BRCA1 (Polish Wikipedia)

Analysis of information sources in references of the Wikipedia article "BRCA1" in Polish language version.

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doi.org

dx.doi.org

  • Y. Miki i inni, A strong candidate for the breast and ovarian cancer susceptibility gene BRCA1, „Science”, 266 (5182), 1994, s. 66–71, DOI10.1126/science.7545954, PMID7545954.
  • J.S. Lee, J.H. Chung, Diverse functions of BRCA1 in the DNA damage response, „Expert Reviews in Molecular Medicine”, 2001, 2001, s. 1–11, DOI10.1017/S1462399401003131, PMID14987363.
  • L.C. Wu i inni, Identification of a RING protein that can interact in vivo with the BRCA1 gene product, „Nature Genetics”, 14 (4), 1996, s. 430–440, DOI10.1038/ng1296-430, PMID8944023.
  • K. Somasundaram i inni, Arrest of the cell cycle by the tumour-suppressor BRCA1 requires the CDK-inhibitor p21WAF1/CiP1, „Nature”, 389 (6647), 1997, s. 187–190, DOI10.1038/38291, PMID9296497.
  • A.N. Monteiro, A. August, H. Hanafusa, Evidence for a transcriptional activation function of BRCA1 C-terminal region, „Proceedings of the National Academy of Sciences of the United States of America”, 93 (24), 1996, s. 13595–13599, DOI10.1073/pnas.93.24.13595, PMID8942979, PMCIDPMC19361.
  • J.E. Thomas i inni, Subcellular localization and analysis of apparent 180-kDa and 220-kDa proteins of the breast cancer susceptibility gene, BRCA1, „Journal of Biological Chemistry”, 271 (45), 1996, s. 28630–28635, DOI10.1074/jbc.271.45.28630, PMID8910495.
  • T.F. Lane i inni, Expression of Brca1 is associated with terminal differentiation of ectodermally and mesodermally derived tissues in mice, „Genes & Development”, 9 (21), 1995, s. 2712–2722, DOI10.1101/gad.9.21.2712, PMID7590247.
  • S.T. Marquis i inni, The developmental pattern of Brca1 expression implies a role in differentiation of the breast and other tissues, „Nature Genetics”, 11 (1), 1995, s. 17–26, DOI10.1038/ng0995-17, PMID7550308.
  • J.M. Gudas i inni, Hormone-dependent regulation of BRCA1 in human breast cancer cells, „Cancer Research”, 55 (20), 1995, s. 4561–4565, DOI10.1038/ng0995-17, ISSN 0008-5472, PMID7553629 [dostęp 2019-02-10].
  • C.Y. Liu i inni, Inactivation of the mouse Brca1 gene leads to failure in the morphogenesis of the egg cylinder in early postimplantation development, „Genes & Development”, 10 (14), 1996, s. 1835–1843, DOI10.1101/gad.10.14.1835, PMID8698242.
  • R. Hakem i inni, The tumor suppressor gene Brca1 is required for embryonic cellular proliferation in the mouse, „Cell”, 85 (7), 1996, s. 1009–1023, DOI10.1016/S0092-8674(00)81302-1, ISSN 0092-8674, PMID8674108 [dostęp 2019-02-10].
  • L.C. Gowen i inni, Brca1 deficiency results in early embryonic lethality characterized by neuroepithelial abnormalities, „Nature Genetics”, 12 (2), 1996, s. 191–194, DOI10.1038/ng0296-191, PMID8563759.
  • R. Hakem i inni, Partial rescue of Brca1 (5-6) early embryonic lethality by p53 or p21 null mutation, „Nature Genetics”, 16 (3), 1997, s. 298–302, DOI10.1038/ng0797-298, PMID9207798.
  • X. Xu i inni, Conditional mutation of Brca1 in mammary epithelial cells results in blunted ductal morphogenesis and tumour formation, „Nature Genetics”, 22 (1), 1999, s. 37–43, DOI10.1038/8743, PMID10319859.
  • X. Xu i inni, Centrosome amplification and a defective G2-M cell cycle checkpoint induce genetic instability in BRCA1 exon 11 isoform-deficient cells, „Molecular Cell”, 3 (3), 1999, s. 389–395, DOI10.1016/S1097-2765(00)80466-9, PMID10198641.
  • L.C. Hsu, R.L. White, BRCA1 is associated with the centrosome during mitosis, „Proceedings of the National Academy of Sciences of the United States of America”, 95 (22), 1998, s. 12983–12988, DOI10.1073/pnas.95.22.12983, PMID9789027, PMCIDPMC23679.
  • Bohdan Górski i inni, A high proportion of founder BRCA1 mutations in Polish breast cancer families, „International Journal of Cancer”, 110 (5), 2004, s. 683–686, DOI10.1002/ijc.20162, PMID15146557.
  • S.A. Narod i inni, Tamoxifen and risk of contralateral breast cancer in BRCA1 and BRCA2 mutation carriers: a case-control study. Hereditary Breast Cancer Clinical Study Group, „The Lancet”, 356 (9245), 2000, s. 1876–1881, DOI10.1016/S0140-6736(00)03258-X, PMID11130383.
  • T. Byrski i inni, Response to neo-adjuvant chemotherapy in women with BRCA1-positive breast cancers, „Breast Cancer Research and Treatment”, 108 (2), 2008, s. 289–296, DOI10.1007/s10549-007-9600-1, PMID17492376.

