Haimovitz-Friedman A (1994). „Ionizing radiation acts on cellular membranes to generate ceramide and initiate apoptosis”. J. Exp. Med.180 (2), 525–35. o. DOI:10.1084/jem.180.2.525. PMID8046331. PMC2191598.
Hannun, Y.A. (2008). „Principles of bioactive lipid signalling: lessons from sphingolipids”. Nature Reviews Molecular Cell Biology9 (2), 139–150. o. DOI:10.1038/nrm2329. PMID18216770.
Kitatani K, Idkowiak-Baldys J, Hannun YA (2008). „The sphingolipid salvage pathway in ceramide metabolism and signaling”. Cell Signaling20 (6), 1010–1018. o. DOI:10.1016/j.cellsig.2007.12.006. PMID18191382. PMC2422835.
Wu D, Ren Z, Pae M, Guo W, Cui X, Merrill AH, Meydani SN (2007). „Aging up-regulates expression of inflammatory mediators in mouse adipose tissue”. The Journal of Immunology179 (7), 4829–39. o. DOI:10.4049/jimmunol.179.7.4829. PMID17878382.
Holland WL, Bikman BT, Wang LP, Yuguang G, Sargent KM, Bulchand S, Knotts TA, Shui G, Clegg DJ, Wenk MR, Pagliassotti MJ, Scherer PE, Summers SA (2011). „Lipid-induced insulin resistance mediated by the proinflammatory receptor TLR4 requires saturated fatty acid-induced ceramide biosynthesis in mice”. Journal of Clinical Investigation121 (5), 1858–1870. o. DOI:10.1172/JCI43378. PMID21490391. PMC3083776.
Chavez JA, Siddique MM, Wang ST, Ching J, Shayman JA, Summers SA (2014). „Ceramides and glucosylceramides are independent antagonists of insulin signaling”. Journal of Biological Chemistry289 (2), 723–734. o. DOI:10.1074/jbc.M113.522847. PMID24214972. PMC3887200.
Li Z, Basterr MJ, Hailemariam TK, Hojjati MR, Lu S, Liu J, Liu R, Zhou H, Jiang XC (2005). „The effect of dietary sphingolipids on plasma sphingomyelin metabolism and atherosclerosis”. Biochimica et Biophysica Acta – Molecular and Cell Biology of Lipids1735 (2), 130–134. o. DOI:10.1016/j.bbalip.2005.05.004. PMID15967715.
Mehra VC, Jackson E, Zhang XM, Jiang XC, Dobrucki LW, Yu J, Bernatchez P, Sinusas AJ, Shulman GI, Sessa WC, Yarovinsky TO, Bender JR (2014). „Ceramide-activated phosphatase mediates fatty acid-induced endothelial VEGF resistance and impaired angiogenesis”. The American Journal of Pathology184 (5), 1562–1576. o. DOI:10.1016/j.ajpath.2014.01.009. PMID24606881. PMC4005977.
Dbaibo GS, Pushkareva MY, Rachid RA, Alter N, Smyth MJ, Obeid LM, Hannun YA (1998). „p53-dependent ceramide response to genotoxic stress”. J. Clin. Invest.102 (2), 329–339. o. DOI:10.1172/JCI1180. PMID9664074. PMC508891.
Rotolo JA, Zhang J, Donepudi M, Lee H, Fuks Z, Kolesnick R (2005). „Caspase-dependent and -independent activation of acid sphingomyelinase signaling”. J. Biol. Chem.280 (28), 26425–34. o. DOI:10.1074/jbc.M414569200. PMID15849201.
Dbaibo GS, El-Assaad W, Krikorian A, Liu B, Diab K, Idriss NZ, El-Sabban M, Driscoll TA, Perry DK, Hannun YA (2001). „Ceramide generation by two distinct pathways in tumor necrosis factor alpha-induced cell death”. FEBS Letters503 (1), 7–12. o. DOI:10.1016/S0014-5793(01)02625-4. PMID11513845.
Taha TA, Mullen TD, Obeid LM (2006). „A house divided: ceramide, sphingosine, and sphingosine-1-phosphate in programmed cell death”. Biochimica et Biophysica Acta (BBA) - Biomembranes1758 (12), 2027–36. o. DOI:10.1016/j.bbamem.2006.10.018. PMID17161984. PMC1766198.
Merleev, AA (2022. augusztus 22.). „Biogeographic and disease-specific alterations in epidermal lipid composition and single-cell analysis of acral keratinocytes.”. JCI Insight7 (16). DOI:10.1172/jci.insight.159762. PMID35900871. PMC9462509.
