“The human Werner syndrome protein stimulates repair of oxidative DNA base damage by the DNA glycosylase NEIL1”.J. Biol. Chem.282(36): 26591–602.(2007).doi:10.1074/jbc.M703343200.PMID17611195.
Lebel M(2001).“Werner syndrome: genetic and molecular basis of a premature aging disorder”.Cell. Mol. Life Sci.58(7): 857–67.doi:10.1007/s00018-001-8398-y.PMID11497235.
“Correlation between senescence and DNA repair in cells from young and old individuals and in premature aging syndromes”.Mutat. Res.316(1): 37–48.(1994).doi:10.1016/0921-8734(94)90006-x.PMID7507567.
“Werner helicase relocates into nuclear foci in response to DNA damaging agents and co-localizes with RPA and Rad51”.Genes Cells6(5): 421–30.(2001).doi:10.1046/j.1365-2443.2001.00433.x.PMID11380620.
“Colocalization, physical, and functional interaction between Werner and Bloom syndrome proteins”.J. Biol. Chem.277(24): 22035–44.(June 2002).doi:10.1074/jbc.M200914200.PMID11919194.
“Substrate specificities and identification of putative substrates of ATM kinase family members”.J. Biol. Chem.274(53): 37538–43.(Dec 1999).doi:10.1074/jbc.274.53.37538.PMID10608806.
“Werner protein is a target of DNA-dependent protein kinase in vivo and in vitro, and its catalytic activities are regulated by phosphorylation”.J. Biol. Chem.277(21): 18291–302.(May 2002).doi:10.1074/jbc.M111523200.PMID11889123.
“Stimulation of flap endonuclease-1 by the Bloom's syndrome protein”.J. Biol. Chem.279(11): 9847–56.(March 2004).doi:10.1074/jbc.M309898200.PMID14688284.
“Functional interaction between Ku and the werner syndrome protein in DNA end processing”.J. Biol. Chem.275(37): 28349–52.(September 2000).doi:10.1074/jbc.C000289200.PMID10880505.
“The processing of Holliday junctions by BLM and WRN helicases is regulated by p53”.J. Biol. Chem.277(35): 31980–7.(August 2002).doi:10.1074/jbc.M204111200.PMID12080066.
“p53 Modulates the exonuclease activity of Werner syndrome protein”.J. Biol. Chem.276(37): 35093–102.(September 2001).doi:10.1074/jbc.M103332200.PMID11427532.
“Characterisation of the interaction between WRN, the helicase/exonuclease defective in progeroid Werner's syndrome, and an essential replication factor, PCNA”.Mech. Ageing Dev.124(2): 167–74.(February 2003).doi:10.1016/S0047-6374(02)00131-8.PMID12633936.
“Telomere-binding protein TRF2 binds to and stimulates the Werner and Bloom syndrome helicases”.J. Biol. Chem.277(43): 41110–9.(October 2002).doi:10.1074/jbc.M205396200.PMID12181313.
“A novel protein interacts with the Werner's syndrome gene product physically and functionally”.J. Biol. Chem.276(23): 20364–9.(June 2001).doi:10.1074/jbc.C100035200.PMID11301316.
“The human Werner syndrome protein stimulates repair of oxidative DNA base damage by the DNA glycosylase NEIL1”.J. Biol. Chem.282(36): 26591–602.(2007).doi:10.1074/jbc.M703343200.PMID17611195.
Lebel M(2001).“Werner syndrome: genetic and molecular basis of a premature aging disorder”.Cell. Mol. Life Sci.58(7): 857–67.doi:10.1007/s00018-001-8398-y.PMID11497235.
“Correlation between senescence and DNA repair in cells from young and old individuals and in premature aging syndromes”.Mutat. Res.316(1): 37–48.(1994).doi:10.1016/0921-8734(94)90006-x.PMID7507567.
“Werner helicase relocates into nuclear foci in response to DNA damaging agents and co-localizes with RPA and Rad51”.Genes Cells6(5): 421–30.(2001).doi:10.1046/j.1365-2443.2001.00433.x.PMID11380620.
“Colocalization, physical, and functional interaction between Werner and Bloom syndrome proteins”.J. Biol. Chem.277(24): 22035–44.(June 2002).doi:10.1074/jbc.M200914200.PMID11919194.
“Substrate specificities and identification of putative substrates of ATM kinase family members”.J. Biol. Chem.274(53): 37538–43.(Dec 1999).doi:10.1074/jbc.274.53.37538.PMID10608806.
“Werner protein is a target of DNA-dependent protein kinase in vivo and in vitro, and its catalytic activities are regulated by phosphorylation”.J. Biol. Chem.277(21): 18291–302.(May 2002).doi:10.1074/jbc.M111523200.PMID11889123.
“Stimulation of flap endonuclease-1 by the Bloom's syndrome protein”.J. Biol. Chem.279(11): 9847–56.(March 2004).doi:10.1074/jbc.M309898200.PMID14688284.
“Functional interaction between Ku and the werner syndrome protein in DNA end processing”.J. Biol. Chem.275(37): 28349–52.(September 2000).doi:10.1074/jbc.C000289200.PMID10880505.
“The processing of Holliday junctions by BLM and WRN helicases is regulated by p53”.J. Biol. Chem.277(35): 31980–7.(August 2002).doi:10.1074/jbc.M204111200.PMID12080066.
“p53 Modulates the exonuclease activity of Werner syndrome protein”.J. Biol. Chem.276(37): 35093–102.(September 2001).doi:10.1074/jbc.M103332200.PMID11427532.
“Characterisation of the interaction between WRN, the helicase/exonuclease defective in progeroid Werner's syndrome, and an essential replication factor, PCNA”.Mech. Ageing Dev.124(2): 167–74.(February 2003).doi:10.1016/S0047-6374(02)00131-8.PMID12633936.
“Telomere-binding protein TRF2 binds to and stimulates the Werner and Bloom syndrome helicases”.J. Biol. Chem.277(43): 41110–9.(October 2002).doi:10.1074/jbc.M205396200.PMID12181313.
“A novel protein interacts with the Werner's syndrome gene product physically and functionally”.J. Biol. Chem.276(23): 20364–9.(June 2001).doi:10.1074/jbc.C100035200.PMID11301316.