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非相同末端結合 (Japanese Wikipedia)

Analysis of information sources in references of the Wikipedia article "非相同末端結合" in Japanese language version.

refsWebsite
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29doi.org
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1cshlp.org
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1cell.com
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4,178th place
1jimmunol.org
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cell.com

  • Moshous, D., Callebaut, I., de Chasseval, R., Corneo, B., Cavazzana-Calvo, M., Le Deist, F., Tezcan, I., Sanal, O., Bertrand, Y., Philippe, N., Fischer, A., & de Villartay, J.-P. (2001). Artemis, a novel DNA Double-Strand break Repair/V(D)J recombination protein, is mutated in human severe combined immune deficiency. Cell, 105 (2), 177-186. URL http://www.cell.com//abstract/S0092-8674(01)00309-9

cshlp.org

genesdev.cshlp.org

doi.org

  • Lieber, M. R. (2010). The mechanism of Double-Strand DNA break repair by the nonhomologous DNA End-Joining pathway. Annual Review of Biochemistry, 79 (1), 181-211. URL https://doi.org/10.1146/annurev.biochem.052308.093131
  • Ochi, T., Blackford, A. N., Coates, J., Jhujh, S., Mehmood, S., Tamura, N., Travers, J., Wu, Q., Draviam, V. M., Robinson, C. V., Blundell, T. L., Jackson, S. P., Jan. 2015. PAXX, a paralog of XRCC4 and XLF, interacts with ku to promote DNA double-strand break repair. Science 347 (6218), 185-188. URL https://doi.org/10.1126/science.1261971
  • Williams, G. J., Lees-Miller, S. P., & Tainer, J. A. (2010). Mre11-Rad50-nbs1 conformations and the control of sensing, signaling, and effector responses at DNA double-strand breaks. DNA Repair, 9 (12), 1299-1306. URL https://doi.org/10.1016/j.dnarep.2010.10.001
  • Dimitrova, N., Chen, Y.-C. M., Spector, D. L., & de Lange, T. (2008). 53BP1 promotes non-homologous end joining of telomeres by increasing chromatin mobility. Nature, 456 (7221), 524-528. URL https://doi.org/10.1038/nature07433
  • Difilippantonio, S., Gapud, E., Wong, N., Huang, C.-Y. Y., Mahowald, G., Chen, H. T. T., Kruhlak, M. J., Callen, E., Livak, F., Nussenzweig, M. C., Sleckman, B. P., & Nussenzweig, A. (2008). 53BP1 facilitates long-range DNA end-joining during V(D)J recombination. Nature, 456 (7221), 529-533. URL https://doi.org/10.1038/nature07476
  • Downs, J. A., & Jackson, S. P. (2004). A means to a DNA end: the many roles of ku. Nature Reviews Molecular Cell Biology, 5 (5), 367-378. URL https://doi.org/10.1038/nrm1367
  • Gell, D., Jackson, S. P., Sep. 1999. Mapping of protein-protein interactions within the DNA-dependent protein kinase complex. Nucleic Acids Research 27 (17), 3494-3502. URL https://doi.org/10.1093/nar/27.17.3494
  • Neal, J. A., & Meek, K. (2011). Choosing the right path: Does DNA-PK help make the decision? Mutation Research/Fundamental and Molecular Mechanisms of Mutagenesis, 711 (1-2), 73-86. URL https://doi.org/10.1016/j.mrfmmm.2011.02.010
  • Roy, S., Andres, S. N., Vergnes, A., Neal, J. A., Xu, Y., Yu, Y., Lees-Miller, S. P., Junop, M., Modesti, M., & Meek, K. (2012). XRCC4's interaction with XLF is required for coding (but not signal) end joining. Nucleic Acids Research, 40 (4), 1684-1694. URL https://doi.org/10.1093/nar/gkr1315
