Патобиологија атеросклерозе (Serbian Wikipedia)

Analysis of information sources in references of the Wikipedia article "Патобиологија атеросклерозе" in Serbian language version.

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37doi.org

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32nih.gov

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6worldcat.org

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4archive.org

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4semanticscholar.org

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3ahajournals.org

3,608^th place

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2harvard.edu

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1em-consulte.com

low place

1healthandmedicineinfo.com

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1jvascsurg.org

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1jbc.org

4,455^th place

2,178^th place

ahajournals.org

atvb.ahajournals.org

M. Abedin; Tintut; Demer; et al. (2004). „Vascular Calcification: Mechanisms and Clinical Ramifications”. Arterioscler. Thromb. Vasc. Biol. (на језику: енглески). 24 (7): 1161—1170. PMID 15155384. doi:10.1161/01.ATV.0000133194.94939.42.
Jian-Su, Shao (2006). „Molecular Mechanisms of Vascular Calcification: Lessons Learned From The Aorta”. Arterioscler. Thromb. Vasc. Biol. (на језику: енглески). 26 (7): 1423—1430. PMID 16601233. doi:10.1161/01.ATV.0000220441.42041.20.

circres.ahajournals.org

R.C. Johnson (2006). „Vascular Calcification: Pathobiological Mechanisms and Clinical Implications”. Circ. Res. (на језику: енглески). 99 (10): 1044—1059. PMID 17095733. S2CID 1935982. doi:10.1161/01.RES.0000249379.55535.21.

archive.org

Khalil, M. F.; Wagner, W. D.; Goldberg, I. J. (2004). „Molecular interactions leading to lipoprotein retention and the initiation of atherosclerosis”. Arteriosclerosis, Thrombosis, and Vascular Biology. 24 (12): 2211—2218. PMID 15472124. S2CID 2761937. doi:10.1161/01.ATV.0000147163.54024.70.
Chiu, J. J.; Chien, S. (2011). „Effects of disturbed flow on vascular endothelium: Pathophysiological basis and clinical perspectives”. Physiological Reviews. 91 (1): 327—387. PMC 3844671 . PMID 21248169. doi:10.1152/physrev.00047.2009.
Cheng, C.; Tempel, D.; Van Haperen, R.; Van Der Baan, A.; Grosveld, F.; Daemen, M. J.; Krams, R.; De Crom, R. (2006). „Atherosclerotic lesion size and vulnerability are determined by patterns of fluid shear stress”. Circulation. 113 (23): 2744—2753. PMID 16754802. S2CID 14376205. doi:10.1161/CIRCULATIONAHA.105.590018.
Jongstra-Bilen, J.; Haidari, M.; Zhu, S. N.; Chen, M.; Guha, D.; Cybulsky, M. I. (2006). „Low-grade chronic inflammation in regions of the normal mouse arterial intima predisposed to atherosclerosis”. The Journal of Experimental Medicine. 203 (9): 2073—2083. PMC 2118407 . PMID 16894012. doi:10.1084/jem.20060245.

