Sodium voltage-gated channel alpha subunit 9 (English Wikipedia)

Analysis of information sources in references of the Wikipedia article "Sodium voltage-gated channel alpha subunit 9" in English language version.

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40nih.gov

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26doi.org

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10semanticscholar.org

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10books.google.com

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7harvard.edu

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2ensembl.org

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1pitt.edu

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1nature.com

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books.google.com

Allerton C, Fox D (2013). Pain Therapeutics: Current and Future Treatment Paradigms. Royal Society of Chemistry. pp. 146–148. ISBN 978-1-84973-645-9.
Kew JN, Davies CH (2010). Ion Channels: From Structure to Function. Oxford University Press. pp. 153–154. ISBN 978-0-19-929675-0.
Mashour GA, Lydic R (7 September 2011). Neuroscientific Foundations of Anesthesiology. Oxford University Press. p. 154. ISBN 978-0-19-987546-7.
Chahine M. Recent advances in voltage-gated sodium channels, their pharmacology and related diseases. Frontiers E-books. p. 90. ISBN 978-2-88919-128-4.
Lamberth C, Dinges J (9 August 2012). Bioactive Heterocyclic Compound Classes: Pharmaceuticals. John Wiley & Sons. p. 127. ISBN 978-3-527-66448-1.
Cairns BE (1 September 2009). Peripheral Receptor Targets for Analgesia: Novel Approaches to Pain Management. John Wiley & Sons. pp. 66–68. ISBN 978-0-470-52221-9.
James WD, Berger T, Elston D (12 April 2015). "Cutaneous Vascular Diseases". Andrews' Diseases of the Skin: Clinical Dermatology. Elsevier Health Sciences. pp. 810–. ISBN 978-0-323-31969-0.
Altman RB, Flockhart D, Goldstein DB (23 January 2012). Principles of Pharmacogenetics and Pharmacogenomics. Cambridge University Press. p. 224. ISBN 978-1-107-37747-9.
McMahon S, Koltzenburg M, Tracey I, Turk DC (2013). Wall & Melzack's Textbook of Pain (6 ed.). Elsevier Health Sciences. p. 508. ISBN 978-0-7020-4059-7. OCLC 1008841684.
Simpson DM, McArthur JC, Dworkin RH (21 June 2012). Neuropathic Pain: Mechanisms, Diagnosis and Treatment. Oxford University Press. pp. 40–. ISBN 978-0-19-539470-2.

