Analysis of information sources in references of the Wikipedia article "Gulf War" in English language version.
SAS first units ground January into iraq.
For Gulf War veterans with the QQ genotype, hearing nerve agent alarms—a proxy for chemical exposure—raised their chance of developing GWI by 3.75 times. For those with the QR genotype, the alarms raised their chance of GWI by 4.43 times. And for those with two copies of the R gene, inefficient at breaking down sarin, the chance of GWI increased by 8.91 times. Those soldiers with both the RR genotype and low-level sarin exposure were over seven times more likely to get GWI due to the interaction per se, over and above the increase in risk from both risk factors acting alone. For genetic epidemiologists, this number leads to a high degree of confidence that sarin is a causative agent of GWI.
For Gulf War veterans with the QQ genotype, hearing nerve agent alarms—a proxy for chemical exposure—raised their chance of developing GWI by 3.75 times. For those with the QR genotype, the alarms raised their chance of GWI by 4.43 times. And for those with two copies of the R gene, inefficient at breaking down sarin, the chance of GWI increased by 8.91 times. Those soldiers with both the RR genotype and low-level sarin exposure were over seven times more likely to get GWI due to the interaction per se, over and above the increase in risk from both risk factors acting alone. For genetic epidemiologists, this number leads to a high degree of confidence that sarin is a causative agent of GWI.
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: CS1 maint: location (link)For Gulf War veterans with the QQ genotype, hearing nerve agent alarms—a proxy for chemical exposure—raised their chance of developing GWI by 3.75 times. For those with the QR genotype, the alarms raised their chance of GWI by 4.43 times. And for those with two copies of the R gene, inefficient at breaking down sarin, the chance of GWI increased by 8.91 times. Those soldiers with both the RR genotype and low-level sarin exposure were over seven times more likely to get GWI due to the interaction per se, over and above the increase in risk from both risk factors acting alone. For genetic epidemiologists, this number leads to a high degree of confidence that sarin is a causative agent of GWI.
For Gulf War veterans with the QQ genotype, hearing nerve agent alarms—a proxy for chemical exposure—raised their chance of developing GWI by 3.75 times. For those with the QR genotype, the alarms raised their chance of GWI by 4.43 times. And for those with two copies of the R gene, inefficient at breaking down sarin, the chance of GWI increased by 8.91 times. Those soldiers with both the RR genotype and low-level sarin exposure were over seven times more likely to get GWI due to the interaction per se, over and above the increase in risk from both risk factors acting alone. For genetic epidemiologists, this number leads to a high degree of confidence that sarin is a causative agent of GWI.
Oil formations frequently run beneath political boundaries, whether they involve unfriendly leaseholders in West Texas or neighboring Arab states, and procedures have existed for years to settle disputes that arise. Typically, participants in the same field share both production costs and revenues, using a formula that sets percentages of ownership. But Iraq refused to negotiate with Kuwait on such an agreement. So Kuwait produced oil from Rumaila without any agreement, and then adopted a policy of producing far more oil than it was allowed under the quota system of the Organization of Petroleum Exporting Countries. ... But during the [Iran–Iraq War] Iraq mined its giant share of the Rumaila field to keep it from falling into Iranian hands, Western political experts say. Kuwait stepped up its total oil production, capturing some of Iraq's customers and pumping millions of barrels from the Rumaila field. ... Some Iraqi officials have accused Kuwait in the past of using advanced drilling techniques developed by American oilfield specialists to siphon oil from the Rumaila field, a charge that American drillers deny, noting that the oil flows easily from the Rumaila field without any need for these techniques. ... W. C. Goins, senior vice president of OGE Drilling Inc., a Houston company that provided oilfield supervisors and workers for Kuwait in the same area, said he was 'positive' all of the wells his employees drilled and operated ran vertically down to the Rumaila pay zone. 'That field crosses the border in north Kuwait,' he added. 'Iraqis were drilling on one side, and Kuwaitis on the other side.'
Sources: Gulf War Airpower Survey, Vol. 5; Norman Friedman, Desert Victory; World Air Power Journal. Additionally, Mark Bovankovich and LT Chuck Chase offered corrections and several intriguing details on these incidents. All errors, however, remain entirely mine.
For Gulf War veterans with the QQ genotype, hearing nerve agent alarms—a proxy for chemical exposure—raised their chance of developing GWI by 3.75 times. For those with the QR genotype, the alarms raised their chance of GWI by 4.43 times. And for those with two copies of the R gene, inefficient at breaking down sarin, the chance of GWI increased by 8.91 times. Those soldiers with both the RR genotype and low-level sarin exposure were over seven times more likely to get GWI due to the interaction per se, over and above the increase in risk from both risk factors acting alone. For genetic epidemiologists, this number leads to a high degree of confidence that sarin is a causative agent of GWI.
For Gulf War veterans with the QQ genotype, hearing nerve agent alarms—a proxy for chemical exposure—raised their chance of developing GWI by 3.75 times. For those with the QR genotype, the alarms raised their chance of GWI by 4.43 times. And for those with two copies of the R gene, inefficient at breaking down sarin, the chance of GWI increased by 8.91 times. Those soldiers with both the RR genotype and low-level sarin exposure were over seven times more likely to get GWI due to the interaction per se, over and above the increase in risk from both risk factors acting alone. For genetic epidemiologists, this number leads to a high degree of confidence that sarin is a causative agent of GWI.