doi.org

Sandow, S. L.; Gzik, D. J.; Lee, R. M. (2009). „Arterial internal elastic lamina holes: Relationship to function?”. Journal of Anatomy. 214 (2): 258—266. PMC 2667883 . PMID 19207987. doi:10.1111/j.1469-7580.2008.01020.x.
Frank, J. S.; Fogelman, A. M. (1989). „Ultrastructure of the intima in WHHL and cholesterol-fed rabbit aortas prepared by ultra-rapid freezing and freeze-etching”. Journal of Lipid Research. 30 (7): 967—978. PMID 2794795. doi:10.1016/S0022-2275(20)38299-7.
Rosenson, R. S.; Hurt-Camejo, E. (2012). „Phospholipase A2 enzymes and the risk of atherosclerosis”. European Heart Journal. 33 (23): 2899—3709. PMID 22802388. doi:10.1093/eurheartj/ehs148.
Wight, T. N.; Merrilees, M. J. (2004). „Proteoglycans in atherosclerosis and restenosis: Key roles for versican”. Circulation Research. 94 (9): 1158—1167. PMID 15142969. doi:10.1161/01.RES.0000126921.29919.51.
Moreth, K.; Iozzo, R. V.; Schaefer, L. (2012). „Small leucine-rich proteoglycans orchestrate receptor crosstalk during inflammation”. Cell Cycle (Georgetown, Tex.). 11 (11): 2084—2091. PMC 3368860 . PMID 22580469. doi:10.4161/cc.20316.
Khalil, M. F.; Wagner, W. D.; Goldberg, I. J. (2004). „Molecular interactions leading to lipoprotein retention and the initiation of atherosclerosis”. Arteriosclerosis, Thrombosis, and Vascular Biology. 24 (12): 2211—2218. PMID 15472124. S2CID 2761937. doi:10.1161/01.ATV.0000147163.54024.70.
Baeyens, N.; Mulligan-Kehoe, M. J.; Corti, F.; Simon, D. D.; Ross, T. D.; Rhodes, J. M.; Wang, T. Z.; Mejean, C. O.; Simons, M.; Humphrey, J.; Schwartz, M. A. (2014). „Syndecan 4 is required for endothelial alignment in flow and atheroprotective signaling”. Proceedings of the National Academy of Sciences of the United States of America. 111 (48): 17308—17313. Bibcode:2014PNAS..11117308B. PMC 4260558 . PMID 25404299. doi:10.1073/pnas.1413725111 .
Nam, D.; Ni, C. W.; Rezvan, A.; Suo, J.; Budzyn, K.; Llanos, A.; Harrison, D.; Giddens, D.; Jo, H. (2009). „Partial carotid ligation is a model of acutely induced disturbed flow, leading to rapid endothelial dysfunction and atherosclerosis”. American Journal of Physiology. Heart and Circulatory Physiology. 297 (4): H1535—43. PMC 2770764 . PMID 19684185. doi:10.1152/ajpheart.00510.2009. . URL consultato il 4 giugno 2016 (archiviato dall'url originale il 25 giugno 2016).
Cunningham, K. S.; Gotlieb, A. I. (2005). „The role of shear stress in the pathogenesis of atherosclerosis”. Laboratory Investigation; A Journal of Technical Methods and Pathology. 85 (1): 9—23. PMID 15568038. S2CID 33316555. doi:10.1038/labinvest.3700215.
Chatzizisis, Y. S.; Coskun, A. U.; Jonas, M.; Edelman, E. R.; Feldman, C. L.; Stone, P. H. (2007). „Role of endothelial shear stress in the natural history of coronary atherosclerosis and vascular remodeling: Molecular, cellular, and vascular behavior”. Journal of the American College of Cardiology. 49 (25): 2379—2393. PMID 17599600. doi:10.1016/j.jacc.2007.02.059.
Tarbell, J. M.; Shi, Z. D.; Dunn, J.; Jo, H. (2014). „Fluid Mechanics, Arterial Disease, and Gene Expression”. Annual Review of Fluid Mechanics. 46: 591—614. Bibcode:2014AnRFM..46..591T. PMC 4211638 . PMID 25360054. doi:10.1146/annurev-fluid-010313-141309.
Lu, D.; Kassab, G. S. (2011). „Role of shear stress and stretch in vascular mechanobiology”. Journal of the Royal Society, Interface. 8 (63): 1379—1385. PMC 3163429 . PMID 21733876. doi:10.1098/rsif.2011.0177.