doi.org

Klugbauer N, Lacinova L, Flockerzi V, Hofmann F (March 1995). "Structure and functional expression of a new member of the tetrodotoxin-sensitive voltage-activated sodium channel family from human neuroendocrine cells". The EMBO Journal. 14 (6): 1084–1090. doi:10.1002/j.1460-2075.1995.tb07091.x. PMC 398185. PMID 7720699.
Plummer NW, Meisler MH (April 1999). "Evolution and diversity of mammalian sodium channel genes". Genomics. 57 (2): 323–331. doi:10.1006/geno.1998.5735. PMID 10198179.
Catterall WA, Goldin AL, Waxman SG (December 2005). "International Union of Pharmacology. XLVII. Nomenclature and structure-function relationships of voltage-gated sodium channels". Pharmacological Reviews. 57 (4): 397–409. doi:10.1124/pr.57.4.4. PMID 16382098. S2CID 7332624.
Raymond CK, Castle J, Garrett-Engele P, Armour CD, Kan Z, Tsinoremas N, et al. (October 2004). "Expression of alternatively spliced sodium channel alpha-subunit genes. Unique splicing patterns are observed in dorsal root ganglia". The Journal of Biological Chemistry. 279 (44): 46234–46241. doi:10.1074/jbc.M406387200. PMID 15302875. S2CID 17753425.
Rush AM, Dib-Hajj SD, Liu S, Cummins TR, Black JA, Waxman SG (May 2006). "A single sodium channel mutation produces hyper- or hypoexcitability in different types of neurons". Proceedings of the National Academy of Sciences of the United States of America. 103 (21): 8245–8250. Bibcode:2006PNAS..103.8245R. doi:10.1073/pnas.0602813103. PMC 1472458. PMID 16702558.
Catterall WA (2000). "Structure and regulation of voltage-gated Ca2+ channels". Annual Review of Cell and Developmental Biology. 16: 521–555. doi:10.1146/annurev.cellbio.16.1.521. PMID 11031246.
Cummins TR, Howe JR, Waxman SG (December 1998). "Slow closed-state inactivation: a novel mechanism underlying ramp currents in cells expressing the hNE/PN1 sodium channel". The Journal of Neuroscience. 18 (23): 9607–9619. doi:10.1523/JNEUROSCI.18-23-09607.1998. PMC 6793269. PMID 9822722.
Renganathan M, Cummins TR, Waxman SG (August 2001). "Contribution of Na(v)1.8 sodium channels to action potential electrogenesis in DRG neurons". Journal of Neurophysiology. 86 (2): 629–640. doi:10.1152/jn.2001.86.2.629. PMID 11495938. S2CID 11579149.
Shekdar K, Langer J, Venkatachalan S, Schmid L, Anobile J, Shah P, et al. (May 2021). "Cell engineering method using fluorogenic oligonucleotide signaling probes and flow cytometry". Biotechnology Letters. 43 (5): 949–958. doi:10.1007/s10529-021-03101-5. PMC 7937778. PMID 33683511.
Nassar MA, Stirling LC, Forlani G, Baker MD, Matthews EA, Dickenson AH, et al. (August 2004). "Nociceptor-specific gene deletion reveals a major role for Nav1.7 (PN1) in acute and inflammatory pain". Proceedings of the National Academy of Sciences of the United States of America. 101 (34): 12706–12711. Bibcode:2004PNAS..10112706N. doi:10.1073/pnas.0404915101. PMC 515119. PMID 15314237.
Minett MS, Nassar MA, Clark AK, Passmore G, Dickenson AH, Wang F, et al. (April 2012). "Distinct Nav1.7-dependent pain sensations require different sets of sensory and sympathetic neurons". Nature Communications. 3 (4): 791. Bibcode:2012NatCo...3..791M. doi:10.1038/ncomms1795. PMC 3337979. PMID 22531176.
Dib-Hajj SD, Yang Y, Black JA, Waxman SG (January 2013). "The Na(V)1.7 sodium channel: from molecule to man". Nature Reviews. Neuroscience. 14 (1): 49–62. doi:10.1038/nrn3404. PMID 23232607. S2CID 5489010.
