“Molecular mechanism of monoamine oxidase A gene regulation under inflammation and ischemia-like conditions: key roles of the transcription factors GATA2, Sp1 and TBP”. Journal of Neurochemistry134 (1): 21–38. (July 2015). doi:10.1111/jnc.13099. PMID25810277.
“A unique demographic history exists for the MAO-A gene in Polynesians”. Journal of Human Genetics57 (5): 294–300. (10 January 2012). doi:10.1038/jhg.2012.19. PMID22377710.
“The structure of monoamine oxidases: past, present, and future”. Journal of Neural Transmission125 (11): 1567–1579. (November 2018). doi:10.1007/s00702-018-1915-z. PMID30167931.
“Structure-function relationships in flavoenzyme-dependent amine oxidations: A comparison of polyamine oxidase and monoamine oxidase”. Journal of Biological Chemistry277 (27): 23973–23976. (July 5, 2002). doi:10.1074/jbc.R200005200.
“Elevated monoamine oxidase a levels in the brain: an explanation for the monoamine imbalance of major depression”. Archives of General Psychiatry63 (11): 1209–16. (November 2006). doi:10.1001/archpsyc.63.11.1209. PMID17088501.
“MAO-A gene polymorphisms are associated with major depression and sleep disturbance in males”. NeuroReport15 (13): 2097–101. (September 2004). doi:10.1097/00001756-200409150-00020. PMID15486489.
“Association study of a monoamine oxidase a gene promoter polymorphism with major depressive disorder and antidepressant response”. Neuropsychopharmacology30 (9): 1719–23. (September 2005). doi:10.1038/sj.npp.1300785. PMID15956990.
“Family-based association study of 5-HTTLPR, TPH, MAO-A, and DRD4 polymorphisms in mood disorders”. American Journal of Medical Genetics114 (4): 361–9. (May 2002). doi:10.1002/ajmg.10356. PMID11992558.
“Association of monoamine oxidase A (MAOA) polymorphisms and clinical subgroups of major depressive disorders in the Han Chinese population”. The World Journal of Biological Psychiatry10 (4 Pt 2): 544–51. (2009). doi:10.1080/15622970701816506. PMID19224413.
“Monoamine oxidase a and catechol-o-methyltransferase functional polymorphisms and the placebo response in major depressive disorder”. Journal of Clinical Psychopharmacology29 (4): 372–7. (August 2009). doi:10.1097/JCP.0b013e3181ac4aaf. PMID19593178.
“The integration of genetic propensities into social-control models of delinquency and violence among male youths”. American Sociological Review73 (4): 543–568. (August 2008). doi:10.1177/000312240807300402.
“Exploring the association between the 2-repeat allele of the MAOA gene promoter polymorphism and psychopathic personality traits, arrests, incarceration, and lifetime antisocial behavior”. Personality and Individual Differences54 (2): 164–168. (2012). doi:10.1016/j.paid.2012.08.014.
“Candidate genes for aggression and antisocial behavior: a meta-analysis of association studies of the 5HTTLPR and MAOA-uVNTR”. Behavior Genetics44 (5): 427–44. (September 2014). doi:10.1007/s10519-014-9661-y. PMID24902785.
“MAOA genotype, social exclusion and aggression: an experimental test of a gene-environment interaction”. Genes, Brain and Behavior12 (1): 140–5. (February 2013). doi:10.1111/j.1601-183X.2012.00868.x. PMID23067570.
“State v. Yepez: Admissibility and Relevance of Behavioral Genetic Evidence in a Criminal Trial”. Psychiatric Services72 (7): 853–855. (July 2021). doi:10.1176/appi.ps.202100226. PMID34074149.
“Effect of MAO A deficiency on different kinds of aggression and social investigation in mice”. Aggressive Behavior33 (1): 1–6. (2007). doi:10.1002/ab.20161. PMID17441000.
“Effect of MAO A deficiency on different kinds of aggression and social investigation in mice”. Aggressive Behavior33 (1): 1–6. (1 January 2007). doi:10.1002/ab.20161. PMID17441000.
“Specification of the phenotype required for men with monoamine oxidase type A deficiency”. Human Genetics96 (3): 372–6. (September 1995). doi:10.1007/BF00210430. PMID7649563.