Chiu, J. J.; Chien, S. (2011). „Effects of disturbed flow on vascular endothelium: Pathophysiological basis and clinical perspectives”. Physiological Reviews. 91 (1): 327—387. PMC 3844671 . PMID 21248169. doi:10.1152/physrev.00047.2009.
Cheng, C.; Tempel, D.; Van Haperen, R.; Van Der Baan, A.; Grosveld, F.; Daemen, M. J.; Krams, R.; De Crom, R. (2006). „Atherosclerotic lesion size and vulnerability are determined by patterns of fluid shear stress”. Circulation. 113 (23): 2744—2753. PMID 16754802. S2CID 14376205. doi:10.1161/CIRCULATIONAHA.105.590018.
Guo, X.; Kassab, G. S. (2004). „Distribution of stress and strain along the porcine aorta and coronary arterial tree”. American Journal of Physiology. Heart and Circulatory Physiology. 286 (6): H2361—8. PMID 15148060. doi:10.1152/ajpheart.01079.2003.
Kwak, B. R.; Bäck, M.; Bochaton-Piallat, M. L.; Caligiuri, G.; Daemen, M. J.; Davies, P. F.; Hoefer, I. E.; Holvoet, P.; Jo, H.; Krams, R.; Lehoux, S.; Monaco, C.; Steffens, S.; Virmani, R.; Weber, C.; Wentzel, J. J.; Evans, P. C. (2014). „Biomechanical factors in atherosclerosis: Mechanisms and clinical implications”. European Heart Journal. 35 (43): 3013—20, 3020a—3020d. PMC 4810806 . PMID 25230814. doi:10.1093/eurheartj/ehu353.
Yoshio, S.; Tokumasu, K.; Fujino, A.; Naganuma, H.; Noguti, H.; Nitta, K.; Hoshino, I. (1991). „Neuro-otological study on Behçet disease”. Nihon Jibiinkoka Gakkai Kaiho. 94 (11): 1716—1726. PMID 1757846. doi:10.3950/jibiinkoka.94.11_1716.
Van Der Heiden, K.; Cuhlmann, S.; Luong Le, A.; Zakkar, M.; Evans, P. C. (2010). „Role of nuclear factor kappaB in cardiovascular health and disease”. Clinical Science (London, England : 1979). 118 (10): 593—605. PMID 20175746. doi:10.1042/CS20090557.
Davies, P. F. (2009). „Hemodynamic shear stress and the endothelium in cardiovascular pathophysiology”. Nature Clinical Practice. Cardiovascular Medicine. 6 (1): 16—26. PMC 2851404 . PMID 19029993. doi:10.1038/ncpcardio1397.
Gimbrone Jr, M. A.; García-Cardeña, G. (2013). „Vascular endothelium, hemodynamics, and the pathobiology of atherosclerosis”. Cardiovascular Pathology : The Official Journal of the Society for Cardiovascular Pathology. 22 (1): 9—15. PMC 4564111 . PMID 22818581. doi:10.1016/j.carpath.2012.06.006.
Le, N. T.; Sandhu, U. G.; Quintana-Quezada, R. A.; Hoang, N. M.; Fujiwara, K.; Abe, J. I. (2017). „Flow signaling and atherosclerosis”. Cellular and Molecular Life Sciences : CMLS. 74 (10): 1835—1858. PMC 5391278 . PMID 28039525. doi:10.1007/s00018-016-2442-4.
Bryan, M. T.; Duckles, H.; Feng, S.; Hsiao, S. T.; Kim, H. R.; Serbanovic-Canic, J.; Evans, P. C. (2014). „Mechanoresponsive networks controlling vascular inflammation”. Arteriosclerosis, Thrombosis, and Vascular Biology. 34 (10): 2199—2305. PMID 24947523. doi:10.1161/ATVBAHA.114.303424.
Wang, C.; Baker, B. M.; Chen, C. S.; Schwartz, M. A. (2013). „Endothelial cell sensing of flow direction”. Arteriosclerosis, Thrombosis, and Vascular Biology. 33 (9): 2130—2136. PMC 3812824 . PMID 23814115. doi:10.1161/ATVBAHA.113.301826.
Jongstra-Bilen, J.; Haidari, M.; Zhu, S. N.; Chen, M.; Guha, D.; Cybulsky, M. I. (2006). „Low-grade chronic inflammation in regions of the normal mouse arterial intima predisposed to atherosclerosis”. The Journal of Experimental Medicine. 203 (9): 2073—2083. PMC 2118407 . PMID 16894012. doi:10.1084/jem.20060245.