nih.gov

ncbi.nlm.nih.gov

  • Y. Miki i inni, A strong candidate for the breast and ovarian cancer susceptibility gene BRCA1, „Science”, 266 (5182), 1994, s. 66–71, DOI10.1126/science.7545954, PMID7545954.
  • J.S. Chamberlain i inni, BRCA1 maps proximal to D17S579 on chromosome 17q21 by genetic analysis, „American Journal of Human Genetics”, 52 (4), 1993, s. 792–798, PMID8460646, PMCIDPMC1682065.
  • J.S. Lee, J.H. Chung, Diverse functions of BRCA1 in the DNA damage response, „Expert Reviews in Molecular Medicine”, 2001, 2001, s. 1–11, DOI10.1017/S1462399401003131, PMID14987363.
  • L.C. Wu i inni, Identification of a RING protein that can interact in vivo with the BRCA1 gene product, „Nature Genetics”, 14 (4), 1996, s. 430–440, DOI10.1038/ng1296-430, PMID8944023.
  • K. Somasundaram i inni, Arrest of the cell cycle by the tumour-suppressor BRCA1 requires the CDK-inhibitor p21WAF1/CiP1, „Nature”, 389 (6647), 1997, s. 187–190, DOI10.1038/38291, PMID9296497.
  • A.N. Monteiro, A. August, H. Hanafusa, Evidence for a transcriptional activation function of BRCA1 C-terminal region, „Proceedings of the National Academy of Sciences of the United States of America”, 93 (24), 1996, s. 13595–13599, DOI10.1073/pnas.93.24.13595, PMID8942979, PMCIDPMC19361.
  • J.E. Thomas i inni, Subcellular localization and analysis of apparent 180-kDa and 220-kDa proteins of the breast cancer susceptibility gene, BRCA1, „Journal of Biological Chemistry”, 271 (45), 1996, s. 28630–28635, DOI10.1074/jbc.271.45.28630, PMID8910495.
  • Y. Chen i inni, BRCA1 is a 220-kDa nuclear phosphoprotein that is expressed and phosphorylated in a cell cycle-dependent manner, „Cancer Research”, 56 (14), 1996, s. 3168–3172, PMID8764100.
  • T.F. Lane i inni, Expression of Brca1 is associated with terminal differentiation of ectodermally and mesodermally derived tissues in mice, „Genes & Development”, 9 (21), 1995, s. 2712–2722, DOI10.1101/gad.9.21.2712, PMID7590247.
  • S.T. Marquis i inni, The developmental pattern of Brca1 expression implies a role in differentiation of the breast and other tissues, „Nature Genetics”, 11 (1), 1995, s. 17–26, DOI10.1038/ng0995-17, PMID7550308.
  • J.M. Gudas i inni, Hormone-dependent regulation of BRCA1 in human breast cancer cells, „Cancer Research”, 55 (20), 1995, s. 4561–4565, DOI10.1038/ng0995-17, ISSN 0008-5472, PMID7553629 [dostęp 2019-02-10].
  • J.M. Gudas i inni, Cell cycle regulation of BRCA1 messenger RNA in human breast epithelial cells, „Cell Growth & Differentiation”, 7 (6), 1996, s. 717–723, PMID8780885.
  • J.P. Vaughn i inni, BRCA1 expression is induced before DNA synthesis in both normal and tumor-derived breast cells, „Cell Growth & Differentiation”, 7 (6), 1996, s. 711–715, PMID8780884.
  • C.Y. Liu i inni, Inactivation of the mouse Brca1 gene leads to failure in the morphogenesis of the egg cylinder in early postimplantation development, „Genes & Development”, 10 (14), 1996, s. 1835–1843, DOI10.1101/gad.10.14.1835, PMID8698242.
