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کمای القایی (Persian Wikipedia)

Analysis of information sources in references of the Wikipedia article "کمای القایی" in Persian language version.

refsWebsite
Global rank Persian rank
7doi.org
2nd place
2nd place
7nih.gov
4th place
5th place
3web.archive.org
1st place
1st place
1scahq.org
low place
low place
1neuroanesthesia.info
low place
low place
1trauma.org
low place
low place

doi.org

  • Jackson AC (2016). "Human Rabies: a 2016 Update". Curr Infect Dis Rep (Review). 18 (11): 38. doi:10.1007/s11908-016-0540-y. PMID 27730539.
  • Zeiler FA, Jackson AC (2016). "Critical Appraisal of the Milwaukee Protocol for Rabies: This Failed Approach Should Be Abandoned". Can J Neurol Sci (Review). 43 (1): 44–51. doi:10.1017/cjn.2015.331. PMID 26639059.
  • "Use of barbiturates in the control of intracranial hypertension". Journal of Neurotrauma. Mary Ann Liebert, Inc. 17 (6–7): 527–30. 2000. doi:10.1089/neu.2000.17.527. PMID 10937896.
  • Lee MW, Deppe SA, Sipperly ME, Barrette RR, Thompson DR (1994). "The efficacy of barbiturate coma in the management of uncontrolled intracranial hypertension following neurosurgical trauma". J. Neurotrauma. 11 (3): 325–31. doi:10.1089/neu.1994.11.325. PMID 7996586.
  • Nordby HK, Nesbakken R (1984). "The effect of high dose barbiturate decompression after severe head injury. A controlled clinical trial". Acta Neurochir (Wien). 72 (3–4): 157–66. doi:10.1007/BF01406868. PMID 6382945.
  • Schwartz ML, Tator CH, Rowed DW, Reid SR, Meguro K, Andrews DF (1984). "The University of Toronto head injury treatment study: a prospective, randomized comparison of pentobarbital and mannitol". Can J Neurol Sci. 11 (4): 434–40. doi:10.1017/s0317167100045960. PMID 6440704.
  • Schalén W, Sonesson B, Messeter K, Nordström G, Nordström CH (1992). "Clinical outcome and cognitive impairment in patients with severe head injuries treated with barbiturate coma". Acta Neurochir (Wien). 117 (3–4): 153–9. doi:10.1007/BF01400613. PMID 1414516.

neuroanesthesia.info

  • "Cerebral protection and resuscitation". CNS Clinic – Jordan – Amman. Archived from the original on 4 November 2020. Retrieved 16 April 2016. The primary mechanism of protection involves a reduction in CMRo2 of up to 55% to 60% at which point the EEG becomes isoelectric.

nih.gov

pubmed.ncbi.nlm.nih.gov

  • Jackson AC (2016). "Human Rabies: a 2016 Update". Curr Infect Dis Rep (Review). 18 (11): 38. doi:10.1007/s11908-016-0540-y. PMID 27730539.
  • Zeiler FA, Jackson AC (2016). "Critical Appraisal of the Milwaukee Protocol for Rabies: This Failed Approach Should Be Abandoned". Can J Neurol Sci (Review). 43 (1): 44–51. doi:10.1017/cjn.2015.331. PMID 26639059.
  • "Use of barbiturates in the control of intracranial hypertension". Journal of Neurotrauma. Mary Ann Liebert, Inc. 17 (6–7): 527–30. 2000. doi:10.1089/neu.2000.17.527. PMID 10937896.
  • Lee MW, Deppe SA, Sipperly ME, Barrette RR, Thompson DR (1994). "The efficacy of barbiturate coma in the management of uncontrolled intracranial hypertension following neurosurgical trauma". J. Neurotrauma. 11 (3): 325–31. doi:10.1089/neu.1994.11.325. PMID 7996586.
  • Nordby HK, Nesbakken R (1984). "The effect of high dose barbiturate decompression after severe head injury. A controlled clinical trial". Acta Neurochir (Wien). 72 (3–4): 157–66. doi:10.1007/BF01406868. PMID 6382945.
  • Schwartz ML, Tator CH, Rowed DW, Reid SR, Meguro K, Andrews DF (1984). "The University of Toronto head injury treatment study: a prospective, randomized comparison of pentobarbital and mannitol". Can J Neurol Sci. 11 (4): 434–40. doi:10.1017/s0317167100045960. PMID 6440704.
  • Schalén W, Sonesson B, Messeter K, Nordström G, Nordström CH (1992). "Clinical outcome and cognitive impairment in patients with severe head injuries treated with barbiturate coma". Acta Neurochir (Wien). 117 (3–4): 153–9. doi:10.1007/BF01400613. PMID 1414516.

scahq.org

trauma.org

  • "Barbiturate Coma". Trauma.org. Archived from the original on 19 August 2016. Retrieved 16 April 2016. Therapeutic EEG response: burst suppression or cortical electrical silence (with preservation of SSEP and BAEF).

web.archive.org

  • Grocott HP. "Update on Techniques for Neuroprotection during Hypothermic Arrest" (PDF). Society of Cardiovascular Anesthesiologists. Archived from the original (PDF) on 23 April 2016. Retrieved 14 April 2016. approximately 60% of CMRO2 is utilized for neuronal function (with the remainder being required for cellular integrity)
  • "Cerebral protection and resuscitation". CNS Clinic – Jordan – Amman. Archived from the original on 4 November 2020. Retrieved 16 April 2016. The primary mechanism of protection involves a reduction in CMRo2 of up to 55% to 60% at which point the EEG becomes isoelectric.
  • "Barbiturate Coma". Trauma.org. Archived from the original on 19 August 2016. Retrieved 16 April 2016. Therapeutic EEG response: burst suppression or cortical electrical silence (with preservation of SSEP and BAEF).