Tang Z, Chen Z, Tang B, Jiang H (September 2015). "Primary erythromelalgia: a review". Orphanet Journal of Rare Diseases. 10: 127. doi:10.1186/s13023-015-0347-1. PMC 4589109. PMID 26419464.
Drenth JP, Michiels JJ (June 1994). "Erythromelalgia and erythermalgia: diagnostic differentiation". International Journal of Dermatology. 33 (6): 393–397. doi:10.1111/j.1365-4362.1994.tb04037.x. PMID 8056469. S2CID 3033396.
Golshani AE, Kamdar AA, Spence SC, Beckmann NM (August 2014). "Congenital indifference to pain: an illustrated case report and literature review". Journal of Radiology Case Reports. 8 (8): 16–23. doi:10.3941/jrcr.v8i8.2194. PMC 4242143. PMID 25426241.
Fertleman CR, Baker MD, Parker KA, Moffatt S, Elmslie FV, Abrahamsen B, et al. (December 2006). "SCN9A mutations in paroxysmal extreme pain disorder: allelic variants underlie distinct channel defects and phenotypes". Neuron. 52 (5): 767–774. doi:10.1016/j.neuron.2006.10.006. PMID 17145499. S2CID 11715780.
Peddareddygari LR, Oberoi K, Grewal RP (2014-09-18). "Congenital insensitivity to pain: a case report and review of the literature". Case Reports in Neurological Medicine. 2014: 141953. doi:10.1155/2014/141953. PMC 4182687. PMID 25309764.
Devigili G, Eleopra R, Pierro T, Lombardi R, Rinaldo S, Lettieri C, et al. (September 2014). "Paroxysmal itch caused by gain-of-function Nav1.7 mutation". Pain. 155 (9): 1702–1707. doi:10.1016/j.pain.2014.05.006. PMID 24820863. S2CID 24462006.
Gingras J, Smith S, Matson DJ, Johnson D, Nye K, Couture L, et al. (2014). "Global Nav1.7 knockout mice recapitulate the phenotype of human congenital indifference to pain". PLOS ONE. 9 (9): e105895. Bibcode:2014PLoSO...9j5895G. doi:10.1371/journal.pone.0105895. PMC 4154897. PMID 25188265.
Waxman SG (December 2006). "Neurobiology: a channel sets the gain on pain". Nature. 444 (7121): 831–832. Bibcode:2006Natur.444..831W. doi:10.1038/444831a. PMID 17167466. S2CID 4300362.
Dib-Hajj SD, Cummins TR, Black JA, Waxman SG (November 2007). "From genes to pain: Na v 1.7 and human pain disorders". Trends in Neurosciences. 30 (11): 555–563. doi:10.1016/j.tins.2007.08.004. PMID 17950472. S2CID 15131517.
Bagal SK, Chapman ML, Marron BE, Prime R, Storer RI, Swain NA (August 2014). "Recent progress in sodium channel modulators for pain". Bioorganic & Medicinal Chemistry Letters. 24 (16): 3690–3699. doi:10.1016/j.bmcl.2014.06.038. PMID 25060923.
Martz L (2014). "Nav-i-gating antibodies for pain". Science-Business EXchange. 7 (23): 662. doi:10.1038/scibx.2014.662. ISSN 1945-3477.
Minett MS, Pereira V, Sikandar S, Matsuyama A, Lolignier S, Kanellopoulos AH, et al. (December 2015). "Endogenous opioids contribute to insensitivity to pain in humans and mice lacking sodium channel Nav1.7". Nature Communications. 6: 8967. Bibcode:2015NatCo...6.8967M. doi:10.1038/ncomms9967. PMC 4686868. PMID 26634308.
Flinspach M, Xu Q, Piekarz AD, Fellows R, Hagan R, Gibbs A, et al. (January 2017). "Insensitivity to pain induced by a potent selective closed-state Nav1.7 inhibitor". Scientific Reports. 7: 39662. Bibcode:2017NatSR...739662F. doi:10.1038/srep39662. PMC 5206724. PMID 28045073.
Moreno AM, Alemán F, Catroli GF, Hunt M, Hu M, Dailamy A, et al. (March 2021). "Long-lasting analgesia via targeted in situ repression of Na_V1.7 in mice". Science Translational Medicine. 13 (584): eaay9056. doi:10.1126/scitranslmed.aay9056. PMC 8830379. PMID 33692134. S2CID 232170826.

