CHEK1 (Japanese Wikipedia)

Analysis of information sources in references of the Wikipedia article "CHEK1" in Japanese language version.

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cell.com

“A Surveillance Mechanism Ensures Repair of DNA Lesions during Zygotic Reprogramming”. Cell 167 (7): 1774–1787.e13. (December 2016). doi:10.1016/j.cell.2016.11.009. PMC 5161750. PMID 27916276.

doi.org

“Conservation of the Chk1 checkpoint pathway in mammals: linkage of DNA damage to Cdk regulation through Cdc25”. Science 277 (5331): 1497–501. (September 1997). doi:10.1126/science.277.5331.1497. PMID 9278511.
“Atm-dependent interactions of a mammalian chk1 homolog with meiotic chromosomes”. Current Biology 7 (12): 977–86. (December 1997). doi:10.1016/S0960-9822(06)00417-9. PMID 9382850.
“CHEK again: revisiting the development of Chk1 inhibitors for cancer therapy”. Pharmacology & Therapeutics 142 (1): 1–10. (April 2014). doi:10.1016/j.pharmthera.2013.10.005. PMID 24140082.
“Roles of Chk1 in cell biology and cancer therapy”. International Journal of Cancer 134 (5): 1013–23. (March 2014). doi:10.1002/ijc.28226. PMC 3852170. PMID 23613359.
“Checkpoint kinase 1 in DNA damage response and cell cycle regulation”. Cellular and Molecular Life Sciences 70 (21): 4009–21. (November 2013). doi:10.1007/s00018-013-1307-3. PMC 3731415. PMID 23508805.
“Regulatory motifs in Chk1”. Cell Cycle 12 (6): 916–22. (March 2013). doi:10.4161/cc.23881. PMC 3637350. PMID 23422000.
“Chk1 suppressed cell death”. Cell Division 5: 21. (September 2010). doi:10.1186/1747-1028-5-21. PMC 2939633. PMID 20813042.
“A Surveillance Mechanism Ensures Repair of DNA Lesions during Zygotic Reprogramming”. Cell 167 (7): 1774–1787.e13. (December 2016). doi:10.1016/j.cell.2016.11.009. PMC 5161750. PMID 27916276.
“Chk1 is an essential kinase that is regulated by Atr and required for the G(2)/M DNA damage checkpoint”. Genes & Development 14 (12): 1448–59. (June 2000). doi:10.1101/gad.840500. PMC 316686. PMID 10859164.
“Novel regulation of checkpoint kinase 1: Is checkpoint kinase 1 a good candidate for anti-cancer therapy?”. Cancer Science 103 (7): 1195–200. (July 2012). doi:10.1111/j.1349-7006.2012.02280.x. PMID 22435685.
“Chk1 targeting reactivates PP2A tumor suppressor activity in cancer cells”. Cancer Research 73 (22): 6757–69. (November 2013). doi:10.1158/0008-5472.CAN-13-1002. PMC 3870284. PMID 24072747.
The ATM-Chk2 and ATR-Chk1 pathways in DNA damage signaling and cancer. 108. (2010). 73–112. doi:10.1016/B978-0-12-380888-2.00003-0. ISBN 9780123808882. PMID 21034966
“DNA damage response pathways in tumor suppression and cancer treatment”. World Journal of Surgery 33 (4): 661–6. (April 2009). doi:10.1007/s00268-008-9840-1. PMID 19034564.
“Validation of RNAi Silencing Efficiency Using Gene Array Data shows 18.5% Failure Rate across 429 Independent Experiments” (英語). Molecular Therapy. Nucleic Acids 5 (9): e366. (September 2016). doi:10.1038/mtna.2016.66. PMC 5056990. PMID 27673562.
“Targeting ATR and Chk1 kinases for cancer treatment: a new model for new (and old) drugs”. Molecular Oncology 5 (4): 368–73. (August 2011). doi:10.1016/j.molonc.2011.07.002. PMC 3590794. PMID 21820372.
“Selective Chk1 inhibitors differentially sensitize p53-deficient cancer cells to cancer therapeutics”. International Journal of Cancer 119 (12): 2784–94. (December 2006). doi:10.1002/ijc.22198. PMID 17019715.
“Checkpoint kinase 1 inhibitors for potentiating systemic anticancer therapy”. Cancer Treatment Reviews 39 (5): 525–33. (August 2013). doi:10.1016/j.ctrv.2012.10.007. PMID 23207059.
“Chk1 inhibitors for novel cancer treatment”. Anti-Cancer Agents in Medicinal Chemistry 6 (4): 377–88. (July 2006). doi:10.2174/187152006777698132. PMID 16842237.
“Death by releasing the breaks: CHK1 inhibitors as cancer therapeutics”. Trends in Molecular Medicine 17 (2): 88–96. (February 2011). doi:10.1016/j.molmed.2010.10.009. PMC 6905465. PMID 21087899.
“Status of p53 in human cancer cells does not predict efficacy of Chk1 kinase inhibitors combined with chemotherapeutic agents”. Oncogene 29 (46): 6149–59. (November 2010). doi:10.1038/onc.2010.343. PMID 20729914.
“The cancer therapeutic potential of Chk1 inhibitors: how mechanistic studies impact on clinical trial design”. British Journal of Clinical Pharmacology 76 (3): 358–69. (September 2013). doi:10.1111/bcp.12139. PMC 3769664. PMID 23593991.
“CHK1 inhibitors in combination chemotherapy: thinking beyond the cell cycle”. Molecular Interventions 11 (2): 133–40. (April 2011). doi:10.1124/mi.11.2.11. PMC 3109860. PMID 21540473.
“Atm-dependent interactions of a mammalian chk1 homolog with meiotic chromosomes”. Current Biology 7 (12): 977–86. (December 1997). doi:10.1016/s0960-9822(06)00417-9. PMID 9382850.
“Checkpoint kinase 1 is essential for meiotic cell cycle regulation in mouse oocytes”. Cell Cycle 11 (10): 1948–55. (May 2012). doi:10.4161/cc.20279. PMID 22544319.

