Analysis of information sources in references of the Wikipedia article "Vasoconstriction" in English language version.
These results suggest that U-46619 elicits contraction of rat caudal arterial smooth muscle by activating Ca2+ entry from the extracellular space, which may or may not involve Ca2+-induced Ca2+ release from the SR (sarcoplasmic reticulum). ... A key step in the contractile response to U-46619 appears to be the entry of extracellular Ca2+, since it was abolished by removal of extracellular Ca2+ (Figure 2A). ... In the rat caudal artery, U-46619-mediated contractile responses have an absolute requirement for Ca2+, which enters from the extracellular pool, is independent of intracellular Ca2+ stores and is blocked by ROK inhibition.
These results suggest that U-46619 elicits contraction of rat caudal arterial smooth muscle by activating Ca2+ entry from the extracellular space, which may or may not involve Ca2+-induced Ca2+ release from the SR (sarcoplasmic reticulum). ... A key step in the contractile response to U-46619 appears to be the entry of extracellular Ca2+, since it was abolished by removal of extracellular Ca2+ (Figure 2A). ... In the rat caudal artery, U-46619-mediated contractile responses have an absolute requirement for Ca2+, which enters from the extracellular pool, is independent of intracellular Ca2+ stores and is blocked by ROK inhibition.
These results suggest that U-46619 elicits contraction of rat caudal arterial smooth muscle by activating Ca2+ entry from the extracellular space, which may or may not involve Ca2+-induced Ca2+ release from the SR (sarcoplasmic reticulum). ... A key step in the contractile response to U-46619 appears to be the entry of extracellular Ca2+, since it was abolished by removal of extracellular Ca2+ (Figure 2A). ... In the rat caudal artery, U-46619-mediated contractile responses have an absolute requirement for Ca2+, which enters from the extracellular pool, is independent of intracellular Ca2+ stores and is blocked by ROK inhibition.