Stary, H C; Blankenhorn, D H; Chandler, A B; Glagov, S; Insull, W; Richardson, M; Rosenfeld, M E; Schaffer, S A; Schwartz, C J (1992). „A definition of the intima of human arteries and of its atherosclerosis-prone regions. A report from the Committee on Vascular Lesions of the Council on Arteriosclerosis, American Heart Association.”. Circulation. 85 (1): 391—405. ISSN 0009-7322. doi:10.1161/01.cir.85.1.391.
Hopkins, Paul N. (2013). „Molecular Biology of Atherosclerosis”. Physiological Reviews. 93 (3): 1317—1542. ISSN 0031-9333. doi:10.1152/physrev.00004.2012.
Dardik, Alan; Yamashita, Akimasa; Aziz, Faisal; Asada, Hidenori; Sumpio, Bauer E. (2005). „Shear stress-stimulated endothelial cells induce smooth muscle cell chemotaxis via platelet-derived growth factor-BB and interleukin-1α”. Journal of Vascular Surgery. 41 (2): 321—331. ISSN 0741-5214. doi:10.1016/j.jvs.2004.11.016.
Stary, Herbert C (2000). „Lipid and macrophage accumulations in arteries of children and the development of atherosclerosis”. The American Journal of Clinical Nutrition. 72 (5): 1297s—1306s. ISSN 0002-9165. doi:10.1093/ajcn/72.5.1297s.
Mitra, R.; O'Neil, G. L.; Harding, I. C.; Cheng, M. J.; Mensah, S. A.; Ebong, E. E. (2017). „Glycocalyx in Atherosclerosis-Relevant Endothelium Function and as a Therapeutic Target”. Current Atherosclerosis Reports. 19 (12): 63. PMC 5681608 . PMID 29127504. doi:10.1007/s11883-017-0691-9.
Moore, Kathryn J.; Sheedy, Frederick J.; Fisher, Edward A. (2013). „Macrophages in atherosclerosis: A dynamic balance”. Nature Reviews Immunology. 13 (10): 709—721. PMC 4357520 . PMID 23995626. doi:10.1038/nri3520.
Proudfoot, Diane; Skepper, Jeremy N.; Hegyi, Laszlo; Bennett, Martin R.; Shanahan, Catherine M.; Weissberg, Peter L. (2000-11-24). „Apoptosis Regulates Human Vascular Calcification In Vitro”. Circulation Research. 87 (11): 1055—1062. ISSN 0009-7330. doi:10.1161/01.res.87.11.1055.
M. Abedin; Tintut; Demer; et al. (2004). „Vascular Calcification: Mechanisms and Clinical Ramifications”. Arterioscler. Thromb. Vasc. Biol. (на језику: енглески). 24 (7): 1161—1170. PMID 15155384. doi:10.1161/01.ATV.0000133194.94939.42.
Jian-Su, Shao (2006). „Molecular Mechanisms of Vascular Calcification: Lessons Learned From The Aorta”. Arterioscler. Thromb. Vasc. Biol. (на језику: енглески). 26 (7): 1423—1430. PMID 16601233. doi:10.1161/01.ATV.0000220441.42041.20.
R.C. Johnson (2006). „Vascular Calcification: Pathobiological Mechanisms and Clinical Implications”. Circ. Res. (на језику: енглески). 99 (10): 1044—1059. PMID 17095733. S2CID 1935982. doi:10.1161/01.RES.0000249379.55535.21.
R.J. Guzman (2007). „Clinical, cellular, and molecular aspects of arterial calcification”. J. Vasc. Surg. (на језику: енглески). 45 (Suppl A): A57—A63. PMC 2435088 . PMID 17544025. doi:10.1016/j.jvs.2007.02.049.
C.H., Byon; Javed; Dai; Kappes; Clemens; Darley-Usmar; McDonald; Chen (2008). „Oxidative stress induces vascular calcification through modulation of the osteogenic transcription factor Runx2 by AKT signaling”. J. Biol. Chem. (на језику: енглески). 283 (22): 15319—15327. PMC 2397455 . PMID 18378684. doi:10.1074/jbc.M800021200 .
Guzman, Raul J. (2007). „Clinical, cellular, and molecular aspects of arterial calcification”. Journal of Vascular Surgery. 45 (6): A57—A63. ISSN 0741-5214. doi:10.1016/j.jvs.2007.02.049.

