«Multiple endocrine neoplasia type 1 (MEN1)». Best Practice & Research. Clinical Endocrinology & Metabolism24 (3): 355-70. June 2010. PMID20833329. doi:10.1016/j.beem.2010.07.003.
«Multiple endocrine neoplasia type 1: new clinical and basic findings». Trends in Endocrinology and Metabolism12 (4): 173-8. 1 de junio de 2001. PMID11295574. doi:10.1016/s1043-2760(00)00372-6.
«Clonality of parathyroid tumors in familial multiple endocrine neoplasia type 1». The New England Journal of Medicine321 (4): 213-8. July 1989. PMID2568586. doi:10.1056/nejm198907273210402.
«Multiple endocrine neoplasia type 1 (MEN1): analysis of 1336 mutations reported in the first decade following identification of the gene». Human Mutation29 (1): 22-32. January 2008. PMID17879353. doi:10.1002/humu.20605.
«Menin interacts with the AP1 transcription factor JunD and represses JunD-activated transcription». Cell96 (1): 143-52. January 1999. PMID9989505. doi:10.1016/s0092-8674(00)80967-8.
«Menin, the multiple endocrine neoplasia type 1 gene product, exhibits GTP-hydrolyzing activity in the presence of the tumor metastasis suppressor nm23». The Journal of Biological Chemistry277 (41): 38197-204. 2002. PMID12145286. doi:10.1074/jbc.M204132200.
«Identification of MEN1 gene mutations in sporadic carcinoid tumors of the lung». Human Molecular Genetics6 (13): 2285-90. December 1997. PMID9361035. doi:10.1093/hmg/6.13.2285.
«The tumor suppressor protein menin interacts with NF-kappaB proteins and inhibits NF-kappaB-mediated transactivation». Oncogene20 (36): 4917-25. August 2001. PMID11526476. doi:10.1038/sj.onc.1204529.
«Menin interacts with the AP1 transcription factor JunD and represses JunD-activated transcription». Cell96 (1): 143-52. January 1999. PMID9989505. doi:10.1016/S0092-8674(00)80967-8.
«Menin's interaction with glial fibrillary acidic protein and vimentin suggests a role for the intermediate filament network in regulating menin activity». Experimental Cell Research278 (2): 175-83. August 2002. PMID12169273. doi:10.1006/excr.2002.5575.
«Multiple endocrine neoplasia type 1 (MEN1)». Best Practice & Research. Clinical Endocrinology & Metabolism24 (3): 355-70. June 2010. PMID20833329. doi:10.1016/j.beem.2010.07.003.
«Multiple endocrine neoplasia type 1: new clinical and basic findings». Trends in Endocrinology and Metabolism12 (4): 173-8. 1 de junio de 2001. PMID11295574. doi:10.1016/s1043-2760(00)00372-6.
«Clonality of parathyroid tumors in familial multiple endocrine neoplasia type 1». The New England Journal of Medicine321 (4): 213-8. July 1989. PMID2568586. doi:10.1056/nejm198907273210402.
«Multiple endocrine neoplasia type 1 (MEN1): analysis of 1336 mutations reported in the first decade following identification of the gene». Human Mutation29 (1): 22-32. January 2008. PMID17879353. doi:10.1002/humu.20605.
«Menin interacts with the AP1 transcription factor JunD and represses JunD-activated transcription». Cell96 (1): 143-52. January 1999. PMID9989505. doi:10.1016/s0092-8674(00)80967-8.
«Menin, the multiple endocrine neoplasia type 1 gene product, exhibits GTP-hydrolyzing activity in the presence of the tumor metastasis suppressor nm23». The Journal of Biological Chemistry277 (41): 38197-204. 2002. PMID12145286. doi:10.1074/jbc.M204132200.
«Mutations of the MEN1 tumor suppressor gene in pituitary tumors». Cancer Research57 (24): 5446-51. December 1997. PMID9407947.
«Identification of MEN1 gene mutations in sporadic carcinoid tumors of the lung». Human Molecular Genetics6 (13): 2285-90. December 1997. PMID9361035. doi:10.1093/hmg/6.13.2285.
«Menin associates with FANCD2, a protein involved in repair of DNA damage». Cancer Research63 (14): 4204-10. July 2003. PMID12874027.
«The tumor suppressor protein menin interacts with NF-kappaB proteins and inhibits NF-kappaB-mediated transactivation». Oncogene20 (36): 4917-25. August 2001. PMID11526476. doi:10.1038/sj.onc.1204529.
«Menin interacts with the AP1 transcription factor JunD and represses JunD-activated transcription». Cell96 (1): 143-52. January 1999. PMID9989505. doi:10.1016/S0092-8674(00)80967-8.
«Menin's interaction with glial fibrillary acidic protein and vimentin suggests a role for the intermediate filament network in regulating menin activity». Experimental Cell Research278 (2): 175-83. August 2002. PMID12169273. doi:10.1006/excr.2002.5575.