D. Mansour, K. Gemzell-Danielsson, P. Inki, J.T. Jensen. Fertility after discontinuation of contraception: a comprehensive review of the literature. „Contraception”. 84 (5), s. 465–477, Nov 2011. DOI: 10.1016/j.contraception.2011.04.002. PMID: 22018120.
P.C. Hannaford, L. Iversen, T.V. Macfarlane, A.M. Elliott i inni. Mortality among contraceptive pill users: cohort evidence from Royal College of General Practitioners’ Oral Contraception Study. „BMJ”. 340, s. c927, Mar 2010. DOI: 10.1136/bmj.c927. PMID: 20223876. PMCID: PMC2837145.
D. Cibula, A. Gompel, A.O. Mueck, C. La Vecchia i inni. Hormonal contraception and risk of cancer. „Hum Reprod Update”. 16 (6). s. 631–650. DOI: 10.1093/humupd/dmq022. PMID: 20543200. (ang.).
J.C. Huber, E.K. Bentz, J. Ott, C.B. Tempfer. Non-contraceptive benefits of oral contraceptives. „Expert Opin Pharmacother”. 9 (13), s. 2317–2325, Sep 2008. DOI: 10.1517/14656566.9.13.2317. PMID: 18710356.
M.F. Gallo, L.M. Lopez, D.A. Grimes, K.F. Schulz i inni. Combination contraceptives: effects on weight. „Cochrane Database Syst Rev”. 7 (9), s. CD003987, Sep 2011. DOI: 10.1002/14651858.CD003987.pub4. PMID: 21901687. (ang.).
J.H. Ford, L. MacCormac. Pregnancy and lifestyle study: the long-term use of the contraceptive pill and the risk of age-related miscarriage. „Hum Reprod”. 10 (6), s. 1397–1402, Jun 1995. DOI: 10.1093/HUMREP/10.6.1397. PMID: 7593504. (ang.).
A. García-Enguídanos, D. Martínez, M.E. Calle, S. Luna i inni. Long-term use of oral contraceptives increases the risk of miscarriage. „Fertil Steril”. 83 (6), s. 1864–1866, Jun 2005. DOI: 10.1016/j.fertnstert.2004.11.085. PMID: 15950668.
R. Jellesen, K. Strandberg-Larsen, T. Jørgensen, J. Olsen i inni. Maternal use of oral contraceptives and risk of fetal death. „Paediatr Perinat Epidemiol”. 22 (4), s. 334–340, Jul 2008. DOI: 10.1111/j.1365-3016.2008.00942.x. PMID: 18578746. (ang.).
J.M. Kemmeren, A. Algra, D.E. Grobbee. Third generation oral contraceptives and risk of venous thrombosis: meta-analysis. „BMJ”. 323 (7305), s. 131–134, Jul 2001. DOI: 10.1136/bmj.323.7305.131. PMID: 11463678. PMCID: PMC34722. (ang.).
M.B. Brito, F. Nobre, C.S. Vieira. Hormonal contraception and cardiovascular system. „Arq Bras Cardiol”. 96 (4), s. e81-9, 2011. DOI: 10.1590/S0066-782X2011005000022. PMID: 21359483. (ang. • port. • hiszp.).
C. Sultan, P. Balaguer, B. Terouanne, V. Georget i inni. Environmental xenoestrogens, antiandrogens and disorders of male sexual differentiation. „Mol Cell Endocrinol”. 178 (1–2), s. 99–105, 2001. DOI: 10.1016/s0303-7207(01)00430-0. PMID: 11403899.
JJ.ToppariJJ. i inni, Male reproductive health and environmental xenoestrogens, „Environmental Health Perspectives”, 104 Suppl 4 (Suppl 4), 1996, s. 741–803, DOI: 10.1289/ehp.96104s4741, PMID: 8880001, PMCID: PMC1469672.
R. Rivera, I. Yacobson, D. Grimes. The mechanism of action of hormonal contraceptives and intrauterine contraceptive devices. „Am J Obstet Gynecol”. 181 (5 Pt 1), s. 1263–1269, Nov 1999. PMID: 10561657.
W.L. Larimore, J.B. Stanford. Postfertilization effects of oral contraceptives and their relationship to informed consent. „Arch Fam Med”. 9 (2), s. 126–133, Feb 2000. PMID: 10693729.
D. Mansour, K. Gemzell-Danielsson, P. Inki, J.T. Jensen. Fertility after discontinuation of contraception: a comprehensive review of the literature. „Contraception”. 84 (5), s. 465–477, Nov 2011. DOI: 10.1016/j.contraception.2011.04.002. PMID: 22018120.