em-consulte.com

Masson, Elsevier. „Patogenesi dell'aterosclerosi”. EM-Consulte (на језику: италијански). Приступљено 2022-12-17.

harvard.edu

adsabs.harvard.edu

Baeyens, N.; Mulligan-Kehoe, M. J.; Corti, F.; Simon, D. D.; Ross, T. D.; Rhodes, J. M.; Wang, T. Z.; Mejean, C. O.; Simons, M.; Humphrey, J.; Schwartz, M. A. (2014). „Syndecan 4 is required for endothelial alignment in flow and atheroprotective signaling”. Proceedings of the National Academy of Sciences of the United States of America. 111 (48): 17308—17313. Bibcode:2014PNAS..11117308B. PMC 4260558 . PMID 25404299. doi:10.1073/pnas.1413725111 .
Tarbell, J. M.; Shi, Z. D.; Dunn, J.; Jo, H. (2014). „Fluid Mechanics, Arterial Disease, and Gene Expression”. Annual Review of Fluid Mechanics. 46: 591—614. Bibcode:2014AnRFM..46..591T. PMC 4211638 . PMID 25360054. doi:10.1146/annurev-fluid-010313-141309.

healthandmedicineinfo.com

sr.healthandmedicineinfo.com

„Туника интима - Структура, функција и болести - Анатомија”. sr.healthandmedicineinfo.com. Приступљено 2022-06-18. ^{[мртва веза]}

jbc.org

C.H., Byon; Javed; Dai; Kappes; Clemens; Darley-Usmar; McDonald; Chen (2008). „Oxidative stress induces vascular calcification through modulation of the osteogenic transcription factor Runx2 by AKT signaling”. J. Biol. Chem. (на језику: енглески). 283 (22): 15319—15327. PMC 2397455 . PMID 18378684. doi:10.1074/jbc.M800021200 .

jvascsurg.org

R.J. Guzman (2007). „Clinical, cellular, and molecular aspects of arterial calcification”. J. Vasc. Surg. (на језику: енглески). 45 (Suppl A): A57—A63. PMC 2435088 . PMID 17544025. doi:10.1016/j.jvs.2007.02.049.

