Hirose T, Smith RJ, Jetten AM (1994). „ROR gamma: the third member of ROR/RZR orphan receptor subfamily that is highly expressed in skeletal muscle”. Biochemical and biophysical research communications. 205 (3): 1976—83. PMID7811290. doi:10.1006/bbrc.1994.2902.
He YW, Deftos ML, Ojala EW, Bevan MJ (1998). „RORgamma t, a novel isoform of an orphan receptor, negatively regulates Fas ligand expression and IL-2 production in T cells”. Immunity. 9 (6): 797—806. PMID9881970. doi:10.1016/S1074-7613(00)80645-7.
Villey I, de Chasseval R, de Villartay JP (1999). „RORgammaT, a thymus-specific isoform of the orphan nuclear receptor RORgamma / TOR, is up-regulated by signaling through the pre-T cell receptor and binds to the TEA promoter”. European journal of immunology. 29 (12): 4072—80. PMID10602018. doi:10.1002/(SICI)1521-4141(199912)29:12<4072::AID-IMMU4072>3.0.CO;2-E.
Eberl G, Littman DR (2003). „The role of the nuclear hormone receptor RORgammat in the development of lymph nodes and Peyer's patches”. Immunological reviews. 195: 81—90. PMID12969312. doi:10.1034/j.1600-065X.2003.00074.x.
Medvedev A, Chistokhina A, Hirose T, Jetten AM (1997). „Genomic structure and chromosomal mapping of the nuclear orphan receptor ROR gamma (RORC) gene”. Genomics. 46 (1): 93—102. PMID9403063. doi:10.1006/geno.1997.4980.
Ortiz MA, Piedrafita FJ, Pfahl M, Maki R (1995). „TOR: a new orphan receptor expressed in the thymus that can modulate retinoid and thyroid hormone signals”. Molecular endocrinology (Baltimore, Md.). 9 (12): 1679—91. PMID8614404. doi:10.1210/me.9.12.1679.
Huang Z, Xie H, Wang R, Sun Z (2007). „Retinoid-related orphan receptor gamma t is a potential therapeutic target for controlling inflammatory autoimmunity”. Expert opinion on therapeutic targets. 11 (6): 737—43. PMID17504012. doi:10.1517/14728222.11.6.737.
Eberl G, Marmon S, Sunshine MJ, Rennert PD, Choi Y, Littman DR (2004). „An essential function for the nuclear receptor RORgamma(t) in the generation of fetal lymphoid tissue inducer cells”. Nature Immunology. 5 (1): 64—73. PMID14691482. doi:10.1038/ni1022.
Villey I, de Chasseval R, de Villartay JP (1999). „RORgammaT, a thymus-specific isoform of the orphan nuclear receptor RORgamma / TOR, is up-regulated by signaling through the pre-T cell receptor and binds to the TEA promoter”. European journal of immunology. 29 (12): 4072—80. PMID10602018. doi:10.1002/(SICI)1521-4141(199912)29:12<4072::AID-IMMU4072>3.0.CO;2-E.
Eberl G, Littman DR (2004). „Thymic origin of intestinal alphabeta T cells revealed by fate mapping of RORgammat+ cells”. Science (New York, N.Y.). 305 (5681): 248—51. PMID15247480. doi:10.1126/science.1096472.
Benoit G, Cooney A, Giguere V, Ingraham H, Lazar M, Muscat G, Perlmann T, Renaud JP, Schwabe J, Sladek F, Tsai MJ, Laudet V (2006). „International Union of Pharmacology. LXVI. Orphan nuclear receptors”. Pharmacological reviews. 58 (4): 798—836. PMID17132856. doi:10.1124/pr.58.4.10.
Eberl G, Littman DR (2003). „The role of the nuclear hormone receptor RORgammat in the development of lymph nodes and Peyer's patches”. Immunological reviews. 195: 81—90. PMID12969312. doi:10.1034/j.1600-065X.2003.00074.x.
Sun Z, Unutmaz D, Zou YR, Sunshine MJ, Pierani A, Brenner-Morton S, Mebius RE, Littman DR (2000). „Requirement for RORgamma in thymocyte survival and lymphoid organ development”. Science (New York, N.Y.). 288 (5475): 2369—73. PMID10875923. doi:10.1126/science.288.5475.2369.
Dong C (2008). „TH17 cells in development: an updated view of their molecular identity and genetic programming”. Nature reviews. Immunology. 8 (5): 337—48. PMID18408735. doi:10.1038/nri2295.
Akashi M, Takumi T (2005). „The orphan nuclear receptor RORalpha regulates circadian transcription of the mammalian core-clock Bmal1”. Nature structural & molecular biology. 12 (5): 441—8. PMID15821743. doi:10.1038/nsmb925.
Gréchez-Cassiau A, Rayet B, Guillaumond F, Teboul M, Delaunay F (2008). „The circadian clock component BMAL1 is a critical regulator of p21WAF1/CIP1 expression and hepatocyte proliferation”. The Journal of biological chemistry. 283 (8): 4535—42. PMID18086663. doi:10.1074/jbc.M705576200.
