Induced coma (English Wikipedia)

Analysis of information sources in references of the Wikipedia article "Induced coma" in English language version.

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bmj.com

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harvard.edu

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accessmedicine.mhmedical.com

  • Mariano GL, Fink ME, Hoffman C, Rosengart A (2014). "Intracranial pressure: monitoring and management.". In Hall JB, Schmidt GA, Kress JP (eds.). Principles of Critical Care (4th ed.). McGraw Hill. ISBN 978-0-07-173881-1.

neuroanesthesia.info

  • "Cerebral protection and resuscitation". CNS Clinic – Jordan – Amman. Archived from the original on 4 November 2020. Retrieved 16 April 2016. The primary mechanism of protection involves a reduction in CMRo2 of up to 55% to 60% at which point the EEG becomes isoelectric.

nih.gov

pubmed.ncbi.nlm.nih.gov

ncbi.nlm.nih.gov

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semanticscholar.org

api.semanticscholar.org

  • Nordby HK, Nesbakken R (1984). "The effect of high dose barbiturate decompression after severe head injury. A controlled clinical trial". Acta Neurochirurgica. 72 (3–4): 157–166. doi:10.1007/BF01406868. PMID 6382945. S2CID 12215655.
  • Schalén W, Sonesson B, Messeter K, Nordström G, Nordström CH (1992). "Clinical outcome and cognitive impairment in patients with severe head injuries treated with barbiturate coma". Acta Neurochirurgica. 117 (3–4): 153–159. doi:10.1007/BF01400613. PMID 1414516. S2CID 23032307.

thelancet.com

trauma.org

  • "Barbiturate Coma". Trauma.org. Archived from the original on 19 August 2016. Retrieved 16 April 2016. Therapeutic EEG response: burst suppression or cortical electrical silence (with preservation of SSEP and BAEF).

web.archive.org

  • Grocott HP. "Update on Techniques for Neuroprotection during Hypothermic Arrest" (PDF). Society of Cardiovascular Anesthesiologists. Archived from the original (PDF) on 23 April 2016. Retrieved 14 April 2016. approximately 60% of CMRO2 is utilized for neuronal function (with the remainder being required for cellular integrity)
  • "Cerebral protection and resuscitation". CNS Clinic – Jordan – Amman. Archived from the original on 4 November 2020. Retrieved 16 April 2016. The primary mechanism of protection involves a reduction in CMRo2 of up to 55% to 60% at which point the EEG becomes isoelectric.
  • "Barbiturate Coma". Trauma.org. Archived from the original on 19 August 2016. Retrieved 16 April 2016. Therapeutic EEG response: burst suppression or cortical electrical silence (with preservation of SSEP and BAEF).

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