Kaminski AM, Bebenek K, Pedersen LC, Kunkel TA (2021. december 28.). „DNA polymerase mu: An inflexible scaffold for substrate flexibility”. DNA Repair93, 102932. o. DOI:10.1016/j.dnarep.2020.102932. PMID33087269. PMC8713560.
Daley JM, Laan RL, Suresh A, Wilson TE (2005. augusztus 1.). „DNA joint dependence of pol X family polymerase action in nonhomologous end joining”. J. Biol. Chem280 (32), 29030–29037. o. DOI:10.1074/jbc.M505277200. PMID15964833.
Nick McElhinny SA, Ramsden DA (2004. augusztus 1.). „Sibling rivalry: competition between Pol X family members in V(D)J recombination and general double strand break repair”. Immunol. Rev200, 156–64. o. DOI:10.1111/j.0105-2896.2004.00160.x. PMID15242403.
Nick McElhinny SA, Havener JM, Garcia-Diaz M (2005. augusztus 1.). „A gradient of template dependence defines distinct biological roles for family X polymerases in nonhomologous end joining”. Mol. Cell19 (3), 357–66. o. DOI:10.1016/j.molcel.2005.06.012. PMID16061182.
Bertocci B, De Smet A, Berek C, Weill JC, Reynaud CA (2003. augusztus 1.). „Immunoglobulin kappa light chain gene rearrangement is impaired in mice deficient for DNA polymerase mu”. Immunity19 (2), 203–11. o. DOI:10.1016/S1074-7613(03)00203-6. PMID12932354.
Kaminski AM, Chiruvella KK, Ramsden DA, Kunkel TA, Bebenek K, Pedersen LC (2019. szeptember 26.). „Unexpected behavior of DNA polymerase Mu opposite template 8-oxo-7,8-dihydro-2'-guanosine”. Nucleic Acids Res47 (17), 9410-9422. o. DOI:10.1093/nar/gkz680. PMID31435651. PMC6755092.
Lucas D, Escudero B, Ligos JM, Segovia JC, Estrada JC, Terrados G, Blanco L, Samper E, Bernad A (2009. február 1.). „Altered hematopoiesis in mice lacking DNA polymerase mu is due to inefficient double-strand break repair”. PLOS Genet5 (2), e1000389. o. DOI:10.1371/journal.pgen.1000389. PMID19229323. PMC2638008.
Lucas D, Delgado-García JM, Escudero B, Albo C, Aza A, Acín-Pérez R, Torres Y, Moreno P, Enríquez JA, Samper E, Blanco L, Fairén A, Bernad A, Gruart A (2013. január 3.). „Increased learning and brain long-term potentiation in aged mice lacking DNA polymerase μ”. PLoS One8 (1), e53243. o. DOI:10.1371/journal.pone.0053243. PMID23301049. PMC3536760.
Kaminski AM, Bebenek K, Pedersen LC, Kunkel TA (2021. december 28.). „DNA polymerase mu: An inflexible scaffold for substrate flexibility”. DNA Repair93, 102932. o. DOI:10.1016/j.dnarep.2020.102932. PMID33087269. PMC8713560.
Daley JM, Laan RL, Suresh A, Wilson TE (2005. augusztus 1.). „DNA joint dependence of pol X family polymerase action in nonhomologous end joining”. J. Biol. Chem280 (32), 29030–29037. o. DOI:10.1074/jbc.M505277200. PMID15964833.
Nick McElhinny SA, Ramsden DA (2004. augusztus 1.). „Sibling rivalry: competition between Pol X family members in V(D)J recombination and general double strand break repair”. Immunol. Rev200, 156–64. o. DOI:10.1111/j.0105-2896.2004.00160.x. PMID15242403.
Nick McElhinny SA, Havener JM, Garcia-Diaz M (2005. augusztus 1.). „A gradient of template dependence defines distinct biological roles for family X polymerases in nonhomologous end joining”. Mol. Cell19 (3), 357–66. o. DOI:10.1016/j.molcel.2005.06.012. PMID16061182.
Bertocci B, De Smet A, Berek C, Weill JC, Reynaud CA (2003. augusztus 1.). „Immunoglobulin kappa light chain gene rearrangement is impaired in mice deficient for DNA polymerase mu”. Immunity19 (2), 203–11. o. DOI:10.1016/S1074-7613(03)00203-6. PMID12932354.
Kaminski AM, Chiruvella KK, Ramsden DA, Kunkel TA, Bebenek K, Pedersen LC (2019. szeptember 26.). „Unexpected behavior of DNA polymerase Mu opposite template 8-oxo-7,8-dihydro-2'-guanosine”. Nucleic Acids Res47 (17), 9410-9422. o. DOI:10.1093/nar/gkz680. PMID31435651. PMC6755092.
Lucas D, Escudero B, Ligos JM, Segovia JC, Estrada JC, Terrados G, Blanco L, Samper E, Bernad A (2009. február 1.). „Altered hematopoiesis in mice lacking DNA polymerase mu is due to inefficient double-strand break repair”. PLOS Genet5 (2), e1000389. o. DOI:10.1371/journal.pgen.1000389. PMID19229323. PMC2638008.
Lucas D, Delgado-García JM, Escudero B, Albo C, Aza A, Acín-Pérez R, Torres Y, Moreno P, Enríquez JA, Samper E, Blanco L, Fairén A, Bernad A, Gruart A (2013. január 3.). „Increased learning and brain long-term potentiation in aged mice lacking DNA polymerase μ”. PLoS One8 (1), e53243. o. DOI:10.1371/journal.pone.0053243. PMID23301049. PMC3536760.
Kaminski AM, Bebenek K, Pedersen LC, Kunkel TA (2021. december 28.). „DNA polymerase mu: An inflexible scaffold for substrate flexibility”. DNA Repair93, 102932. o. DOI:10.1016/j.dnarep.2020.102932. PMID33087269. PMC8713560.
Kaminski AM, Chiruvella KK, Ramsden DA, Kunkel TA, Bebenek K, Pedersen LC (2019. szeptember 26.). „Unexpected behavior of DNA polymerase Mu opposite template 8-oxo-7,8-dihydro-2'-guanosine”. Nucleic Acids Res47 (17), 9410-9422. o. DOI:10.1093/nar/gkz680. PMID31435651. PMC6755092.
Lucas D, Escudero B, Ligos JM, Segovia JC, Estrada JC, Terrados G, Blanco L, Samper E, Bernad A (2009. február 1.). „Altered hematopoiesis in mice lacking DNA polymerase mu is due to inefficient double-strand break repair”. PLOS Genet5 (2), e1000389. o. DOI:10.1371/journal.pgen.1000389. PMID19229323. PMC2638008.
Lucas D, Delgado-García JM, Escudero B, Albo C, Aza A, Acín-Pérez R, Torres Y, Moreno P, Enríquez JA, Samper E, Blanco L, Fairén A, Bernad A, Gruart A (2013. január 3.). „Increased learning and brain long-term potentiation in aged mice lacking DNA polymerase μ”. PLoS One8 (1), e53243. o. DOI:10.1371/journal.pone.0053243. PMID23301049. PMC3536760.