ensembl.org

May2017.archive.ensembl.org

GRCh38: Ensembl release 89: ENSG00000149554 - Ensembl, May 2017
GRCm38: Ensembl release 89: ENSMUSG00000032113 - Ensembl, May 2017

nih.gov

pubmed.ncbi.nlm.nih.gov

“Conservation of the Chk1 checkpoint pathway in mammals: linkage of DNA damage to Cdk regulation through Cdc25”. Science 277 (5331): 1497–501. (September 1997). doi:10.1126/science.277.5331.1497. PMID 9278511.
“Atm-dependent interactions of a mammalian chk1 homolog with meiotic chromosomes”. Current Biology 7 (12): 977–86. (December 1997). doi:10.1016/S0960-9822(06)00417-9. PMID 9382850.
“CHEK again: revisiting the development of Chk1 inhibitors for cancer therapy”. Pharmacology & Therapeutics 142 (1): 1–10. (April 2014). doi:10.1016/j.pharmthera.2013.10.005. PMID 24140082.
“Roles of Chk1 in cell biology and cancer therapy”. International Journal of Cancer 134 (5): 1013–23. (March 2014). doi:10.1002/ijc.28226. PMC 3852170. PMID 23613359.
“Checkpoint kinase 1 in DNA damage response and cell cycle regulation”. Cellular and Molecular Life Sciences 70 (21): 4009–21. (November 2013). doi:10.1007/s00018-013-1307-3. PMC 3731415. PMID 23508805.
“Regulatory motifs in Chk1”. Cell Cycle 12 (6): 916–22. (March 2013). doi:10.4161/cc.23881. PMC 3637350. PMID 23422000.
“Chk1 suppressed cell death”. Cell Division 5: 21. (September 2010). doi:10.1186/1747-1028-5-21. PMC 2939633. PMID 20813042.
“A Surveillance Mechanism Ensures Repair of DNA Lesions during Zygotic Reprogramming”. Cell 167 (7): 1774–1787.e13. (December 2016). doi:10.1016/j.cell.2016.11.009. PMC 5161750. PMID 27916276.
“Chk1 is an essential kinase that is regulated by Atr and required for the G(2)/M DNA damage checkpoint”. Genes & Development 14 (12): 1448–59. (June 2000). doi:10.1101/gad.840500. PMC 316686. PMID 10859164.
“Novel regulation of checkpoint kinase 1: Is checkpoint kinase 1 a good candidate for anti-cancer therapy?”. Cancer Science 103 (7): 1195–200. (July 2012). doi:10.1111/j.1349-7006.2012.02280.x. PMID 22435685.
“Chk1 targeting reactivates PP2A tumor suppressor activity in cancer cells”. Cancer Research 73 (22): 6757–69. (November 2013). doi:10.1158/0008-5472.CAN-13-1002. PMC 3870284. PMID 24072747.
The ATM-Chk2 and ATR-Chk1 pathways in DNA damage signaling and cancer. 108. (2010). 73–112. doi:10.1016/B978-0-12-380888-2.00003-0. ISBN 9780123808882. PMID 21034966
“DNA damage response pathways in tumor suppression and cancer treatment”. World Journal of Surgery 33 (4): 661–6. (April 2009). doi:10.1007/s00268-008-9840-1. PMID 19034564.