P.C. Hannaford, L. Iversen, T.V. Macfarlane, A.M. Elliott i inni. Mortality among contraceptive pill users: cohort evidence from Royal College of General Practitioners’ Oral Contraception Study. „BMJ”. 340, s. c927, Mar 2010. DOI: 10.1136/bmj.c927. PMID: 20223876. PMCID: PMC2837145.
D. Cibula, A. Gompel, A.O. Mueck, C. La Vecchia i inni. Hormonal contraception and risk of cancer. „Hum Reprod Update”. 16 (6). s. 631–650. DOI: 10.1093/humupd/dmq022. PMID: 20543200. (ang.).
J.C. Huber, E.K. Bentz, J. Ott, C.B. Tempfer. Non-contraceptive benefits of oral contraceptives. „Expert Opin Pharmacother”. 9 (13), s. 2317–2325, Sep 2008. DOI: 10.1517/14656566.9.13.2317. PMID: 18710356.
M.F. Gallo, L.M. Lopez, D.A. Grimes, K.F. Schulz i inni. Combination contraceptives: effects on weight. „Cochrane Database Syst Rev”. 7 (9), s. CD003987, Sep 2011. DOI: 10.1002/14651858.CD003987.pub4. PMID: 21901687. (ang.).
J.H. Ford, L. MacCormac. Pregnancy and lifestyle study: the long-term use of the contraceptive pill and the risk of age-related miscarriage. „Hum Reprod”. 10 (6), s. 1397–1402, Jun 1995. DOI: 10.1093/HUMREP/10.6.1397. PMID: 7593504. (ang.).
A. García-Enguídanos, D. Martínez, M.E. Calle, S. Luna i inni. Long-term use of oral contraceptives increases the risk of miscarriage. „Fertil Steril”. 83 (6), s. 1864–1866, Jun 2005. DOI: 10.1016/j.fertnstert.2004.11.085. PMID: 15950668.
R. Jellesen, K. Strandberg-Larsen, T. Jørgensen, J. Olsen i inni. Maternal use of oral contraceptives and risk of fetal death. „Paediatr Perinat Epidemiol”. 22 (4), s. 334–340, Jul 2008. DOI: 10.1111/j.1365-3016.2008.00942.x. PMID: 18578746. (ang.).
J.M. Kemmeren, A. Algra, D.E. Grobbee. Third generation oral contraceptives and risk of venous thrombosis: meta-analysis. „BMJ”. 323 (7305), s. 131–134, Jul 2001. DOI: 10.1136/bmj.323.7305.131. PMID: 11463678. PMCID: PMC34722. (ang.).
M.B. Brito, F. Nobre, C.S. Vieira. Hormonal contraception and cardiovascular system. „Arq Bras Cardiol”. 96 (4), s. e81-9, 2011. DOI: 10.1590/S0066-782X2011005000022. PMID: 21359483. (ang. • port. • hiszp.).
C. Sultan, P. Balaguer, B. Terouanne, V. Georget i inni. Environmental xenoestrogens, antiandrogens and disorders of male sexual differentiation. „Mol Cell Endocrinol”. 178 (1–2), s. 99–105, 2001. DOI: 10.1016/s0303-7207(01)00430-0. PMID: 11403899.
JJ.ToppariJJ. i inni, Male reproductive health and environmental xenoestrogens, „Environmental Health Perspectives”, 104 Suppl 4 (Suppl 4), 1996, s. 741–803, DOI: 10.1289/ehp.96104s4741, PMID: 8880001, PMCID: PMC1469672.
Nelson, Anita L.; Cwiak, Carrie (2011). „Combined oral contraceptives (COCs)”. In Robert Hatcher: Contraceptive technology. New York, N.Y: Ardent Media, 2011, s. 257–258. ISBN 978-1-59708-004-0. OCLC781956734. Cytat: „Mechanism of action COCs prevent fertilization and, therefore, qualify as contraceptives. There is no significant evidence that they work after fertilization. The progestins in all COCs provide most of the contraceptive effect by suppressing ovulation and thickening cervical mucus, although the estrogens also make a small contribution to ovulation suppression. Cycle control is enhanced by the estrogen. Because COCs so effectively suppress ovulation and block ascent of sperm into the upper genital tract, the potential impact on endometrial receptivity to implantation is almost academic. When the two primary mechanisms fail, the fact that pregnancy occurs despite the endometrial changes demonstrates that those endometrial changes do not significantly contribute to the pill’s mechanism of action.”. (ang.).