“Pluripotency redux--advances in stem-cell research”.The New England Journal of Medicine357(15): 1469–72.(October 2007).doi:10.1056/NEJMp078126.PMID17928593.
“Comprehensive genomic profiling of epithelial ovarian cancer by next generation sequencing-based diagnostic assay reveals new routes to targeted therapies”.Gynecologic Oncology130(3): 554–9.(September 2013).doi:10.1016/j.ygyno.2013.06.019.PMID23791828.
“Tumor induction by ras and myc oncogenes in fetal and neonatal brain: modulating effects of developmental stage and retroviral dose”.Acta Neuropathologica86(5): 456–65.(1993).doi:10.1007/bf00228580.PMID8310796.
Takahaski,Kazutoshi;Yamanaka,Shinya(17 Feb 2016).“A decade of transcription factor-mediated reprogramming to pluripotency”.Nature Reviews. Molecular Cell Biology17(3): 183–193.doi:10.1038/nrm.2016.8.PMID26883003.
“A novel tricomplex of BRCA1, Nmi, and c-Myc inhibits c-Myc-induced human telomerase reverse transcriptase gene (hTERT) promoter activity in breast cancer”.The Journal of Biological Chemistry277(23): 20965–73.(June 2002).doi:10.1074/jbc.M112231200.PMID11916966.
“Inhibition of human telomerase reverse transcriptase gene expression by BRCA1 in human ovarian cancer cells”.Biochemical and Biophysical Research Communications303(1): 130–6.(March 2003).doi:10.1016/s0006-291x(03)00318-8.PMID12646176.
“BRCA1 binds c-Myc and inhibits its transcriptional and transforming activity in cells”.Oncogene17(15): 1939–48.(October 1998).doi:10.1038/sj.onc.1202403.PMID9788437.
“Tobacco-specific nitrosamine 4-(methylnitrosamino)-1-(3-pyridyl)-1-butanone promotes functional cooperation of Bcl2 and c-Myc through phosphorylation in regulating cell survival and proliferation”.The Journal of Biological Chemistry279(38): 40209–19.(September 2004).doi:10.1074/jbc.M404056200.PMID15210690.
“c-Myc recruits P-TEFb for transcription, cellular proliferation and apoptosis”.Oncogene22(36): 5707–11.(August 2003).doi:10.1038/sj.onc.1206800.PMID12944920.
“BioID identifies novel c-MYC interacting partners in cultured cells and xenograft tumors”.Journal of Proteomics118(12): 95–111.(April 2015).doi:10.1016/j.jprot.2014.09.029.PMID25452129.
“CRD-BP/IMP1 expression characterizes cord blood CD34+ stem cells and affects c-myc and IGF-II expression in MCF-7 cancer cells”.The Journal of Biological Chemistry280(20): 20086–93.(May 2005).doi:10.1074/jbc.M410036200.PMID15769738.
“Regulation of c-Myc through phosphorylation at Ser-62 and Ser-71 by c-Jun N-terminal kinase”.The Journal of Biological Chemistry274(46): 32580–7.(November 1999).doi:10.1074/jbc.274.46.32580.PMID10551811.
“The novel ATM-related protein TRRAP is an essential cofactor for the c-Myc and E2F oncoproteins”.Cell94(3): 363–74.(August 1998).doi:10.1016/s0092-8674(00)81479-8.PMID9708738.
“c-MYC interacts with INI1/hSNF5 and requires the SWI/SNF complex for transactivation function”.Nature Genetics22(1): 102–5.(May 1999).doi:10.1038/8811.PMID10319872.
“Interactions of the DNA mismatch repair proteins MLH1 and MSH2 with c-MYC and MAX”.Oncogene22(6): 819–25.(February 2003).doi:10.1038/sj.onc.1206252.PMID12584560.
“Mlx, a novel Max-like BHLHZip protein that interacts with the Max network of transcription factors”.The Journal of Biological Chemistry274(51): 36344–50.(December 1999).doi:10.1074/jbc.274.51.36344.PMID10593926.
“Mmip1: a novel leucine zipper protein that reverses the suppressive effects of Mad family members on c-myc”.Oncogene16(9): 1149–59.(March 1998).doi:10.1038/sj.onc.1201634.PMID9528857.
“X-ray structures of Myc-Max and Mad-Max recognizing DNA. Molecular bases of regulation by proto-oncogenic transcription factors”.Cell112(2): 193–205.(January 2003).doi:10.1016/s0092-8674(02)01284-9.PMID12553908.
