Hara, J., Higuchi, K., Okamoto, R., Kawashima, M. and Imokawa, G. (2000). “High-expression of sphingomyelin deacylase is an important determinant of ceramide deficiency leading to barrier disruption in atopic dermatitis”. J. Invest. Dermatol.115: 406-413. doi:10.1046/j.1523-1747.2000.00072.x. PMID10951276.
Higuchi, K., Hara, J., Okamoto, R., Kawashima, M. and Imokawa, G. (2000). “The skin of atopic dermatitis patients contains a novel enzyme, glucosylceramide sphingomyelin deacylase, which cleaves the N-acyl linkage of sphingomyelin and glucosylceramide”. Biochem. J.350: 747-756. doi:10.1042/0264-6021:3500747. PMID10970788.
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Hara, J., Higuchi, K., Okamoto, R., Kawashima, M. and Imokawa, G. (2000). “High-expression of sphingomyelin deacylase is an important determinant of ceramide deficiency leading to barrier disruption in atopic dermatitis”. J. Invest. Dermatol.115: 406-413. doi:10.1046/j.1523-1747.2000.00072.x. PMID10951276.
Higuchi, K., Hara, J., Okamoto, R., Kawashima, M. and Imokawa, G. (2000). “The skin of atopic dermatitis patients contains a novel enzyme, glucosylceramide sphingomyelin deacylase, which cleaves the N-acyl linkage of sphingomyelin and glucosylceramide”. Biochem. J.350: 747-756. doi:10.1042/0264-6021:3500747. PMID10970788.
Ishibashi, M., Arikawa, J., Okamoto, R., Kawashima, M., Takagi, Y., Ohguchi, K. and Imokawa, G. (2003). “Abnormal expression of the novel epidermal enzyme, glucosylceramide deacylase, and the accumulation of its enzymatic reaction product, glucosylsphingosine, in the skin of patients with atopic dermatitis”. Lab. Invest.83: 397-408. PMID12649340.