디하이드로아스코르브산 (Korean Wikipedia)

Analysis of information sources in references of the Wikipedia article "디하이드로아스코르브산" in Korean language version.

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6doi.org

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5nih.gov

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1harvard.edu

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doi.org

dx.doi.org

May, J. M. (1998). “Ascorbate function and metabolism in the human erythrocyte”. 《Frontiers in Bioscience》 3 (4): d1–10. doi:10.2741/a262. PMID 9405334.
Welch, R. W.; Wang, Y.; Crossman, A. Jr.; Park, J. B.; Kirk, K. L.; Levine, M. (1995). “Accumulation of Vitamin C (Ascorbate) and Its Oxidized Metabolite Dehydroascorbic Acid Occurs by Separate Mechanisms”. 《Journal of Biological Chemistry》 270 (21): 12584–12592. doi:10.1074/jbc.270.21.12584. PMID 7759506.
Lee, Y. C.; Huang, H. Y.; Chang, C. J.; Cheng, C. H.; Chen, Y. T. (2010). “Mitochondrial GLUT10 facilitates dehydroascorbic acid import and protects cells against oxidative stress: Mechanistic insight into arterial tortuosity syndrome”. 《Human Molecular Genetics》 19 (19): 3721–33. doi:10.1093/hmg/ddq286. PMID 20639396.
Kerber, R. C. (2008). “"As Simple as Possible, but Not Simpler"—The Case of Dehydroascorbic Acid”. 《Journal of Chemical Education》 85 (9): 1237. Bibcode:2008JChEd..85.1237K. doi:10.1021/ed085p1237.
Kimoto, E.; Tanaka, H.; Ohmoto, T.; Choami, M. (1993). “Analysis of the transformation products of dehydro-L-ascorbic acid by ion-pairing high-performance liquid chromatography”. 《Analytical Biochemistry》 214 (1): 38–44. doi:10.1006/abio.1993.1453. PMID 8250252.
Montel-Hagen, A.; Kinet, S.; Manel, N.; Mongellaz, C.; Prohaska, R.; Battini, J. L.; Delaunay, J.; Sitbon, M.; Taylor, N. (2008). “Erythrocyte Glut1 triggers dehydroascorbic acid uptake in mammals unable to synthesize vitamin C”. 《Cell》 132 (6): 1039–48. doi:10.1016/j.cell.2008.01.042. PMID 18358815.

harvard.edu

adsabs.harvard.edu

Kerber, R. C. (2008). “"As Simple as Possible, but Not Simpler"—The Case of Dehydroascorbic Acid”. 《Journal of Chemical Education》 85 (9): 1237. Bibcode:2008JChEd..85.1237K. doi:10.1021/ed085p1237.

nih.gov

ncbi.nlm.nih.gov

May, J. M. (1998). “Ascorbate function and metabolism in the human erythrocyte”. 《Frontiers in Bioscience》 3 (4): d1–10. doi:10.2741/a262. PMID 9405334.
Welch, R. W.; Wang, Y.; Crossman, A. Jr.; Park, J. B.; Kirk, K. L.; Levine, M. (1995). “Accumulation of Vitamin C (Ascorbate) and Its Oxidized Metabolite Dehydroascorbic Acid Occurs by Separate Mechanisms”. 《Journal of Biological Chemistry》 270 (21): 12584–12592. doi:10.1074/jbc.270.21.12584. PMID 7759506.
Lee, Y. C.; Huang, H. Y.; Chang, C. J.; Cheng, C. H.; Chen, Y. T. (2010). “Mitochondrial GLUT10 facilitates dehydroascorbic acid import and protects cells against oxidative stress: Mechanistic insight into arterial tortuosity syndrome”. 《Human Molecular Genetics》 19 (19): 3721–33. doi:10.1093/hmg/ddq286. PMID 20639396.
Kimoto, E.; Tanaka, H.; Ohmoto, T.; Choami, M. (1993). “Analysis of the transformation products of dehydro-L-ascorbic acid by ion-pairing high-performance liquid chromatography”. 《Analytical Biochemistry》 214 (1): 38–44. doi:10.1006/abio.1993.1453. PMID 8250252.
Montel-Hagen, A.; Kinet, S.; Manel, N.; Mongellaz, C.; Prohaska, R.; Battini, J. L.; Delaunay, J.; Sitbon, M.; Taylor, N. (2008). “Erythrocyte Glut1 triggers dehydroascorbic acid uptake in mammals unable to synthesize vitamin C”. 《Cell》 132 (6): 1039–48. doi:10.1016/j.cell.2008.01.042. PMID 18358815.