Hill JR, Wertz PW (2009). „Structures of the ceramides from porcine palatal stratum corneum”. Lipids44 (3), 291–295. o. DOI:10.1007/s11745-009-3283-9. PMID19184160.
Garidel P, Fölting B, Schaller I, Kerth A (2010). „The microstructure of the stratum corneum lipid barrier: mid-infrared spectroscopic studies of hydrated ceramide:palmitic acid:cholesterol model systems”. Biophysical Chemistry150 (1–3), 144–156. o. DOI:10.1016/j.bpc.2010.03.008. PMID20457485.
Motta, S (1993. szeptember 8.). „Ceramide composition of the psoriatic scale.”. Biochimica et Biophysica Acta (BBA) - Molecular Basis of Disease1182 (2), 147–51. o. DOI:10.1016/0925-4439(93)90135-n. PMID8357845.
Merleev, Alexander A. (2022. augusztus 22.). „Biogeographic and disease-specific alterations in epidermal lipid composition and single-cell analysis of acral keratinocytes”. JCI Insight7 (16). DOI:10.1172/jci.insight.159762. PMID35900871. PMC9462509.
Febbraio, Mark (2014). „Role of interleukins in obesity: implications for metabolic disease”. Trends in Endocrinology and Metabolism25 (6), 312–319. o. DOI:10.1016/j.tem.2014.02.004. PMID24698032.
Hallahan DE (1996). „Radiation-mediated gene expression in the pathogenesis of the clinical radiation response”. Sem. Radiat. Oncol.6 (4), 250–267. o. DOI:10.1016/S1053-4296(96)80021-X. PMID10717183.
Siskind LJ, Kolesnick RN, Colombini M (2002). „Ceramide Channels Increase the Permeability of the Mitochondrial Outer Membrane to Small Proteins”. J. Biol. Chem.277 (30), 26796–803. o. DOI:10.1074/jbc.M200754200. PMID12006562. PMC2246046.
Stiban J, Fistere D, Colombini M (2006). „Dihydroceramide hinders ceramide channel formation: Implications on apoptosis”. Apoptosis11 (5), 773–80. o. DOI:10.1007/s10495-006-5882-8. PMID16532372.
Siskind LJ, Kolesnick RN, Colombini M (2006). „Ceramide forms channels in mitochondrial outer membranes at physiologically relevant concentrations”. Mitochondrion6 (3), 118–25. o. DOI:10.1016/j.mito.2006.03.002. PMID16713754. PMC2246045.
Minamino, Miki (2003). „Bacterial ceramides and sphingophospholipids induce apoptosis of human leukaemic cells”. Microbiology149 (8), 2071–2081. o. DOI:10.1099/mic.0.25922-0. PMID12904547.
Haimovitz-Friedman A (1994). „Ionizing radiation acts on cellular membranes to generate ceramide and initiate apoptosis”. J. Exp. Med.180 (2), 525–35. o. DOI:10.1084/jem.180.2.525. PMID8046331. PMC2191598.
Hannun, Y.A. (2008). „Principles of bioactive lipid signalling: lessons from sphingolipids”. Nature Reviews Molecular Cell Biology9 (2), 139–150. o. DOI:10.1038/nrm2329. PMID18216770.
Kitatani K, Idkowiak-Baldys J, Hannun YA (2008). „The sphingolipid salvage pathway in ceramide metabolism and signaling”. Cell Signaling20 (6), 1010–1018. o. DOI:10.1016/j.cellsig.2007.12.006. PMID18191382. PMC2422835.
Wu D, Ren Z, Pae M, Guo W, Cui X, Merrill AH, Meydani SN (2007). „Aging up-regulates expression of inflammatory mediators in mouse adipose tissue”. The Journal of Immunology179 (7), 4829–39. o. DOI:10.4049/jimmunol.179.7.4829. PMID17878382.
Holland WL, Bikman BT, Wang LP, Yuguang G, Sargent KM, Bulchand S, Knotts TA, Shui G, Clegg DJ, Wenk MR, Pagliassotti MJ, Scherer PE, Summers SA (2011). „Lipid-induced insulin resistance mediated by the proinflammatory receptor TLR4 requires saturated fatty acid-induced ceramide biosynthesis in mice”. Journal of Clinical Investigation121 (5), 1858–1870. o. DOI:10.1172/JCI43378. PMID21490391. PMC3083776.
Chavez JA, Siddique MM, Wang ST, Ching J, Shayman JA, Summers SA (2014). „Ceramides and glucosylceramides are independent antagonists of insulin signaling”. Journal of Biological Chemistry289 (2), 723–734. o. DOI:10.1074/jbc.M113.522847. PMID24214972. PMC3887200.