“Differential effects of the widely expressed dMax splice variant of Max on E-box vs initiator element-mediated regulation by c-Myc”.Oncogene18(15): 2489–98.(April 1999).doi:10.1038/sj.onc.1202611.PMID10229200.
“Mlx, a new Max-like bHLHZip family member: the center stage of a novel transcription factors regulatory pathway?”.Oncogene19(29): 3266–77.(July 2000).doi:10.1038/sj.onc.1203634.PMID10918583.
“Cell cycle-dependent switch of up-and down-regulation of human hsp70 gene expression by interaction between c-Myc and CBF/NF-Y”.The Journal of Biological Chemistry274(34): 24270–9.(August 1999).doi:10.1074/jbc.274.34.24270.PMID10446203.
“p73 Interacts with c-Myc to regulate Y-box-binding protein-1 expression”.The Journal of Biological Chemistry277(35): 31694–702.(August 2002).doi:10.1074/jbc.M200266200.PMID12080043.
“MM-1, a novel c-Myc-associating protein that represses transcriptional activity of c-Myc”.The Journal of Biological Chemistry273(45): 29794–800.(November 1998).doi:10.1074/jbc.273.45.29794.PMID9792694.
“MM-1, a c-Myc-binding protein, is a candidate for a tumor suppressor in leukemia/lymphoma and tongue cancer”.The Journal of Biological Chemistry276(48): 45137–44.(November 2001).doi:10.1074/jbc.M106127200.PMID11567024.
“Direct interaction of c-Myc with Smad2 and Smad3 to inhibit TGF-beta-mediated induction of the CDK inhibitor p15(Ink4B)”.Molecular Cell9(1): 133–43.(January 2002).doi:10.1016/s1097-2765(01)00430-0.PMID11804592.
“Guanine nucleotide exchange factor, Tiam1, directly binds to c-Myc and interferes with c-Myc-mediated apoptosis in rat-1 fibroblasts”.The Journal of Biological Chemistry278(7): 5132–40.(February 2003).doi:10.1074/jbc.M206733200.PMID12446731.
“Repression of p15INK4b expression by Myc through association with Miz-1”.Nature Cell Biology3(4): 392–9.(April 2001).doi:10.1038/35070076.PMID11283613.
“Pluripotency redux--advances in stem-cell research”.The New England Journal of Medicine357(15): 1469–72.(October 2007).doi:10.1056/NEJMp078126.PMID17928593.
“Comprehensive genomic profiling of epithelial ovarian cancer by next generation sequencing-based diagnostic assay reveals new routes to targeted therapies”.Gynecologic Oncology130(3): 554–9.(September 2013).doi:10.1016/j.ygyno.2013.06.019.PMID23791828.
“Tumor induction by ras and myc oncogenes in fetal and neonatal brain: modulating effects of developmental stage and retroviral dose”.Acta Neuropathologica86(5): 456–65.(1993).doi:10.1007/bf00228580.PMID8310796.
Takahaski,Kazutoshi;Yamanaka,Shinya(17 Feb 2016).“A decade of transcription factor-mediated reprogramming to pluripotency”.Nature Reviews. Molecular Cell Biology17(3): 183–193.doi:10.1038/nrm.2016.8.PMID26883003.
“A novel tricomplex of BRCA1, Nmi, and c-Myc inhibits c-Myc-induced human telomerase reverse transcriptase gene (hTERT) promoter activity in breast cancer”.The Journal of Biological Chemistry277(23): 20965–73.(June 2002).doi:10.1074/jbc.M112231200.PMID11916966.
“Inhibition of human telomerase reverse transcriptase gene expression by BRCA1 in human ovarian cancer cells”.Biochemical and Biophysical Research Communications303(1): 130–6.(March 2003).doi:10.1016/s0006-291x(03)00318-8.PMID12646176.
“BRCA1 binds c-Myc and inhibits its transcriptional and transforming activity in cells”.Oncogene17(15): 1939–48.(October 1998).doi:10.1038/sj.onc.1202403.PMID9788437.
“Tobacco-specific nitrosamine 4-(methylnitrosamino)-1-(3-pyridyl)-1-butanone promotes functional cooperation of Bcl2 and c-Myc through phosphorylation in regulating cell survival and proliferation”.The Journal of Biological Chemistry279(38): 40209–19.(September 2004).doi:10.1074/jbc.M404056200.PMID15210690.
“c-Myc recruits P-TEFb for transcription, cellular proliferation and apoptosis”.Oncogene22(36): 5707–11.(August 2003).doi:10.1038/sj.onc.1206800.PMID12944920.
