スフィンゴミエリンデアシラーゼ (Japanese Wikipedia)

Analysis of information sources in references of the Wikipedia article "スフィンゴミエリンデアシラーゼ" in Japanese language version.

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doi.org

  • Hara, J., Higuchi, K., Okamoto, R., Kawashima, M. and Imokawa, G. (2000). “High-expression of sphingomyelin deacylase is an important determinant of ceramide deficiency leading to barrier disruption in atopic dermatitis”. J. Invest. Dermatol. 115: 406-413. doi:10.1046/j.1523-1747.2000.00072.x. PMID 10951276. 
  • Higuchi, K., Hara, J., Okamoto, R., Kawashima, M. and Imokawa, G. (2000). “The skin of atopic dermatitis patients contains a novel enzyme, glucosylceramide sphingomyelin deacylase, which cleaves the N-acyl linkage of sphingomyelin and glucosylceramide”. Biochem. J. 350: 747-756. doi:10.1042/0264-6021:3500747. PMID 10970788. 

nih.gov

pubmed.ncbi.nlm.nih.gov

  • Hara, J., Higuchi, K., Okamoto, R., Kawashima, M. and Imokawa, G. (2000). “High-expression of sphingomyelin deacylase is an important determinant of ceramide deficiency leading to barrier disruption in atopic dermatitis”. J. Invest. Dermatol. 115: 406-413. doi:10.1046/j.1523-1747.2000.00072.x. PMID 10951276. 
  • Higuchi, K., Hara, J., Okamoto, R., Kawashima, M. and Imokawa, G. (2000). “The skin of atopic dermatitis patients contains a novel enzyme, glucosylceramide sphingomyelin deacylase, which cleaves the N-acyl linkage of sphingomyelin and glucosylceramide”. Biochem. J. 350: 747-756. doi:10.1042/0264-6021:3500747. PMID 10970788. 
  • Ishibashi, M., Arikawa, J., Okamoto, R., Kawashima, M., Takagi, Y., Ohguchi, K. and Imokawa, G. (2003). “Abnormal expression of the novel epidermal enzyme, glucosylceramide deacylase, and the accumulation of its enzymatic reaction product, glucosylsphingosine, in the skin of patients with atopic dermatitis”. Lab. Invest. 83: 397-408. PMID 12649340.