Li Z, Basterr MJ, Hailemariam TK, Hojjati MR, Lu S, Liu J, Liu R, Zhou H, Jiang XC (2005). „The effect of dietary sphingolipids on plasma sphingomyelin metabolism and atherosclerosis”. Biochimica et Biophysica Acta – Molecular and Cell Biology of Lipids1735 (2), 130–134. o. DOI:10.1016/j.bbalip.2005.05.004. PMID15967715.
Mehra VC, Jackson E, Zhang XM, Jiang XC, Dobrucki LW, Yu J, Bernatchez P, Sinusas AJ, Shulman GI, Sessa WC, Yarovinsky TO, Bender JR (2014). „Ceramide-activated phosphatase mediates fatty acid-induced endothelial VEGF resistance and impaired angiogenesis”. The American Journal of Pathology184 (5), 1562–1576. o. DOI:10.1016/j.ajpath.2014.01.009. PMID24606881. PMC4005977.
Dbaibo GS, Pushkareva MY, Rachid RA, Alter N, Smyth MJ, Obeid LM, Hannun YA (1998). „p53-dependent ceramide response to genotoxic stress”. J. Clin. Invest.102 (2), 329–339. o. DOI:10.1172/JCI1180. PMID9664074. PMC508891.
Rotolo JA, Zhang J, Donepudi M, Lee H, Fuks Z, Kolesnick R (2005). „Caspase-dependent and -independent activation of acid sphingomyelinase signaling”. J. Biol. Chem.280 (28), 26425–34. o. DOI:10.1074/jbc.M414569200. PMID15849201.
Dbaibo GS, El-Assaad W, Krikorian A, Liu B, Diab K, Idriss NZ, El-Sabban M, Driscoll TA, Perry DK, Hannun YA (2001). „Ceramide generation by two distinct pathways in tumor necrosis factor alpha-induced cell death”. FEBS Letters503 (1), 7–12. o. DOI:10.1016/S0014-5793(01)02625-4. PMID11513845.
Taha TA, Mullen TD, Obeid LM (2006). „A house divided: ceramide, sphingosine, and sphingosine-1-phosphate in programmed cell death”. Biochimica et Biophysica Acta (BBA) - Biomembranes1758 (12), 2027–36. o. DOI:10.1016/j.bbamem.2006.10.018. PMID17161984. PMC1766198.
Merleev, AA (2022. augusztus 22.). „Biogeographic and disease-specific alterations in epidermal lipid composition and single-cell analysis of acral keratinocytes.”. JCI Insight7 (16). DOI:10.1172/jci.insight.159762. PMID35900871. PMC9462509.
Hill JR, Wertz PW (2009). „Structures of the ceramides from porcine palatal stratum corneum”. Lipids44 (3), 291–295. o. DOI:10.1007/s11745-009-3283-9. PMID19184160.
Garidel P, Fölting B, Schaller I, Kerth A (2010). „The microstructure of the stratum corneum lipid barrier: mid-infrared spectroscopic studies of hydrated ceramide:palmitic acid:cholesterol model systems”. Biophysical Chemistry150 (1–3), 144–156. o. DOI:10.1016/j.bpc.2010.03.008. PMID20457485.
Motta, S (1993. szeptember 8.). „Ceramide composition of the psoriatic scale.”. Biochimica et Biophysica Acta (BBA) - Molecular Basis of Disease1182 (2), 147–51. o. DOI:10.1016/0925-4439(93)90135-n. PMID8357845.
Merleev, Alexander A. (2022. augusztus 22.). „Biogeographic and disease-specific alterations in epidermal lipid composition and single-cell analysis of acral keratinocytes”. JCI Insight7 (16). DOI:10.1172/jci.insight.159762. PMID35900871. PMC9462509.
Febbraio, Mark (2014). „Role of interleukins in obesity: implications for metabolic disease”. Trends in Endocrinology and Metabolism25 (6), 312–319. o. DOI:10.1016/j.tem.2014.02.004. PMID24698032.
Hallahan DE (1996). „Radiation-mediated gene expression in the pathogenesis of the clinical radiation response”. Sem. Radiat. Oncol.6 (4), 250–267. o. DOI:10.1016/S1053-4296(96)80021-X. PMID10717183.
Siskind LJ, Kolesnick RN, Colombini M (2002). „Ceramide Channels Increase the Permeability of the Mitochondrial Outer Membrane to Small Proteins”. J. Biol. Chem.277 (30), 26796–803. o. DOI:10.1074/jbc.M200754200. PMID12006562. PMC2246046.