duke.edu

today.duke.edu

Yong S (May 22, 2014). "One Molecule To Block Both Pain And Itch".

ensembl.org

May2017.archive.ensembl.org

GRCh38: Ensembl release 89: ENSG00000169432 – Ensembl, May 2017
GRCm38: Ensembl release 89: ENSMUSG00000075316 – Ensembl, May 2017

harvard.edu

ui.adsabs.harvard.edu

Rush AM, Dib-Hajj SD, Liu S, Cummins TR, Black JA, Waxman SG (May 2006). "A single sodium channel mutation produces hyper- or hypoexcitability in different types of neurons". Proceedings of the National Academy of Sciences of the United States of America. 103 (21): 8245–8250. Bibcode:2006PNAS..103.8245R. doi:10.1073/pnas.0602813103. PMC 1472458. PMID 16702558.
Nassar MA, Stirling LC, Forlani G, Baker MD, Matthews EA, Dickenson AH, et al. (August 2004). "Nociceptor-specific gene deletion reveals a major role for Nav1.7 (PN1) in acute and inflammatory pain". Proceedings of the National Academy of Sciences of the United States of America. 101 (34): 12706–12711. Bibcode:2004PNAS..10112706N. doi:10.1073/pnas.0404915101. PMC 515119. PMID 15314237.
Minett MS, Nassar MA, Clark AK, Passmore G, Dickenson AH, Wang F, et al. (April 2012). "Distinct Nav1.7-dependent pain sensations require different sets of sensory and sympathetic neurons". Nature Communications. 3 (4): 791. Bibcode:2012NatCo...3..791M. doi:10.1038/ncomms1795. PMC 3337979. PMID 22531176.
Gingras J, Smith S, Matson DJ, Johnson D, Nye K, Couture L, et al. (2014). "Global Nav1.7 knockout mice recapitulate the phenotype of human congenital indifference to pain". PLOS ONE. 9 (9): e105895. Bibcode:2014PLoSO...9j5895G. doi:10.1371/journal.pone.0105895. PMC 4154897. PMID 25188265.
Waxman SG (December 2006). "Neurobiology: a channel sets the gain on pain". Nature. 444 (7121): 831–832. Bibcode:2006Natur.444..831W. doi:10.1038/444831a. PMID 17167466. S2CID 4300362.
Minett MS, Pereira V, Sikandar S, Matsuyama A, Lolignier S, Kanellopoulos AH, et al. (December 2015). "Endogenous opioids contribute to insensitivity to pain in humans and mice lacking sodium channel Nav1.7". Nature Communications. 6: 8967. Bibcode:2015NatCo...6.8967M. doi:10.1038/ncomms9967. PMC 4686868. PMID 26634308.
Flinspach M, Xu Q, Piekarz AD, Fellows R, Hagan R, Gibbs A, et al. (January 2017). "Insensitivity to pain induced by a potent selective closed-state Nav1.7 inhibitor". Scientific Reports. 7: 39662. Bibcode:2017NatSR...739662F. doi:10.1038/srep39662. PMC 5206724. PMID 28045073.

nature.com

Martz L. "Nav-i-gating antibodies for pain". SciBX.

newatlas.com

"Unique CRISPR gene therapy offers opioid-free chronic pain treatment". New Atlas. 11 March 2021. Retrieved 18 April 2021.

