:2012Txcn...60.1159C. doi:10.1016/j.toxicon.2012.07.169. PMID22885173.
Papapetropoulos S (June 2007). "Is there a role for naturally occurring cyanobacterial toxins in neurodegeneration? The beta-N-methylamino-L-alanine (BMAA) paradigm". Neurochemistry International. 50 (7–8): 998–1003. doi:10.1016/j.neuint.2006.12.011. PMID17296249. S2CID24476846.
Yang DD, Kuan CY, Whitmarsh AJ, Rincón M, Zheng TS, Davis RJ, et al. (October 1997). "Absence of excitotoxicity-induced apoptosis in the hippocampus of mice lacking the Jnk3 gene". Nature. 389 (6653): 865–870. Bibcode:1997Natur.389..865Y. doi:10.1038/39899. PMID9349820. S2CID4430535.
Chiu AS, Gehringer MM, Braidy N, Guillemin GJ, Welch JH, Neilan BA (November 2012). "Excitotoxic potential of the cyanotoxin β-methyl-amino-L-alanine (BMAA) in primary human neurons". Toxicon. 60 (6): 1159–1165. Bibcode:2012Txcn...60.1159C. doi:10.1016/j.toxicon.2012.07.169. PMID22885173.
Manev H, Favaron M, Guidotti A, Costa E (July 1989). "Delayed increase of Ca2+ influx elicited by glutamate: role in neuronal death". Molecular Pharmacology. 36 (1): 106–112. PMID2568579.
Camacho A, Massieu L (January 2006). "Role of glutamate transporters in the clearance and release of glutamate during ischemia and its relation to neuronal death". Archives of Medical Research. 37 (1): 11–18. doi:10.1016/j.arcmed.2005.05.014. PMID16314180.
Lucas DR, Newhouse JP (August 1957). "The toxic effect of sodium L-glutamate on the inner layers of the retina". A.M.A. Archives of Ophthalmology. 58 (2): 193–201. doi:10.1001/archopht.1957.00940010205006. PMID13443577.
Yang DD, Kuan CY, Whitmarsh AJ, Rincón M, Zheng TS, Davis RJ, et al. (October 1997). "Absence of excitotoxicity-induced apoptosis in the hippocampus of mice lacking the Jnk3 gene". Nature. 389 (6653): 865–870. Bibcode:1997Natur.389..865Y. doi:10.1038/39899. PMID9349820. S2CID4430535.
Stavrovskaya IG, Kristal BS (March 2005). "The powerhouse takes control of the cell: is the mitochondrial permeability transition a viable therapeutic target against neuronal dysfunction and death?". Free Radical Biology & Medicine. 38 (6): 687–697. doi:10.1016/j.freeradbiomed.2004.11.032. PMID15721979.
Li S, Stys PK (2001). "Na(+)-K(+)-ATPase inhibition and depolarization induce glutamate release via reverse Na(+)-dependent transport in spinal cord white matter". Neuroscience. 107 (4): 675–683. doi:10.1016/s0306-4522(01)00385-2. PMID11720790. S2CID25693141.
Hardingham GE, Fukunaga Y, Bading H (May 2002). "Extrasynaptic NMDARs oppose synaptic NMDARs by triggering CREB shut-off and cell death pathways". Nature Neuroscience. 5 (5): 405–414. doi:10.1038/nn835. PMID11953750. S2CID659716.
Chiu AS, Gehringer MM, Braidy N, Guillemin GJ, Welch JH, Neilan BA (November 2012). "Excitotoxic potential of the cyanotoxin β-methyl-amino-L-alanine (BMAA) in primary human neurons". Toxicon. 60 (6): 1159–1165. Bibcode:2012Txcn...60.1159C. doi:10.1016/j.toxicon.2012.07.169. PMID22885173.
Papapetropoulos S (June 2007). "Is there a role for naturally occurring cyanobacterial toxins in neurodegeneration? The beta-N-methylamino-L-alanine (BMAA) paradigm". Neurochemistry International. 50 (7–8): 998–1003. doi:10.1016/j.neuint.2006.12.011. PMID17296249. S2CID24476846.
Hardingham GE, Fukunaga Y, Bading H (May 2002). "Extrasynaptic NMDARs oppose synaptic NMDARs by triggering CREB shut-off and cell death pathways". Nature Neuroscience. 5 (5): 405–414. doi:10.1038/nn835. PMID11953750. S2CID659716.
Yan J, Bengtson CP, Buchthal B, Hagenston AM, Bading H (October 2020). "Coupling of NMDA receptors and TRPM4 guides discovery of unconventional neuroprotectants". Science. 370 (6513): eaay3302. doi:10.1126/science.aay3302. PMID33033186. S2CID222210921.
Yang DD, Kuan CY, Whitmarsh AJ, Rincón M, Zheng TS, Davis RJ, et al. (October 1997). "Absence of excitotoxicity-induced apoptosis in the hippocampus of mice lacking the Jnk3 gene". Nature. 389 (6653): 865–870. Bibcode:1997Natur.389..865Y. doi:10.1038/39899. PMID9349820. S2CID4430535.
Li S, Stys PK (2001). "Na(+)-K(+)-ATPase inhibition and depolarization induce glutamate release via reverse Na(+)-dependent transport in spinal cord white matter". Neuroscience. 107 (4): 675–683. doi:10.1016/s0306-4522(01)00385-2. PMID11720790. S2CID25693141.
Hardingham GE, Fukunaga Y, Bading H (May 2002). "Extrasynaptic NMDARs oppose synaptic NMDARs by triggering CREB shut-off and cell death pathways". Nature Neuroscience. 5 (5): 405–414. doi:10.1038/nn835. PMID11953750. S2CID659716.
Papapetropoulos S (June 2007). "Is there a role for naturally occurring cyanobacterial toxins in neurodegeneration? The beta-N-methylamino-L-alanine (BMAA) paradigm". Neurochemistry International. 50 (7–8): 998–1003. doi:10.1016/j.neuint.2006.12.011. PMID17296249. S2CID24476846.