“Interactions of nitrogen-containing xenobiotics with monoamine oxidase (MAO) isozymes A and B: SAR studies on MAO substrates and inhibitors”. Chemical Research in Toxicology14 (9): 1139–62. (September 2001). doi:10.1021/tx010073b. PMID11559028.
“Decreased blood platelet MAO activity in unmedicated chronic schizophrenic patients”. The American Journal of Psychiatry133 (3): 323–6. (March 1976). doi:10.1176/ajp.133.3.323. PMID943955.
“Reduced platelet monoamine oxidase activity in a subgroup of schizophrenic patients”. The American Journal of Psychiatry133 (4): 438–40. (April 1976). doi:10.1176/ajp.133.4.438. PMID1267046.
“Elevated monoamine oxidase a levels in the brain: an explanation for the monoamine imbalance of major depression”. Archives of General Psychiatry63 (11): 1209–16. (November 2006). doi:10.1001/archpsyc.63.11.1209. PMID17088501.
“Association analysis of the monoamine oxidase A and B genes with attention deficit hyperactivity disorder (ADHD) in an Irish sample: preferential transmission of the MAO-A 941G allele to affected children”. American Journal of Medical Genetics. Part B, Neuropsychiatric Genetics134B (1): 110–4. (April 2005). doi:10.1002/ajmg.b.30158. PMID15717295.
“Platelet monoamine oxidase, personality and alcoholism: the rise, fall and resurrection”. Neurotoxicology25 (1–2): 79–89. (January 2004). doi:10.1016/S0161-813X(03)00115-3. PMID14697883.
“Monoamine oxidases A and B gene polymorphisms in migraine patients”. Journal of the Neurological Sciences228 (2): 149–53. (February 2005). doi:10.1016/j.jns.2004.11.045. PMID15694196.
“Neuropharmacological actions of cigarette smoke: brain monoamine oxidase B (MAO B) inhibition”. Journal of Addictive Diseases17 (1): 23–34. (1998). doi:10.1300/J069v17n01_03. PMID9549600.
“Species differences in the deamination of dopamine and other substrates for monoamine oxidase in brain”. Psychopharmacology72 (1): 27–33. (1980). doi:10.1007/bf00433804. PMID6781004.
“Cloning, after cloning, knock-out mice, and physiological functions of MAO A and B”. Neurotoxicology25 (1–2): 21–30. (January 2004). doi:10.1016/s0161-813x(03)00112-8. PMID14697877.
“Monoamine oxidase activity in the rat pineal gland: Comparison with brain areas and alteration during aging”. Advances in Gerontology6 (2): 111–116. (2016-07-14). doi:10.1134/S2079057016020120. ISSN2079-0570.
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“Monoamine oxidases: certainties and uncertainties”. Current Medicinal Chemistry11 (15): 1965–82. (August 2004). doi:10.2174/0929867043364810. PMID15279561.
“Interactions of nitrogen-containing xenobiotics with monoamine oxidase (MAO) isozymes A and B: SAR studies on MAO substrates and inhibitors”. Chemical Research in Toxicology14 (9): 1139–62. (September 2001). doi:10.1021/tx010073b. PMID11559028.
“Decreased blood platelet MAO activity in unmedicated chronic schizophrenic patients”. The American Journal of Psychiatry133 (3): 323–6. (March 1976). doi:10.1176/ajp.133.3.323. PMID943955.
“Reduced platelet monoamine oxidase activity in a subgroup of schizophrenic patients”. The American Journal of Psychiatry133 (4): 438–40. (April 1976). doi:10.1176/ajp.133.4.438. PMID1267046.
“Elevated monoamine oxidase a levels in the brain: an explanation for the monoamine imbalance of major depression”. Archives of General Psychiatry63 (11): 1209–16. (November 2006). doi:10.1001/archpsyc.63.11.1209. PMID17088501.
“Association analysis of the monoamine oxidase A and B genes with attention deficit hyperactivity disorder (ADHD) in an Irish sample: preferential transmission of the MAO-A 941G allele to affected children”. American Journal of Medical Genetics. Part B, Neuropsychiatric Genetics134B (1): 110–4. (April 2005). doi:10.1002/ajmg.b.30158. PMID15717295.
“Platelet monoamine oxidase, personality and alcoholism: the rise, fall and resurrection”. Neurotoxicology25 (1–2): 79–89. (January 2004). doi:10.1016/S0161-813X(03)00115-3. PMID14697883.
“Monoamine oxidase activities in patients with migraine or with cluster headache during the acute phases and after treatment with L-5-hydroxytryptophan”. Rivista di Patologia Nervosa e Mentale100 (5): 269–74. (1 October 2016). PMID318025.
“Monoamine oxidases A and B gene polymorphisms in migraine patients”. Journal of the Neurological Sciences228 (2): 149–53. (February 2005). doi:10.1016/j.jns.2004.11.045. PMID15694196.
“Neuropharmacological actions of cigarette smoke: brain monoamine oxidase B (MAO B) inhibition”. Journal of Addictive Diseases17 (1): 23–34. (1998). doi:10.1300/J069v17n01_03. PMID9549600.
“Species differences in the deamination of dopamine and other substrates for monoamine oxidase in brain”. Psychopharmacology72 (1): 27–33. (1980). doi:10.1007/bf00433804. PMID6781004.
“Cloning, after cloning, knock-out mice, and physiological functions of MAO A and B”. Neurotoxicology25 (1–2): 21–30. (January 2004). doi:10.1016/s0161-813x(03)00112-8. PMID14697877.
“Monoamine oxidase activity in the rat pineal gland: Comparison with brain areas and alteration during aging”. Advances in Gerontology6 (2): 111–116. (2016-07-14). doi:10.1134/S2079057016020120. ISSN2079-0570.