nih.gov

pubmed.ncbi.nlm.nih.gov

Klugbauer N, Lacinova L, Flockerzi V, Hofmann F (March 1995). "Structure and functional expression of a new member of the tetrodotoxin-sensitive voltage-activated sodium channel family from human neuroendocrine cells". The EMBO Journal. 14 (6): 1084–1090. doi:10.1002/j.1460-2075.1995.tb07091.x. PMC 398185. PMID 7720699.
Plummer NW, Meisler MH (April 1999). "Evolution and diversity of mammalian sodium channel genes". Genomics. 57 (2): 323–331. doi:10.1006/geno.1998.5735. PMID 10198179.
Catterall WA, Goldin AL, Waxman SG (December 2005). "International Union of Pharmacology. XLVII. Nomenclature and structure-function relationships of voltage-gated sodium channels". Pharmacological Reviews. 57 (4): 397–409. doi:10.1124/pr.57.4.4. PMID 16382098. S2CID 7332624.
Raymond CK, Castle J, Garrett-Engele P, Armour CD, Kan Z, Tsinoremas N, et al. (October 2004). "Expression of alternatively spliced sodium channel alpha-subunit genes. Unique splicing patterns are observed in dorsal root ganglia". The Journal of Biological Chemistry. 279 (44): 46234–46241. doi:10.1074/jbc.M406387200. PMID 15302875. S2CID 17753425.
Rush AM, Dib-Hajj SD, Liu S, Cummins TR, Black JA, Waxman SG (May 2006). "A single sodium channel mutation produces hyper- or hypoexcitability in different types of neurons". Proceedings of the National Academy of Sciences of the United States of America. 103 (21): 8245–8250. Bibcode:2006PNAS..103.8245R. doi:10.1073/pnas.0602813103. PMC 1472458. PMID 16702558.
Catterall WA (2000). "Structure and regulation of voltage-gated Ca2+ channels". Annual Review of Cell and Developmental Biology. 16: 521–555. doi:10.1146/annurev.cellbio.16.1.521. PMID 11031246.
Cummins TR, Howe JR, Waxman SG (December 1998). "Slow closed-state inactivation: a novel mechanism underlying ramp currents in cells expressing the hNE/PN1 sodium channel". The Journal of Neuroscience. 18 (23): 9607–9619. doi:10.1523/JNEUROSCI.18-23-09607.1998. PMC 6793269. PMID 9822722.
Renganathan M, Cummins TR, Waxman SG (August 2001). "Contribution of Na(v)1.8 sodium channels to action potential electrogenesis in DRG neurons". Journal of Neurophysiology. 86 (2): 629–640. doi:10.1152/jn.2001.86.2.629. PMID 11495938. S2CID 11579149.
Shekdar K, Langer J, Venkatachalan S, Schmid L, Anobile J, Shah P, et al. (May 2021). "Cell engineering method using fluorogenic oligonucleotide signaling probes and flow cytometry". Biotechnology Letters. 43 (5): 949–958. doi:10.1007/s10529-021-03101-5. PMC 7937778. PMID 33683511.
Nassar MA, Stirling LC, Forlani G, Baker MD, Matthews EA, Dickenson AH, et al. (August 2004). "Nociceptor-specific gene deletion reveals a major role for Nav1.7 (PN1) in acute and inflammatory pain". Proceedings of the National Academy of Sciences of the United States of America. 101 (34): 12706–12711. Bibcode:2004PNAS..10112706N. doi:10.1073/pnas.0404915101. PMC 515119. PMID 15314237.
Minett MS, Nassar MA, Clark AK, Passmore G, Dickenson AH, Wang F, et al. (April 2012). "Distinct Nav1.7-dependent pain sensations require different sets of sensory and sympathetic neurons". Nature Communications. 3 (4): 791. Bibcode:2012NatCo...3..791M. doi:10.1038/ncomms1795. PMC 3337979. PMID 22531176.
Dib-Hajj SD, Yang Y, Black JA, Waxman SG (January 2013). "The Na(V)1.7 sodium channel: from molecule to man". Nature Reviews. Neuroscience. 14 (1): 49–62. doi:10.1038/nrn3404. PMID 23232607. S2CID 5489010.