“BioID identifies novel c-MYC interacting partners in cultured cells and xenograft tumors”.Journal of Proteomics118(12): 95–111.(April 2015).doi:10.1016/j.jprot.2014.09.029.PMID25452129.
“CRD-BP/IMP1 expression characterizes cord blood CD34+ stem cells and affects c-myc and IGF-II expression in MCF-7 cancer cells”.The Journal of Biological Chemistry280(20): 20086–93.(May 2005).doi:10.1074/jbc.M410036200.PMID15769738.
“Regulation of c-Myc through phosphorylation at Ser-62 and Ser-71 by c-Jun N-terminal kinase”.The Journal of Biological Chemistry274(46): 32580–7.(November 1999).doi:10.1074/jbc.274.46.32580.PMID10551811.
“The novel ATM-related protein TRRAP is an essential cofactor for the c-Myc and E2F oncoproteins”.Cell94(3): 363–74.(August 1998).doi:10.1016/s0092-8674(00)81479-8.PMID9708738.
“c-MYC interacts with INI1/hSNF5 and requires the SWI/SNF complex for transactivation function”.Nature Genetics22(1): 102–5.(May 1999).doi:10.1038/8811.PMID10319872.
“Interactions of the DNA mismatch repair proteins MLH1 and MSH2 with c-MYC and MAX”.Oncogene22(6): 819–25.(February 2003).doi:10.1038/sj.onc.1206252.PMID12584560.
“Mlx, a novel Max-like BHLHZip protein that interacts with the Max network of transcription factors”.The Journal of Biological Chemistry274(51): 36344–50.(December 1999).doi:10.1074/jbc.274.51.36344.PMID10593926.
“Mmip1: a novel leucine zipper protein that reverses the suppressive effects of Mad family members on c-myc”.Oncogene16(9): 1149–59.(March 1998).doi:10.1038/sj.onc.1201634.PMID9528857.
“X-ray structures of Myc-Max and Mad-Max recognizing DNA. Molecular bases of regulation by proto-oncogenic transcription factors”.Cell112(2): 193–205.(January 2003).doi:10.1016/s0092-8674(02)01284-9.PMID12553908.
“Differential effects of the widely expressed dMax splice variant of Max on E-box vs initiator element-mediated regulation by c-Myc”.Oncogene18(15): 2489–98.(April 1999).doi:10.1038/sj.onc.1202611.PMID10229200.
“Mlx, a new Max-like bHLHZip family member: the center stage of a novel transcription factors regulatory pathway?”.Oncogene19(29): 3266–77.(July 2000).doi:10.1038/sj.onc.1203634.PMID10918583.
“Mechanism for the transcriptional repression by c-Myc on PDGF beta-receptor”.Journal of Cell Science114(Pt 8): 1533–44.(April 2001).PMID11282029.
“Cell cycle-dependent switch of up-and down-regulation of human hsp70 gene expression by interaction between c-Myc and CBF/NF-Y”.The Journal of Biological Chemistry274(34): 24270–9.(August 1999).doi:10.1074/jbc.274.34.24270.PMID10446203.
“p73 Interacts with c-Myc to regulate Y-box-binding protein-1 expression”.The Journal of Biological Chemistry277(35): 31694–702.(August 2002).doi:10.1074/jbc.M200266200.PMID12080043.
“MM-1, a novel c-Myc-associating protein that represses transcriptional activity of c-Myc”.The Journal of Biological Chemistry273(45): 29794–800.(November 1998).doi:10.1074/jbc.273.45.29794.PMID9792694.
“MM-1, a c-Myc-binding protein, is a candidate for a tumor suppressor in leukemia/lymphoma and tongue cancer”.The Journal of Biological Chemistry276(48): 45137–44.(November 2001).doi:10.1074/jbc.M106127200.PMID11567024.
“Direct interaction of c-Myc with Smad2 and Smad3 to inhibit TGF-beta-mediated induction of the CDK inhibitor p15(Ink4B)”.Molecular Cell9(1): 133–43.(January 2002).doi:10.1016/s1097-2765(01)00430-0.PMID11804592.
“Guanine nucleotide exchange factor, Tiam1, directly binds to c-Myc and interferes with c-Myc-mediated apoptosis in rat-1 fibroblasts”.The Journal of Biological Chemistry278(7): 5132–40.(February 2003).doi:10.1074/jbc.M206733200.PMID12446731.
“Repression of p15INK4b expression by Myc through association with Miz-1”.Nature Cell Biology3(4): 392–9.(April 2001).doi:10.1038/35070076.PMID11283613.