“Validation of RNAi Silencing Efficiency Using Gene Array Data shows 18.5% Failure Rate across 429 Independent Experiments” (英語). Molecular Therapy. Nucleic Acids 5 (9): e366. (September 2016). doi:10.1038/mtna.2016.66. PMC 5056990. PMID 27673562.
“Targeting ATR and Chk1 kinases for cancer treatment: a new model for new (and old) drugs”. Molecular Oncology 5 (4): 368–73. (August 2011). doi:10.1016/j.molonc.2011.07.002. PMC 3590794. PMID 21820372.
“Selective Chk1 inhibitors differentially sensitize p53-deficient cancer cells to cancer therapeutics”. International Journal of Cancer 119 (12): 2784–94. (December 2006). doi:10.1002/ijc.22198. PMID 17019715.
“Checkpoint kinase 1 inhibitors for potentiating systemic anticancer therapy”. Cancer Treatment Reviews 39 (5): 525–33. (August 2013). doi:10.1016/j.ctrv.2012.10.007. PMID 23207059.
“Chk1 inhibitors for novel cancer treatment”. Anti-Cancer Agents in Medicinal Chemistry 6 (4): 377–88. (July 2006). doi:10.2174/187152006777698132. PMID 16842237.
“Death by releasing the breaks: CHK1 inhibitors as cancer therapeutics”. Trends in Molecular Medicine 17 (2): 88–96. (February 2011). doi:10.1016/j.molmed.2010.10.009. PMC 6905465. PMID 21087899.
“Status of p53 in human cancer cells does not predict efficacy of Chk1 kinase inhibitors combined with chemotherapeutic agents”. Oncogene 29 (46): 6149–59. (November 2010). doi:10.1038/onc.2010.343. PMID 20729914.
“The cancer therapeutic potential of Chk1 inhibitors: how mechanistic studies impact on clinical trial design”. British Journal of Clinical Pharmacology 76 (3): 358–69. (September 2013). doi:10.1111/bcp.12139. PMC 3769664. PMID 23593991.
“CHK1 inhibitors in combination chemotherapy: thinking beyond the cell cycle”. Molecular Interventions 11 (2): 133–40. (April 2011). doi:10.1124/mi.11.2.11. PMC 3109860. PMID 21540473.
“Atm-dependent interactions of a mammalian chk1 homolog with meiotic chromosomes”. Current Biology 7 (12): 977–86. (December 1997). doi:10.1016/s0960-9822(06)00417-9. PMID 9382850.
“Checkpoint kinase 1 is essential for meiotic cell cycle regulation in mouse oocytes”. Cell Cycle 11 (10): 1948–55. (May 2012). doi:10.4161/cc.20279. PMID 22544319.

ncbi.nlm.nih.gov

pmc.ncbi.nlm.nih.gov

wikipedia.org

en.wikipedia.org

GRCh38: Ensembl release 89: ENSG00000149554 - Ensembl, May 2017
GRCm38: Ensembl release 89: ENSMUSG00000032113 - Ensembl, May 2017