Tang Z, Chen Z, Tang B, Jiang H (September 2015). "Primary erythromelalgia: a review". Orphanet Journal of Rare Diseases. 10: 127. doi:10.1186/s13023-015-0347-1. PMC 4589109. PMID 26419464.
Drenth JP, Michiels JJ (June 1994). "Erythromelalgia and erythermalgia: diagnostic differentiation". International Journal of Dermatology. 33 (6): 393–397. doi:10.1111/j.1365-4362.1994.tb04037.x. PMID 8056469. S2CID 3033396.
Golshani AE, Kamdar AA, Spence SC, Beckmann NM (August 2014). "Congenital indifference to pain: an illustrated case report and literature review". Journal of Radiology Case Reports. 8 (8): 16–23. doi:10.3941/jrcr.v8i8.2194. PMC 4242143. PMID 25426241.
Fertleman CR, Baker MD, Parker KA, Moffatt S, Elmslie FV, Abrahamsen B, et al. (December 2006). "SCN9A mutations in paroxysmal extreme pain disorder: allelic variants underlie distinct channel defects and phenotypes". Neuron. 52 (5): 767–774. doi:10.1016/j.neuron.2006.10.006. PMID 17145499. S2CID 11715780.
Peddareddygari LR, Oberoi K, Grewal RP (2014-09-18). "Congenital insensitivity to pain: a case report and review of the literature". Case Reports in Neurological Medicine. 2014: 141953. doi:10.1155/2014/141953. PMC 4182687. PMID 25309764.
Devigili G, Eleopra R, Pierro T, Lombardi R, Rinaldo S, Lettieri C, et al. (September 2014). "Paroxysmal itch caused by gain-of-function Nav1.7 mutation". Pain. 155 (9): 1702–1707. doi:10.1016/j.pain.2014.05.006. PMID 24820863. S2CID 24462006.
Gingras J, Smith S, Matson DJ, Johnson D, Nye K, Couture L, et al. (2014). "Global Nav1.7 knockout mice recapitulate the phenotype of human congenital indifference to pain". PLOS ONE. 9 (9): e105895. Bibcode:2014PLoSO...9j5895G. doi:10.1371/journal.pone.0105895. PMC 4154897. PMID 25188265.
Waxman SG (December 2006). "Neurobiology: a channel sets the gain on pain". Nature. 444 (7121): 831–832. Bibcode:2006Natur.444..831W. doi:10.1038/444831a. PMID 17167466. S2CID 4300362.
Dib-Hajj SD, Cummins TR, Black JA, Waxman SG (November 2007). "From genes to pain: Na v 1.7 and human pain disorders". Trends in Neurosciences. 30 (11): 555–563. doi:10.1016/j.tins.2007.08.004. PMID 17950472. S2CID 15131517.
Bagal SK, Chapman ML, Marron BE, Prime R, Storer RI, Swain NA (August 2014). "Recent progress in sodium channel modulators for pain". Bioorganic & Medicinal Chemistry Letters. 24 (16): 3690–3699. doi:10.1016/j.bmcl.2014.06.038. PMID 25060923.
Minett MS, Pereira V, Sikandar S, Matsuyama A, Lolignier S, Kanellopoulos AH, et al. (December 2015). "Endogenous opioids contribute to insensitivity to pain in humans and mice lacking sodium channel Nav1.7". Nature Communications. 6: 8967. Bibcode:2015NatCo...6.8967M. doi:10.1038/ncomms9967. PMC 4686868. PMID 26634308.
Flinspach M, Xu Q, Piekarz AD, Fellows R, Hagan R, Gibbs A, et al. (January 2017). "Insensitivity to pain induced by a potent selective closed-state Nav1.7 inhibitor". Scientific Reports. 7: 39662. Bibcode:2017NatSR...739662F. doi:10.1038/srep39662. PMC 5206724. PMID 28045073.
Moreno AM, Alemán F, Catroli GF, Hunt M, Hu M, Dailamy A, et al. (March 2021). "Long-lasting analgesia via targeted in situ repression of Na_V1.7 in mice". Science Translational Medicine. 13 (584): eaay9056. doi:10.1126/scitranslmed.aay9056. PMC 8830379. PMID 33692134. S2CID 232170826.