  • Xing, M., Yang, M., Huo, W., Feng, F., Wei, L., Jiang, W., Ning, S., Yan, Z., Li, W., Wang, Q., Hou, M., Dong, C., Guo, R., Gao, G., Ji, J., Zha, S., Lan, L., Liang, H., Xu, D., Feb. 2015. Interactome analysis identifies a new paralogue of XRCC4 in non-homologous end joining DNA repair pathway. Nature Communications 6, 6233+. URL https://doi.org/10.1038/ncomms7233
  • Craxton, A., Somers, J., Munnur, D., Jukes-Jones, R., Cain, K., Malewicz, M., Mar. 2015. XLS (c9orf142) is a new component of mammalian DNA double-stranded break repair. Cell Death & Differentiation aop (current). URL https://doi.org/10.1038/cdd.2015.22
  • Ma, Y., Pannicke, U., Schwarz, K., & Lieber, M. R. (2002). Hairpin opening and overhang processing by an Artemis/DNA-dependent protein kinase complex in nonhomologous end joining and V(D)j recombination. Cell, 108 (6), 781-794. URL https://doi.org/10.1016/s0092-8674(02)00671-2
  • Moon, A. F., Garcia-Diaz, M., Batra, V. K., Beard, W. A., Bebenek, K., Kunkel, T. A., Wilson, S. H., & Pedersen, L. C. (2007). The x family portrait: structural insights into biological functions of x family polymerases. DNA repair, 6 (12), 1709-1725. URL https://doi.org/10.1016/j.dnarep.2007.05.009
  • Gellert, M. (2002). V(D)j recombination: RAG proteins, repair factors, and regulation. Annual review of biochemistry, 71 , 101-132. URL https://doi.org/10.1146/annurev.biochem.71.090501.150203
  • Bryans, M., Valenzano, M. C., & Stamato, T. D. (1999). Absence of DNA ligase IV protein in XR-1 cells: evidence for stabilization by XRCC4. Mutation Research/DNA Repair, 433 (1), 53-58. URL https://doi.org/10.1016/s0921-8777(98)00063-9
  • Tsai, C. J., Kim, S. A., & Chu, G. (2007). Cernunnos/XLF promotes the ligation of mismatched and noncohesive DNA ends. Proceedings of the National Academy of Sciences of the United States of America, 104 (19), 7851-7856. URL https://doi.org/10.1073/pnas.0702620104
  • Riballo, E., Woodbine, L., Stiff, T., Walker, S. A., Goodarzi, A. A., & Jeggo, P. A. (2009). XLF-cernunnos promotes DNA ligase IV-XRCC4 re-adenylation following ligation. Nucleic acids research, 37 (2), 482-492. URL https://doi.org/10.1093/nar/gkn957
  • Chen, X., & Tomkinson, A. E. (2011). Yeast nej1 is a key participant in the initial end binding and final ligation steps of nonhomologous end joining. The Journal of biological chemistry, 286 (6), 4931-4940. URL https://doi.org/10.1074/jbc.m110.195024
  • van der Burg, M., Ijspeert, H., Verkaik, N. S., Turul, T., Wiegant, W. W., Morotomi-Yano, K., Mari, P.-O. O., Tezcan, I., Chen, D. J., Zdzienicka, M. Z., van Dongen, J. J., & van Gent, D. C. (2009). A DNA-PKcs mutation in a radiosensitive T-B- SCID patient inhibits artemis activation and nonhomologous end-joining. The Journal of clinical investigation, 119 (1), 91-98. URL https://doi.org/10.1172/jci37141
  • Chistiakov, D. A., Voronova, N. V., & Chistiakov, A. P. (2009). Ligase IV syndrome. European Journal of Medical Genetics, 52 (6), 373-378. URL https://doi.org/10.1016/j.ejmg.2009.05.009
  • Buck, D., Malivert, L., de Chasseval, R., Barraud, A., Fondaneche, M.-C., Sanal, O., Plebani, A., Stephan, J.-L., Hufnagel, M., le Deist, F., Fischer, A., Durandy, A., Jean-Pierre, D., & Revy, P. (2006). Cernunnos, a novel nonhomologous End-Joining factor, is mutated in human immunodeficiency with microcephaly. Cell, 124 (2), 287-299. URL https://doi.org/10.1016/j.cell.2005.12.030