Stiban J, Fistere D, Colombini M (2006). „Dihydroceramide hinders ceramide channel formation: Implications on apoptosis”. Apoptosis11 (5), 773–80. o. DOI:10.1007/s10495-006-5882-8. PMID16532372.
Siskind LJ, Kolesnick RN, Colombini M (2006). „Ceramide forms channels in mitochondrial outer membranes at physiologically relevant concentrations”. Mitochondrion6 (3), 118–25. o. DOI:10.1016/j.mito.2006.03.002. PMID16713754. PMC2246045.
Minamino, Miki (2003). „Bacterial ceramides and sphingophospholipids induce apoptosis of human leukaemic cells”. Microbiology149 (8), 2071–2081. o. DOI:10.1099/mic.0.25922-0. PMID12904547.
Haimovitz-Friedman A (1994). „Ionizing radiation acts on cellular membranes to generate ceramide and initiate apoptosis”. J. Exp. Med.180 (2), 525–35. o. DOI:10.1084/jem.180.2.525. PMID8046331. PMC2191598.
Kitatani K, Idkowiak-Baldys J, Hannun YA (2008). „The sphingolipid salvage pathway in ceramide metabolism and signaling”. Cell Signaling20 (6), 1010–1018. o. DOI:10.1016/j.cellsig.2007.12.006. PMID18191382. PMC2422835.
Holland WL, Bikman BT, Wang LP, Yuguang G, Sargent KM, Bulchand S, Knotts TA, Shui G, Clegg DJ, Wenk MR, Pagliassotti MJ, Scherer PE, Summers SA (2011). „Lipid-induced insulin resistance mediated by the proinflammatory receptor TLR4 requires saturated fatty acid-induced ceramide biosynthesis in mice”. Journal of Clinical Investigation121 (5), 1858–1870. o. DOI:10.1172/JCI43378. PMID21490391. PMC3083776.
Chavez JA, Siddique MM, Wang ST, Ching J, Shayman JA, Summers SA (2014). „Ceramides and glucosylceramides are independent antagonists of insulin signaling”. Journal of Biological Chemistry289 (2), 723–734. o. DOI:10.1074/jbc.M113.522847. PMID24214972. PMC3887200.
Mehra VC, Jackson E, Zhang XM, Jiang XC, Dobrucki LW, Yu J, Bernatchez P, Sinusas AJ, Shulman GI, Sessa WC, Yarovinsky TO, Bender JR (2014). „Ceramide-activated phosphatase mediates fatty acid-induced endothelial VEGF resistance and impaired angiogenesis”. The American Journal of Pathology184 (5), 1562–1576. o. DOI:10.1016/j.ajpath.2014.01.009. PMID24606881. PMC4005977.
Dbaibo GS, Pushkareva MY, Rachid RA, Alter N, Smyth MJ, Obeid LM, Hannun YA (1998). „p53-dependent ceramide response to genotoxic stress”. J. Clin. Invest.102 (2), 329–339. o. DOI:10.1172/JCI1180. PMID9664074. PMC508891.
Taha TA, Mullen TD, Obeid LM (2006). „A house divided: ceramide, sphingosine, and sphingosine-1-phosphate in programmed cell death”. Biochimica et Biophysica Acta (BBA) - Biomembranes1758 (12), 2027–36. o. DOI:10.1016/j.bbamem.2006.10.018. PMID17161984. PMC1766198.
Merleev, AA (2022. augusztus 22.). „Biogeographic and disease-specific alterations in epidermal lipid composition and single-cell analysis of acral keratinocytes.”. JCI Insight7 (16). DOI:10.1172/jci.insight.159762. PMID35900871. PMC9462509.
Merleev, Alexander A. (2022. augusztus 22.). „Biogeographic and disease-specific alterations in epidermal lipid composition and single-cell analysis of acral keratinocytes”. JCI Insight7 (16). DOI:10.1172/jci.insight.159762. PMID35900871. PMC9462509.
Siskind LJ, Kolesnick RN, Colombini M (2002). „Ceramide Channels Increase the Permeability of the Mitochondrial Outer Membrane to Small Proteins”. J. Biol. Chem.277 (30), 26796–803. o. DOI:10.1074/jbc.M200754200. PMID12006562. PMC2246046.
Siskind LJ, Kolesnick RN, Colombini M (2006). „Ceramide forms channels in mitochondrial outer membranes at physiologically relevant concentrations”. Mitochondrion6 (3), 118–25. o. DOI:10.1016/j.mito.2006.03.002. PMID16713754. PMC2246045.