“Piperine from the fruits of Piper longum with inhibitory effect on monoamine oxidase and antidepressant-like activity”. Chemical & Pharmaceutical Bulletin53 (7): 832–5. (July 2005). doi:10.1248/cpb.53.832. PMID15997146.
“Monoamine oxidase inhibition by Rhodiola rosea L. roots”. Journal of Ethnopharmacology122 (2): 397–401. (March 2009). doi:10.1016/j.jep.2009.01.007. PMID19168123.
“Molecular mechanism of monoamine oxidase A gene regulation under inflammation and ischemia-like conditions: key roles of the transcription factors GATA2, Sp1 and TBP”. Journal of Neurochemistry134 (1): 21–38. (July 2015). doi:10.1111/jnc.13099. PMID25810277.
“A unique demographic history exists for the MAO-A gene in Polynesians”. Journal of Human Genetics57 (5): 294–300. (10 January 2012). doi:10.1038/jhg.2012.19. PMID22377710.
“The structure of monoamine oxidases: past, present, and future”. Journal of Neural Transmission125 (11): 1567–1579. (November 2018). doi:10.1007/s00702-018-1915-z. PMID30167931.
“Elevated monoamine oxidase a levels in the brain: an explanation for the monoamine imbalance of major depression”. Archives of General Psychiatry63 (11): 1209–16. (November 2006). doi:10.1001/archpsyc.63.11.1209. PMID17088501.
“MAO-A gene polymorphisms are associated with major depression and sleep disturbance in males”. NeuroReport15 (13): 2097–101. (September 2004). doi:10.1097/00001756-200409150-00020. PMID15486489.
“Association study of a monoamine oxidase a gene promoter polymorphism with major depressive disorder and antidepressant response”. Neuropsychopharmacology30 (9): 1719–23. (September 2005). doi:10.1038/sj.npp.1300785. PMID15956990.
“Family-based association study of 5-HTTLPR, TPH, MAO-A, and DRD4 polymorphisms in mood disorders”. American Journal of Medical Genetics114 (4): 361–9. (May 2002). doi:10.1002/ajmg.10356. PMID11992558.
“Association of monoamine oxidase A (MAOA) polymorphisms and clinical subgroups of major depressive disorders in the Han Chinese population”. The World Journal of Biological Psychiatry10 (4 Pt 2): 544–51. (2009). doi:10.1080/15622970701816506. PMID19224413.
“Monoamine oxidase a and catechol-o-methyltransferase functional polymorphisms and the placebo response in major depressive disorder”. Journal of Clinical Psychopharmacology29 (4): 372–7. (August 2009). doi:10.1097/JCP.0b013e3181ac4aaf. PMID19593178.
“Candidate genes for aggression and antisocial behavior: a meta-analysis of association studies of the 5HTTLPR and MAOA-uVNTR”. Behavior Genetics44 (5): 427–44. (September 2014). doi:10.1007/s10519-014-9661-y. PMID24902785.
“MAOA genotype, social exclusion and aggression: an experimental test of a gene-environment interaction”. Genes, Brain and Behavior12 (1): 140–5. (February 2013). doi:10.1111/j.1601-183X.2012.00868.x. PMID23067570.
“State v. Yepez: Admissibility and Relevance of Behavioral Genetic Evidence in a Criminal Trial”. Psychiatric Services72 (7): 853–855. (July 2021). doi:10.1176/appi.ps.202100226. PMID34074149.
“Effect of MAO A deficiency on different kinds of aggression and social investigation in mice”. Aggressive Behavior33 (1): 1–6. (2007). doi:10.1002/ab.20161. PMID17441000.
“Effect of MAO A deficiency on different kinds of aggression and social investigation in mice”. Aggressive Behavior33 (1): 1–6. (1 January 2007). doi:10.1002/ab.20161. PMID17441000.
“Specification of the phenotype required for men with monoamine oxidase type A deficiency”. Human Genetics96 (3): 372–6. (September 1995). doi:10.1007/BF00210430. PMID7649563.
“Piperine from the fruits of Piper longum with inhibitory effect on monoamine oxidase and antidepressant-like activity”. Chemical & Pharmaceutical Bulletin53 (7): 832–5. (July 2005). doi:10.1248/cpb.53.832. PMID15997146.
“Monoamine oxidase inhibition by Rhodiola rosea L. roots”. Journal of Ethnopharmacology122 (2): 397–401. (March 2009). doi:10.1016/j.jep.2009.01.007. PMID19168123.