Sawzdargo M, Nguyen T, Lee DK, Lynch KR, Cheng R, Heng HH, George SR, O'Dowd BF (1999). „Identification and cloning of three novel human G protein-coupled receptor genes GPR52, PsiGPR53 and GPR55: GPR55 is extensively expressed in human brain”. Brain Res. Mol. Brain Res. 64 (2): 193—8. PMID9931487. doi:10.1016/S0169-328X(98)00277-0.
Oka S, Nakajima K, Yamashita A, Kishimoto S, Sugiura T (2007). „Identification of GPR55 as a lysophosphatidylinositol receptor”. Biochemical and Biophysical Research Communications. 362 (4): 928—34. PMID17765871. doi:10.1016/j.bbrc.2007.08.078.
Henstridge CM, Balenga NA, Ford LA, Ross RA, Waldhoer M, Irving AJ (2009). „The GPR55 ligand L-alpha-lysophosphatidylinositol promotes RhoA-dependent Ca2+ signaling and NFAT activation”. The FASEB Journal : Official Publication of the Federation of American Societies for Experimental Biology. 23 (1): 183—93. PMID18757503. doi:10.1096/fj.08-108670.
Oka S, Toshida T, Maruyama K, Nakajima K, Yamashita A, Sugiura T (2009). „2-Arachidonoyl-sn-glycero-3-phosphoinositol: a possible natural ligand for GPR55”. Journal of Biochemistry. 145 (1): 13—20. PMID18845565. doi:10.1093/jb/mvn136.
Staton PC, Hatcher JP, Walker DJ, Morrison AD, Shapland EM, Hughes JP, Chong E, Mander PK, Green PJ, Billinton A, Fulleylove M, Lancaster HC, Smith JC, Bailey LT, Wise A, Brown AJ, Richardson JC, Chessell IP (2008). „The putative cannabinoid receptor GPR55 plays a role in mechanical hyperalgesia associated with inflammatory and neuropathic pain”. Pain. 139 (1): 225—36. PMID18502582. doi:10.1016/j.pain.2008.04.006.
Kress M, Kuner R (2009). „Mode of action of cannabinoids on nociceptive nerve endings”. Experimental Brain Research. Experimentelle Hirnforschung. Experimentation Cerebrale. 196 (1): 79—88. PMID19306092. doi:10.1007/s00221-009-1762-0.
Sawzdargo M, Nguyen T, Lee DK, Lynch KR, Cheng R, Heng HH, George SR, O'Dowd BF (1999). „Identification and cloning of three novel human G protein-coupled receptor genes GPR52, PsiGPR53 and GPR55: GPR55 is extensively expressed in human brain”. Brain Res. Mol. Brain Res. 64 (2): 193—8. PMID9931487. doi:10.1016/S0169-328X(98)00277-0.
Oka S, Nakajima K, Yamashita A, Kishimoto S, Sugiura T (2007). „Identification of GPR55 as a lysophosphatidylinositol receptor”. Biochemical and Biophysical Research Communications. 362 (4): 928—34. PMID17765871. doi:10.1016/j.bbrc.2007.08.078.
Henstridge CM, Balenga NA, Ford LA, Ross RA, Waldhoer M, Irving AJ (2009). „The GPR55 ligand L-alpha-lysophosphatidylinositol promotes RhoA-dependent Ca2+ signaling and NFAT activation”. The FASEB Journal : Official Publication of the Federation of American Societies for Experimental Biology. 23 (1): 183—93. PMID18757503. doi:10.1096/fj.08-108670.
Oka S, Toshida T, Maruyama K, Nakajima K, Yamashita A, Sugiura T (2009). „2-Arachidonoyl-sn-glycero-3-phosphoinositol: a possible natural ligand for GPR55”. Journal of Biochemistry. 145 (1): 13—20. PMID18845565. doi:10.1093/jb/mvn136.
Staton PC, Hatcher JP, Walker DJ, Morrison AD, Shapland EM, Hughes JP, Chong E, Mander PK, Green PJ, Billinton A, Fulleylove M, Lancaster HC, Smith JC, Bailey LT, Wise A, Brown AJ, Richardson JC, Chessell IP (2008). „The putative cannabinoid receptor GPR55 plays a role in mechanical hyperalgesia associated with inflammatory and neuropathic pain”. Pain. 139 (1): 225—36. PMID18502582. doi:10.1016/j.pain.2008.04.006.
Kress M, Kuner R (2009). „Mode of action of cannabinoids on nociceptive nerve endings”. Experimental Brain Research. Experimentelle Hirnforschung. Experimentation Cerebrale. 196 (1): 79—88. PMID19306092. doi:10.1007/s00221-009-1762-0.