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(2006) „Following the path of the virus: the exploitation of host DNA repair mechanisms by retroviruses”. ACS Chemical Biology1 (4), 217–26. o. DOI:10.1021/cb600131q. PMID17163676.
(2002) „A common mechanism underlying promiscuous inhibitors from virtual and high-throughput screening”. Journal of Medicinal Chemistry45 (8), 1712–22. o. DOI:10.1021/jm010533y. PMID11931626.
(1991. április 1.) „Serine phosphorylation-independent downregulation of cell-surface CD4 by nef”. Nature350 (6318), 508–11. o. DOI:10.1038/350508a0. PMID2014052.
(1996. március 1.) „Endocytosis of major histocompatibility complex class I molecules is induced by the HIV-1 Nef protein”. Nature Medicine2 (3), 338–42. o. DOI:10.1038/nm0396-338. PMID8612235.
(2003) „The impact of host genetics on HIV infection and disease progression in the era of highly active antiretroviral therapy”. AIDS17 (Suppl 4), S51–S60. o. DOI:10.1097/00002030-200317004-00006. PMID15080180.
(1993) „Genotypic and phenotypic characterization of HIV-1 patients with primary infection”. Science261 (5125), 1179–81. o. DOI:10.1126/science.8356453. PMID8356453.
(2005) „The X4 phenotype of HIV type 1 evolves from R5 in two children of mothers, carrying X4, and is not linked to transmission”. AIDS Research and Human Retroviruses21 (5), 371–8. o. DOI:10.1089/aid.2005.21.371. PMID15929699.
(2003) „Transmission, acute HIV-1 infection and the quest for strategies to prevent infection”. Nature Medicine9 (7), 847–52. o. DOI:10.1038/nm0703-847. PMID12835704.
(2008) „In vitro expression of the HIV-2 genomic RNA is controlled by three distinct internal ribosome entry segments that are regulated by the HIV protease and the Gag polyprotein”. RNA14 (7), 1443–55. o. DOI:10.1261/rna.813608. PMID18495939. PMC2441975.
(2008) „HIV-1 envelope protein binds to and signals through integrin alpha(4)beta(7), the gut mucosal homing receptor for peripheral T cells”. Nature Immunology9 (3), 301–9. o. DOI:10.1038/ni1566. PMID18264102.
(2004) „HIV-1 cell to cell transfer across an Env-induced, actin-dependent synapse”. Journal of Experimental Medicine199 (2), 283–293. o. DOI:10.1084/jem.20030648. PMID14734528. PMC2211771.
(2008) „Avoiding the void: cell-to-cell spread of human viruses”. Nature Reviews Microbiology6 (11), 815–826. o. DOI:10.1038/nrmicro1972. PMID18923409.
(2008. október 1.) „Estimating drug efficacy and viral dynamic parameters: HIV and HCV”. Statistics in Medicine27 (23), 4647–57. o. DOI:10.1002/sim.3116. PMID17960579.
(2009) „Toward an AIDS vaccine: lessons from natural simian immunodeficiency virus infections of African nonhuman primate hosts”. Nature Medicine15 (8), 861–865. o. DOI:10.1038/nm.2013. PMID19661993. PMC2782707.
(2001) „High virus loads in naturally and experimentally SIVagm-infected African green monkeys”. Virology283 (2), 324–31. o. DOI:10.1006/viro.2001.0870. PMID11336557.
(1984) „A new type D retrovirus isolated from macaques with an immunodeficiency syndrome”. Science223 (4636), 602–5. o. DOI:10.1126/science.6695172. PMID6695172.
(2009) „Increased mortality and AIDS-like immunopathology in wild chimpanzees infected with SIVcpz”. Nature460 (7254), 515–519. o. DOI:10.1038/nature08200. PMID19626114. PMC2872475.
(2006. január 18.) „Nef-mediated suppression of T cell activation was lost in a lentiviral lineage that gave rise to HIV-1”. Cell125 (6), 1055–67. o. DOI:10.1016/j.cell.2006.04.033. PMID16777597.
(2002) „Molecular epidemiology of HIV-1 genetic forms and its significance for vaccine development and therapy”. The Lancet Infectious Diseases2 (8), 461–471. o. DOI:10.1016/S1473-3099(02)00343-2. PMID12150845.
(2002) „Estimated global distribution and regional spread of HIV-1 genetic subtypes in the year 2000”. Acquired Immune Deficiency Syndrome29 (2), 184–190. o. DOI:10.1097/00042560-200202010-00013. PMID11832690.
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(1983) „Isolation of human T-cell leukemia virus in acquired immune deficiency syndrome (AIDS)”. Science220 (4599), 865–867. o. DOI:10.1126/science.6601823. PMID6601823.
(1983) „Isolation of a T-lymphotropic retrovirus from a patient at risk for acquired immune deficiency syndrome (AIDS)”. Science220 (4599), 868–871. o. DOI:10.1126/science.6189183. PMID6189183.
(2001) „The origins of acquired immune deficiency syndrome viruses: where and when?”. Philosophical Transactions of the Royal Society B356 (1410), 867–76. o. DOI:10.1098/rstb.2001.0863. PMID11405934. PMC1088480.
(2000) „Origin of HIV type 1 in colonial French equatorial Africa?”. AIDS Research and Human Retroviruses16 (1), 5–8. o. DOI:10.1089/088922200309548. PMID10628811.
(2010) „High GUD incidence in the early 20th century created a particularly permissive time window for the origin and initial spread of epidemic HIV strains”. PLOS One5 (4), e9936. o. DOI:10.1371/journal.pone.0009936. PMID20376191. PMC2848574.
(2012) „HIV-Specific Antibodies Capable of ADCC Are Common in Breastmilk and Are Associated with Reduced Risk of Transmission in Women with High Viral Loads”. PLOS Pathogens8 (6), e1002739. o. DOI:10.1371/journal.ppat.1002739. PMID22719248. PMC3375288.
(2008) „In vitro expression of the HIV-2 genomic RNA is controlled by three distinct internal ribosome entry segments that are regulated by the HIV protease and the Gag polyprotein”. RNA14 (7), 1443–55. o. DOI:10.1261/rna.813608. PMID18495939. PMC2441975.
(2004) „HIV-1 cell to cell transfer across an Env-induced, actin-dependent synapse”. Journal of Experimental Medicine199 (2), 283–293. o. DOI:10.1084/jem.20030648. PMID14734528. PMC2211771.
(2009) „Toward an AIDS vaccine: lessons from natural simian immunodeficiency virus infections of African nonhuman primate hosts”. Nature Medicine15 (8), 861–865. o. DOI:10.1038/nm.2013. PMID19661993. PMC2782707.
(2009) „Increased mortality and AIDS-like immunopathology in wild chimpanzees infected with SIVcpz”. Nature460 (7254), 515–519. o. DOI:10.1038/nature08200. PMID19626114. PMC2872475.
(2001) „The origins of acquired immune deficiency syndrome viruses: where and when?”. Philosophical Transactions of the Royal Society B356 (1410), 867–76. o. DOI:10.1098/rstb.2001.0863. PMID11405934. PMC1088480.
(2010) „High GUD incidence in the early 20th century created a particularly permissive time window for the origin and initial spread of epidemic HIV strains”. PLOS One5 (4), e9936. o. DOI:10.1371/journal.pone.0009936. PMID20376191. PMC2848574.