  • Shaheen, R., Faqeih, E., Ansari, S., Abdel-Salam, G., Al-Hassnan, Z. N., Al-Shidi, T., Alomar, R., Sogaty, S., Alkuraya, F. S., Feb. 2014. Genomic analysis of primordial dwarfism reveals novel disease genes. Genome Research 24 (2), 291-299. URL https://doi.org/10.1101/gr.160572.113
  • Murray, J. E., van der Burg, M., IJspeert, H., Carroll, P., Wu, Q., Ochi, T., Leitch, A., Miller, E. S., Kysela, B., Jawad, A., Bottani, A., Brancati, F., Cappa, M., Cormier-Daire, V., Deshpande, C., Faqeih, E. A., Graham, G. E., Ranza, E., Blundell, T. L., Jackson, A. P., Stewart, G. S., Bicknell, L. S., Mar. 2015. Mutations in the NHEJ component XRCC4 cause primordial dwarfism. The American Journal of Human Genetics 96 (3), 412-424. URL https://doi.org/10.1016/j.ajhg.2015.01.013
  • Buck, D., Moshous, D., de Chasseval, R., Ma, Y., Francoise, L. D., Cavazzana-Calvo, M., Fischer, A., Casanova, J.-L., Lieber, M. R., & deVillartay, J.-P. (2006). Severe combined immunodeficiency and microcephaly in siblings with hypomorphic mutations in DNA ligase IV. Eur. J. Immunol., 36 (1), 224-235. URL https://doi.org/10.1002/eji.200535401
  • Grunebaum, E., Bates, A., & Roifman, C. (2008). Omenn syndrome is associated with mutations in DNA ligase IV. Journal of Allergy and Clinical Immunology, 122 (6), 1219-1220. URL https://doi.org/10.1016/j.jaci.2008.08.031
  • Riballo, E., Critchlow, S. E., Teo, S. H., Doherty, A. J., Priestley, A., Broughton, B., Kysela, B., Beamish, H., Plowman, N., & Arlett, C. F. (1999). Identification of a defect in DNA ligase IV in a radiosensitive leukaemia patient. Current Biology, 9 (13), 699-S2. URL https://doi.org/10.1016/s0960-9822(99)80311-x
  • Ben-Omran, T. I., Cerosaletti, K., Concannon, P., Weitzman, S., & Nezarati, M. M. (2005). A patient with mutations in DNA ligase IV: Clinical features and overlap with nijmegen breakage syndrome. Am. J. Med. Genet., 137A (3), 283-287. URL https://doi.org/10.1002/ajmg.a.30869
  • Toita, N., Hatano, N., Ono, S., Yamada, M., Kobayashi, R., Kobayashi, I., Kawamura, N., Okano, M., Satoh, A., Nakagawa, A., Ohshima, K., Shindoh, M., Takami, T., Kobayashi, K., & Ariga, T. (2007). Epstein-Barr virus-associated b-cell lymphoma in a patient with DNA ligase IV (LIG4) syndrome. Am. J. Med. Genet., 143A (7), 742-745. URL https://doi.org/10.1002/ajmg.a.31644
  • Murray, J. E., Bicknell, L. S., Yigit, G., Duker, A. L., van Kogelenberg, M., Haghayegh, S., Wieczorek, D., Kayserili, H., Albert, M. H., Wise, C. A., Brandon, J., Kleefstra, T., Warris, A., van der Flier, M., Bamforth, J. S., Doonanco, K., Adès, L., Ma, A., Field, M., Johnson, D., Shackley, F., Firth, H., Woods, C. G., Nürnberg, P., Gatti, R. A., Hurles, M., Bober, M. B., Wollnik, B., Jackson, A. P., Jan. 2014. Extreme growth failure is a common presentation of ligase IV deficiency. Human Mutation 35 (1), 76-85. URL https://doi.org/10.1002/humu.22461

jimmunol.org

  • Enders, A., Fisch, P., Schwarz, K., Duffner, U., Pannicke, U., Nikolopoulos, E., Peters, A., Orlowska-Volk, M., Schindler, D., Friedrich, W., Selle, B., Niemeyer, C., & Ehl, S. (2006). A severe form of human combined immunodeficiency due to mutations in DNA ligase IV. The Journal of Immunology, 176 (8), 5060-5068. URL http://www.jimmunol.org/content/176/8/5060.abstract