nih.gov

ncbi.nlm.nih.gov

Sandow, S. L.; Gzik, D. J.; Lee, R. M. (2009). „Arterial internal elastic lamina holes: Relationship to function?”. Journal of Anatomy. 214 (2): 258—266. PMC 2667883 . PMID 19207987. doi:10.1111/j.1469-7580.2008.01020.x.
Frank, J. S.; Fogelman, A. M. (1989). „Ultrastructure of the intima in WHHL and cholesterol-fed rabbit aortas prepared by ultra-rapid freezing and freeze-etching”. Journal of Lipid Research. 30 (7): 967—978. PMID 2794795. doi:10.1016/S0022-2275(20)38299-7.
Rosenson, R. S.; Hurt-Camejo, E. (2012). „Phospholipase A2 enzymes and the risk of atherosclerosis”. European Heart Journal. 33 (23): 2899—3709. PMID 22802388. doi:10.1093/eurheartj/ehs148.
Wight, T. N.; Merrilees, M. J. (2004). „Proteoglycans in atherosclerosis and restenosis: Key roles for versican”. Circulation Research. 94 (9): 1158—1167. PMID 15142969. doi:10.1161/01.RES.0000126921.29919.51.
Moreth, K.; Iozzo, R. V.; Schaefer, L. (2012). „Small leucine-rich proteoglycans orchestrate receptor crosstalk during inflammation”. Cell Cycle (Georgetown, Tex.). 11 (11): 2084—2091. PMC 3368860 . PMID 22580469. doi:10.4161/cc.20316.
Khalil, M. F.; Wagner, W. D.; Goldberg, I. J. (2004). „Molecular interactions leading to lipoprotein retention and the initiation of atherosclerosis”. Arteriosclerosis, Thrombosis, and Vascular Biology. 24 (12): 2211—2218. PMID 15472124. S2CID 2761937. doi:10.1161/01.ATV.0000147163.54024.70.
Baeyens, N.; Mulligan-Kehoe, M. J.; Corti, F.; Simon, D. D.; Ross, T. D.; Rhodes, J. M.; Wang, T. Z.; Mejean, C. O.; Simons, M.; Humphrey, J.; Schwartz, M. A. (2014). „Syndecan 4 is required for endothelial alignment in flow and atheroprotective signaling”. Proceedings of the National Academy of Sciences of the United States of America. 111 (48): 17308—17313. Bibcode:2014PNAS..11117308B. PMC 4260558 . PMID 25404299. doi:10.1073/pnas.1413725111 .
Nam, D.; Ni, C. W.; Rezvan, A.; Suo, J.; Budzyn, K.; Llanos, A.; Harrison, D.; Giddens, D.; Jo, H. (2009). „Partial carotid ligation is a model of acutely induced disturbed flow, leading to rapid endothelial dysfunction and atherosclerosis”. American Journal of Physiology. Heart and Circulatory Physiology. 297 (4): H1535—43. PMC 2770764 . PMID 19684185. doi:10.1152/ajpheart.00510.2009. . URL consultato il 4 giugno 2016 (archiviato dall'url originale il 25 giugno 2016).
Cunningham, K. S.; Gotlieb, A. I. (2005). „The role of shear stress in the pathogenesis of atherosclerosis”. Laboratory Investigation; A Journal of Technical Methods and Pathology. 85 (1): 9—23. PMID 15568038. S2CID 33316555. doi:10.1038/labinvest.3700215.
Chatzizisis, Y. S.; Coskun, A. U.; Jonas, M.; Edelman, E. R.; Feldman, C. L.; Stone, P. H. (2007). „Role of endothelial shear stress in the natural history of coronary atherosclerosis and vascular remodeling: Molecular, cellular, and vascular behavior”. Journal of the American College of Cardiology. 49 (25): 2379—2393. PMID 17599600. doi:10.1016/j.jacc.2007.02.059.
Tarbell, J. M.; Shi, Z. D.; Dunn, J.; Jo, H. (2014). „Fluid Mechanics, Arterial Disease, and Gene Expression”. Annual Review of Fluid Mechanics. 46: 591—614. Bibcode:2014AnRFM..46..591T. PMC 4211638 . PMID 25360054. doi:10.1146/annurev-fluid-010313-141309.
Lu, D.; Kassab, G. S. (2011). „Role of shear stress and stretch in vascular mechanobiology”. Journal of the Royal Society, Interface. 8 (63): 1379—1385. PMC 3163429 . PMID 21733876. doi:10.1098/rsif.2011.0177.
Chiu, J. J.; Chien, S. (2011). „Effects of disturbed flow on vascular endothelium: Pathophysiological basis and clinical perspectives”. Physiological Reviews. 91 (1): 327—387. PMC 3844671 . PMID 21248169. doi:10.1152/physrev.00047.2009.
Cheng, C.; Tempel, D.; Van Haperen, R.; Van Der Baan, A.; Grosveld, F.; Daemen, M. J.; Krams, R.; De Crom, R. (2006). „Atherosclerotic lesion size and vulnerability are determined by patterns of fluid shear stress”. Circulation. 113 (23): 2744—2753. PMID 16754802. S2CID 14376205. doi:10.1161/CIRCULATIONAHA.105.590018.