ncbi.nlm.nih.gov

"Human PubMed Reference:". National Center for Biotechnology Information, U.S. National Library of Medicine.
"Mouse PubMed Reference:". National Center for Biotechnology Information, U.S. National Library of Medicine.
Klugbauer N, Lacinova L, Flockerzi V, Hofmann F (March 1995). "Structure and functional expression of a new member of the tetrodotoxin-sensitive voltage-activated sodium channel family from human neuroendocrine cells". The EMBO Journal. 14 (6): 1084–1090. doi:10.1002/j.1460-2075.1995.tb07091.x. PMC 398185. PMID 7720699.
Rush AM, Dib-Hajj SD, Liu S, Cummins TR, Black JA, Waxman SG (May 2006). "A single sodium channel mutation produces hyper- or hypoexcitability in different types of neurons". Proceedings of the National Academy of Sciences of the United States of America. 103 (21): 8245–8250. Bibcode:2006PNAS..103.8245R. doi:10.1073/pnas.0602813103. PMC 1472458. PMID 16702558.
Cummins TR, Howe JR, Waxman SG (December 1998). "Slow closed-state inactivation: a novel mechanism underlying ramp currents in cells expressing the hNE/PN1 sodium channel". The Journal of Neuroscience. 18 (23): 9607–9619. doi:10.1523/JNEUROSCI.18-23-09607.1998. PMC 6793269. PMID 9822722.
Shekdar K, Langer J, Venkatachalan S, Schmid L, Anobile J, Shah P, et al. (May 2021). "Cell engineering method using fluorogenic oligonucleotide signaling probes and flow cytometry". Biotechnology Letters. 43 (5): 949–958. doi:10.1007/s10529-021-03101-5. PMC 7937778. PMID 33683511.
Nassar MA, Stirling LC, Forlani G, Baker MD, Matthews EA, Dickenson AH, et al. (August 2004). "Nociceptor-specific gene deletion reveals a major role for Nav1.7 (PN1) in acute and inflammatory pain". Proceedings of the National Academy of Sciences of the United States of America. 101 (34): 12706–12711. Bibcode:2004PNAS..10112706N. doi:10.1073/pnas.0404915101. PMC 515119. PMID 15314237.
Minett MS, Nassar MA, Clark AK, Passmore G, Dickenson AH, Wang F, et al. (April 2012). "Distinct Nav1.7-dependent pain sensations require different sets of sensory and sympathetic neurons". Nature Communications. 3 (4): 791. Bibcode:2012NatCo...3..791M. doi:10.1038/ncomms1795. PMC 3337979. PMID 22531176.
Tang Z, Chen Z, Tang B, Jiang H (September 2015). "Primary erythromelalgia: a review". Orphanet Journal of Rare Diseases. 10: 127. doi:10.1186/s13023-015-0347-1. PMC 4589109. PMID 26419464.
Golshani AE, Kamdar AA, Spence SC, Beckmann NM (August 2014). "Congenital indifference to pain: an illustrated case report and literature review". Journal of Radiology Case Reports. 8 (8): 16–23. doi:10.3941/jrcr.v8i8.2194. PMC 4242143. PMID 25426241.
Peddareddygari LR, Oberoi K, Grewal RP (2014-09-18). "Congenital insensitivity to pain: a case report and review of the literature". Case Reports in Neurological Medicine. 2014: 141953. doi:10.1155/2014/141953. PMC 4182687. PMID 25309764.
Gingras J, Smith S, Matson DJ, Johnson D, Nye K, Couture L, et al. (2014). "Global Nav1.7 knockout mice recapitulate the phenotype of human congenital indifference to pain". PLOS ONE. 9 (9): e105895. Bibcode:2014PLoSO...9j5895G. doi:10.1371/journal.pone.0105895. PMC 4154897. PMID 25188265.
Minett MS, Pereira V, Sikandar S, Matsuyama A, Lolignier S, Kanellopoulos AH, et al. (December 2015). "Endogenous opioids contribute to insensitivity to pain in humans and mice lacking sodium channel Nav1.7". Nature Communications. 6: 8967. Bibcode:2015NatCo...6.8967M. doi:10.1038/ncomms9967. PMC 4686868. PMID 26634308.
Flinspach M, Xu Q, Piekarz AD, Fellows R, Hagan R, Gibbs A, et al. (January 2017). "Insensitivity to pain induced by a potent selective closed-state Nav1.7 inhibitor". Scientific Reports. 7: 39662. Bibcode:2017NatSR...739662F. doi:10.1038/srep39662. PMC 5206724. PMID 28045073.
Moreno AM, Alemán F, Catroli GF, Hunt M, Hu M, Dailamy A, et al. (March 2021). "Long-lasting analgesia via targeted in situ repression of Na_V1.7 in mice". Science Translational Medicine. 13 (584): eaay9056. doi:10.1126/scitranslmed.aay9056. PMC 8830379. PMID 33692134. S2CID 232170826.

pitt.edu

rosslab.neurobio.pitt.edu

"An SCN9A variant, known to cause pain, is now found to cause itch" (PDF). Pain.

semanticscholar.org

api.semanticscholar.org

Catterall WA, Goldin AL, Waxman SG (December 2005). "International Union of Pharmacology. XLVII. Nomenclature and structure-function relationships of voltage-gated sodium channels". Pharmacological Reviews. 57 (4): 397–409. doi:10.1124/pr.57.4.4. PMID 16382098. S2CID 7332624.
Raymond CK, Castle J, Garrett-Engele P, Armour CD, Kan Z, Tsinoremas N, et al. (October 2004). "Expression of alternatively spliced sodium channel alpha-subunit genes. Unique splicing patterns are observed in dorsal root ganglia". The Journal of Biological Chemistry. 279 (44): 46234–46241. doi:10.1074/jbc.M406387200. PMID 15302875. S2CID 17753425.
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Martz L (2014). "Nav-i-gating antibodies for pain". Science-Business EXchange. 7 (23): 662. doi:10.1038/scibx.2014.662. ISSN 1945-3477.
McMahon S, Koltzenburg M, Tracey I, Turk DC (2013). Wall & Melzack's Textbook of Pain (6 ed.). Elsevier Health Sciences. p. 508. ISBN 978-0-7020-4059-7. OCLC 1008841684.