Analysis of information sources in references of the Wikipedia article "Osteogenesis imperfecta" in English language version.
{{cite journal}}: ISBN / Date incompatibility (help){{cite journal}}: CS1 maint: DOI inactive as of November 2024 (link){{cite book}}: |work= ignored (help){{cite journal}}: CS1 maint: DOI inactive as of November 2024 (link)In 1979 Sillence proposed a classification of Osteogenesis Imperfecta (OI) in OI types I, II, III and IV. In 2004 and 2007, this classification was expanded with OI types V–VIII because of distinct clinical features and/or different causative gene mutations.
For example, it can occur that a fetus is diagnosed with OI type II, the lethal form, on prenatal ultrasound, but after birth turns out to be less severely affected than initially thought and is eventually reclassified as OI type III or even type VI, if adequate management is initiated early on.
The persistent pale blue sclera is uncommon in OI type IV, but can be seen in up to 10% of affected subjects.
{{cite journal}}: CS1 maint: DOI inactive as of December 2024 (link){{cite journal}}: ISBN / Date incompatibility (help){{cite book}}: |website= ignored (help) Last Update: February 3, 2019.{{cite journal}}: CS1 maint: DOI inactive as of November 2024 (link)In 1979 Sillence proposed a classification of Osteogenesis Imperfecta (OI) in OI types I, II, III and IV. In 2004 and 2007, this classification was expanded with OI types V–VIII because of distinct clinical features and/or different causative gene mutations.
For example, it can occur that a fetus is diagnosed with OI type II, the lethal form, on prenatal ultrasound, but after birth turns out to be less severely affected than initially thought and is eventually reclassified as OI type III or even type VI, if adequate management is initiated early on.
The persistent pale blue sclera is uncommon in OI type IV, but can be seen in up to 10% of affected subjects.
{{cite journal}}: CS1 maint: DOI inactive as of December 2024 (link){{cite book}}: |website= ignored (help) Last Update: February 3, 2019.For example, it can occur that a fetus is diagnosed with OI type II, the lethal form, on prenatal ultrasound, but after birth turns out to be less severely affected than initially thought and is eventually reclassified as OI type III or even type VI, if adequate management is initiated early on.
The persistent pale blue sclera is uncommon in OI type IV, but can be seen in up to 10% of affected subjects.
{{cite journal}}: CS1 maint: DOI inactive as of December 2024 (link)In 1979 Sillence proposed a classification of Osteogenesis Imperfecta (OI) in OI types I, II, III and IV. In 2004 and 2007, this classification was expanded with OI types V–VIII because of distinct clinical features and/or different causative gene mutations.
{{cite book}}: |work= ignored (help){{cite journal}}: CS1 maint: DOI inactive as of November 2024 (link){{cite journal}}: ISBN / Date incompatibility (help)Shriners Hospitals for Children is committed to providing specialized care to children with orthopaedic conditions [...] regardless of the families' ability to pay.
{{cite book}}: |work= ignored (help)Dentinogenesis imperfecta (DI) is most frequent in OI types III and IV, and overall, affects about 15% of OI patients among the different phenotypes.
{{cite book}}: |website= ignored (help) Last Update: February 3, 2019.{{cite journal}}: CS1 maint: DOI inactive as of November 2024 (link)In 1979 Sillence proposed a classification of Osteogenesis Imperfecta (OI) in OI types I, II, III and IV. In 2004 and 2007, this classification was expanded with OI types V–VIII because of distinct clinical features and/or different causative gene mutations.
Shriners Hospitals for Children is committed to providing specialized care to children with orthopaedic conditions [...] regardless of the families' ability to pay.
{{cite journal}}: ISBN / Date incompatibility (help)Dentinogenesis imperfecta (DI) is most frequent in OI types III and IV, and overall, affects about 15% of OI patients among the different phenotypes.
Dentinogenesis imperfecta (DI) is most frequent in OI types III and IV, and overall, affects about 15% of OI patients among the different phenotypes.