Perméabilité intestinale (French Wikipedia)

Analysis of information sources in references of the Wikipedia article "Perméabilité intestinale" in French language version.

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  • Fasano, A., « Leaky Gut and Autoimmune Diseases », Clinical Reviews in Allergy & Immunology, vol. 42, no 1,‎ , p. 71–78 (PMID 22109896, DOI 10.1007/s12016-011-8291-x).
  • (en) Suzuki, « Regulation of intestinal epithelial permeability by tight junctions », Cellular and Molecular Life Sciences, vol. 70, no 4,‎ , p. 631–659 (ISSN 1420-682X, PMID 22782113, DOI 10.1007/s00018-012-1070-x).
  • Fasano A, « Zonulin and its regulation of intestinal barrier function: the biological door to inflammation, autoimmunity, and cancer », Physiol. Rev., vol. 91, no 1,‎ , p. 151–75 (PMID 21248165, DOI 10.1152/physrev.00003.2008).
  • O'Hara et Buret, « Mechanisms of intestinal tight junctional disruption during infection », Frontiers in Bioscience, vol. 13, no 13,‎ , p. 7008–21 (PMID 18508712, DOI 10.2741/3206).
  • Groschwitz et Hogan, « Intestinal Barrier Function: Molecular Regulation and Disease Pathogenesis », The Journal of Allergy and Clinical Immunology, vol. 124, no 1,‎ , p. 3–22 (ISSN 0091-6749, PMID 19560575, PMCID 4266989, DOI 10.1016/j.jaci.2009.05.038).
  • Khan et Asif, « Transcriptional Regulators of Claudins in Epithelial Tight Junctions », Mediators of Inflammation, vol. 2015,‎ , p. 1–6 (ISSN 0962-9351, PMID 25948882, PMCID 4407569, DOI 10.1155/2015/219843).
  • Näslund et Hellström, « Appetite signaling: from gut peptides and enteric nerves to brain », Physiology & Behavior, vol. 92, nos 1–2,‎ , p. 256–262 (ISSN 0031-9384, PMID 17582445, DOI 10.1016/j.physbeh.2007.05.017).
  • Mariarosaria Di Pierro, Ruliang Lu, Sergio Uzzau et Wenle Wang, « Zonula Occludens Toxin Structure-Function Analysis », Journal of Biological Chemistry, vol. 276, no 22,‎ , p. 19160–19165 (ISSN 0021-9258 et 1083-351X, DOI 10.1074/jbc.m009674200, lire en ligne, consulté le ).
  • « Intestinal permeability in coeliac disease: insight into mechanisms and relevance to pathogenesis », Gut, vol. 61, no 9,‎ , p. 1355–64 (PMID 21890812, DOI 10.1136/gutjnl-2011-300327) :

    « Changes in intestinal paracellular and transcellular permeability appear secondary to the abnormal immune reaction induced by gluten. Gliadin was suggested to increase junction permeability to small molecules through the release of prehaptoglobin-2. Environmental triggers of CD other than gliadin may also promote changes in permeability. Intestinal infection and iron deficiency can stimulate the expression of the transferrin receptor (TfR) CD71 in enterocytes. ... Once established, the alterations in intestinal permeability, notably the retro-transport of IgA-gliadin peptides, might self-sustain the inflammatory immune responses and perpetuate a vicious circle »

    .
  • Francesco Ciccia, Giuliana Guggino, Aroldo Rizzo et Riccardo Alessandro, « Dysbiosis and zonulin upregulation alter gut epithelial and vascular barriers in patients with ankylosing spondylitis », Annals of the Rheumatic Diseases, vol. 76, no 6,‎ , p. 1123–1132 (ISSN 0003-4967 et 1468-2060, DOI 10.1136/annrheumdis-2016-210000, lire en ligne, consulté le ).

issn.org

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nhs.uk

nih.gov

ncbi.nlm.nih.gov

  • Fasano, A., « Leaky Gut and Autoimmune Diseases », Clinical Reviews in Allergy & Immunology, vol. 42, no 1,‎ , p. 71–78 (PMID 22109896, DOI 10.1007/s12016-011-8291-x).
  • (en) Suzuki, « Regulation of intestinal epithelial permeability by tight junctions », Cellular and Molecular Life Sciences, vol. 70, no 4,‎ , p. 631–659 (ISSN 1420-682X, PMID 22782113, DOI 10.1007/s00018-012-1070-x).
  • Fasano A, « Zonulin and its regulation of intestinal barrier function: the biological door to inflammation, autoimmunity, and cancer », Physiol. Rev., vol. 91, no 1,‎ , p. 151–75 (PMID 21248165, DOI 10.1152/physrev.00003.2008).
  • O'Hara et Buret, « Mechanisms of intestinal tight junctional disruption during infection », Frontiers in Bioscience, vol. 13, no 13,‎ , p. 7008–21 (PMID 18508712, DOI 10.2741/3206).
  • Groschwitz et Hogan, « Intestinal Barrier Function: Molecular Regulation and Disease Pathogenesis », The Journal of Allergy and Clinical Immunology, vol. 124, no 1,‎ , p. 3–22 (ISSN 0091-6749, PMID 19560575, PMCID 4266989, DOI 10.1016/j.jaci.2009.05.038).
  • Khan et Asif, « Transcriptional Regulators of Claudins in Epithelial Tight Junctions », Mediators of Inflammation, vol. 2015,‎ , p. 1–6 (ISSN 0962-9351, PMID 25948882, PMCID 4407569, DOI 10.1155/2015/219843).
  • Näslund et Hellström, « Appetite signaling: from gut peptides and enteric nerves to brain », Physiology & Behavior, vol. 92, nos 1–2,‎ , p. 256–262 (ISSN 0031-9384, PMID 17582445, DOI 10.1016/j.physbeh.2007.05.017).
  • « Intestinal permeability in coeliac disease: insight into mechanisms and relevance to pathogenesis », Gut, vol. 61, no 9,‎ , p. 1355–64 (PMID 21890812, DOI 10.1136/gutjnl-2011-300327) :

    « Changes in intestinal paracellular and transcellular permeability appear secondary to the abnormal immune reaction induced by gluten. Gliadin was suggested to increase junction permeability to small molecules through the release of prehaptoglobin-2. Environmental triggers of CD other than gliadin may also promote changes in permeability. Intestinal infection and iron deficiency can stimulate the expression of the transferrin receptor (TfR) CD71 in enterocytes. ... Once established, the alterations in intestinal permeability, notably the retro-transport of IgA-gliadin peptides, might self-sustain the inflammatory immune responses and perpetuate a vicious circle »

    .