Guo, X.; Kassab, G. S. (2004). „Distribution of stress and strain along the porcine aorta and coronary arterial tree”. American Journal of Physiology. Heart and Circulatory Physiology. 286 (6): H2361—8. PMID 15148060. doi:10.1152/ajpheart.01079.2003.
Kwak, B. R.; Bäck, M.; Bochaton-Piallat, M. L.; Caligiuri, G.; Daemen, M. J.; Davies, P. F.; Hoefer, I. E.; Holvoet, P.; Jo, H.; Krams, R.; Lehoux, S.; Monaco, C.; Steffens, S.; Virmani, R.; Weber, C.; Wentzel, J. J.; Evans, P. C. (2014). „Biomechanical factors in atherosclerosis: Mechanisms and clinical implications”. European Heart Journal. 35 (43): 3013—20, 3020a—3020d. PMC 4810806 . PMID 25230814. doi:10.1093/eurheartj/ehu353.
Yoshio, S.; Tokumasu, K.; Fujino, A.; Naganuma, H.; Noguti, H.; Nitta, K.; Hoshino, I. (1991). „Neuro-otological study on Behçet disease”. Nihon Jibiinkoka Gakkai Kaiho. 94 (11): 1716—1726. PMID 1757846. doi:10.3950/jibiinkoka.94.11_1716.
Van Der Heiden, K.; Cuhlmann, S.; Luong Le, A.; Zakkar, M.; Evans, P. C. (2010). „Role of nuclear factor kappaB in cardiovascular health and disease”. Clinical Science (London, England : 1979). 118 (10): 593—605. PMID 20175746. doi:10.1042/CS20090557.
Davies, P. F. (2009). „Hemodynamic shear stress and the endothelium in cardiovascular pathophysiology”. Nature Clinical Practice. Cardiovascular Medicine. 6 (1): 16—26. PMC 2851404 . PMID 19029993. doi:10.1038/ncpcardio1397.
Gimbrone Jr, M. A.; García-Cardeña, G. (2013). „Vascular endothelium, hemodynamics, and the pathobiology of atherosclerosis”. Cardiovascular Pathology : The Official Journal of the Society for Cardiovascular Pathology. 22 (1): 9—15. PMC 4564111 . PMID 22818581. doi:10.1016/j.carpath.2012.06.006.
Le, N. T.; Sandhu, U. G.; Quintana-Quezada, R. A.; Hoang, N. M.; Fujiwara, K.; Abe, J. I. (2017). „Flow signaling and atherosclerosis”. Cellular and Molecular Life Sciences : CMLS. 74 (10): 1835—1858. PMC 5391278 . PMID 28039525. doi:10.1007/s00018-016-2442-4.
Bryan, M. T.; Duckles, H.; Feng, S.; Hsiao, S. T.; Kim, H. R.; Serbanovic-Canic, J.; Evans, P. C. (2014). „Mechanoresponsive networks controlling vascular inflammation”. Arteriosclerosis, Thrombosis, and Vascular Biology. 34 (10): 2199—2305. PMID 24947523. doi:10.1161/ATVBAHA.114.303424.
Wang, C.; Baker, B. M.; Chen, C. S.; Schwartz, M. A. (2013). „Endothelial cell sensing of flow direction”. Arteriosclerosis, Thrombosis, and Vascular Biology. 33 (9): 2130—2136. PMC 3812824 . PMID 23814115. doi:10.1161/ATVBAHA.113.301826.
Jongstra-Bilen, J.; Haidari, M.; Zhu, S. N.; Chen, M.; Guha, D.; Cybulsky, M. I. (2006). „Low-grade chronic inflammation in regions of the normal mouse arterial intima predisposed to atherosclerosis”. The Journal of Experimental Medicine. 203 (9): 2073—2083. PMC 2118407 . PMID 16894012. doi:10.1084/jem.20060245.
Mitra, R.; O'Neil, G. L.; Harding, I. C.; Cheng, M. J.; Mensah, S. A.; Ebong, E. E. (2017). „Glycocalyx in Atherosclerosis-Relevant Endothelium Function and as a Therapeutic Target”. Current Atherosclerosis Reports. 19 (12): 63. PMC 5681608 . PMID 29127504. doi:10.1007/s11883-017-0691-9.
Moore, Kathryn J.; Sheedy, Frederick J.; Fisher, Edward A. (2013). „Macrophages in atherosclerosis: A dynamic balance”. Nature Reviews Immunology. 13 (10): 709—721. PMC 4357520 . PMID 23995626. doi:10.1038/nri3520.
Zhang, J.; Wang, D.; He, S. (2015). „Roles of antibody against oxygenized low density lipoprotein in atherosclerosis: Recent advances”. International Journal of Clinical and Experimental Medicine. 8 (8): 11922—11929. PMC 4612790 . PMID 26550105.
M. Abedin; Tintut; Demer; et al. (2004). „Vascular Calcification: Mechanisms and Clinical Ramifications”. Arterioscler. Thromb. Vasc. Biol. (на језику: енглески). 24 (7): 1161—1170. PMID 15155384. doi:10.1161/01.ATV.0000133194.94939.42.
Jian-Su, Shao (2006). „Molecular Mechanisms of Vascular Calcification: Lessons Learned From The Aorta”. Arterioscler. Thromb. Vasc. Biol. (на језику: енглески). 26 (7): 1423—1430. PMID 16601233. doi:10.1161/01.ATV.0000220441.42041.20.
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