“CC chemokines induce the generation of killer cells from CD56+ cells”. European Journal of Immunology26 (2): 315–319. (February 1996). doi:10.1002/eji.1830260207. PMID8617297.
“RFLAT-1: a new zinc finger transcription factor that activates RANTES gene expression in T lymphocytes”. Immunity10 (1): 93–103. (January 1999). doi:10.1016/S1074-7613(00)80010-2. PMID10023774.
“JNK MAPK pathway regulates constitutive transcription of CCL5 by human NK cells through SP1”. Journal of Immunology182 (2): 1011–1020. (January 2009). doi:10.4049/jimmunol.182.2.1011. PMID19124744.
“RANTES production by memory phenotype T cells is controlled by a posttranscriptional, TCR-dependent process”. Immunity17 (5): 605–615. (November 2002). doi:10.1016/S1074-7613(02)00456-9. PMID12433367.
“Diverging binding capacities of natural LD78beta isoforms of macrophage inflammatory protein-1alpha to the CC chemokine receptors 1, 3 and 5 affect their anti-HIV-1 activity and chemotactic potencies for neutrophils and eosinophils”. European Journal of Immunology31 (7): 2170–2178. (July 2001). doi:10.1002/1521-4141(200107)31:7<2170::AID-IMMU2170>3.0.CO;2-D. PMID11449371.
“Interaction of RANTES with syndecan-1 and syndecan-4 expressed by human primary macrophages”. Biochimica et Biophysica Acta (BBA) - Biomembranes1617 (1–2): 80–88. (October 2003). doi:10.1016/j.bbamem.2003.09.006. PMID14637022.
“The BBXB motif of RANTES is the principal site for heparin binding and controls receptor selectivity”. The Journal of Biological Chemistry276 (14): 10620–10626. (April 2001). doi:10.1074/jbc.M010867200. PMID11116158.
“Distribution of functional polymorphic variants of inflammation-related genes RANTES and CCR5 in long-lived individuals”. Cytokine58 (1): 10–13. (April 2012). doi:10.1016/j.cyto.2011.12.021. PMID22265023.
“Aggregation of RANTES is responsible for its inflammatory properties. Characterization of nonaggregating, noninflammatory RANTES mutants”. The Journal of Biological Chemistry274 (39): 27505–27512. (September 1999). doi:10.1074/jbc.274.39.27505. PMID10488085.
“CCL5-CCR5-mediated apoptosis in T cells: Requirement for glycosaminoglycan binding and CCL5 aggregation”. The Journal of Biological Chemistry281 (35): 25184–25194. (September 2006). doi:10.1074/jbc.M603912200. PMID16807236.
“CC chemokines induce the generation of killer cells from CD56+ cells”. European Journal of Immunology26 (2): 315–319. (February 1996). doi:10.1002/eji.1830260207. PMID8617297.
“RFLAT-1: a new zinc finger transcription factor that activates RANTES gene expression in T lymphocytes”. Immunity10 (1): 93–103. (January 1999). doi:10.1016/S1074-7613(00)80010-2. PMID10023774.
“JNK MAPK pathway regulates constitutive transcription of CCL5 by human NK cells through SP1”. Journal of Immunology182 (2): 1011–1020. (January 2009). doi:10.4049/jimmunol.182.2.1011. PMID19124744.
“RANTES production by memory phenotype T cells is controlled by a posttranscriptional, TCR-dependent process”. Immunity17 (5): 605–615. (November 2002). doi:10.1016/S1074-7613(02)00456-9. PMID12433367.
“Diverging binding capacities of natural LD78beta isoforms of macrophage inflammatory protein-1alpha to the CC chemokine receptors 1, 3 and 5 affect their anti-HIV-1 activity and chemotactic potencies for neutrophils and eosinophils”. European Journal of Immunology31 (7): 2170–2178. (July 2001). doi:10.1002/1521-4141(200107)31:7<2170::AID-IMMU2170>3.0.CO;2-D. PMID11449371.
“Interaction of RANTES with syndecan-1 and syndecan-4 expressed by human primary macrophages”. Biochimica et Biophysica Acta (BBA) - Biomembranes1617 (1–2): 80–88. (October 2003). doi:10.1016/j.bbamem.2003.09.006. PMID14637022.
“The BBXB motif of RANTES is the principal site for heparin binding and controls receptor selectivity”. The Journal of Biological Chemistry276 (14): 10620–10626. (April 2001). doi:10.1074/jbc.M010867200. PMID11116158.
“Distribution of functional polymorphic variants of inflammation-related genes RANTES and CCR5 in long-lived individuals”. Cytokine58 (1): 10–13. (April 2012). doi:10.1016/j.cyto.2011.12.021. PMID22265023.
“Aggregation of RANTES is responsible for its inflammatory properties. Characterization of nonaggregating, noninflammatory RANTES mutants”. The Journal of Biological Chemistry274 (39): 27505–27512. (September 1999). doi:10.1074/jbc.274.39.27505. PMID10488085.
“CCL5-CCR5-mediated apoptosis in T cells: Requirement for glycosaminoglycan binding and CCL5 aggregation”. The Journal of Biological Chemistry281 (35): 25184–25194. (September 2006). doi:10.1074/jbc.M603912200. PMID16807236.