Hirose T, Smith RJ, Jetten AM (1994). „ROR gamma: the third member of ROR/RZR orphan receptor subfamily that is highly expressed in skeletal muscle”. Biochemical and biophysical research communications. 205 (3): 1976—83. PMID7811290. doi:10.1006/bbrc.1994.2902.
He YW, Deftos ML, Ojala EW, Bevan MJ (1998). „RORgamma t, a novel isoform of an orphan receptor, negatively regulates Fas ligand expression and IL-2 production in T cells”. Immunity. 9 (6): 797—806. PMID9881970. doi:10.1016/S1074-7613(00)80645-7.
Villey I, de Chasseval R, de Villartay JP (1999). „RORgammaT, a thymus-specific isoform of the orphan nuclear receptor RORgamma / TOR, is up-regulated by signaling through the pre-T cell receptor and binds to the TEA promoter”. European journal of immunology. 29 (12): 4072—80. PMID10602018. doi:10.1002/(SICI)1521-4141(199912)29:12<4072::AID-IMMU4072>3.0.CO;2-E.
Eberl G, Littman DR (2003). „The role of the nuclear hormone receptor RORgammat in the development of lymph nodes and Peyer's patches”. Immunological reviews. 195: 81—90. PMID12969312. doi:10.1034/j.1600-065X.2003.00074.x.
Medvedev A, Chistokhina A, Hirose T, Jetten AM (1997). „Genomic structure and chromosomal mapping of the nuclear orphan receptor ROR gamma (RORC) gene”. Genomics. 46 (1): 93—102. PMID9403063. doi:10.1006/geno.1997.4980.
Ortiz MA, Piedrafita FJ, Pfahl M, Maki R (1995). „TOR: a new orphan receptor expressed in the thymus that can modulate retinoid and thyroid hormone signals”. Molecular endocrinology (Baltimore, Md.). 9 (12): 1679—91. PMID8614404. doi:10.1210/me.9.12.1679.
Huang Z, Xie H, Wang R, Sun Z (2007). „Retinoid-related orphan receptor gamma t is a potential therapeutic target for controlling inflammatory autoimmunity”. Expert opinion on therapeutic targets. 11 (6): 737—43. PMID17504012. doi:10.1517/14728222.11.6.737.
Eberl G, Marmon S, Sunshine MJ, Rennert PD, Choi Y, Littman DR (2004). „An essential function for the nuclear receptor RORgamma(t) in the generation of fetal lymphoid tissue inducer cells”. Nature Immunology. 5 (1): 64—73. PMID14691482. doi:10.1038/ni1022.
Villey I, de Chasseval R, de Villartay JP (1999). „RORgammaT, a thymus-specific isoform of the orphan nuclear receptor RORgamma / TOR, is up-regulated by signaling through the pre-T cell receptor and binds to the TEA promoter”. European journal of immunology. 29 (12): 4072—80. PMID10602018. doi:10.1002/(SICI)1521-4141(199912)29:12<4072::AID-IMMU4072>3.0.CO;2-E.
Eberl G, Littman DR (2004). „Thymic origin of intestinal alphabeta T cells revealed by fate mapping of RORgammat+ cells”. Science (New York, N.Y.). 305 (5681): 248—51. PMID15247480. doi:10.1126/science.1096472.
Benoit G, Cooney A, Giguere V, Ingraham H, Lazar M, Muscat G, Perlmann T, Renaud JP, Schwabe J, Sladek F, Tsai MJ, Laudet V (2006). „International Union of Pharmacology. LXVI. Orphan nuclear receptors”. Pharmacological reviews. 58 (4): 798—836. PMID17132856. doi:10.1124/pr.58.4.10.
Eberl G, Littman DR (2003). „The role of the nuclear hormone receptor RORgammat in the development of lymph nodes and Peyer's patches”. Immunological reviews. 195: 81—90. PMID12969312. doi:10.1034/j.1600-065X.2003.00074.x.
Sun Z, Unutmaz D, Zou YR, Sunshine MJ, Pierani A, Brenner-Morton S, Mebius RE, Littman DR (2000). „Requirement for RORgamma in thymocyte survival and lymphoid organ development”. Science (New York, N.Y.). 288 (5475): 2369—73. PMID10875923. doi:10.1126/science.288.5475.2369.
Dong C (2008). „TH17 cells in development: an updated view of their molecular identity and genetic programming”. Nature reviews. Immunology. 8 (5): 337—48. PMID18408735. doi:10.1038/nri2295.
Akashi M, Takumi T (2005). „The orphan nuclear receptor RORalpha regulates circadian transcription of the mammalian core-clock Bmal1”. Nature structural & molecular biology. 12 (5): 441—8. PMID15821743. doi:10.1038/nsmb925.
Gréchez-Cassiau A, Rayet B, Guillaumond F, Teboul M, Delaunay F (2008). „The circadian clock component BMAL1 is a critical regulator of p21WAF1/CIP1 expression and hepatocyte proliferation”. The Journal of biological chemistry. 283 (8): 4535—42. PMID18086663. doi:10.1074/jbc.M705576200.