  • R. Hakem i inni, The tumor suppressor gene Brca1 is required for embryonic cellular proliferation in the mouse, „Cell”, 85 (7), 1996, s. 1009–1023, DOI10.1016/S0092-8674(00)81302-1, ISSN 0092-8674, PMID8674108 [dostęp 2019-02-10].
  • L.C. Gowen i inni, Brca1 deficiency results in early embryonic lethality characterized by neuroepithelial abnormalities, „Nature Genetics”, 12 (2), 1996, s. 191–194, DOI10.1038/ng0296-191, PMID8563759.
  • R. Hakem i inni, Partial rescue of Brca1 (5-6) early embryonic lethality by p53 or p21 null mutation, „Nature Genetics”, 16 (3), 1997, s. 298–302, DOI10.1038/ng0797-298, PMID9207798.
  • X. Xu i inni, Conditional mutation of Brca1 in mammary epithelial cells results in blunted ductal morphogenesis and tumour formation, „Nature Genetics”, 22 (1), 1999, s. 37–43, DOI10.1038/8743, PMID10319859.
  • X. Xu i inni, Centrosome amplification and a defective G2-M cell cycle checkpoint induce genetic instability in BRCA1 exon 11 isoform-deficient cells, „Molecular Cell”, 3 (3), 1999, s. 389–395, DOI10.1016/S1097-2765(00)80466-9, PMID10198641.
  • L.C. Hsu, R.L. White, BRCA1 is associated with the centrosome during mitosis, „Proceedings of the National Academy of Sciences of the United States of America”, 95 (22), 1998, s. 12983–12988, DOI10.1073/pnas.95.22.12983, PMID9789027, PMCIDPMC23679.
  • J.S. Larson, J.L. Tonkinson, M.T. Lai, A BRCA1 mutant alters G2-M cell cycle control in human mammary epithelial cells, „Cancer Research”, 57 (16), 1997, s. 3351–3355, ISSN 0008-5472, PMID9269994 [dostęp 2019-02-10].
  • Bohdan Górski i inni, A high proportion of founder BRCA1 mutations in Polish breast cancer families, „International Journal of Cancer”, 110 (5), 2004, s. 683–686, DOI10.1002/ijc.20162, PMID15146557.
  • S.A. Narod i inni, Tamoxifen and risk of contralateral breast cancer in BRCA1 and BRCA2 mutation carriers: a case-control study. Hereditary Breast Cancer Clinical Study Group, „The Lancet”, 356 (9245), 2000, s. 1876–1881, DOI10.1016/S0140-6736(00)03258-X, PMID11130383.
  • T. Byrski i inni, Response to neo-adjuvant chemotherapy in women with BRCA1-positive breast cancers, „Breast Cancer Research and Treatment”, 108 (2), 2008, s. 289–296, DOI10.1007/s10549-007-9600-1, PMID17492376.

worldcat.org

  • J.M. Gudas i inni, Hormone-dependent regulation of BRCA1 in human breast cancer cells, „Cancer Research”, 55 (20), 1995, s. 4561–4565, DOI10.1038/ng0995-17, ISSN 0008-5472, PMID7553629 [dostęp 2019-02-10].
  • R. Hakem i inni, The tumor suppressor gene Brca1 is required for embryonic cellular proliferation in the mouse, „Cell”, 85 (7), 1996, s. 1009–1023, DOI10.1016/S0092-8674(00)81302-1, ISSN 0092-8674, PMID8674108 [dostęp 2019-02-10].
  • J.S. Larson, J.L. Tonkinson, M.T. Lai, A BRCA1 mutant alters G2-M cell cycle control in human mammary epithelial cells, „Cancer Research”, 57 (16), 1997, s. 3351–3355, ISSN 0008-5472, PMID9269994 [dostęp 2019-02-10].